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1 dantly in pectin methylesterification in the Golgi apparatus.
2 arked by Rab5, Rab7 and Rab9, as well as the Golgi apparatus.
3 GE synthase-1 (mPGES-1) but not COX-1 in the Golgi apparatus.
4 eases AAV2 transduction and transport to the Golgi apparatus.
5 IP probes, it triggered fragmentation of the Golgi apparatus.
6 ticipate in membrane-tethering events at the Golgi apparatus.
7 olved in heparan sulfate substitution in the Golgi apparatus.
8 d abnormal swelling and fragmentation of the Golgi apparatus.
9 downstream docking and fusion events at the Golgi apparatus.
10 autophagic response that required an intact Golgi apparatus.
11 g protein located at both the centrosome and Golgi apparatus.
12 he activity of glycosyltransferases from the Golgi apparatus.
13 nized interphase distribution of Mad1 at the Golgi apparatus.
14 tributions with varying association with the Golgi apparatus.
15 two distinct populations of vesicles at the Golgi apparatus.
16 rane--xylan is synthesized by enzymes in the Golgi apparatus.
17 e secretory pathways and was retained in the Golgi apparatus.
18 the organization of membrane traffic at the Golgi apparatus.
19 oteins from the endoplasmic reticulum to the Golgi apparatus.
20 nsferases and glycosyl hydrolases within the Golgi apparatus.
21 inds phosphatidylinositol 4-phosphate in the Golgi apparatus.
22 needed for glycosylation in the lumen of the Golgi apparatus.
23 newly synthesized proteins and lipids to the Golgi apparatus.
24 s shuttled away from phagosomes and into the Golgi apparatus.
25 ed Ca(2+) transport pathway localized in the Golgi apparatus.
26 e core in the endoplasmic reticulum (ER) and Golgi apparatus.
27 is demonstrated here to be localized at the Golgi apparatus.
28 bidirectional traffic between the ER and the Golgi apparatus.
29 es that regulate cargo transport through the Golgi apparatus.
30 alpha/beta-subunit precursor protein in the Golgi apparatus.
31 is trapped in the endoplasmic reticulum and Golgi apparatus.
32 uality control of proteins in the ER and the Golgi apparatus.
33 lucosylceramide (GlcCer) in the lumen of the Golgi apparatus.
34 termediate compartment that matures into the Golgi apparatus.
35 nd that could be exchanged for sterol in the Golgi apparatus.
36 trograde transport of endosomes to the trans-Golgi apparatus.
37 herin is not degraded but accumulates in the Golgi apparatus.
38 regulated by reversible association with the Golgi apparatus.
39 isassembly and cPLA(2)alpha release from the Golgi apparatus.
40 arly secretory pathway, primarily within the Golgi apparatus.
41 transferase reactions in the cytosol and the Golgi apparatus.
42 g a giant secretory vesicle derived from the Golgi apparatus.
43 aled that SAG113 protein is localized in the Golgi apparatus.
44 mutation, Kit localizes predominantly on the Golgi apparatus.
45 s cPLA(2)alpha from the adhesion complex and Golgi apparatus.
46 that proteins routed to CRs pass through the Golgi apparatus.
47 ifications that are expected to occur in the Golgi apparatus.
48 als abnormal accumulation of HP1alpha in the Golgi apparatus.
49 of INF2-non-CAAX causes fragmentation of the Golgi apparatus.
50 brane vesicles that transport cargo from the Golgi apparatus.
51 trapped in the cytoplasm associated with the Golgi apparatus.
52 to engorgement of the endoplasmic reticulum/Golgi apparatus.
53 eavage of the gp160 precursor protein in the Golgi apparatus.
54 thway regulating precise localization of the Golgi apparatus.
55 subunit, which binds ganglioside GM1, to the Golgi apparatus.
56 ction in vesicle movement from the ER to the Golgi apparatus.
57 suggestive of the endoplasmic reticulum and Golgi apparatus.
58 eins that regulate CSC assembly in the plant Golgi apparatus.
59 rt between the endoplasmic reticulum and the Golgi apparatus.
60 s selective defects both in mitochondria and Golgi apparatus.
61 ains, while the LamG domain localized to the Golgi apparatus.
62 ibuted to a bypass or a hypo-function of the Golgi apparatus.
63 ls, coincident with the flagellar pocket and Golgi apparatus.
64 the endoplasmic reticulum and matures in the Golgi apparatus.
65 rect role in GPI anchor glycosylation in the Golgi apparatus.
66 E1 interacts and colocalizes with APP in the Golgi apparatus.
67 ce of these connections linking WPBs and the Golgi apparatus.
68 x phase polysaccharides are assembled in the Golgi apparatus.
69 n within the highly dynamic membranes of the Golgi apparatus.
70 lycoproteins by sequestering them within the Golgi apparatus.
71 t (ERQC), and, surprisingly, recently to the Golgi apparatus.
72 VPS13B gene, which encodes a protein of the Golgi apparatus.
73 s from the endoplasmic reticulum (ER) to the Golgi apparatus.
74 sponse that in mammalian cells relies on the Golgi apparatus.
75 -biological ingredients needed to generate a Golgi apparatus?
76 particles and disrupts the structure of the Golgi apparatus, a key cellular organelle involved in se
80 ed in transporting cytoplasmic zinc into the Golgi apparatus and a ZnT7-containing vesicular compartm
81 plex is formed at, and transits through, the Golgi apparatus and also exists as a soluble complex in
82 was due to their pronounced retention in the Golgi apparatus and also to their rapid internalization
86 rting protein that binds ligands both in the Golgi apparatus and at the plasma membrane and traffics
87 ated vesicles mediate trafficking within the Golgi apparatus and between the Golgi and the endoplasmi
88 protein that resides on the cis face of the Golgi apparatus and binds alpha-SNAP-like proteins, but
89 pressed in HeLa cells, can both fragment the Golgi apparatus and block secretion, whereas viral infec
92 s of the cell extract, Endoplasmic Reticulum/Golgi apparatus and conditioned medium of T24 vs. its me
93 ng of proteins are critical functions of the Golgi apparatus and depend on its highly complex and com
94 that unlike oBST2A, oBST2B is limited to the Golgi apparatus and disrupts JSRV envelope (Env) traffic
95 ail to undergo proteolytic processing in the Golgi apparatus and drive IFN-gamma-induced gene express
97 the primary cell wall, is synthesized in the Golgi apparatus and exported to the cell wall in a highl
98 phosphatidylinositol 4-phosphate within the Golgi apparatus and failure to maintain residence of a m
99 ated vesicles mediate trafficking within the Golgi apparatus and from the Golgi to the endoplasmic re
100 causes an ERK-dependent fragmentation of the Golgi apparatus and inhibits Golgi polarization and dire
102 tially sorted into different vesicles in the Golgi apparatus and inserted into distinct domains of th
103 interacting (GORAB) protein localizes to the Golgi apparatus and interacts with the small GTPase RAB6
105 to an increase in cytoplasmic, cytoskeletal, Golgi apparatus and lipid metabolism proteins associated
106 This protein was shown to locate on the Golgi apparatus and mainly catalyze the Nt-acetylation o
107 with incorrectly positioned centrosomes and Golgi apparatus and mislocalized molecules of the slit d
108 clumping of mitochondria, disruption of the Golgi apparatus and missorting of synaptic proteins.
109 ellular matrix, and for the formation of the Golgi apparatus and organization of F-actin bundles in m
110 a golgin that localizes predominantly at the Golgi apparatus and physically interacts with small guan
111 The same complexes were detected both in Golgi apparatus and plasma membrane by using FRET micros
113 yme indicates that GXMT1 is localized in the Golgi apparatus and requires Co(2+) for optimal activity
115 bitor of these processes that resides in the Golgi apparatus and shows higher prenatal brain expressi
118 on of viral particles are transported to the Golgi apparatus and that Golgi apparatus disruption caus
119 (YFP) fusion of BdCSLF6 is localized to the Golgi apparatus and that the Golgi localization of BdCSL
120 ed a higher level of colocalization with the Golgi apparatus and the endoplasmic reticulum (ER).
121 vesicles that transport contents between the Golgi apparatus and the endoplasmic reticulum, and they
124 diates bidirectional trafficking between the Golgi apparatus and the plasma membrane, but nothing is
125 veal that the sorting of ion channels at the Golgi apparatus and their subsequent trafficking by uniq
126 , which promptly accumulated in the cellular Golgi apparatus and then translocated to the extracellul
127 tomannan probably occurs in the lumen of the Golgi apparatus and thus contrasts with the biosynthesis
129 predicts the size of experimentally measured Golgi apparatus and vacuole abundance fluctuations, sugg
130 flammasomes required its localization to the Golgi apparatus and was dependent on the pH gradient.
133 g is found in the endoplasmic reticulum, the Golgi apparatus, and at the plasma membrane but not the
135 neoformans species complex, localized to the Golgi apparatus, and functions in the O-glycosylation of
136 t localizes to organelles, in particular the Golgi apparatus, and has a preference for acetylating N
139 se of dopamine release, the fragmentation of Golgi apparatus, and the impairments of autophagy/lysoso
140 ed in the endoplasmic reticulum (ER) and the Golgi apparatus, and the lateral motility of apoE4 was s
141 icking from the endoplasmic reticulum to the Golgi apparatus, and the nuclear pore complex (NPC), whi
142 ergo also prevents export from the ER to the Golgi apparatus, and this traffic block results in break
143 surface of mitochondria, peroxisomes, or the Golgi apparatus, and thus prevention of plasma membrane
145 ulum (ER)-to-Golgi intermediate compartment, Golgi apparatus, and trans-Golgi network form a ring tha
147 D1 system surveys the endoplasmic reticulum, Golgi apparatus, and/or secretory compartments, in addit
150 ntified the intermediate compartment and the Golgi apparatus as the precursors of the replication "or
151 olled by various HS sulfotransferases in the Golgi apparatus as well as extracellular 6-O-endosulfata
152 tracellular APOB-containing particles in the Golgi apparatus as well as extracellular LDL at the plas
153 tubule organizing centre, or centrosome, the Golgi apparatus, associated endosomes and the nucleus du
154 ligases localize to the nucleus, centrosome, Golgi apparatus, axon and dendrite cytoskeleton, and syn
157 interactions as well as repositioning of the Golgi apparatus, both of which can be controlled by the
158 chondria, endoplasmic reticulum, nucleus and Golgi apparatus but its functional role remains largely
159 lity of apoE4 was significantly lower in the Golgi apparatus (but not in the ER) than that of apoE3.
161 t enables prolonged live-cell imaging of the Golgi apparatus by 3D confocal and STED microscopy.
162 synthesized in the endoplasmic reticulum and Golgi apparatus by a series of enzymatic transformations
163 abidopsis thaliana, is biosynthesized in the Golgi apparatus by a series of glycan synthases and glyc
164 rmore, FAM21 prevents cargo transport to the Golgi apparatus by controlling levels of phosphatidylino
165 on of proteins and lipids takes place in the Golgi apparatus by the consecutive actions of functional
166 ifications are initiated subsequently in the Golgi apparatus by the UDP-GalNAc polypeptide N-acetylga
167 oform coatomer protein complex zeta2, caused Golgi apparatus collapse, blocked autophagy, and induced
168 d that human fVIII was notably absent in the Golgi apparatus, confirming that endoplasmic reticulum t
169 toward the trans-Golgi network (TGN) and the Golgi apparatus correlates with transduction efficiency
170 of the N-glycosylation machinery within the Golgi apparatus; deletion of either gene results in N-gl
171 PR:MHC class I complex trafficks through the Golgi apparatus, demonstrating a function of TAPBPR beyo
172 ation of Gbetagamma, a PKD activator, to the Golgi apparatus, determined by bioluminescence resonance
173 2014) show that the size and topology of the Golgi apparatus determines the size and functionality of
175 of cells with HRV16 also caused significant Golgi apparatus dispersal; however, this did not result
176 transported to the Golgi apparatus and that Golgi apparatus disruption caused by the drug brefeldin
177 udies; consequently, to address the issue of Golgi apparatus disruption for HRV16, we have systematic
178 3A protein from HRV14 or HRV2 did not cause Golgi apparatus disruption or a block in secretion, wher
179 that infection of cells with HRV1A did cause Golgi apparatus disruption which was replicated by the e
180 ture, cellular location, and function of the Golgi apparatus during male germ cell differentiation is
187 the functional relevance of transport to the Golgi apparatus for AAV transduction remains to be estab
190 ound adjacent to the single ER exit site and Golgi apparatus, forming both stable and dynamic associa
191 atively examined the effect of HRV16 on both Golgi apparatus fragmentation and protein secretion in H
192 ll polysaccharides are biosynthesized in the Golgi apparatus from cytosolic-derived nucleotide sugars
193 ng proteins to the endoplasmic reticulum and Golgi apparatus from membrane-bound ribosomes were not t
194 ell wall polysaccharides are produced in the Golgi apparatus from nucleotide sugars that are predomin
197 ral disruption and cisternal swelling of the Golgi apparatus (GA) in the cortex of AS (UBE3A(m-/p+))
198 ndritic translation, most dendrites lack the Golgi apparatus (GA), an essential organelle for convent
201 PAT4 is predominantly associated with the Golgi apparatus in a range of cell types, and in situ pr
202 , we provide evidence for a dual role of the Golgi apparatus in controlling the size of these secreto
205 glycosylated, is partially retained into the Golgi apparatus in vitro, and disrupts intercellular adh
206 nerate diacylglycerol (DAG) from PI4P in the Golgi apparatus, in close proximity to the nuclear envel
207 es of posttranslational modifications in the Golgi apparatus, including attachment of carbohydrate mo
208 m (without endoplasmic reticulum stress) and Golgi apparatus, increased vesicular trafficking, and a
209 However, invasive pathogens can disrupt the Golgi apparatus, interfering with secretion and compromi
210 In mammalian cells, the inheritance of the Golgi apparatus into the daughter cells during each cycl
211 taxin 5-mediated retrograde transport to the Golgi apparatus is a broadly conserved feature of AAV tr
215 on of the Arabidopsis (Arabidopsis thaliana) Golgi apparatus is currently reasonably well documented;
217 at in SCYL1-deficient human fibroblasts, the Golgi apparatus is massively enlarged, which is in line
221 ss through the endoplasmic reticulum and the Golgi apparatus - is one of the most frequent protein mo
222 erized regulator of vesicle formation at the Golgi apparatus, is also a component of the synaptic rib
223 oteins from the endoplasmic reticulum to the Golgi apparatus, leading to the lysosomal degradation of
224 unique view on the organization of the plant Golgi apparatus, leading toward unique hypotheses center
225 P and cathepsin B was observed in a distinct Golgi apparatus-like pattern, which required a 1-h OA tr
229 the mode of cytosolic Naa60 anchoring to the Golgi apparatus, most likely occurring post-translationa
232 b) induction of morphological changes of the Golgi apparatus of plant and mammalian cells, and (c) in
233 Q6 was located within cell processes and the Golgi apparatus of renal glomerular podocytes and in str
234 affics through the endoplasmic reticulum and Golgi apparatus on its way to the cell membrane and is h
238 r, localize to endoplasmic reticulum and the Golgi apparatus, presumably through the recycling endoso
239 is traffic block results in breakdown of the Golgi apparatus, primarily due to maintenance of the con
240 ortholog of the gene encoding human STK16, a Golgi apparatus protein kinase with undefined function.
242 In filamentous fungi, organization of the Golgi apparatus reflects the unique challenges brought a
243 ve shown that binding of UL20 to GODZ in the Golgi apparatus regulates trafficking of UL20 and its su
244 red Golgi morphology and fewer cisternae per Golgi apparatus relative to wild-type cells, indicative
246 point that a spatially organized and dynamic Golgi apparatus represents an adaptation that is as impo
247 se genes, TbGT11, and show that it encodes a Golgi apparatus resident UDP-GlcNAc:alpha3-D-mannoside b
248 equence and prodomain cleavage in the ER and Golgi apparatus, respectively, activates their adhesive
249 tion of the spatial separation of the ER and Golgi apparatus restored cleavage of ATF6alpha in the pr
251 However, electron micrographs of WPBs at the Golgi apparatus show that these forming WPBs contain ver
252 in RAB7L1 or LRRK2 led to endolysosomal and Golgi apparatus sorting defects and deficiency of the VP
253 UL20 binds to GODZ (also known as DHHC3), a Golgi apparatus-specific Asp-His-His-Cys (DHHC) zinc fin
254 ly to GODZ (also known as DHHC3), a cellular Golgi apparatus-specific Asp-His-His-Cys (DHHC) zinc fin
255 nsport from the endoplasmic reticulum to the Golgi apparatus, suggesting that P2Y(2)R/filamin-mediate
256 trafficking route differs from that of known Golgi apparatus-targeted cargos, and we raise the possib
257 BLOC-1 interactions with the COG complex, a Golgi apparatus tether, and antioxidant enzymes peroxire
259 phosphatase of the endoplasmic reticulum and Golgi apparatus that controls organelle membrane composi
260 er variable delay times, are captured by the Golgi apparatus that is reached either by random diffusi
261 of onset and evolution of PNG components in Golgi apparatus that shaped diversity of eukaryotic prot
262 organelles (early endosomes, lysosomes, the Golgi apparatus, the endoplasmic reticulum or the nucleu
263 ellular compartment at the trans side of the Golgi apparatus, the TOR-autophagy spatial coupling comp
264 , from the endoplasmic reticulum (ER) to the Golgi apparatus, thereby triggering their secretion by e
265 me of the GmExo70J proteins are localized to Golgi apparatus through a novel N-terminal transmembrane
267 al stages of membrane trafficking, including Golgi apparatus to endosome and vacuole, peroxisomal fis
268 enous TLR3 is transported through the ER and Golgi apparatus to endosomes, where it is rapidly cleave
269 o form a multiprotein complex that links the Golgi apparatus to F-actin, which regulates muscle integ
270 Notch1 is first cleaved by furin in the Golgi apparatus to produce the biologically active heter
271 of casein kinase 1alpha (CK1alpha) from the Golgi apparatus to the cytoplasm where CK1alpha could ph
272 is essential in vesicle trafficking from the Golgi apparatus to the endoplasmic reticulum (ER) throug
273 to retrograde vesicular trafficking from the Golgi apparatus to the endoplasmic reticulum in interpha
277 holera toxin from the plasma membrane to the Golgi apparatus, Tpcn2(-/-) MEFs show slower kinetics of
278 at label the nucleus, endoplasmic reticulum, Golgi apparatus, trans-Golgi network, plasma membrane, a
280 ng the G2-M transition, the highly organized Golgi apparatus undergoes reversible fragmentation throu
282 liana) membrane proteome with a focus on the Golgi apparatus using localization of organelle proteins
283 nsists of a stable endoplasmic reticulum and Golgi apparatus, using discrete transport vesicles to ex
284 cytoplasmic domain targets receptors to the Golgi apparatus, vesicular structures, and the cell surf
285 ganelle fractionation approach targeting the Golgi apparatus was combined with a label-free quantitat
286 e neurogenic zone were eliminated, a compact Golgi apparatus was positioned exclusively at the base o
287 best-known examples is that of the mammalian Golgi apparatus, where constant inward movement of Golgi
288 sol, it is transported into the lumen of the Golgi apparatus, where it is converted to UDP-galacturon
289 on of this polytopic membrane protein is the Golgi apparatus, where its accumulation is strictly regu
290 and 2-furoyl-LIGRLO-NH2 activated PKD in the Golgi apparatus, where PKD regulates protein trafficking
291 LR and PDGFRbeta are then transported to the Golgi apparatus, where those complexes trigger Galphai3-
292 tUXS1, AtUXS2, and AtUXS4 are located in the Golgi apparatus whereas AtUXS3, AtUXS5, and AtUXS6 are l
294 in cell lines, mutant Kit accumulates on the Golgi apparatus, whereas normal Kit localizes to the pla
295 onsistent with virion assembly in the medial Golgi apparatus, whereas oligomannose-type glycans requi
296 tin ligase for c-Met, was sequestered in the Golgi apparatus with alphabeta integrin, resulting in th
297 nhibition of COPI complex recruitment to the Golgi apparatus with pharmacological compounds failed to
298 Isoforms Tm1J and Tm2A colocalize around the Golgi apparatus with the formin-family protein Diaphanou
299 ex regulates cPLA(2)alpha recruitment to the Golgi apparatus, with functional consequences for vascul
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