ライフサイエンスコーパス: Goldman

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1 Goldman shows that such feedback is not necessary for me

2 Goldman-Rakic discovered the circuit basis for working m

3 ion, we distinguished area 6Ds of Preuss and Goldman-Rakic between PMV and PMDc, and a more posterior

4 ls, Pf was approximately 17% as predicted by Goldman-Hodgkin-Katz (GHK) theory.

5 A new study by Goldman et al. has now provided the first evidence that

6 a2+ channels and can be described by classic Goldman flux analysis with a Ca2+/Cs+ selectivity of the

7 etter estimate these localized ion currents, Goldman-Hodgkin-Katz (GHK) theory was used to model ion

8 tance (reversal, -95+/-4 mV) which displayed Goldman-Hodgkin-Katz outward rectification, supportive o

9 eurons and symmetrical chloride to eliminate Goldman rectification, which results in chloride flow in

10 ntial to extreme negative values, and follow Goldman-Hodgkin-Katz constant field equation at extreme

11 Its permeability to Na(+), estimated from Goldman-Hodgkin-Katz voltage equation, was negligible.

12 for channel selectivity by using a modified Goldman-Hodgkin-Katz (GHK) configuration that employs vo

13 is perspective, we summarize the findings of Goldman et al. and discuss the prospect that nanoRNA-med

14 hermore, this correlation was independent of Goldman rectification and persisted under altered chlori

15 ences (> 600 nucleotides), and the method of Goldman et al. for shorter sequences (< 600 nucleotides)

16 U and emergency department patients, and one Goldman's Class I unexpected finding in the ward patient

17 When Pat Goldman-Rakic described the circuitry and function of pr

18 diac complications, according to the revised Goldman index, who were treated for at least 36 hrs in t

19 on an alter ego of the stem cell itself (S. Goldman, this issue of TINS).

20 occurring model of enhanced S-cone syndrome, Goldman-Favre syndrome and clumped pigmentary retinopath

21 retina, a model of enhanced S-cone syndrome, Goldman-Favre syndrome, and clumped pigmentary retinopat

22 ng SPQ fluorescence quenching by I-) and the Goldman flux equation (for modeling the electrodiffusive

23 r clinical diagnoses were categorized by the Goldman method and compared with post mortem findings to

24 ale, and family history as quantified by the Goldman Scale.

25 o +30 mV, which could be approximated by the Goldman-Hodgkin-Katz current equation.

26 on properties that could be described by the Goldman-Hodgkin-Katz current equation.

27 ion that is reasonably well described by the Goldman-Hodgkin-Katz current equation.

28 A modification of the Goldman criteria was used to classify diagnostic error.

29 REV) followed closely the predictions of the Goldman-Hodgkin-Katz constant field equation at low conc

30 abilities were estimated on the basis of the Goldman-Hodgkin-Katz equation as follows: PH:PK:PNa = 40

31 or polarized epithelia the equivalent of the Goldman-Hodgkin-Katz equation for single isolated cells.

32 inations of Em by previous work based on the Goldman-Hodgkin-Katz equation and its derivatives and th

33 Because rTASK currents satisfy the Goldman-Hodgkin-Katz current equation for an open channe

34 cardial infarction < or =7% according to the Goldman algorithm, no biochemical evidence of infarction

35 Finally, we used the Goldman-Hodgkin-Katz equation to examine how EGABA would

36 e model the device characteristics using the Goldman-Hodgkin-Katz (GHK) solution to the Nernst-Planck

37 (-)-free Ringer solution consistent with the Goldman-Hodgkin-Katz (GHK) equation.

38 Though Goldman applanation tonometry (GAT) is the "gold standar

39 There were two Goldman's Class I unexpected findings in the pediatric I

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