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1 y urinary (29.2%), gastrointestinal (20.4%), Gram negative (29.9%), Gram positive (16.8%), and cultur
2                                          Two gram-negative anaerobic rod taxa, Prevotella and Porphyr
3         Antibacterial activity against model Gram negative and Gram positive bacteria is reported for
4 microbial susceptibility in a wider range of Gram negative and Gram positive bacteria.
5 hloramphenicol, and co-trimoxazole; 68.3% of Gram-negative and 6.6% of Gram-positive pathogens.
6 00 cells per run in a synthetic community of Gram-negative and Gram-positive bacteria and fungi.
7 et, the difference in mechanism of action on gram-negative and gram-positive bacteria may be less pro
8 ed on four bacterial strains, including both Gram-negative and Gram-positive bacteria, showing great
9 re associated with secretion systems in many Gram-negative and Gram-positive bacteria.
10                       Vibrio vulnificus is a Gram-negative aquatic bacterium first isolated by the Un
11                                       Eighty Gram-negative bacilli (54 Enterobacteriaceae and 26 nonf
12 arbapenemase-producing glucose-nonfermenting Gram-negative bacilli (CPNFs), including Pseudomonas aer
13                                              Gram-negative bacilli (GNB) bacteremia is typically tran
14 tion being significantly shorter for enteric Gram-negative bacilli and enterococci (means, 3.6 h and
15 l stewardship in patients with less frequent Gram-negative bacilli bloodstream infections.
16 ce characteristics for the identification of Gram-negative bacilli commonly isolated from blood cultu
17                  Infections due to resistant gram-negative bacilli continue to cause unacceptable mor
18 uter membrane, C10OOc12O was able to enhance gram-negative bacilli susceptibility to antibacterial co
19 peptides may therefore be useful in fighting gram-negative bacilli threats through sensitization to e
20 es to antibiotics, we attempted to sensitize gram-negative bacilli to innate antibacterial protagonis
21  A total of 210 Bactec bottles demonstrating Gram-negative bacilli were prospectively enrolled for th
22  (54 Enterobacteriaceae and 26 nonfermenting Gram-negative bacilli) obtained from multiple institutio
23                                              Gram-negative bacilli, Staphylococcus aureus, Chlamydia,
24 ., Northfield, IL) for the identification of Gram-negative bacilli.
25                                              Gram-negative bacteremia (GNB) is a major cause of illne
26                 Currently, the management of gram-negative bacteremia is determined by clinical judgm
27 rains 0.3-8 mug/mL) than for the majority of Gram negative bacteria (Pseudomonas aeruginosa, 16-32 mu
28 um chemotaxis towards live gram positive and gram negative bacteria and demonstrate high sensitivity
29 e potentially useful biological functions in Gram negative bacteria.
30 s on the cell wall of both gram positive and gram negative bacteria.
31 e active against Gram positive bacteria than Gram negative bacteria; however zerumbone showed highest
32             Mechanisms of drug resistance in gram-negative bacteria (GNB) are numerous; beta-lactamas
33       Predicting antimicrobial resistance in gram-negative bacteria (GNB) could balance the need for
34                                              Gram-negative bacteria (GNBs) are common pathogens causi
35 es for the prevention of multidrug-resistant gram-negative bacteria (MDR-GNB) in adult intensive care
36 occus aureus and Streptococcus pyogenes) and gram-negative bacteria (Pseudomonas aeruginosa and Esche
37 togenes, Geobacillus stearothermophilus) and gram-negative bacteria (Pseudomonas aeruginosa, Pseudomo
38 ated molecular patterns in gram-positive and gram-negative bacteria activate IL-1beta release from im
39 urpose, we chose the pilus protein FimG from Gram-negative bacteria and a disulfide-bonded variant of
40 le to rapidly traverse the outer membrane of Gram-negative bacteria and accumulate inside these cells
41 acteria that are obligate predators of other Gram-negative bacteria and are considered potential alte
42 he periplasmic side of the inner membrane of Gram-negative bacteria and are then extracted by the Lpt
43 biofilm resistance to both Gram-positive and Gram-negative bacteria and fungi: it remains almost "zer
44 ed protein H-NS is a key global regulator in Gram-negative bacteria and is believed to be a crucial p
45 a six-component system that is widespread in Gram-negative bacteria and is thought to mediate retrogr
46 lass of molecules found in Gram-positive and Gram-negative bacteria and most archaea(1-5).
47                              The hallmark of gram-negative bacteria and organelles such as mitochondr
48  line for pilus production at the surface of Gram-negative bacteria and the archetypical protein-poly
49 ericidal protein that limits contact between Gram-negative bacteria and the colonic epithelial surfac
50 iens, Viridiplantae, Gram-positive Bacteria, Gram-negative Bacteria and Virus .
51                                              Gram-negative bacteria are common in pneumonia and incre
52 udies have shown that conjugation systems of Gram-negative bacteria are composed of distinct inner an
53                         Although a number of Gram-negative bacteria are known to catabolize quinate a
54 ling and export of amyloid protein sequences.Gram-negative bacteria assemble biofilms from amyloid fi
55 f the greatest threats to human health, with gram-negative bacteria being of major concern.
56  Mouse and human RELMbeta selectively killed Gram-negative bacteria by forming size-selective pores t
57      Bioinformatic screens reveal that these gram-negative bacteria carry genes coding for thiol-disu
58 90 bloodstream isolates of the 4 most common gram-negative bacteria causing bloodstream infections in
59 evalence of MCRPE infection from isolates of Gram-negative bacteria collected at the hospitals from 2
60                     The cell surface of most Gram-negative bacteria contains lipopolysaccharide that
61  release (ER) in severe infections caused by gram-negative bacteria could be a matter of concern.
62 pime-tazobactam when tested against clinical Gram-negative bacteria during clinical studies and routi
63 nder a low light dose (0.6 J cm(-2) ) toward Gram-negative bacteria E. coli, making it a remarkably e
64 h TLR4, as well as through activation by the Gram-negative bacteria E. coli, results in reduced NET p
65 s and Enterococcus faecalis, and against the Gram-negative bacteria Escherichia coli, Escherichia col
66 y that targets a highly conserved protein of Gram-negative bacteria essential for the fitness of V. c
67 anism of inter-cellular competition in which Gram-negative bacteria exchange polymorphic toxins using
68                                              Gram-negative bacteria express a diverse array of lipopr
69  of any known membrane-embedded insertase in Gram-negative bacteria fold into a prepore before membra
70 , III, IV, and VI), which are widely used by Gram-negative bacteria for pathogenesis.
71 bat multidrug resistant bacteria, especially Gram-negative bacteria for which the situation is partic
72 will be the effectiveness of EDP on reducing Gram-negative bacteria growth and the opposite trend was
73                    Multidrug-resistant (MDR) gram-negative bacteria have increased the prevalence of
74 ed to invade, reseal, kill, and digest other gram-negative bacteria in soils and water environments.
75 AMP responses against both Gram-positive and Gram-negative bacteria in T. molitor.
76 he biosensor construct was tested in several Gram-negative bacteria including Pseudomonas, Shewanella
77 an effective permeability barrier that makes Gram-negative bacteria inherently resistant to many anti
78 machines used for injection of proteins from Gram-negative bacteria into eukaryotic cells.
79 genesis of outer-membrane proteins (OMPs) in gram-negative bacteria involves delivery by periplasmic
80                   The outer membrane (OM) of Gram-negative bacteria is a permeability barrier and an
81 eactivity to antigens from Gram-positive and Gram-negative bacteria is common in patients suffering f
82                   The Cu tolerance system in Gram-negative bacteria is composed minimally of a Cu sen
83                   The outer membrane (OM) of Gram-negative bacteria is composed of lipopolysaccharide
84 olysaccharide (LPS) in the outer membrane of Gram-negative bacteria is critical for the assembly of t
85       Antimicrobial resistance in pathogenic gram-negative bacteria is one of the most pressing chall
86 he mechanism of action of promysalin against Gram-negative bacteria is still not clarified, even if a
87                        Activation of TLR4 by Gram-negative bacteria or lipopolysaccharide accelerates
88                                              Gram-negative bacteria possess specialised biogenesis ma
89 lex (Bcc) are a group of multidrug-resistant gram-negative bacteria rarely reported in patients witho
90                                              Gram-negative bacteria remodel their surfaces to interac
91 ctivity against a panel of Gram-positive and Gram-negative bacteria revealed structure-activity relat
92                                              Gram-negative bacteria secrete proteins using a type III
93 represent a major mechanism of resistance in Gram-negative bacteria showing multi-drug or extensively
94                                              Gram-negative bacteria such as Escherichia coli are prot
95 c children, nasopharyngeal colonization with Gram-negative bacteria such as Haemophilus influenzae an
96                  Images of gram-positive and gram-negative bacteria taken with this technique show ex
97 Lipopolysaccharide (LPS) is the component of Gram-negative bacteria that activates Toll-like receptor
98  Brucella spp. are facultative intracellular Gram-negative bacteria that cause the zoonotic disease b
99 imeric channels across the outer membrane of Gram-negative bacteria that mediate the import or export
100 Proteins (PGRPs) kill both Gram-positive and Gram-negative bacteria through simultaneous induction of
101 complexes constitute a primary mechanism for Gram-negative bacteria to expel toxic molecules for surv
102 and utilization of enterobactin permits many Gram-negative bacteria to thrive in environments where l
103 acter baumannii is one of the most difficult Gram-negative bacteria to treat and eradicate.
104 an discriminate between viable and nonviable Gram-negative bacteria to tune the immune response, ther
105                              Many pathogenic Gram-negative bacteria use the type III secretion system
106                                         Many Gram-negative bacteria use type 2 secretion systems (T2S
107 omysalin is active against Gram-positive and Gram-negative bacteria using a microdilution assay.
108                    Colonization by resistant gram-negative bacteria was significantly associated with
109 sceptibility profiles of clinically relevant Gram-negative bacteria within two hours of antibiotic in
110                         The cell envelope of gram-negative bacteria, a structure comprising an outer
111 io aeruginosavorus are obligate predators of Gram-negative bacteria, and have been proposed to be use
112 enable gene exchange between five species of Gram-negative bacteria, and that the identity of the gen
113 acellular sensors for both Gram-positive and Gram-negative bacteria, but their role in steady-state h
114                                           In Gram-negative bacteria, efflux pumps are able to prevent
115  HFM and showed that HFM increases rat fecal Gram-negative bacteria, elevates lipopolysaccharides (LP
116 espread antibiotic resistance, especially of Gram-negative bacteria, has become a severe concern for
117 id development of resistance particularly in Gram-negative bacteria, illustrates the urgent need for
118  it is widespread in more than 25 species of Gram-negative bacteria, including enterohemorrhagic E. c
119                                      In many Gram-negative bacteria, including Rhodobacter capsulatus
120                                        These Gram-negative bacteria, including the species Vibrio vul
121    The zauPzapA operon is present in diverse Gram-negative bacteria, indicating a common mechanism fo
122                                           In Gram-negative bacteria, lipid modification of proteins i
123 multitude of essential cellular functions in Gram-negative bacteria, mitochondria and chloroplasts.
124                 The T6SS is widespread among Gram-negative bacteria, mostly within the Proteobacteriu
125 n across the cell envelope are widespread in Gram-negative bacteria, NBs are found exclusively in gam
126                                           In Gram-negative bacteria, outer membrane phospholipase A (
127 ride (LPS), which is a membrane component of gram-negative bacteria, secrete more EVs than cholangioc
128                                           In Gram-negative bacteria, some of these pumps form multi-p
129 light that, although BAM is conserved across Gram-negative bacteria, structural and functional differ
130                                      In many Gram-negative bacteria, the peptidoglycan synthase PBP1A
131 le for detecting lipopolysaccharide (LPS) of Gram-negative bacteria, was immobilized on both a large
132 ted excellent sensitivity to trace levels of Gram-negative bacteria, while remaining insensitive to b
133  is markedly induced by avirulent strains of Gram-negative bacteria, Yersinia and Klebsiella, and les
134 sidues that occurs in most Gram-positive and Gram-negative bacteria.
135 anders are multifaceted infections caused by gram-negative bacteria.
136 mbrane has long defined the cell envelope of Gram-negative bacteria.
137  with the virulence of medically significant Gram-negative bacteria.
138 fections caused by multidrug-resistant (MDR) Gram-negative bacteria.
139 iaminopimelic-type peptidoglycans present in Gram-negative bacteria.
140  communication within and between species of Gram-negative bacteria.
141 LPS biosynthesis, transport, and assembly in Gram-negative bacteria.
142 idal activity against both Gram-positive and Gram-negative bacteria.
143 ity of specific Gram-positive antibiotics to Gram-negative bacteria.
144  and TonB-dependent transporters (TBDT) from Gram-negative bacteria.
145  both bacterial lipopolysaccharide (LPS) and gram-negative bacteria.
146 il microbial biomass, primarily by fungi and Gram-negative bacteria.
147 overy and development of antibiotics against Gram-negative bacteria.
148 isms of action of Ctn and Ctn(15-34) against Gram-negative bacteria.
149 lated inhibitor of DsbB enzymes from several Gram-negative bacteria.
150 similar to the HJ migration helicase RuvB in Gram-negative bacteria.
151 e bacterial cell surface was demonstrated in Gram-negative bacteria.
152 against multidrug-resistant tuberculosis and Gram-negative bacteria.
153 ride (LPS) constituting the outer leaflet of Gram-negative bacteria.
154 for developing countermeasures in pathogenic Gram-negative bacteria.
155 ed from work with Escherichia coli and other Gram-negative bacteria.
156 essential outer membrane glycolipids in most gram-negative bacteria.
157 nserved cell-cell communication mechanism in Gram-negative bacteria.
158 ycobacteria, and anaerobic Gram-positive and Gram-negative bacteria.
159 change of acyl-homoserine lactones (AHLs) by Gram-negative bacteria.
160 (LPS), the major outer-membrane component of Gram-negative bacteria.
161 ne" antibiotic against extensively-resistant Gram-negative bacteria.
162 t commensal and pathogenic Gram-positive and Gram-negative bacteria.
163 her MDR pathogens, such as malaria, HIV, and Gram-negative bacteria.
164 upies the space between the two membranes of Gram-negative bacteria.
165 purposing candidate to treat infections with Gram-negative bacteria.
166 otein in the extracellular matrix of enteric Gram-negative bacteria.
167 eta-barrel outer membrane proteins (OMPs) in Gram-negative bacteria.
168 tic cell-cell interactions between competing Gram-negative bacteria.
169 rel proteins into the outer membrane (OM) of Gram-negative bacteria.
170 t clinical needs in treating infections with Gram-negative bacteria.
171  are also relevant for other T3SS-containing Gram-negative bacteria.
172 e role of Type Vd secreted phospholipases in Gram-negative bacteria.
173 domains are widespread in toxins that target Gram-negative bacteria.
174 g highly effective against Gram-positive and Gram-negative bacteria; and (iii) the concentration of t
175 imics the structural moieties of its natural Gram negative bacterial pathogen-associated molecular pa
176 s and show that constriction in a variety of Gram-negative bacterial cells, including Proteus mirabil
177                                              Gram-negative bacterial endotoxin lipopolysaccharide (LP
178 nflammasome-based surveillance machinery for Gram-negative bacterial infections has been recently dis
179        In Drosophila the Imd pathway detects Gram-negative bacterial infections through recognition o
180 gram-positive bacterial isolates (57.9%), 17 gram-negative bacterial isolates (22.4%), and 15 fungal
181                                          The Gram-negative bacterial outer membrane (OM) is a unique
182 component of LPS in the outer leaflet of the Gram-negative bacterial outer membrane.
183                  Pseudomonas aeruginosa is a Gram-negative bacterial pathogen associated with acute a
184                   Tularemia is caused by the Gram-negative bacterial pathogen Francisella tularensis
185 enterica serovar Typhi is a human-restricted Gram-negative bacterial pathogen responsible for causing
186 ss termed "effector-triggered immunity." The Gram-negative bacterial pathogen Yersinia inactivates cr
187  response of Sts(-/-) mice to infection by a Gram-negative bacterial pathogen.
188                                         Many Gram-negative bacterial pathogens use a syringe-like app
189                                              Gram-negative bacterial pathogens utilize virulence-asso
190 locks into the backbone of Gram-positive and Gram-negative bacterial PG utilizing metabolic cell wall
191 ibility profiling for both Gram-positive and Gram-negative bacterial species requires at least 48-72
192 pe 6 secretion system (T6SS) is used by many Gram-negative bacterial species to deliver toxic effecto
193 e III and type IV effector proteins from six Gram-negative bacterial species to interact with the euk
194 ncludes well-characterized gram-positive and gram-negative bacterial strains published by ARLG invest
195 riose was taken up in both gram-positive and gram-negative bacterial strains.
196 needle-tip invasin proteins SipD and IpaD of Gram-negative bacterial type-3 secretion systems that br
197                                          The Gram-negative bacterium Bordetella pertussis is the caus
198 de to understand morphology in the dimorphic Gram-negative bacterium Caulobacter crescentus.
199 ions with model biological membranes and the Gram-negative bacterium Shewanella oneidensis MR-1.
200              Granulibacter bethesdensis is a Gram-negative bacterium that infects patients with chron
201 arainfluenzae is a nutritionally fastidious, Gram-negative bacterium with an oropharyngeal/nasopharyn
202    This method was successfully applied to a Gram-negative bacterium; it has yet to be implemented in
203                                  Predominant Gram-negative baseline pathogens in the microbiologicall
204                                          The Gram-negative Bdellovibrio bacteriovorus (BV) is a model
205 tudies support a uniquely nuanced pathway of Gram-negative CAMPs resistance and provide a more detail
206    However, recent evidence points towards a Gram-negative cell plan for Planctomycetes, although in-
207 ns and proteomic analyses support an altered Gram-negative cell plan for Planctomycetes, including a
208   Aggregatibacter actinomycetemcomitans is a Gram-negative commensal bacterium of the oral cavity whi
209                           The mission of the Gram-Negative Committee is to advance our knowledge of t
210 eins retaining functions similar to those in Gram-negative counterparts.
211 ro susceptibilities, inappropriate (initial) gram-negative coverage was given in 9 of 245 (4%) and 18
212 tial distribution of the main colonizer, the Gram-negative Curvibacter sp., along the body axis.
213             Neisseria meningitidis (Nm) is a Gram-negative diplococcus that normally colonizes the na
214 lling efficiency toward both B. subtilis and Gram negative E. coli, features that demonstrate the pro
215 CD/linalool-IC-NFs inhibited growth of model Gram-negative (E. coli) and Gram-positive (S. aureus) ba
216 in vitro susceptibility of Gram-positive and Gram-negative endophthalmitis bacterial isolates to vanc
217  and the crystal structure of a PRF from the Gram-negative endophytic bacterium Burkholderia phytofir
218                                          The Gram-negative entomopathogenic bacterium Photorhabdus lu
219  the gastric pathogen Helicobacter pylori, a Gram-negative epsilonproteobacterium, encodes two protei
220                                        Mixed Gram-negative flora, suggesting fecal contamination was,
221 nce with previously identified DSDs from the Gram-negative genus, Acinetobacter, but instead shows li
222 Gram-positive (other-GP) bacteria (7.4%) and Gram-negative (GN) bacteria (7.1%).
223  vitro activity against clinically important gram-negative, gram-positive aerobic, and facultative ba
224 gardless of the inciting bacterial stimulus (gram-negative, gram-positive, or bacterial products).
225 of antimicrobial resistance, particularly in Gram-negative hospital pathogens, which has led to renew
226  TTOT (48.21 versus 11.75 h; P < 0.001), the Gram-negative infection (GNI) TTOT (71.83 versus 35.98 h
227 ovel antibiotics to treat drug-resistant and Gram-negative infections.
228 pment of novel therapeutic options to manage Gram-negative infections.
229 cal development for the treatment of serious Gram-negative infections.
230                   The outer membrane (OM) of Gram-negative is a unique lipid bilayer containing LPS i
231  Pseudomonas aeruginosa MexAB-OprM pump in a Gram-negative membrane model that contained both inner a
232     EVs were harvested from three species of Gram-negative microbes carrying different plasmids.
233                                 Depletion of Gram-negative microbiota, hematopoietic cell deletion of
234 Here, we have addressed this question in the Gram-negative model bacterium Burkholderia thailandensis
235                             Twenty-one small Gram-negative motile coccobacilli were isolated from 15
236 teriaceae isolates, 5 Gram-positive cocci, 5 Gram-negative nonfermenting species, 9 Mycobacterium tub
237 f the Burkholderia cepacia complex (Bcc) are Gram-negative opportunisitic bacteria that are capable o
238                  Pseudomonas aeruginosa is a Gram-negative opportunistic pathogen that requires iron
239 is a potent phospholipase A2 secreted by the Gram-negative opportunistic pathogen, Pseudomonas aerugi
240                  Pseudomonas aeruginosa is a Gram-negative, opportunistic pathogen that infects immun
241 vided into two groups by their hosts, either Gram-negative or Gram-positive bacteria.
242        Emerging evidence also indicates that gram-negative oral bacteria, such as Porphyromonas gingi
243       Pseudomonas aeruginosa is a pathogenic gram-negative organism that has the ability to cause bli
244 ram-positive (OR, 0.73; 95% CI, .55-.97) and gram-negative organisms (0.51; .33-.78) but not yeast (0
245 ms predominate, whereas later more resistant Gram-negative organisms are found.
246 ens, novobiocin has limited activity against Gram-negative organisms due to the presence of the lipop
247                              Among neonates, Gram-negative organisms were the predominant cause of ea
248 e report that GA is antibacterial, targeting Gram-negative organisms with higher activity towards Pse
249                                The nefarious Gram-negative pathogen Pseudomonas aeruginosa encodes el
250                                          The Gram-negative pathogen Pseudomonas aeruginosa has three
251                 Here we demonstrate that the Gram-negative pathogen Pseudomonas aeruginosa is suscept
252                         Vibrio cholerae is a Gram-negative pathogen that can use its T6SS during anta
253 icle considers the cases of the non-invasive Gram-negative pathogen Vibrio cholerae and the invasive
254 he preferential selectivity of the method is gram negative pathogenic species E. coli O157:H7.
255 hyllococcus aureus and Enterococcus) and two gram negative pathogens (E. coli and Salmonella).
256 cterial activities against Gram-positive and Gram-negative pathogens (Staphylococcus aureus, Enteroba
257     CDI systems are distributed widely among Gram-negative pathogens and are thought to mediate inter
258 vel E3 ligase (NEL) domain that is unique to Gram-negative pathogens and whose activity is repressed
259 bacterial clearance in, animal models of the Gram-negative pathogens Haemophilus influenzae and Neiss
260                                     Numerous Gram-negative pathogens infect eukaryotes and use the ty
261 ype II secretion (T2S) is one means by which Gram-negative pathogens secrete proteins into the extrac
262 bapenemase-producing organisms, or CPOs, are Gram-negative pathogens that produce a transmissible car
263 d apparatus functions in the injectisomes of gram-negative pathogens to export virulence factors into
264  and fluoroquinolone resistance among common Gram-negative pathogens, and the emergence of MRSA, high
265 e of the major contributors to resistance in Gram-negative pathogens, by efficiently expelling a broa
266 on (T3S), a protein export pathway common to Gram-negative pathogens, comprises a trans-envelope syri
267 rio bacteriovorus bacteria naturally prey on Gram-negative pathogens, including antibiotic-resistant
268 r process that occurs in the injectisomes of gram-negative pathogens, is termed type-III secretion.
269 class of molecules toward difficult-to-treat Gram-negative pathogens.
270 e- and multidrug-resistant Gram-positive and Gram-negative pathogens.
271 beta-lactam activity in a broad range of MDR Gram-negative pathogens.
272 inst a diverse panel of multi-drug-resistant Gram-negative pathogens.
273 g ventilator-associated pneumonia) caused by Gram-negative pathogens.
274 at are active against both Gram-positive and Gram-negative pathogens.
275 of inter-bacterial competition found in many Gram-negative pathogens.
276 only associated with antimicrobial-resistant Gram-negative pathogens.
277 cterial activity, including activity against Gram-negative pathogens.
278  LyeTxI/betaCD was determined for planktonic Gram-negative periodontopathogens.
279 ival of microbiota members from the dominant Gram-negative phylum Bacteroidetes depends on their abil
280 of broad-spectrum compounds transcending the Gram negative-positive borderline.
281 g, modifying and finally destroying walls of Gram-negative prey bacteria, modifying their own PG as t
282 lipopolysaccharide, a cell wall component of Gram-negative Proteobacteria and known inducer of lupus
283 eriovorus and Micavibrio aeruginosavorus are Gram-negative proteobacteria that are obligate predators
284 ons: gram-positive Staphylococcus aureus and gram-negative Pseudomonas aeruginosa (99.3 +/- 1.9% and
285 rom both Gram-positive Bacillus subtilis and Gram-negative Pseudomonas aeruginosa.
286 ic Gram-positive rods and other uncultivable Gram-negative rods, and, rarely, opportunistic microorga
287 at recurrent nonlethal gastric infections of Gram-negative Salmonella enterica Typhimurium (ST), a ma
288 NAs in monocytes isolated from patients with Gram-negative sepsis compared with healthy control subje
289 endent Ags, and they are more susceptible to Gram-negative sepsis.
290 ression as a key modulator of mortality from Gram-negative sepsis.
291        A total of 78 clinical isolates of 13 Gram-negative species collected between April 2013 and N
292 e a number of clinical isolates of important Gram-negative species-Enterobacter cloacae, Escherichia
293  multidrug resistant (MDR) Gram-positive and Gram-negative species.
294 s composed of 15 conserved proteins in model gram-negative species.
295 r consideration as a vaccine target for some Gram-negative species.
296 orter that is expressed in gram-positive and gram-negative strains of bacteria.
297  13 showed moderate activity against the MDR Gram-negative strains, with MICs in the range of 16-32 m
298                                         This Gram-negative system bears striking resemblance to a rel
299 al NADases predicted to transit not only the Gram-negative T6SS but also the Gram-positive type VII s
300                                          Six gram-negative waterborne pathogens were used to demonstr

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