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1 the Apis mellifera karyotype as revealed by Giemsa stain.
2 immunofluorescence, electron microscopy, and Giemsa staining.
3 by flow cytometric analysis and May-Grunwald Giemsa staining.
4 Biopsy-based tests (i.e., culture, histology Giemsa stain and rapid urease test) and non-invasive tes
6 ted PCR assay and microscopic examination of Giemsa-stained blood films for detection and identificat
7 mated blood culture, malaria microscopy with Giemsa-stained blood films, and human immunodeficiency v
8 was as sensitive and specific as the use of Giemsa-stained blood smears and inoculation of hamsters.
9 d objective supplement to the examination of Giemsa-stained blood smears and may replace microscopy f
10 ts for duplicate microscopic examinations of Giemsa-stained blood smears as the reference diagnostic
14 roperties of cells, morphological studies of Giemsa-stained cells, annexin V binding, and DNA fragmen
15 hin 2 weeks, infected cells were detected in Giemsa-stained culture samples, and the organisms subseq
17 osinophils, they were purified (>95% pure by Giemsa-stained cytospin preparations) from liver granulo
19 itivity of 54% and a specificity of 87%; and Giemsa stain (> 2% ICO) had a sensitivity of 46% and a s
20 nal method, i.e., microscopic examination of Giemsa-stained lesion scraping (46.7%), biopsy culture (
23 ted 4 to 5 hours earlier than it was seen in Giemsa-stained preparations and 8 hours earlier than it
28 icroscopic identification of the organism in Giemsa-stained thin blood smears, detection of babesial
29 s with atrophy, the sensitivity of histology Giemsa stain was 100%, 100%, 88%, and 66%, respectively,
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