ライフサイエンスコーパス: GST

1 GST activity can be further controlled by adding specifi

2 GST is widely studied in the metabolism of xenobiotics a

3 GST pull-down indicated that nesprin-1/lamin/SUN interac

4 GST pulldown and coimmunoprecipitation studies reveal th

5 GST pulldown assays demonstrate that the dimerization do

6 GST pulldown assays demonstrated that vIRF1 interacts wi

7 GST-AE1 pull-down assays using human kidney membrane pro

8 GST-B1B2 inhibited CLL cell migration as effectively as

9 GST-PEX9 inhibited MMP-9-driven gelatin proteolysis, mea

10 GSTs with the highest affinity (Tyr-based GSTs) are also

11 sessed for cross-reactivity with the other 3 GSTs using antibody binding assays.

12 ents from tropical Colombia had IgE to all 4 GSTs.

13 significant IgE cross-reactivity among the 4 GSTs is in agreement with the low shared amino acid iden

14 nge in a single-crystalline Ge(2)Sb(2)Te(5) (GST) nanowire memory device by in situ transmission elec

15 sion of the SMPL-engineered Mtd variant as a GST-bound fusion protein demonstrated specific binding t

16 d Ca(2+) sensitization of force induced by a GST-MYPT1(654-880) fragment inhibiting myosin light chai

17 n when IRE1alpha is forced to dimerise, by a GST-tag, phospho-enhancement of activity is still observ

18 (ii) A GST fusion protein comprising the C terminus of SERT pul

19 We conclude that a GST protein anchor can be used to immobilize functional

20 e inhibiting the function of KDM1A maps to a GST-tag.

21 otagmin-1 C2B domain fragment but not with a GST-mutant synaptotagmin-1 C2B domain fragment that poor

22 assay, the His-tagged Myb1 interacted with a GST-TvCyP1 fusion protein, which had an enzymatic profic

23 tantly, preabsorption of the antibody with a GST-WT synaptotagmin-1 C2B domain fragment but not with

24 hence represents a bcc-type polyamorph of a-GST.

25 mouse hearts, Gstp1/2 was the most abundant GST transcript followed by Gsta4 and Gstm4.1, and GSTP a

26 Accordingly, GST-PEX9 also abolished the degradation of gelatin by MM

27 based probes (ABPs) that characterize active GSTs in mammalian tissues.

28 Arabidopsis lines overexpressing TNT-active GSTs AtGST-U24 and AtGST-U25 were compromised in biomass

29 at increased levels of endogenous TNT-active GSTs catalyse excessive glutathionylation of endogenous

30 he hepatic antioxidant enzymatic activities (GST and GPx), clearly showing that this combination incr

31 We hypothesized that altered enzyme activity GST genotypes influence the susceptibility for esophagea

32 c and adopts a disordered conformation after GST removal.

33 ein mixture containing bovine serum albumin, GST, and ubiquitin could be specifically probed in paral

34 lly flexible building units of the amorphous GST network, intimately linked to the presence of distin

35 aining with phospho-specific antibodies, and GST pull-down assays showed that Nck determines spatiote

36 rdingly, ELISAs demonstrated that GST-B4 and GST-B1 specifically bound to gelatin.

37 capacities and flexibilities of the EROD and GST activities in Antarctic fish were significantly lowe

38 As both, fission and GST activities of GDAP1, are critically dependent on HD1

39 y increase in glutathione reductase (GR) and GST activities.

40 Furthermore, Immunoprecipitation and GST pull-down assays demonstrated that TRIM28 interacts

41 Here, using immunoprecipitation and GST pull-down experiments, we demonstrated that SET inte

42 chain (KLC-1/2) and immunoprecipitation and GST pull-down showed that these proteins interacted via

43 Co-immunoprecipitation and GST pulldown experiments provided evidence that EPLIN in

44 Mutation analysis, immunoprecipitation, and GST pulldown assays based on the theoretical predictions

45 ncy for NO transport and storage by MRP1 and GST P1-1, respectively.

46 und this gene organization of a split RO and GST gene cluster to occur more broadly, implying a large

47 , Asc s 1 (ABA-1), Asc l 3 (tropomyosin) and GST (glutathione transferase).

48 rabidopsis plants overexpressing GST-U24 and GST-U25 exhibit significantly enhanced ability to withst

49 physiological concentrations of GST-U24 and GST-U25 result in only a limited innate ability to cope

50 glutathione transferases (GSTs), GST-U24 and GST-U25, from Arabidopsis (Arabidopsis thaliana) that ar

51 To assess the role of GST-U24 and GST-U25, we purified and characterized recombinant forms

52 ctions interrupted by excess monovalent anti-GST-EMI F(ab) fragments.

53 NT-active Arabidopsis thaliana (Arabidopsis) GSTs.

54 hibited CLL cell migration as effectively as GST-B3B4.

55 otyrosine-recognition sequences expressed as GST-fusion proteins.

56 s confirmed through hemagglutination assays; GST-VP8* P[11] hemagglutinates type O, A, and B red bloo

57 boundary resistance of 28 +/- 8 m(2)K/GW at GST-SiO2 interfaces and an effective thermopower 350 +/-

58 effective thermopower 350 +/- 50 microV/K at GST-Pt interfaces.

59 S-transferase omega 1 (GSTO1) is an atypical GST isoform that is overexpressed in several cancers and

60 transferase Omega 1 (GSTO1-1) is an atypical GST reported to play a pro-inflammatory role in response

61 ere, we identify novel features of bacterial GSTs that cleave beta-aryl ether bonds typically found i

62 H is a derivative of a known mechanism-based GST inhibitor that binds within the active site and inhi

63 vity of bacteria that express only Cys-based GSTs could be related to the low or null affinity of the

64 Ser-based GSTs and Cys-based GSTs show affinities 10(2)-10(4) times lower, not suffic

65 Ser-based GSTs and Cys-based GSTs show affinities 10(2)-10(4) time

66 hat only the more recently evolved Tyr-based GSTs display enough affinity for DNDGIC (KD < 10(-9) M)

67 GSTs with the highest affinity (Tyr-based GSTs) are also over-represented in the perinuclear regio

68 larities to the body-centered-cubic GST (bcc-GST) it eventually crystallizes into at 28 GPa, and henc

69 ne-S-transferase-fused recombinant biglycan (GST-BGN) on craniofacial bone regeneration.

70 Furthermore, our results from both GST pulldown assays and analytical ultracentrifugation s

71 Vav3-Cdc37 interaction was confirmed by GST pulldown and, for native proteins, by co-immunopreci

72 The interaction was confirmed by GST pulldown, blot overlay, and co-immunoprecipitation a

73 s identified as the 14-3-3 binding region by GST-14-3-3 pulldown assays.

74 rminal 44 amino acids of PDZD11, as shown by GST-pulldown assays.

75 ic features by comparison with the canonical GST structure.

76 mSNAREs mimic (electronically) the canonical GST substrate 1-chloro-2,4-dinitrobenzene (CDNB), and be

77 SOD, CAT, GST, GSH, vitamin E and C levels were high in combinatio

78 Quantitative proteomic analysis of Cav1-GST (amino acids 1-101) pull downs showed sixfold-increa

79 cCPE.GST or GST (GST is glutathione S-transferase) was conjug

80 plastic lesions and accumulated (111)In-cCPE.GST (3.17 +/- 0.51 %ID/g) but not (111)In-GST (0.99 +/-

81 The uptake of (111)In-cCPE.GST in claudin-4-positive MDA-MB-468 xenograft tumors in

82 Taken together, (111)In-cCPE.GST targets claudin-4 expression in frank tumors and pre

83 rs were claudin-4-positive, and (111)In-cCPE.GST uptake was 3.2 +/- 0.70 %ID/g, significantly higher

84 The affinity of radiolabeled cCPE.GST for claudin-4 was confirmed using claudin-4-expressi

85 mparative sequence analysis of characterized GSTs of the PHYA1, PHYB, and HY5 genes of G. hirsutum an

86 se from Alzheimer disease brain could cleave GST-I2(PP2A), except when I2(PP2A) was mutated at the cl

87 Immunohistochemical colocalization, GST pulldown, and surface plasmon resonance studies reve

88 report here experimental results confirming GST-like activity for 82 of them, along with 37 new 3D s

89 bear similarities to the body-centered-cubic GST (bcc-GST) it eventually crystallizes into at 28 GPa,

90 of the presumed common ancestor of cytosolic GSTs (i.e. glutaredoxin 2).

91 oreover, this isoform is the first described GST that contains all secondary structural elements, inc

92 on the lens plane by assigning the designed GST crystallization levels to the corresponding slits, a

93 literature along with the lowest detectable GST concentration (200pmolL(-1)) for GST label-free sens

94 blocking JAK2 function increases detoxifying GSTs in hepatocytes and protects against oxidative liver

95 To investigate, we studied 16 different GSTs, revealing that negative cooperativity is present o

96 By examining 42 different GSTs we discovered that only the more recently evolved T

97 rality of this behavior across the divergent GST family and its evolutionary significance were unclea

98 Each GST is needed for accurate molecular diagnosis in differ

99 It revealed that elevated GST and heat loss from basements are dominant factors in

100 hanced dephosphorylation of recombinant ERK2-GST in an in vitro phosphatase assay.

101 ity is present only in more recently evolved GSTs, indicating evolutionary drift in this direction.

102 ylation catalyzed by a bacterially expressed GST-tagged human PRMT7 fusion protein with a broad range

103 Moreover, GST-B4 and, to a lesser extent, GST-B1 also inhibited gelatin degradation by MMP-9, indi

104 eins and could particularly detect extrinsic GST proteins added in crude Escherichia coli or 293T cel

105 s between subunits may be proposed for a few GSTs.

106 First, GST-DMAP pulled down several acid hydrolases, but not no

107 -2) and detection limit of 1.3 ng mL(-1) for GST-p16 protein which is equivalent to 0.49 ng mL(-1) fo

108 ectable GST concentration (200pmolL(-1)) for GST label-free sensors.

109 cis-acting regulatory elements required for GST-specific expression.

110 tional polymorphisms in the genes coding for GSTs alter their enzyme activity in vitro, and were repo

111 Furthermore, GST and SOD activities of trout exposed to both Se-Met a

112 Purified PP1c bound to recombinant Gbeta1-GST protein, and PP1c co-immunoprecipitated with Gbeta1

113 t Raman spectra of HfO2, TiO2 and Ge2Sb2Te5 (GST) films, and demonstrate direct measurements of tempe

114 e phases of the PCM section (here Ge2Sb2Te5 (GST)) allows for designing a tunable plasmonic switch fo

115 filled with phase-change material Ge2Sb2Te5 (GST).

116 cular basis of this inhibition, we generated GST fusion proteins containing PEX9 or truncated forms c

117 cifically for a presumed sensitive genotype (GST-theta-1+/+), and PBPK modeling accounting for human

118 , GTL; glucobrassicin, GBC; gluconasturtiin, GST; glucoalyssin, ALY; 4-hydroxyglucobrassicin, 4-OH; 4

119 cCPE.GST or GST (GST is glutathione S-transferase) was conjugated to the

120 ance of two glutathione transferases (GSTs), GST-U24 and GST-U25, from Arabidopsis (Arabidopsis thali

121 sotropy (kappaa/kappac~2) of kappa in bulk h-GST, with the dominant contribution to kappa from optic

122 nductivity (kappa) of hexagonal Ge2Sb2Te5 (h-GST) is studied via direct first-principles calculations

123 s to the thermal conductivity in low-kappa h-GST is unusual, and development of fundamental physical

124 The phonon spectrum of h-GST has very dispersive optic branches with higher group

125 dels for lower GST and validate under higher GST, with three calibration experiments.

126 However, GST-PEX9 did not prevent the degradation of other MMP-9

127 Similar effects were observed with human GST isozymes A1-1 and M1-1.

128 Using yeast two-hybrid, GST pull-down, co-immunoprecipitation and bimolecular fl

129 United States, Bla g 5 is the most important GST allergen and lack of coexposure to GSTs from certain

130 Importantly, GST-U25 catalyzed the denitration of TNT to form 2-gluta

131 re selected and genotypes were determined in GST classes Alpha, Mu, Theta and Pi by means of polymera

132 Functional genotypes in GST genes are not involved in EAC or ESCC susceptibility

133 uld play a role in negative cooperativity in GSTs.

134 PE.GST (3.17 +/- 0.51 %ID/g) but not (111)In-GST (0.99 +/- 0.39 %ID/g; P < 0.001).

135 .70 %ID/g, significantly higher than (111)In-GST (1.00 +/- 0.60 %ID/g; P < 0.05).

136 ice was significantly higher than in (111)In-GST or claudin-4-negative HT1080 tumors (6.72 +/- 0.18 v

137 at binds within the active site and inhibits GST activity.

138 zed immobilization for Schistosoma japonicum GST.

139 se new data, along with experimentally known GST reactions and structures reported in the literature,

140 Furthermore, full-length GST-mu1A and the GST-mu1A C-terminal domain, but not the

141 However, full-length GST-mu1A did not bind to the phospho-L-selectin tail or

142 All three Lig GSTs produced the ketone product (beta-S-glutathionyl-al

143 onstrate for the first time theta-class-like GST activity for GDAP1, and it's activity being regulate

144 terminal secretion of reporter proteins like GST and YopE lacking its native secretion signal.

145 allowed us to calibrate the models for lower GST and validate under higher GST, with three calibratio

146 ontrol experiments, we showed that mammalian GST-PRMT1 and Myc-PRMT5 were, unlike PRMT7, able to dime

147 ompletely blocked upon formation of the MDM2/GST-p53TAD complex owing to charge conversion.

148 indicating that Nutlin-3 disrupted the MDM2/GST-p53TAD complex, thereby releasing MDM2.

149 t is likely to be present also in microsomal GST-1 based on sequence similarity.

150 Moreover, GST-B4 and, to a lesser extent, GST-B1 also inhibited ge

151 Similarly, mGsta4, the murine GST with the highest catalytic efficiency for 4HNE, is d

152 This gave the sensitivity of 1.78 muA mL(ng GST-p16)(-1) cm(-2) and detection limit of 1.3 ng mL(-1)

153 maintain a residual conjugating activity of GST against toxins even in the presence of high DNDGIC c

154 tivity were both enhanced by the addition of GST-BGN.

155 Analysis of GST structures identified elements that could play a rol

156 A new fungal specific class of GST has been highlighted by genomic approaches.

157 y in plants, physiological concentrations of GST-U24 and GST-U25 result in only a limited innate abil

158 asily regenerated, allowing the detection of GST with the very same device.

159 is NO-mediated 59Fe efflux and the effect of GST P1-1 on preventing this were observed with NO-genera

160 Here, we investigated the effects of GST-BGN lacking any posttranslational modifications on B

161 By preparing truncated forms of GST-PEX9 containing structural blades B1B2 or B3B4, we h

162 Thus, the DNIC storage function of GST P1-1 and ability of MRP1 to efflux DNICs are vital i

163 By varying the crystallization level of GST from 0% to 90%, the Fabry-Perot resonance supported

164 optimal BMP-2 combined with 4 or 8 microg of GST-BGN.

165 BMP-2 combined with 0, 2, 4, or 8 microg of GST-BGN.

166 ive investigation, we constructed a panel of GST-Zinc finger (ZnF) array chimera from 21 selected ZAD

167 ty of cyclin D1/Cdk4 upon phosphorylation of GST-Rb.

168 Both the enzymatic proficiency of GST-TvCyP1 and its binding to His-Myb1 were eliminated b

169 However, a proof of GST activity and its possible impact on membrane dynamic

170 To assess the role of GST-U24 and GST-U25, we purified and characterized recom

171 e is known about the regulatory functions of GSTs.

172 As DNDGIC is also a strong inhibitor of GSTs, we suggest negative cooperativity might have evolv

173 acks a signal sequence but contains an omega GST fold.

174 cCPE.GST or GST (GST is glutathione S-transferase) was conjugated to

175 d not covalently react with UbcH7, USP08, or GST-tagged human rhinovirus 3C protease.

176 trate that Arabidopsis plants overexpressing GST-U24 and GST-U25 exhibit significantly enhanced abili

177 of toxic compounds against which particular GSTs provide protection include 4-hydroxynonenal and ort

178 g GAM, the cutoff value of 14.7 mug/L for pi-GST showed the best performance to predict composite out

179 The addition of pi-GST to the SOFA score improved risk stratification (tota

180 Urinary pi-GST differentiated patients with/without advanced AKI or

181 Thus, urinary pi-GST levels predict advanced AKI or hospital mortality af

182 Urinary pi-GST predicted advanced AKI at 3 hrs post-surgery (p = 0.

183 yzed for urinary alpha-(alpha-) and pi-(pi-) GSTs.

184 ype and pfhrp2-deleted parasite populations (GST = .046, p </= .00001).

185 y potential therapeutic targets, we prepared GST-PEX9 forms containing structural blades B1B2 or B3B4

186 lB, YidC, and LepI) and one soluble protein (GST), each fused to GFP, was examined.

187 Recombinant GST-theta with monochlorobimane substrate had an optimum

188 Furthermore recombinant GST-theta activity was abolished by the denaturants trit

189 The bonafide of 40.8 kDa recombinant GST+Alr1105 fusion protein was confirmed by immunoblotti

190 Products of recombinant GSTs (LigE, LigP, and LigF) are beta-S-glutathionyl-alph

191 domain prevents the formation of the regular GST dimer and acts as a lid, which closes upon glutathio

192 e broadly, implying a larger function for RO-GST protein partners.

193 suggest a potential role for M. rosenbergii GST-theta in detoxification and possibly conferring immu

194 m to discriminate among structurally similar GST isozymes.

195 ies with an elevation in levels of GSH, SOD, GST and t-GPx activities.

196 ass that we name GSTFuA, for fungal specific GST class A.

197 rization of enzyme- and active site-specific GST activity in mammalian model systems.

198 pairs, of the prototypical PCM, Ge2 Sb2 Te5 (GST).

199 he difference in growing season temperature (GST) varied between 2.2 and 8.2 degrees C in different t

200 ch as increased ground surface temperatures (GSTs) at artificial surfaces and heat losses from baseme

201 e Ca(2+)-dependent interaction of N-terminal GST-tagged RcCDPK1 with BTPC.

202 Accordingly, ELISAs demonstrated that GST-B4 and GST-B1 specifically bound to gelatin.

203 n the basis of these results we propose that GST evolution in higher organisms could be linked to the

204 The GST active site is composed of a GSH binding "G site" an

205 The GST)-effector pull down assay was used to study the pres

206 Furthermore, full-length GST-mu1A and the GST-mu1A C-terminal domain, but not the GST-mu1A N-termi

207 etic background, a Th1 polarization, and the GST-tag.

208 Changing the GST phase to crystalline via optical heating allows for

209 -dinitrobenzene (CDNB) and the drugs for the GST enzyme in the electrochemical potential at 0.1V vs.

210 bination of events previously unknown in the GST protein superfamily and potentially explaining the d

211 5, 6, 7, 11, 12, and 13 participated in the GST-catalyzed conjugation, indicating the substrate tole

212 tion with vWFA2 fused to intein (lacking the GST-tag).

213 the GST-mu1A C-terminal domain, but not the GST-mu1A N-terminal domain, bind to L-selectin tail pept

214 cal properties of the amorphous phase of the GST section we adjusted the extinction peak of the dipol

215 The sensor can quantify the GST enzyme concentration through its biospecific interac

216 overall capacitance values according to the GST concentration.

217 to the surface of the microspheres using the GST anchor.

218 fication of postprandial xenobiotics via the GST action mediated hepatic GSH conjugation.

219 related to the low or null affinity of their GSTs for DNDGIC.

220 the absence of TNT, overexpression of these GSTs reduces root and shoot biomass, and although glutat

221 inal features of the reaction cycle of these GSTs, including stereospecific substrate recognition and

222 mite were tested for IgE reactivity to these GSTs.

223 rization of glutathione s-transferase-theta (GST-theta) from freshwater prawn Macrobrachium rosenberg

224 onal properties lead us to propose that this GST belongs to a new class that we name GSTFuA, for fung

225 ties, and various molecules showed that this GST binds glutathionylated and sulfated compounds but al

226 anti-P3 antibody also inhibited adhesion to GST-PEX9 and proMMP-9.

227 CLL cells bound to GST-B1B2 and CD44 was the primary receptor.

228 y replaced by fusing the catalytic domain to GST to promote dimerization.

229 a short GW protein-derived peptide fused to GST and demonstrate that it binds to Ago proteins with h

230 ast ubiquitin-binding protein Dsk2, fused to GST.

231 ve moiety for UV-induced covalent binding to GSTs and GSH-binding enzymes.

232 lution but completely harmless when bound to GSTs.

233 rtant GST allergen and lack of coexposure to GSTs from certain species allows a better assessment of

234 in flower buds was specifically localized to GSTs.

235 mGstp1/2 deletion reduced total GST activity, but no compensatory increase in GSTA and G

236 vity was a significant fraction of the total GST activity.

237 ity occurs in human glutathione transferase (GST) GSTP1-1 when it binds and neutralizes a toxic nitri

238 itrosation of human glutathione transferase (GST) P1-1, a major detoxification enzyme and key regulat

239 e reductase (GR), Glutathione-S-Transferase (GST) activities, and reduced glutathione (GSH) content,

240 Glutathione S-transferase (GST) affinity pulldowns also suggest that p18 and Gag in

241 , catalase (CAT), glutathione-s-transferase (GST) and non-enzymatic antioxidant - reduced glutathione

242 he stimulation of glutathione S-transferase (GST) and superoxide dismutase (SOD) activities.

243 dismutase (SOD), glutathione S-transferase (GST) and total glutathione peroxidase (t-GPx) were decre

244 s evaluated using glutathione S-transferase (GST) as a model enzyme that utilizes two distinct substr

245 rsible label-free glutathione-S-transferase (GST) biosensor is demonstrated.

246 ch containing two glutathione-S-transferase (GST) domains.

247 were subjected to glutathione S-transferase (GST) E-cadherin pulldown and immunoblot analysis to asse

248 nt members of the glutathione-S-transferase (GST) enzyme family.

249 ties of bacterial glutathione-S-transferase (GST) enzymes that cleave beta-aryl ether bonds in lignin

250 e electrodes with glutathione-s-transferase (GST) enzymes.

251 Considering that glutathione S-transferase (GST) is a broadly employed enzymatic fusion tag, we repo

252 ity of functional glutathione S-transferase (GST) metabolic activity, the key activation pathway for

253 ified recombinant glutathione-S-transferase (GST) proteins and could particularly detect extrinsic GS

254 spectrometry and glutathione S-transferase (GST) pulldown assays identified integrin alpha5 as a nov

255 precipitation and glutathione S-transferase (GST) pulldown assays revealed that GBP1 interacted with

256 ction assays, and glutathione S-transferase (GST) pulldown assays, we show that NR2A subunits interac

257 tion and in vitro glutathione S-transferase (GST) pulldown assays.

258 By using glutathione S-transferase (GST) pulldowns, we identified an essential role of lysin

259 r, enzymes in the glutathione S-transferase (GST) superfamily function in stress responses, defense s

260 re the use of the glutathione s-transferase (GST) to anchor the bactericidal peptide, melittin, to th

261 hylase (EROD) and glutathione-S-transferase (GST), and (ii) the metabolic clearance of benzo(a)pyrene

262 us, activities of glutathione-S-transferase (GST), general esterases (ESTs) and phenol oxidase (PO) d

263 modifier (Sumo), glutathione S-transferase (GST), maltose-binding protein (MBP), N-utilisation subst

264 oxidative stress (glutathione-S-transferase (GST), superoxide dismutase (SOD)), and fish health (cond

265 lytic activity of glutathione-s-transferase (GST), which is not its natural enzyme partner.

266 n host cells with glutathione S-transferase (GST)-Asp14 significantly inhibited infection of host cel

267 bound directly to glutathione S-transferase (GST)-fused M3R intracellular loops 2 and 3 (M3Ri2 and M3

268 P particles, and glutathione S-transferase (GST)-P domain fusion proteins to sialic acid-containing

269 Glutathione S-transferase (GST)-RP2 pulled down Gbeta from retinal lysates and the

270 d the activity of glutathione S-transferase (GST).

271 and NdmE, a novel glutathione-S-transferase (GST).

272 lytic activity of glutathione-S-transferase (GST).

273 ic translation of glutathione S-transferase (GST-3) from constitutive mRNA levels in vivo is dependen

274 Glutathione S-transferases (GST) were evaluated as biomarkers of AKI.

275 e omega class of Glutathione S-transferases (GST), yet differ from them in their ability to form ion

276 Glutathione transferases (GSTs) are protection enzymes capable of conjugating glut

277 importance of two glutathione transferases (GSTs), GST-U24 and GST-U25, from Arabidopsis (Arabidopsi

278 enzymes such as Glutathione S-Transferases (GSTs ) detoxify mutagenic and genotoxic compounds and th

279 Although glutathione S-transferases (GSTs) are thought to play major roles in oxidative stres

280 Glutathione S-transferases (GSTs) comprise a diverse family of phase II drug metabol

281 sensitization to glutathione S-transferases (GSTs) occurs in tropical and subtropical environments.

282 nooxygenases and glutathione S-transferases (GSTs) was detected in populations from both central and

283 and activity of glutathione S-transferases (GSTs).

284 ties of GDAP1 to glutathione S-transferases (GSTs).

285 localized to glandular secretory trichomes (GSTs) of leaf primordia and top expanded leaves.

286 s parallels with similar roles for unrelated GSTs in MDR in humans and shows their potential as targe

287 Using GST-CTD fusion protein substrates we find that CDK12/Cyc

288 In addition, using GST affinity purification combined with mass spectrometr

289 l and the TM 4,5-loop was demonstrated using GST pulldown assays.

290 By utilizing GST pull-down and immunoprecipitation assays, we validat

291 forms a stable complex with DAT in vivo via GST pulldown and co-immunoprecipitation assays.

292 interacts with ERK1/2 by using both in vitro GST pulldown and in vivo co-immunoprecipitation assays.

293 Moreover, preincubating host cells with GST-tagged forms of both Asp14 and outer membrane protei

294 s of individual domains and comparisons with GST classes suggest that TDR1 evolved by gene duplicatio

295 a free protein or as a fusion construct with GST, is monomeric and adopts a disordered conformation a

296 xperimental EBA induced by immunization with GST-COL7, disease manifestation depended on the genetic

297 B-CLL cell incubation with GST-PEX9 induced intracellular survival signals, namely

298 ell invasion, but increased interaction with GST-B-Raf as compared with wild-type-FLAG-MLK3 in H2O2-t

299 Notably, 59Fe accumulated in cells within GST P1-1-containing fractions, indicating an alteration

300 proline-rich motif reduced SFK binding to WT GST-TRAF6 compared with the Pro --> Ala-substituted pept