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1 ere positively selected in medium containing geneticin.
2 nce of high concentrations of the antibiotic geneticin.
3 thine (d-Orn), a substrate analog, and G418 (Geneticin), a weak non-competitive inhibitor, was determ
5 ht PTC-containing XP-C cells, treatment with Geneticin and gentamicin resulted in (i) stabilized XPC-
8 Chinese hamster ovary cells, selection with Geneticin, and amplification with methotrexate, a cell l
10 for selection of stable transfectants using Geneticin, and the presence of plasmid in transfectants
11 2-DOS aminoglycosides paromomycin and G418 (geneticin) are compared, using both human and Escherichi
12 om complexes of paromomycin, tobramycin, and Geneticin bound to an A-site oligonucleotide, and paromo
15 aminoglycoside antibiotic read-through agent geneticin (G418) across a diverse range of in vitro repo
16 vidence is now available that gentamicin and Geneticin (G418) can induce the mammalian ribosome to su
17 found that doxycycline, chloramphenicol, and Geneticin (G418) interfered with insertion of selenocyst
19 a model of the Leishmania ribosomal A site: Geneticin (G418), a potent aminoglycoside for the treatm
20 he 80S ribosome in complex with paromomycin, geneticin (G418), gentamicin, and TC007, solved at 3.3-
25 rt that paromomycin and the related compound geneticin manifest their major in vitro anti-C. parvum a
26 ponsiveness to the readthrough-inducing drug geneticin of 11 rationally selected factor IX (FIX) nons
27 two aminoglycoside antibiotics, neomycin and Geneticin, on the endocytic pathway were studied using a
30 slation fidelity antibiotics paromomycin and geneticin, to high salt and calcium concentrations, to p
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