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1 ert's syndrome vs 461 [15%] who did not have Gilbert's syndrome).
2 ether or not they had laboratory evidence of Gilbert's syndrome.
3 roximately 10% of the population manifesting Gilbert's syndrome.
4 he (TA)7 allele was considered diagnostic of Gilbert's syndrome.
5 ia in liver transplant recipients was due to Gilbert's syndrome acquired through the liver allograft.
6                        211 (6%) patients had Gilbert's syndrome and 3168 (94%) did not have this cond
7 ndrome vs 617 [19%] of 3168 who did not have Gilbert's syndrome), and for non-relapse mortality 499 (
8  recurrent jaundice patients (with suspected Gilbert's syndrome), and nine clinically diagnosed cases
9 rwent a 24 h fasting test to see if they had Gilbert's syndrome, and all four positives had the 7/7 g
10 A1 promoter variant sequence associated with Gilbert's syndrome, and the fourth was a heterozygote.
11 xistence of a mild and a more severe form of Gilbert's syndrome, depending on whether the gene defect
12 from 12 patients with confirmed or suspected Gilbert's syndrome, from 6 members of a family with 4 Gi
13     Upon reactive pathway progression, as in Gilbert's Syndrome (GS; UGT1A1*28 polymorphism), aggrava
14  melphalan conditioning regimens, those with Gilbert's syndrome had similar outcomes to those without
15 curonosyltransferase-1A1 deficiency, causing Gilbert's syndrome, has been attributed to two extra (TA
16                                              Gilbert's syndrome is a common inherited disorder of bil
17                         The genetic basis of Gilbert's syndrome is ill-defined.
18 east milk jaundice, and the realization that Gilbert's syndrome may play a greater role in neonatal j
19 with low UGT1A1 activity, such as those with Gilbert's syndrome, may be at an increased risk for irin
20 curonosyltransferase-1A1 deficiency, causing Gilbert's syndrome, may be carried by the donor liver an
21 ality by day 200 compared with those without Gilbert's syndrome (n=1071; hazard ratio [HR] 2.30, 95%
22 that contained busulfan (n=1131), those with Gilbert's syndrome (n=60) were at a significantly increa
23                       However, the effect of Gilbert's syndrome on the disposition of some drugs can
24                                The effect of Gilbert's syndrome on the risk of overall mortality and
25 ndrome had similar outcomes to those without Gilbert's syndrome (overall mortality at day 200 HR 0.90
26 ive conditioning regimens, compared with non-Gilbert's syndrome patients.
27                                Patients with Gilbert's syndrome should receive busulfan-containing my
28 efore relapse was recorded (38 [18%] who had Gilbert's syndrome vs 461 [15%] who did not have Gilbert
29 00 after transplant (47 [22%] of 211 who had Gilbert's syndrome vs 617 [19%] of 3168 who did not have
30                                Patients with Gilbert's syndrome were defined as having laboratory val
31 dition associated with jaundice in adults is Gilbert's syndrome, which is characterized by an allelic
32 nt were significantly worse in patients with Gilbert's syndrome who received busulfan-containing myel
33               We assessed the association of Gilbert's syndrome with overall mortality and non-relaps

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