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1 increase in the precursor activity of serum Gc protein.
2 -acetylgalactosaminidase that deglycosylates Gc protein.
3 ion with precursor activities of their serum Gc protein.
5 8-kilobase transcript on Northern blots, and GCS protein, a approximately 38-kDa polypeptide detected
6 h was preceded by the depletion of the gamma-GCS protein and RNA and by the loss of the cellular pool
8 found to be due to deglycosylation of plasma Gc protein by alpha-N-acetylgalactosaminidase detected i
9 analysis and the ER relocation of endogenous GC proteins by phospholipase A2 inhibitors demonstrate t
12 atients with low precursor activity of serum Gc protein had high serum alpha-N-acetylgalactosaminidas
13 idase activity and MAF precursor activity of Gc protein in patient bloodstream can serve as diagnosti
15 -binding protein (DBP; human DBP is known as Gc protein) is the precursor of macrophage activating fa
16 versus 298 +/- 90, respectively; P = 0.004); GC protein levels decreased with increasing severity of
20 ivating factor requires a precursor protein, Gc protein (serum vitamin D3-binding protein), as well a
21 emonstrate that Golgi-ER cycling of resident GC proteins takes place in both myoblasts and myotubes.
23 the MAF precursor activity of patient plasma Gc protein was found to be severely reduced in about 25%
25 sing of BUNV GPC and found that both NSm and Gc proteins were cleaved at their own internal signal pe
26 zed beta-galactosidase or treatment of human Gc protein with immobilized beta-galactosidase and siali
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