ライフサイエンスコーパス: Golgi compartment

1 lowed by subsequent lipidation in the ER and Golgi compartment.

2 ransport of G(AE) in a Rab43-positive medial Golgi compartment.

3 ocalization of hGIIA to the cell surface and Golgi compartment.

4 icles or a merged endoplasmic reticulum (ER)-Golgi compartment.

5 ing pathway initiated by ATP uptake into the Golgi compartment.

6 mplicated in retrograde transport to the cis-Golgi compartment.

7 for CHIP in protein trafficking through the Golgi compartment.

8 E is inactive until it reaches this terminal Golgi compartment.

9 catalyzes the palmitoylation of PRCD in the Golgi compartment.

10 palmitoylated, in either the Golgi or a post-Golgi compartment.

11 RA1 co-localizes with Ha-Ras and RhoA in the Golgi compartment.

12 nomeric form within the late Golgi or a post-Golgi compartment.

13 but only partial retention of K(b), in a pre-Golgi compartment.

14 nant VIIMT was partially degraded in the pre-Golgi compartment.

15 f pro-alphaf is also initiated in a distinct Golgi compartment.

16 olgi and is cleaved and secreted from a post-Golgi compartment.

17 al to the endoplasmic reticulum from the pre-Golgi compartment.

18 SV)-G protein to a mannosidase II-containing Golgi compartment.

19 rgo protein to the mannosidase II-containing Golgi compartment.

20 he ER-Golgi intermediate compartment and cis-Golgi compartment.

21 quired for protein sorting at the yeast late Golgi compartment.

22 e two mutated proteins to concentrate in the Golgi compartment.

23 embrane protein located predominantly in the Golgi compartment.

24 a serine endoprotease localized to the late Golgi compartment.

25 fusion of recycling vesicles with the trans-Golgi compartment.

26 e protein have been further processed in the Golgi compartment.

27 sicles must target to, and fuse with the cis-Golgi compartment.

28 sting that their interaction occurs in a pre-Golgi compartment.

29 sing it to localize to the ER instead of the Golgi compartment.

30 nism of retention of soluble proteins in the Golgi compartment.

31 ytoplasmic domain for localization to a late Golgi compartment.

32 the ER or causes retrograde transport from a Golgi compartment.

33 s and predominantly localized in vivo to the Golgi compartment.

34 nd causes accumulation of aquaporin-2 in the Golgi compartment.

35 ound that Cav3 and Kir2.1 accumulated in the Golgi compartment.

36 a- and beta-subunits failed to reach the cis-Golgi compartment.

37 ssociates with vesicular membranes in a post-Golgi compartment.

38 gi and the accumulation of cargo in an early Golgi compartment.

39 21p complex from the ER for transport to the Golgi compartment.

40 from bound receptors at the lower pH of the Golgi compartment.

41 elope from the endoplasmic reticulum (ER) or Golgi compartment.

42 ling the flow of ceramide from the ER to the Golgi compartment.

43 nction in retrograde transport between early Golgi compartments.

44 FTR efficiently process this protein to post-Golgi compartments.

45 PLA(2)beta-EGFP fluorescence with the ER and Golgi compartments.

46 s that can be activated by proteases in post-Golgi compartments.

47 eolysis and peptide binding to MHC-I in post-Golgi compartments.

48 the endoplasmic reticulum to the cis-medial Golgi compartments.

49 ifications characteristic of transit through Golgi compartments.

50 plications for coronavirus assembly in early Golgi compartments.

51 icles and actively cycles between the ER and Golgi compartments.

52 orting of integral membrane proteins in post-Golgi compartments.

53 ic cycling of SNARE machinery between ER and Golgi compartments.

54 fusion in transport between the yeast ER and Golgi compartments.

55 the endoplasmic reticulum and the cis/medial Golgi compartments.

56 embled KATP channels fail to exit the ER/cis-Golgi compartments.

57 y retrieval of processing enzymes from later Golgi compartments.

58 icular trafficking within the Golgi and post-Golgi compartments.

59 and a concomitant increase of Mnn1p in later Golgi compartments.

60 greatly reduced levels of mutant DM in post-Golgi compartments.

61 etween the endoplasmic reticulum and the cis-Golgi compartments.

62 d to the Sia) is taken up by isolated intact Golgi compartments.

63 ated in protein localization to similar post-Golgi compartments.

64 e secreted via the endoplasmic reticulum and Golgi compartments.

65 ssociated with plasma membrane (PM) and post-Golgi compartments.

66 between endoplasmic reticulum and cisternal Golgi compartments.

67 ansmembrane-spanning protein associated with Golgi compartments.

68 ter a retrograde trafficking through the TGN/Golgi compartments.

69 ccelerating its clearance in the ER and post-Golgi compartments.

70 kinesis stimulated PSKs are present in trans-Golgi compartments.

71 reticulum to the Golgi apparatus and within Golgi compartments.

72 d anterograde transport from the ER to early Golgi compartments.

73 diate sorting of transmembrane cargo in post-Golgi compartments.

74 he protein is located in both the ER and cis-Golgi compartments.

75 e between the endoplasmic reticulum (ER) and Golgi compartments.

76 f the receptors in the endoplasmic reticulum/Golgi compartments.

77 MTP deficiency occurred distal to the ER and Golgi compartments.

78 COPII vesicles and cycled between the ER and Golgi compartments.

79 fferential localization to cis- versus trans-Golgi compartment, a mechanism that was compromised by o

80 sp-Erf2 localizes to the trans-Golgi compartment, a process which is mediated by its th

81 quality control system and aggregate in post-Golgi compartments, a significant proportion of PrP(217)

82 ical and basolateral cargoes traversing post-Golgi compartments accessible to endocytic ligands befor

83 y glycosyltransferases localized to specific Golgi compartments according to the step in which each e

84 cipal counter ion accompanying endosomal and Golgi compartment acidification, and that an interior-ne

85 targeted to the RER but is retained in a pre-Golgi compartment and cannot be secreted.

86 domains but requires maturation of TF in the Golgi compartment and cell surface expression.

87 the K314 and D319 for it to localize in the Golgi compartment and function in membrane envelopment o

88 ORF7b localizes to the Golgi compartment and is incorporated into SARS-CoV part

89 ex assembles in the endoplasmic reticulum or Golgi compartment and is transported to the plasma membr

90 t of vesicular stomatitis virus G from a pre-Golgi compartment and the exit of Shiga toxin from an en

91 N to the ERGIC prior to its movement to the Golgi compartment and the requirement of an intact ERGIC

92 The Golgi compartment and the secretion of apolipoprotein E

93 ned, but is less stable, after cleavage in a Golgi compartment and transport to the surface of infect

94 ure of Env is maintained after cleavage in a Golgi compartment and transport to the surfaces of infec

95 ressed in 293T cells, were retained in a pre-Golgi compartment and were not transported to the cell s

96 tact Mnn1p, resulting in its loss from early Golgi compartments and a concomitant increase of Mnn1p i

97 transfer into the endoplasmic reticulum and Golgi compartments and entry into classical secretory pa

98 16 was found to take place in the Golgi/post-Golgi compartments and is dependent on the acidic pH in

99 d presented through early/late endosomal and Golgi compartments and stimulated antigen-specific CD4+

100 ion protein (YFP-PUB13) localizes to TGN and Golgi compartments and that PUB13, PI4Kbeta1, and PI4Kbe

101 port of HA from the ER to the cis and medial golgi compartments and the TGN indicated that transport

102 equired for KATP channels to exit the ER/cis-Golgi compartments and transit to the cell surface.

103 ng occurred in the endoplasmic reticulum-cis Golgi compartments and was dependent on an R at the -4 p

104 he FXFXD motif directs retrieval from a post-Golgi compartment, and a second that slows the rate at w

105 plasmic reticulum and retrieval from the pre-Golgi compartment, and although structural requirements

106 -) cells nicastrin fails to reach the medial Golgi compartment, and as a consequence, is incompletely

107 e H indicated trafficking of S to the medial Golgi compartment, and confocal microscopy showed that S

108 m152 blocks the export of class I from a pre-Golgi compartment, and MCMV m6 directs class I to the ly

109 o between the endoplasmic reticulum, the cis-Golgi compartment, and possibly the plasma membrane.

110 eceptors release bound cargo in pre-Golgi or Golgi compartments, and receptors are then recycled back

111 ernalization and targeting to different post-Golgi compartments; and (c) YXXO- and LL-mediated target

112 icles that carry proteins from the ER to the Golgi compartment are encapsulated by COPII coat protein

113 ys, microtubules nucleated at the peripheral Golgi compartment are preferentially oriented toward the

114 rent origins to selectively access different Golgi compartments are incompletely understood.

115 tics specific to transport between different Golgi compartments are reconstituted in the cell-free sy

116 Recent results show that Golgi compartments are spatially segregated within hypha

117 reduction in the association of rab2 with a Golgi compartment as compared with controls.

118 We show that pre-Golgi and Golgi compartments, as well as secretory vesicles, are s

119 idered a late protein, gE accumulated in the Golgi compartment at as early as 4 h.

120 We show that Stv1p localizes to a late Golgi compartment at steady state and cycles continuousl

121 structural aberrations in early but not late Golgi compartments at nonpermissive temperature.

122 f being secreted, they are retained in a pre-Golgi compartment, at least partly in a misfolded state.

123 hese protein complexes formed in a premedial Golgi compartment before trimming of sugar chains.

124 aries among endomembranes with pre- and post-Golgi compartments being poor and rich in sphingolipids,

125 required for a step that occurs in the trans-Golgi compartment, between the reactions regulated by Yp

126 f targeting a Us9-EGFP fusion protein to the Golgi compartment but also is able to direct efficient i

127 t between the endoplasmic reticulum (ER) and Golgi compartments but is not essential for cell growth.

128 n as a vesicle-tethering complex on an early Golgi compartment, but its role is not fully understood.

129 mistry to the endoplasmic reticulum (ER) and Golgi compartments, but previous studies could not diffe

130 hway as well as retrograde traffic from post-Golgi compartments, but the machinery that regulates the

131 sed fragmentation of early, medial, and late Golgi compartments, but the most marked effect was on ea

132 COPI coated vesicles carry material between Golgi compartments, but the role of COPI in the secretor

133 i localization through retrieval from a post-Golgi compartment by detecting a post-Golgi processed fo

134 Maturation continues at terminal Golgi compartments by retrieval of transport components

135 subunit-labeled Golgi complex in Vero cells; Golgi compartment Cl- accumulation and acidification wer

136 ng virus infection, ORF7b accumulates in the Golgi compartment, colocalizing with both cis- and trans

137 rter was roughly 2-fold greater in the early Golgi compartments compared with that of the TGN.

138 n the ER and Golgi, often accumulated in the Golgi compartments distal from the nucleus.

139 on of most of the Kell glycoprotein in a pre-Golgi compartment due to differential processing, thereb

140 ein chimera, were rapidly degraded in a post-Golgi compartment following normal glycosylation.

141 traverse the endoplasmic reticulum (ER) and Golgi compartments for final maturation prior to reachin

142 This smaller G protein represents a post-Golgi compartment form that is lacking its C terminus, i

143 were restricted to an endoplasmic reticulum/Golgi compartment identified by co-localization with flu

144 specific cargo by the Erv41-Erv46 complex in Golgi compartments identifies escaped ER resident protei

145 ined in the endoplasmic reticulum and middle Golgi compartment, (ii) fails to undergo late Golgi proc

146 and/or assembled proteins to transit to the Golgi compartment in a process termed "quality control."

147 aveolin-2, which is present primarily in the Golgi compartment in all macrophages studied.

148 rescence microscopy localized hSec34p to the Golgi compartment in cells of all species examined, wher

149 ssemble in the ER and are transported to the Golgi compartment in COPII vesicles to embark on the Gol

150 articles budding from the ER en route to the Golgi compartment in COPII vesicles.

151 (apoB) undergoes rapid degradation in a pre-Golgi compartment in HepG2 cells.

152 ORF7 is a virion component localized to the Golgi compartment in infected cells, whose deletion caus

153 stably within the endoplasmic reticulum/cis-Golgi compartment in peptide-processing deficient cells,

154 nt trafficking of the Menkes ATPase from the Golgi compartment in response to copper were observed be

155 Tropoelastin accumulates in the fused ER/Golgi compartment in the presence of BFA if its degradat

156 p appears to be restricted to a late or post-Golgi compartment in the secretory pathway.

157 AtPEP12 is likely to be localized to a post-Golgi compartment in the vacuolar pathway.

158 e family of P-type ATPases, localizes to the Golgi compartment in yeast where it provides Ca(2+) and

159 localization of cycling proteins to the late Golgi compartment in yeast.

160 GFP-B2 subunit is enriched at distal Golgi compartments in HeLa cells.

161 components of the endoplasmic reticulum and Golgi compartments in LPS-stimulated B-cells.

162 GM130 is responsible for connecting distinct Golgi compartments in soma and dendritic branch points,

163 oplasmic reticulum-derived vesicles to early Golgi compartments in yeast.

164 nts, but the most marked effect was on early Golgi compartments, indicated by redistribution of ERGIC

165 We show that Pmel17 is cleaved in a post-Golgi compartment into two disulfide-linked subunits: a

166 with the ER, presumably within a pre- or cis-Golgi compartment involved in KDEL-substrate retention.

167 1 and Arf1 and that its glycosylation in the Golgi compartment is necessary for efficient post-Golgi

168 ibility that the ST tyr is cleaved in a post-Golgi compartment is supported by the observation that a

169 Expansion of the endoplasmic reticulum and Golgi compartments is a prerequisite for high rate synth

170 the Erv25p-Emp24p complex between the ER and Golgi compartments is discussed.

171 sport between endoplasmic reticulum (ER) and Golgi compartments is poorly understood.

172 of PI4KIIalpha blocked trafficking at a post-Golgi compartment, leading to accumulation of LIMP-2 in

173 transmembrane domain (TMD) is essential for Golgi compartment localization.

174 Because this association occurs in a pre-Golgi compartment, m4/gp34 might also interfere with Ag

175 is partially colocalized with the cis/medial Golgi compartment markers such as GM130 and Vti1b but no

176 Tctex-1 colocalization with Golgi compartment markers, its distribution upon treatme

177 in which GBF1 localized within pre-Golgi and Golgi compartments mediates ARF activation to facilitate

178 uration of the alpha-mating factor in a late Golgi compartment, most likely the yeast trans-Golgi net

179 e alpha-GalNAc-transferases expressed in the Golgi compartment of animal cells.

180 rion-associated gO complexes arise in a post-Golgi compartment of infected cells.

181 amic vesicle transport processes at the late-Golgi compartment of Saccharomyces cerevisiae (TGN) requ

182 Binding occurred primarily in the Golgi compartment of T cells, unlike with AP-2 binding t

183 t it occurs in the cytoplasm rather than the Golgi compartment of the cell.

184 In a late-Golgi compartment of the yeast Saccharomyces cerevisiae,

185 tion occurs in the endoplasmic reticulum and Golgi compartments of endothelial cells.

186 dimers form in the endoplasmic reticulum and Golgi compartments of human umbilical vein endothelial c

187 was present in the endoplasmic reticulum and Golgi compartments of nonpolarized cells.

188 nsport between the endoplasmic reticulum and Golgi compartments of the cell.

189 After cleavage in a Golgi compartment, oligomeric env complexes are transpor

190 could act by directing retrieval from a post-Golgi compartment or by preventing exit from the TGN.

191 TNV-infected Vero E6 cells, not with the ER, Golgi compartment, or early endosomes.

192 lar compartment (PVC) (vps27Delta) or a post-Golgi compartment (PGC) (vps21Delta).

193 plicated in endosomal interactions with post-Golgi compartments, plays a role in phagosomal maturatio

194 ement, with intracellular Env localized to a Golgi compartment proximal to the polarized MTOC.

195 here transport from the ER to the cis/medial-Golgi compartments requires the action of p115, GM130, a

196 ng between the endoplasmic reticulum and the Golgi compartments, resulted in an almost complete suppr

197 eceptor, misdirected APP into a distinct non-Golgi compartment, resulting in increased amyloid proces

198 ascent VLDL particle in either the ER or the Golgi compartment, resulting in the generation of LVPs.

199 rmally in the endoplasmic reticulum (ER) and Golgi compartments, resulting in impaired Notch signalin

200 rnover and were retained in the Golgi or pre-Golgi compartments(s).

201 cessed by a pro-protein convertase in a late Golgi compartment, since (i) addition of brefeldin A or

202 World hantaviruses assemble and bud into the Golgi compartment, some studies with New World hantaviru

203 esulted in accumulation of GFP-tpn-aa in the Golgi compartment, suggesting that the double lysine is

204 nin, a process normally restricted to a post-Golgi compartment termed the melanosome.

205 degraded by a proteolytic process in the pre-Golgi compartment that can be inhibited by N-acetyl-L-le

206 is further supports the presence of an early Golgi compartment that houses an alpha1,6-mannosyltransf

207 possibly through activities mediated by post-Golgi compartments that coordinate membrane traffic and

208 weak signal for retention within the ER/cis-Golgi compartments that is strengthened by oligomerizati

209 ning vesicles derived from the ER form early Golgi compartments that then mature by retrieval of proc

210 The resulting fragment trafficked to the Golgi compartment, the site of virus assembly.

211 one determines the retention of BACE1 to the Golgi compartments, through examination of recombinant p

212 position in the trans-Golgi network and post-Golgi compartments, thus counteracting fluctuations in t

213 cquire ATPase activity in a post-ER, but pre-Golgi, compartment, thus avoiding unproductive ATP hydro

214 The t-SNARE in a late Golgi compartment (Tlg2p) syntaxin is required for endoc

215 s to implicate AP-1 in transport from a post-Golgi compartment to a mature secretory organelle and su

216 ns in a highly selective manner in the trans-Golgi compartment to facilitate PM targeting via the tra

217 is required for its transport from the trans-Golgi compartment to the plasma membrane.

218 .Ser175Phe mutation displaced GORAB from the Golgi compartment to vesicular structures and selectivel

219 coalesce with vesicles derived from existing Golgi compartments to generate new Golgi cisternae.

220 ctive redistribution of alpha2C-ARs from the Golgi compartments to the cell surface, allowing the res

221 The movement of melanosomes from post-Golgi compartments to the periphery of melanocytes is kn

222 pts the insertion of alpha2delta-2 from post-Golgi compartments to the plasma membrane, and this effe

223 g (28 degrees C), alpha2C-ARs relocated from Golgi compartments to the plasma membrane, whereas the a

224 g of a 21-kDa propeptide (proSP-C21) in post-Golgi compartments to yield a 3.7-kDa mature form.

225 The post-Golgi compartment trans-Golgi Network (TGN) is a central

226 PH domains detect PI(4)P formation in extra-Golgi compartments under dynamic conditions and that var

227 Maturation and transit from the ER to the Golgi compartment was facilitated by lowering the temper

228 -G in downstream pre-Golgi intermediates and Golgi compartments was lost.

229 ivate PDGFR within the thin membranes of the Golgi compartment, we suggest that the intrinsic hydroph

230 elanogaster CSAS protein was targeted to the Golgi compartment when expressed in both heterologous ma

231 s they exited the ER and distributed in post-Golgi compartments when beta2m was reconstituted in thes

232 Vps10p has been immunolocalized to the late-Golgi compartment where CPY is sorted away from the secr

233 +)/Mn(2+) pump and has been localized to the Golgi compartment where it is important for protein sort

234 -binding protein, Rab6, is localized in late Golgi compartments where it mediates intra-Golgi vesicul

235 ents and accompanies them from the ER to the Golgi compartment, where it releases them and is recycle

236 ragments are subsequently transported to the Golgi compartment, where their turnover rate is much slo

237 titutes a major skeletal component of distal Golgi compartments, where it is necessary to maintain it

238 t of them remain as immature proteins in pre-Golgi compartments, where they are degraded by the prote

239 ted at a reduced rate and retained in a late Golgi compartment, whereas the plasma membrane H(+) ATPa

240 r distribution with a signal increase in the Golgi compartment, whereas the wild-type showed a widesp

241 alpha2C-ARs were localized predominantly to Golgi compartments, whereas alpha2A-ARs localized predom

242 GBF1 recruits COPI to pre-Golgi and Golgi compartments, whereas BIG1 and BIG2 recruit AP1 an

243 PI4KIIIbeta was enriched in early Golgi compartments, whereas PI4KIIalpha colocalized with

244 ic-derived proteins in a multivesicular post-Golgi compartment, which establishes the existence of a

245 M protein interacted with N protein in a pre-Golgi compartment, which is part of the MHV budding site

246 ate chains formed during biosynthesis in the Golgi compartment will determine the interaction potenti

247 nsport from the endoplasmic reticulum to the Golgi compartment with brefeldin A abolishes the mature,

248 the HCV NS5A protein and colocalized to the Golgi compartment with NS5A.

249 , are anchored to the membranes of the trans-Golgi compartment with the catalytic domain oriented to

250 might be a candidate protein integrating the Golgi compartment with the IF cytoskeleton.

251 ng of Trojan Ags can also occur in the trans-Golgi compartment, with the participation of the endopep