ライフサイエンスコーパス: GI

1 GI bleeding events among patient aged 75 years or older

2 GI bleeding occurred more frequently in patients given r

3 GI GVHD was increased in patients with vitamin A levels

4 GI leakage with ATB, DSS, and DSS plus ATB (DSS+ATB) was

5 GI stromal tumors (GISTs) are neoplasms with a varying m

6 GI symptoms are associated with a modest reduction in Pr

7 GI TB commonly affects the small bowel but is difficult

8 GI, GL, and insulin index (II) values were calculated wi

9 GIs disproportionately encode factors that enhance the a

10 resulted in lower glucose AUCi (P < 0.0001), GI (P = 0.0096), and GL (P = 0.0101) values than did the

11 Conversely, YC-1 + GI increased intra-tumoral 8-OHdG and levels of apoptosi

12 Low-dose YC-1 + GI is a unique therapy targeting hypoxic GC cells that g

13 tochemical analysis demonstrated that YC-1 + GI reduced HIF-1alpha expression and pimonidazole accumu

14 YC-1 + GI therapy strongly inhibited tumour growth.

15 5% and 12%, respectively, from 1995 to 2012 (GI on glucose standard: 56.5 +/- 6.2 compared with 53.9

16 17% of outbreaks in a season, included GI.3, GI.5, GII.2, GII.3, GII.6, GII.13, and GII.17 Kawasaki 3

17 ch Suc affects the circadian oscillator in a GI-dependent manner was unknown.

18 In response to acute GI mucosal injury, TFF peptides accelerate cell migratio

19 Additional GI accelerated membranous GLUT1 translocation, elevating

20 patients with metastatic or locally advanced GI stromal tumors (GISTs).

21 encapsulation to protect probiotics against GI tract insults and improve their adhesion and growth o

22 ide has just completed Phase 1 trial against GI cancers and is currently awaiting Phase 2 trials.

23 46.7%) and of grade 3/4, hepatic (17.0%) and GI (16.3%); 30.1% developed a grade 2 to 4 select AE in

24 elect AEs of any grade were skin (64.3%) and GI (46.7%) and of grade 3/4, hepatic (17.0%) and GI (16.

25 GCF endocan and TNF-alpha, VEGF-A, CAL, and GI for all groups (P <0.05).

26 r-old man with renal impairment, cardiac and GI symptoms, and peripheral neuropathy.

27 negative correlation between RS contents and GI values was found (R(2)=-0.747, P<0.01).

28 arms for grade >/= 3 late genitourinary and GI toxicity.

29 C) in patients with newly diagnosed lung and GI cancer.

30 h the control group (P <0.001): lower PI and GI, less BOP, less increase in GCF volume, and lower IL-

31 mic analysis of Congo red-positive renal and GI amyloid deposits detected abundant lysozyme C protein

32 he mutant peptide in the proband's renal and GI amyloid deposits.

33 ary octreotide prophylaxis developed another GI bleed, whereas 39 (76%) did not.

34 As GI TB can cause morphological alterations in and around

35 Lactobacillus rhamnosus L34 attenuated GI leakage in these models, as shown by the reductions o

36 gative correlation (r>-0.81; P<0.01) between GI value with amylose, crude fiber, crude fat, crude pro

37 We examined the relationship between GI group and IOP and medications at one year with a line

38 incorporation of 8% PSC powder in biscuits (GI=49) could be an effective way of developing a nutriti

39 EC cells in the human and mouse small bowel GI epithelium selectively express the mechanosensitive i

40 symptoms in a murine sepsis model induced by GI leakage and the administration of pathogenic bacteria

41 All cases were reviewed by GI gastrointestinal pathologists, and pathologic feature

42 tep in GI mobilization, producing a circular GI and a deletion site in the chromosome; circular forms

43 12 than in 1995.Although the average dietary GI and GL declined between 1995 and 2012, trends in spec

44 influences the prediction of average dietary GI and GL values for eating patterns.

45 time.The aim was to compare average dietary GI and glycemic load (GL), and contributing carbohydrate

46 ed less interindividual variation in dietary GI (R(2): 0.376 compared with 0.290) and GL (R(2): 0.825

47 ups to interindividual variations in dietary GI and GL.Overall, dietary GI and GL decreased by 5% and

48 The differences in mean dietary GI and GL between surveys were compared by using 1-facto

49 nutrient composition influences mean dietary GI and GL values that are calculated for eating patterns

50 ations in dietary GI and GL.Overall, dietary GI and GL decreased by 5% and 12%, respectively, from 19

51 lowing in vitro gastro-intestinal digestion (GI), was investigated systematically and coupled with ph

52 with diagnosis of gastrointestinal diseases (GI).

53 d with clopidogrel were matched to emergency GI surgery patients not treated with clopidogrel.

54 In total, 1240 patients undergoing emergent GI surgery while treated with clopidogrel were matched t

55 , we identified patients undergoing emergent GI surgery.

56 mic load (GL) of 13 while the rest exhibited GI ranging from 70 by Savitri to 80 by Salivahana.

57 microbial genomics analysis, while expanding GI predictions and improving its flexible visualization

58 Apixaban had the most favorable GI safety profile among all age groups.

59 vestigated which DOAC had the most favorable GI safety profile and compared differences among these d

60 ts, we found apixaban had the most favorable GI safety profile and rivaroxaban the least favorable pr

61 w-up testing should be limited and FilmArray GI panel should not be used as a test of cure.

62 Follow-up testing with FilmArray GI panel within 4 weeks of a negative result rarely chan

63 he yield of follow-up testing with FilmArray GI panel within 4 weeks of an initial test.

64 f FKF1 with the optimization of a split FKF1/GI dimerized Gal4-VP16 transcriptional system, we identi

65 The improvements regarding the FKF1/GI- and CRY2/CIB1-based systems will be widely applicabl

66 e associated with cessation is difficult for GI surgeons.

67 All patients had a hospitalization for GI bleed and received octreotide after discharge.

68 The negative predictive values (NPV) for GI and GII were 100% and 99.9%, respectively.

69 linking them to a glucuronic acid sugar for GI excretion.

70 event were not associated with ER visits for GI illness overall, and associations by county or age we

71 ciation between SSO events and ER visits for GI illness using a case-crossover study design.

72 he overall odds ratio (OR) for ER visits for GI illness was 1.09 [95% confidence interval (CI): 1.03,

73 ER visits for GI were considered exposed if an SSO event occurred in t

74 astrointestinal (GI) (patho)physiology; from GI motility and epithelial barrier function to enteric n

75 nd a CRISPR-based tool to dissect functional GI networks.

76 Gastrointestinal (GI) anthrax is the most prevalent form of naturally acqu

77 Gastrointestinal (GI) bacterial translocation in sepsis is well known, but

78 Gastrointestinal (GI) bleeding is one of the most common complications aft

79 Gastrointestinal (GI) colonization of two-day-old (P2) rats leads to invas

80 mmals and their associated gastrointestinal (GI) microbes contain complementary systems that are idea

81 taking clopidogrel before gastrointestinal (GI) surgery, to aid surgeons in decisions regarding clop

82 g the correlations between gastrointestinal (GI) bacterial biota and diseases.

83 h adverse post-colonoscopy gastrointestinal (GI) and non-gastrointestinal events (cardiovascular, pul

84 with Parkinson's disease, gastrointestinal (GI) dysfunctions, such as gastroparesis and constipation

85 The FilmArray gastrointestinal (GI) panel (BioFire Diagnostics, Salt Lake City, UT) is a

86 est, the BioFire FilmArray gastrointestinal (GI) panel, which tests for many of the most common agent

87 rbed onto SWCNTs in a fish gastrointestinal (GI) tract.

88 rug stability in the harsh gastrointestinal (GI) tract environment, providing opportunities for targe

89 gestion in a dynamic human gastrointestinal (GI) model that simulates gut digestive conditions to stu

90 nsal organism of the human gastrointestinal (GI) tract primarily transmissible via the fecal-oral rou

91 sts with renal impairment, gastrointestinal (GI) symptoms, and sicca syndrome, whereas cardiac involv

92 their regulatory roles in gastrointestinal (GI) (patho)physiology; from GI motility and epithelial b

93 activated KIT mutations in gastrointestinal (GI) stromal tumors (GISTs) in 1998 triggered a sea chang

94 Lower gastrointestinal (GI) tract graft-versus-host disease (GVHD) is the predom

95 gents increase the risk of gastrointestinal (GI) bleeding.

96 ith a primary diagnosis of gastrointestinal (GI) illness in Massachusetts for 2006-2007.

97 en SSO events and rates of gastrointestinal (GI) illness in Massachusetts.

98 spondents with overlapping gastrointestinal (GI) symptom complexes have significantly higher odds of

99 ase (GVHD) through reduced gastrointestinal (GI) permeability, reduced mucosal injury, and reduced ly

100 that maintain and restore gastrointestinal (GI) mucosal homeostasis.

101 n subjected to a simulated gastrointestinal (GI) digestion, and transport studies were conducted usin

102 r (PAM) contributes to the gastrointestinal (GI) and cardiovascular (CV) cholinergic adverse events (

103 nents that result from the gastrointestinal (GI) conditions, which would be bioaccessible and availab

104 drugs (NSAIDs) damage the gastrointestinal (GI) epithelial cell membranes by inducing several signal

105 hromaffin (EC) cell in the gastrointestinal (GI) epithelium is the source of nearly all systemic sero

106 In the gastrointestinal (GI) epithelium, enterochromaffin (EC) cells are enteroen

107 s and interaction with the gastrointestinal (GI) membrane.

108 The gastrointestinal (GI) microbiome is widely investigated for its role in ma

109 cted to tissues within the gastrointestinal (GI) tract (jejunum, ileum and colon).

110 on-induced toxicity in the gastrointestinal (GI) tract and the main cellular compartments studied in

111 e dynamic ecosystem of the gastrointestinal (GI) tract by translating chemical cues from the environm

112 onic devices placed in the gastrointestinal (GI) tract for prolonged periods have the potential to tr

113 iated drug delivery in the gastrointestinal (GI) tract is a bourgeoning area of study.

114 The gastrointestinal (GI) tract is essential for the absorption of nutrients,

115 ompartmentalization of the gastrointestinal (GI) tract of metazoans is critical for health.

116 to epithelial cells of the gastrointestinal (GI) tract, mediating toxicities restricting the therapeu

117 m the genital tract to the gastrointestinal (GI) tract.

118 es is important within the gastrointestinal (GI) tract.

119 oxemia) originating in the gastrointestinal (GI) tract.

120 ons encountered throughout gastrointestinal (GI) transit, leading to degradation of the delivery vect

121 ancreatitis, predispose to gastrointestinal (GI) adenocarcinoma by reprogramming differentiated cells

122 o deliver nucleic acids to gastrointestinal (GI) tissues due to their size and need for intracellular

123 (>/= 18 years) undergoing gastrointestinal (GI) operations was assessed in a nationwide cohort using

124 ovel cluster (n = 5) within four genogroups (GI, GII, GIV, and GV) were identified by phylogenetic an

125 ey are classified into two major genogroups (GI and GII), with each genogroup further divided into mu

126 x 1 (FKF1) and its binding partner GIGANTEA (GI) as well as CRY2/CIB1.

127 We have previously shown that GIGANTEA (GI) is required to sustain Suc-dependent circadian rhyth

128 udied here can be classified as medium- high GI foods, not to be considered as a proper food ingredie

129 can be expected in individuals with a higher GI group that indicates a clinically more challenging gl

130 Individuals in the next higher GI group had a 1.69 +/- 0.2 mmHg larger IOP decrease.

131 in women, whereas the consumption of higher-GI fruit, such as bananas, was associated with higher ri

132 T) values up to 450 mg-min/L except for hNoV GI, where 1 log10 reduction was observed at CT values of

133 in equivalent or greater reductions for hNoV GI.

134 In PB, hNoV GI and MNV exhibited comparable resistance to PAA and mo

135 y was to characterize human norovirus (hNoV) GI and GII reductions during disinfection by peracetic a

136 applied CDKO to generate a large-scale human GI map, comprising 490,000 double-sgRNAs directed agains

137 ed prevalence of C. trachomatis in the human GI tract.

138 diated by ILC2 production of IL-13, improved GI tract barrier function, and a preserved graft-versus-

139 atekeeper function of omega-3 PUFAs improves GI safety when administered with NSAID.

140 Mean change in GI was 1.89 +/- 0.32 and 1.68 +/- 0.58 in the control gr

141 ell lines that have been used for decades in GI research.

142 There was no significant difference in GI incidence rate between periods of dabigatran and warf

143 rials provided information on differences in GI between control and intervention arms.

144 A median reduction in GI of 10 units reduced the overall pooled estimates for

145 selection, dramatic reductions were seen in GI, especially the SB and SC paths.

146 Excision is an early step in GI mobilization, producing a circular GI and a deletion

147 5 to 17% of outbreaks in a season, included GI.3, GI.5, GII.2, GII.3, GII.6, GII.13, and GII.17 Kawa

148 ith more than one polymerase type, including GI.3, GII.2, GII.3, GII.4 Sydney, GII.13, and GII.17, fo

149 rophylaxis with octreotide after their index GI bleed from 2009 to 2015.

150 and after 6 months were: 1) gingival index (GI), 2) probing depth (PD), 3) clinical attachment level

151 ures were plaque index (PI), gingival index (GI), bleeding on probing (BOP), PD, gingival crevicular

152 four groups according to the Glaucoma Index (GI) that incorporated preoperative intraocular pressure

153 elopment time (DDT), LASRC and gluten index (GI) were positively related to polymeric protein and neg

154 arch digestibility, in vivo glycaemic index (GI) and sensorial properties of biscuits.

155 rrelated negatively with the glycemic index (GI) (r=-0.674; p</=0.05) and contributed for 45.5% of GI

156 patterns on meal or dietary glycemic index (GI) and glycemic load (GL) value determinations has rema

157 andial glycemic response and glycemic index (GI) and glycemic load (GL) value determinations remains

158 the associations of dietary glycemic index (GI) and glycemic load (GL) with systolic blood pressure

159 men was observed for higher-glycemic index (GI) fruit [HR: 1.51 (95% CI: 1.22, 1.86) for >/=1 servin

160 Australians have used the glycemic index (GI) since 1995; however, there are no data on changes in

161 their nutrient composition, glycemic index (GI), total phenolic content (TPC), total anthocyanin con

162 pth [PD], plaque index [PI], gingival index [GI], bleeding on probing [BOP], and clinical attachment

163 contributing to the acute radiation-induced GI syndrome (RIGS).

164 medial hypothalamus (VMH) glucose-inhibited (GI) neurons by low glucose after recurrent hypoglycemia

165 thyrocytes with a mean genetic instability (GI) index of 35.8+/-2.6%, as well as significant inducti

166 In addition, IslandViewer's integrated GI predictions from multiple methods have been improved

167 method for calculating genetic interactions (GIs) from CRISPR-deleted gene pairs.

168 event Heinrich 3 and Greenland Interstadial (GI) 5.1 in the North Atlantic ( 30,400 to 28,400 years a

169 tative information on the gastro-intestinal (GI) absorption of prions (i.e. the bioavailability and s

170 le genetic elements such as genomic islands (GIs) have been pivotal in the evolution of O1/O139 V. ch

171 nteractive visualization of genomic islands (GIs, regions of probable horizontal origin) in bacterial

172 4 of whom were from a Dutch genetic isolate (GI), and 5 patients had a Leber congenital amaurosis phe

173 t irradiation with these doses caused lethal GI syndrome, focal (5 mm) radiation of the intestine did

174 Identification of Lalat as a low GI variety is of significance in the dietary prevention

175 e basmati varieties can be classified as low GI and medium GI rice, respectively.

176 fore, red basmati varieties can serve as low GI sources of functional food.

177 27.9% amylose was the only one eliciting low GI of 50 and glycemic load (GL) of 13 while the rest exh

178 ctive way of developing a nutritious and low-GI biscuit without jeopardizing its desirable sensorial

179 f they reported author-defined high- and low-GI or -GL diets and blood pressure, were of >/=6 wk dura

180 intima-media thickness was lower in the low-GI group than in the HF group (657 +/-12 compared with 6

181 ht and length z scores were lower in the low-GI group than in the HF group (birth weight z score: 0.2

182 ular outcomes after aspirin-associated lower GI bleeding.

183 scuss new insights into the biology of lower GI tract GVHD and focus on intrinsic pathways and regula

184 essed the long-term risks of recurrent lower GI bleeding and serious cardiovascular outcomes after as

185 stasis may improve treatment of severe lower GI tract aGVHD.

186 is with octreotide had a significantly lower GI bleed recurrence compared with historical controls no

187 Recent data indicate that lower GI tract GVHD is a complicated process mediated by donor

188 2 innate lymphoid cells (ILC2s) in the lower GI tract are sensitive to conditioning therapy and show

189 with grade III or greater aGVHD of the lower GI tract, who do not respond to therapy with corticoster

190 diversity increased from the upper to lower GI segments and that the stratification of microbial com

191 the lethality of aGVHD and in treating lower GI tract disease.

192 sed glucose AUCi (P < 0.0001), measured meal GI (P = 0.0066), and mean GL (P < 0.0001).

193 sed glucose AUCi (P = 0.0026), measured meal GI (P = 0.0139), and meal GL (P = 0.0140).

194 blood was sampled for 2 h, and measured meal GI and GL and insulin index (II) values were calculated

195 of macronutrients and fiber on measured meal GI and GL values.Four studies were conducted during whic

196 hat uncertainty in the determination of meal GI and GL values is introduced when carbohydrate-contain

197 ained 15% and 16% of the variability in mean GI value for white bread, respectively.

198 eties can be classified as low GI and medium GI rice, respectively.

199 trend = 0.001] but not for lower or moderate GI fruit.

200 mplications led to hospitalization, and most GI complications occurred within 14 days of colonoscopy.

201 tiple different cancer types, including most GI cancers, providers will frequently encounter patients

202 or four synbodies against VLPs from multiple GI and GII genotypes and found that the synbodies were b

203 After vaccination with a multivalent GI.1 and GII.4c norovirus virus-like particle (VLP) vacc

204 atis causes "opportunistic" infection at non-GI sites under conditions driven by improved sanitation/

205 , which was significantly faster than in non-GI patients (P < 0.05).

206 ectal surgery, whereas 45% had noncolorectal GI surgery.

207 ly diagnosed incurable lung or noncolorectal GI cancer to receive either early integrated PC and onco

208 Notably, GI tract clones display extensive sharing of sequence va

209 .674; p</=0.05) and contributed for 45.5% of GI variability.

210 clude depletion and functional alteration of GI-resident CD4(+) T cells, loss of antigen-presenting c

211 e glycemic response and the determination of GI and GL values for white bread.

212 n the glycemic response and determination of GI and GL values of a subsequent standard test food.Twen

213 riteria were revised to permit management of GI toxicities.

214 gression of infection using a mouse model of GI infection.

215 ity of symptoms and the common occurrence of GI disorders in transplant recipients may delay diagnosi

216 er adults through the preoperative period of GI surgery does not significantly increase bleeding even

217 associated with increased incidence rate of GI bleeding compared with non-exposed period among naive

218 We did not detect an increased risk of GI bleeding over dabigatran vs warfarin risk period.

219 Apixaban was associated with a lower risk of GI bleeding than dabigatran (HR, 0.39; 95% CI, 0.27-0.58

220 ces among these drugs in age-related risk of GI bleeding.

221 y enhancements to facilitate the analysis of GIs and better understand their role in the evolution of

222 ality for patients suspected of abdominal or GI TB.

223 ween amylose contents and the RS contents or GI values, while a strong negative correlation between R

224 n Scale) within 8 weeks of incurable lung or GI cancer diagnosis.

225 ients with newly diagnosed incurable lung or GI cancer.

226 not sustained at day 35 after vaccination or GI.1 infection, as measured from archived sera.

227 + 0.5% AZM showed enhanced reductions in PI, GI, mSBI, and PD and gains in CAL (P <0.05) over 9 month

228 was noticed with respect to reduction in PI, GI, OHI-S, and microbiologic counts in group I compared

229 Significant reduction in mean PI, GI, PD, and BOP were found after treatment in all groups

230 , significantly more bleeders had a previous GI bleeding history before left ventricular assist devic

231 hat low levels of vitamin A actively promote GI GVHD and are not simply a marker of poor nutritional

232 inal crypts and that ATM inhibition promotes GI syndrome after TBI.

233 st comprehensive characterization of the rat GI microbiota landscape for the research community, layi

234 tients in this study experienced a recurrent GI bleed compared with a matched historical control grou

235 th incident bleed go on to develop recurrent GI bleeding.

236 is proposed to reduce the risk of recurrent GI bleeding in this population.

237 of the Notch signalling pathway to regulate GI stem cell function in adult small intestine and stoma

238 patients) were most frequently skin related, GI, endocrine, and hepatic; grade 3 to 4 select AEs occu

239 ditional on the effects of Suc and requiring GI These findings reveal that Suc affects the stability

240 administered UAPs could survive the animal's GI tracts for as long as 18h.

241 ound that following S-colo, rates of serious GI adverse events were low but clinically relevant, and

242 d in encapsulates were released in simulated GI fluids.

243 uded enteric nervous system hypoplasia, slow GI transit, diminished peristaltic reflex activity, and

244 ng emission X-ray fluorescence spectroscopy (GI/GE-XRF) are techniques that enable nondestructive, qu

245 sustains rhythms in the dark by stabilizing GI protein, dependent on the F-box protein ZEITLUPE, and

246 ohorts of individuals have linked suboptimal GI reconstitution to residual inflammation and heightene

247 are located in the gastrointestinal system (GI), followed by the bronchi, endocrine glands-like C ce

248 The GI mucosal intestinal fatty acid-binding protein was dec

249 The GI panel improved patient care by rapidly identifying a

250 o electronics at various locations along the GI tract.

251 The most common organisms detected by the GI panel were enteropathogenic Escherichia coli (EPEC, n

252 When comparing the GI microbiota from different hosts, we found that the ra

253 d cellular mechanisms of TFF peptides in the GI epithelium remain largely unknown.

254 collection to discharge was 3.4 days in the GI panel group versus 3.9 days in the controls (P = 0.04

255 OR is crucial for T-cell accumulation in the GI tract and for establishing local adaptive immunity ag

256 Infectious organisms produced in the GI tract and reaching the rectum may then chronically co

257 n regulating the inflammatory process in the GI tract during aGVHD are needed.

258 rapies that might restore homeostasis in the GI tract during GVHD are highlighted.

259 The use of low-frequency ultrasound in the GI tract represents a novel tool for the delivery of a w

260 tunities for targeting specific sites in the GI tract, increasing drug solubility and bioavailability

261 In the GI tract, the microbiota express beta-glucuronidase enzy

262 Amyloid was also detected in the GI tract.

263 on by nucleases, which are ubiquitous in the GI tract.

264 lity, and providing sustained release in the GI tract.

265 nisms, focusing on defense mechanisms in the GI tract.

266 tanding of the anatomy and physiology of the GI tract by focusing on the ENS and the mucosal immune s

267 In this region of the GI tract, the protective mucus barrier is poorly develop

268 stool cultures submitted were tested on the GI panel (n = 241) and were compared with control patien

269 Patients tested on the GI panel had an average of 0.58 other infectious stool t

270 The degree of polishing had no effect on the GI.

271 starch hydrolysis rate markedly reduced the GI of biscuits.

272 unique and diverse physiology throughout the GI tract, including wide variation in pH, mucus that var

273 did not induce hydrosalpinx or spread to the GI tract even when delivered to the oviduct by intraburs

274 Blockade Ab avidity to the GI.1 vaccine component peaked at day 35 (7 days after do

275 Patients with the GI panel had 0.18 abdomen and/or pelvic imaging studies

276 pants maintained high avidity blockade Ab to GI.1 at day 180.

277 hemical penetration enhancers on delivery to GI tissue using ultrasound.

278 olyzed collagen showed greater resistance to GI digestion and greater transport efficiency than the u

279 peptides play important roles in response to GI mucosal injury and inflammation.

280 Median times to GI bleeding were <90 days for apixaban and rivaroxaban a

281 by age, were used to estimate rates of total GI bleeding.

282 Gastrointestinal tuberculosis (GI TB) is an important manifestation of abdominal tuberc

283 dentifying known and previously unidentified GIs between modifiers of ricin toxicity.

284 2011 and 2015, registered in the Dutch Upper GI Cancer Audit.

285 The severity of upper GI symptoms was assessed and measurements of height, wei

286 in 897 consecutive patients undergoing upper GI tract endoscopy.

287 human norovirus virus-like-particles (VLPs), GI.1 and GII.4 VLPs.

288 ized both the CRR and glucose sensing by VMH GI neurons in STZ rats.

289 AC does not normalize glucose sensing by VMH GI neurons when RH occurs during diabetes.

290 prevent HAAF or normalize activation of VMH GI neurons by low glucose in STZ rats after RH.

291 poglycemia in vivo and the activation of VMH GI neurons in low glucose using membrane potential sensi

292 Massachusetts and 66,460 ER admissions with GI illness listed as the primary diagnostic code.

293 pixaban had a lower risk of association with GI bleeding in the very elderly than dabigatran (HR, 0.4

294 l administration of pathogenic bacteria with GI leakage induced by either an antibiotic cocktail (ATB

295 trated within 24 h in 50 to 88% of mice with GI leakage plus the administration of pathogenic bacteri

296 stration of pathogenic bacteria but not with GI leakage induction alone or bacterial gavage alone.

297 ders will frequently encounter patients with GI cancer who are overweight or obese.

298 Patients with GI cancers in both study groups reported improvements in

299 al review on MDCTE findings in patients with GI tuberculosis proved on FNAC and clinical and/or imagi

300 appears to have occurred synchronously with GI-5.1 warming and decreased precipitation over the west