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3 oacylation, from kinetic analysis, activated GPI-PLC by accelerating the conversion of a GPI-PLC.VSG
6 phosphatidylinositol (GPI)-phospholipase C (GPI-PLC) from the protozoan parasite Trypanosoma brucei,
7 osphatidylinositol-specific phospholipase C (GPI-PLC) have been implicated in releasing the old VSG c
8 osphatidylinositol-specific phospholipase C (GPI-PLC) is an integral membrane protein in the protozoa
9 n of Trypanosoma brucei GPI-phospholipase C (GPI-PLC) results in decreased expression of major surfac
11 has been considered unimportant because (i) GPI-PLC null mutants are fully viable and (ii) cytosolic
16 Leishmania where heterologous expression of GPI-PLC causes a GPI deficiency, the enzyme existed as a
17 in Nonidet P-40, and dimers and monomers of GPI-PLC were the major species in 3-[(3-cholamidopropyl)
24 monomers, dimers, and tetramers of purified GPI-PLC was detected by molecular sieving and shown to b
27 erentiation has multiple mechanisms and that GPI-PLC plays a more significant role in VSG release tha
28 Surface biotinylation assays indicate that GPI-PLC does gain access to extracellular VSG, suggestin
30 rted that the differential expression of the GPI-PLC mRNA also results from a 10-fold difference in h
31 specifically blocking the translation of the GPI-PLC mRNA in procyclic forms by the inclusion of a ha
33 tein synthesis inhibitors in stabilizing the GPI-PLC mRNA operates in trans through a short-lived fac
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