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1 nse is not well characterized, especially in Gram-positives.
2 an identify 24 etiologic agents of sepsis (8 Gram-positive, 11 Gram-negative, and 5 yeast species) an
3 rointestinal (20.4%), Gram negative (29.9%), Gram positive (16.8%), and culture negative (30.7%).
4  against clinically important gram-negative, gram-positive aerobic, and facultative bacteria includin
5                                    These new Gram-positive agents are reviewed here.
6 tified Dictyostelium chemotaxis towards live gram positive and gram negative bacteria and demonstrate
7 ith cis-diol groups on the cell wall of both gram positive and gram negative bacteria.
8 and RNA viruses including oncogenic viruses, gram positive and negative bacteria, fungi and parasites
9 amines were shown to exhibit potency against Gram-positive and -negative bacteria despite a contrary
10 LPPO II display excellent activities against Gram-positive and -negative bacteria, including pathogen
11    Notably, sTLR2 treatment markedly reduced Gram-positive and -negative bacteria-induced fibrosis in
12  posttrauma pneumonia susceptibility to both Gram-positive and -negative pathogens.
13    Pathogen-associated molecular patterns in gram-positive and gram-negative bacteria activate IL-1be
14 eported long-term biofilm resistance to both Gram-positive and Gram-negative bacteria and fungi: it r
15 ns are a diverse class of molecules found in Gram-positive and Gram-negative bacteria and most archae
16 monic nanoparticles can be delivered to both Gram-positive and Gram-negative bacteria boosting both p
17 munity, mediating AMP responses against both Gram-positive and Gram-negative bacteria in T. molitor.
18              IgE reactivity to antigens from Gram-positive and Gram-negative bacteria is common in pa
19       The outermost protective layer of both Gram-positive and Gram-negative bacteria is composed of
20 of antibacterial activity against a panel of Gram-positive and Gram-negative bacteria revealed struct
21                                    Images of gram-positive and gram-negative bacteria taken with this
22 lycan Recognition Proteins (PGRPs) kill both Gram-positive and Gram-negative bacteria through simulta
23 ureus and Escherichia coli as representative Gram-positive and Gram-negative bacteria under dark and
24 emonstrate that promysalin is active against Gram-positive and Gram-negative bacteria using a microdi
25                             After two hours, Gram-positive and Gram-negative bacteria were reduced to
26  and NOD2 are intracellular sensors for both Gram-positive and Gram-negative bacteria, but their role
27             Using eight different species of Gram-positive and Gram-negative bacteria, we demonstrate
28 iviridae can infect commensal and pathogenic Gram-positive and Gram-negative bacteria.
29 ainst parasites, mycobacteria, and anaerobic Gram-positive and Gram-negative bacteria.
30 of community diversity and the ratio between Gram-positive and Gram-negative bacteria.
31 nstrated inhibitory activity against several gram-positive and gram-negative bacteria.
32 tion and antibacterial property against both Gram-positive and Gram-negative bacteria.
33 elicit potent antibacterial activity against Gram-positive and Gram-negative bacteria.
34 powerful antibacterial surfaces against both Gram-positive and Gram-negative bacteria.
35 ation of MurNAc residues that occurs in most Gram-positive and Gram-negative bacteria.
36 s a rapid bactericidal activity against both Gram-positive and Gram-negative bacteria.
37 olic extracts being highly effective against Gram-positive and Gram-negative bacteria; and (iii) the
38 f these building blocks into the backbone of Gram-positive and Gram-negative bacterial PG utilizing m
39 cation and susceptibility profiling for both Gram-positive and Gram-negative bacterial species requir
40 strain catalogue includes well-characterized gram-positive and gram-negative bacterial strains publis
41 (18)F-fluoromaltotriose was taken up in both gram-positive and gram-negative bacterial strains.
42 o investigate the in vitro susceptibility of Gram-positive and Gram-negative endophthalmitis bacteria
43 nd nonsmokers) demonstrated higher levels of gram-positive and gram-negative facultatives, and lower
44  displaying antibacterial activities against Gram-positive and Gram-negative pathogens (Staphylococcu
45 els of tetracycline- and multidrug-resistant Gram-positive and Gram-negative pathogens.
46 mical scaffolds that are active against both Gram-positive and Gram-negative pathogens.
47 l activity against multidrug resistant (MDR) Gram-positive and Gram-negative species.
48 altodextrin transporter that is expressed in gram-positive and gram-negative strains of bacteria.
49                                              Gram-positive and negative bacterial glycolipids were ex
50 s with three different bacteria species, one gram-positive and two gram-negative species, allowing th
51 acteria, while remaining insensitive to both Gram-positive and viral challenges.
52 trategy can also extend activity of specific Gram-positive antibiotics to Gram-negative bacteria.
53  by a toxin produced by Clostridium tetani-a Gram-positive bacillus found in high concentrations in s
54 structures for flagellar filaments from both Gram-positive Bacillus subtilis and Gram-negative Pseudo
55        Both extracts inhibited the growth of gram-positive (Bacillus cereus, Staphylococcus aureus, L
56 fferent stirring conditions on the growth of Gram-positive (Bacillus subtilis), Gram-negative (Escher
57 itory concentration (MIC) was much lower for Gram positive bacteria (Enterococcus spp. and Staphyloco
58                                              Gram positive bacteria are the major contributor of bact
59 ial activity against model Gram negative and Gram positive bacteria is reported for selected compound
60                 They are more active against Gram positive bacteria than Gram negative bacteria; howe
61  covalently attached to the peptidoglycan of gram positive bacteria.
62     Its antibacterial activity is limited to gram positive bacteria.
63 bility in a wider range of Gram negative and Gram positive bacteria.
64                           The highest ranked Gram-positive bacteria (high priority) were vancomycin-r
65  the formation of competence-induced pili in Gram-positive bacteria and corroborate the remarkable st
66 n a synthetic community of Gram-negative and Gram-positive bacteria and fungi.
67 uld be used for the quantitative analysis of Gram-positive bacteria and might be applied potentially
68 us of Zg16(-/-) animals had a higher load of Gram-positive bacteria and showed bacteria with higher m
69                          Surface proteins in Gram-positive bacteria are incorporated into the cell wa
70   However, the mechanisms of MV formation in Gram-positive bacteria are unclear, as these cells posse
71 anscription in innate immunity, and identify Gram-positive bacteria as extracellular stimuli of Hippo
72 ver, treated cattle had reduced abundance of gram-positive bacteria at the genus level.
73                                              Gram-positive bacteria cause the majority of skin and so
74 al functions of specific compounds, and that Gram-positive bacteria considered to be obligate aerobes
75                             The cell wall of Gram-positive bacteria contains abundant surface-exposed
76                        The cell wall of most Gram-positive bacteria contains equal amounts of peptido
77  call NDA-1, contributes to the reduction of Gram-positive bacteria during early development and thus
78 ased on self-assembly of vancomycin (Van) on Gram-positive bacteria for imaging bacterial infection.
79                                   Given that Gram-positive bacteria genomes encode a variety of sorta
80                Regulators of PBP activity in Gram-positive bacteria have yet to be discovered but are
81                              The presence of Gram-positive bacteria in foodstuffs is a chronic worldw
82  antibiotic with activity against a range of Gram-positive bacteria including drug-resistant pathogen
83 at cyclic-di-adenosine monophosphate in live Gram-positive bacteria is a vita-PAMP, engaging the inna
84 c IgG booster responses to Gram-negative and Gram-positive bacteria is contained solely within the B
85   Surface display of proteins by sortases in Gram-positive bacteria is crucial for bacterial fitness
86 or example, increasing drug resistance among gram-positive bacteria is responsible for approximately
87      The mechanism of persister formation in Gram-positive bacteria is unknown.
88             By contrast, Ntox28 domains from Gram-positive bacteria lack C-terminal Gly-Tyr-Gly-Ile m
89  in mechanism of action on gram-negative and gram-positive bacteria may be less pronounced than forme
90 sensitive and resistant Gram-negative and/or Gram-positive bacteria of new amphiphilic 3',4'-dialkyl
91             The DnaB primosomal protein from Gram-positive bacteria plays a key role in DNA replicati
92                                  Most low GC Gram-positive bacteria possess an essential walKR two-co
93 here enhances our understanding of how other Gram-positive bacteria produce essential components of t
94                  Antimicrobial resistance in gram-positive bacteria remains a challenge in infectious
95  of complement by FH6-7/Fc on the surface of Gram-positive bacteria such as S. pyogenes will enable p
96  Streptococcus agalactiae are beta-hemolytic gram-positive bacteria that colonize the lower genital t
97 tibody-conjugated AuNPs can readily identify Gram-positive bacteria through antibody-antigen recognit
98 ns that mediate the attachment of pathogenic Gram-positive bacteria to host cells.
99 rticles (AuNP) modified with monoclonal anti-Gram-positive bacteria to produce an immune-sensor.
100  The more penetrable Zg16(-/-) mucus allowed Gram-positive bacteria to translocate to systemic tissue
101                                              Gram-positive bacteria use reactive thioester-containing
102                 ZG16 bound to and aggregated Gram-positive bacteria via binding to the bacterial cell
103         delta, a small protein found in most Gram-positive bacteria was, for a long time, thought to
104                                              Gram-positive bacteria were responsible for a high propo
105                                              Gram-positive bacteria were sensitive to vancomycin (27/
106                           Promysalin acts on Gram-positive bacteria with a mechanism of action involv
107 or of coproporphyrinogen oxidase (CgoX) from Gram-positive bacteria, an enzyme essential for heme bio
108 n shows broad antibacterial activity against Gram-positive bacteria, but is also hemolytic and cytoto
109 er regulator of carbon source utilization in gram-positive bacteria, but the CcpA regulon remains ill
110                                           In Gram-positive bacteria, CPS linkage is to either the cyt
111                             However, in many Gram-positive bacteria, especially in fermicutes, RNAP i
112                                           In Gram-positive bacteria, extracellular protein appendages
113 ding Eukaryota, Homo sapiens, Viridiplantae, Gram-positive Bacteria, Gram-negative Bacteria and Virus
114 numerous antibiotic resistance plasmids from Gram-positive bacteria, Gram-negative phage and the mobi
115  shown to play a role in the pathogenesis of Gram-positive bacteria, including E. faecium We previous
116 t activity and antibacterial effects against Gram-positive bacteria, namely methicillin-susceptible S
117                                              Gram-positive bacteria, particularly coagulase-negative
118 basis for formation of the 100S complexes in Gram-positive bacteria, shedding light on the mechanism
119 al strains, including both Gram-negative and Gram-positive bacteria, showing great potential for appl
120                           Pheromones of many gram-positive bacteria, such as Bacillus and Streptococc
121 s exhibited antibacterial activities against gram-positive bacteria, such as Bacillus subtilis and St
122                                     In other gram-positive bacteria, such as Enterococcus faecalis, d
123  environmental conditions, including, within Gram-positive bacteria, the stressosome complex that reg
124 glycopolymers found on the cell wall of many Gram-positive bacteria, whose diverse surface structures
125 edicine and nanotechnology.It is unclear how Gram-positive bacteria, with a thick cell wall, can rele
126 ss regulator Spx is ubiquitously found among Gram-positive bacteria.
127 oups by their hosts, either Gram-negative or Gram-positive bacteria.
128 xhibits potent antimicrobial effects against Gram-positive bacteria.
129 iscuous plasmids and their preference toward Gram-positive bacteria.
130 anding the biology of tectiviruses infecting Gram-positive bacteria.
131 hat displays nanomolar activity against many Gram-positive bacteria.
132 e opposite trend was observed in the case of Gram-positive bacteria.
133 vidin, recognizing almost none of the tested Gram-positive bacteria.
134 obal regulator of transcription in low G + C Gram-positive bacteria.
135 wn function that is well conserved in low-GC Gram-positive bacteria.
136 he CTC protein, a feature typical to various Gram-positive bacteria.
137 ive peptide/protein pairs we engineered from Gram-positive bacteria.
138 wn tropism of RNA bacteriophages may include gram-positive bacteria.
139 nificant antimicrobial effect, especially in gram-positive bacteria.
140 uses (bacteriophages) specifically infecting Gram-positive bacteria.
141 izes many types of PAMPs that originate from gram-positive bacteria.
142  peptides, and vulnerability to infection by Gram-positive bacteria.
143 epeat glycoproteins (SRRPs) are conserved in Gram-positive bacteria.
144 ich is linked to cell growth and division in Gram-positive bacteria.
145 , Staphylococci, and Streptococci, and other Gram-positive bacteria.
146 ates initiation of DNA replication in low-GC Gram-positive bacteria.
147  The most commonly identified organisms were gram-positive bacteria.
148 negative bacteria, but neither is present in Gram-positive bacteria.
149 gh which the stringent response functions in Gram-positive bacteria.
150 ossessed weak antibacterial activity against Gram-positive bacteria.
151  secretion systems in many Gram-negative and Gram-positive bacteria.
152 omplementing the method's proven utility for Gram-positive bacteria.
153 ring elongation in rod-shaped and ovococcoid Gram-positive bacteria.
154 tial target for evaluating antimicrobials in gram-positive bacteria.
155  whose hosts are Bacillus cereus and related Gram-positive bacteria.
156 dable in vitro activity against a variety of Gram-positive bacteria.
157 protein family, which is spread widely among gram-positive bacteria; and suggests approaches to targe
158                                          The Gram positive bacterial isolates were susceptible to cip
159                        However, 65% of these Gram positive bacterial pathogens showed resistance to p
160                            The proportion of Gram positive bacterial pathogens was (88%), and Staphyl
161 al design, through splitting a domain from a Gram-positive bacterial adhesin.
162      Recent structural studies on a class of Gram-positive bacterial adhesins have revealed an intram
163                               Degradation of Gram-positive bacterial cell wall peptidoglycan in macro
164 polymers are omnipresent constituents of the Gram-positive bacterial cell wall where they fulfill a v
165 60-RNAi) enhanced drosomycin expression upon Gram-positive bacterial challenge but the basal drosomyc
166 rtant role in regulating innate immunity and gram-positive bacterial clearance by functioning, in par
167      Fatty acid kinase (Fak) is a ubiquitous Gram-positive bacterial enzyme consisting of an ATP-bind
168 ammatory response and host defense against a gram-positive bacterial infection.
169 ration of novel vaccine modalities to combat Gram-positive bacterial infections.
170 pposed to protective, role for IL-17A during Gram-positive bacterial infections.
171                                   Forty-four gram-positive bacterial isolates (57.9%), 17 gram-negati
172 stent with the idea that CcpA is critical to gram-positive bacterial pathogenesis.
173                             Sepsis caused by Gram-positive bacterial pathogens is a major fatal disea
174 ococcus aureus and Streptococcus pneumoniae, Gram-positive bacterial pathogens of significant clinica
175 ogous pore-forming proteins secreted by many Gram-positive bacterial pathogens.
176 of peptidoglycan is integral to detection of gram-positive bacterial pathogens.
177 lated to the amounts of total microbial, and Gram-positive bacterial PLFAs, but not to the chemical c
178 t impacts on the amounts of total microbial, Gram-positive bacterial, and actinomycic PLFAs, but not
179              WMG1 shows dark toxicity to the Gram positive bacterium B. subtilis and good phototherma
180 ids as a positive determinant of size in the Gram-positive bacterium Bacillus subtilis and the single
181                       Biofilms formed by the Gram-positive bacterium Bacillus subtilis depend on the
182                            When starved, the Gram-positive bacterium Bacillus subtilis forms durable
183 troduce an isotopic labeling strategy in the gram-positive bacterium Bacillus subtilis to investigate
184  the solution structure of sigma1.1 from the Gram-positive bacterium Bacillus subtilis We found that
185 ment of oriC during spore development in the Gram-positive bacterium Bacillus subtilis.
186                     Staphylococcus aureus, a Gram-positive bacterium colonizing nares, skin, and the
187                                          The Gram-positive bacterium Enterococcus faecalis is both a
188 lethanolamine halve during elongation of the Gram-positive bacterium Listeria innocua.
189                                          The Gram-positive bacterium Listeria monocytogenes transitio
190 or the stringent response nucleotides in the Gram-positive bacterium Staphylococcus aureus We demonst
191 terial immunotherapeutic protein against the Gram-positive bacterium Streptococcus pyogenes This prot
192   Listeria monocytogenes is an intracellular Gram-positive bacterium that induces expression of type
193 treptococcus agalactiae is a beta-hemolytic, Gram-positive bacterium that is a leading cause of neona
194          Bacillus anthracis is a sporulating Gram-positive bacterium that is the causative agent of a
195 te the properties of an ion channel from the Gram-positive bacterium Tsukamurella paurometabola with
196                     Enterococcus faecalis, a Gram-positive bacterium, and Candida albicans, a fungus,
197 aphylococcus epidermidis (S. epidermidis), a Gram-positive bacterium, live inside the human nasal cav
198 ococcus suis, an important emerging zoonotic Gram-positive bacterium, while only RelA is functional u
199 ailed molecular characterisation of Tfp in a Gram-positive bacterium.
200  determinant of functional properties of the Gram-positive cell envelope.
201 stem bears striking resemblance to a related Gram-positive cell-wall remodeling strategy that also pr
202 iated with higher rates of positive FUBC for gram-positive cocci (GPC) but not GNB.
203  A total of 765 Bactec bottles demonstrating Gram-positive cocci in singles or clusters were tested d
204 comprising 10 Enterobacteriaceae isolates, 5 Gram-positive cocci, 5 Gram-negative nonfermenting speci
205 shes it from other staphylococci and related Gram-positive cocci.
206                           The mission of the Gram-Positive Committee of the Antibacterial Resistance
207  TTOT (75.17 versus 43.06 h; P < 0.001), the Gram-positive contaminant TTOT (48.21 versus 11.75 h; P
208 ptide antibiotic with potent activity toward Gram-positive drug-resistant bacteria.
209 timal antibiotic choice for the treatment of Gram-positive endophthalmitis.
210 zers generally enhanced photoinactivation of Gram-positive facultative anaerobes (Ent. faecalis, Stap
211 o the virulence of Listeria monocytogenes, a Gram-positive facultative intracellular pathogen.
212  first physiological barrier breached by the Gram-positive facultative pathogen Listeria monocytogene
213 ication of novel periodontopathogens such as Gram-positive Filifactor alocis, but its virulence mecha
214 tics of the ADEP class inhibit the growth of Gram-positive firmicutes by activating ClpP and causing
215 tructurally conserved in other amidases from Gram-positive Firmicutes, are pivotal for enzymatic acti
216  and J showed antimicrobial activity against Gram positive foodborne pathogens (Listeria monocytogene
217                              In recent years gram-positive (G(+)) bacteria, most commonly staphylococ
218 meningitidis, capsular serogroup C (MenC) or Gram-positive group B Streptococcus, capsular type III (
219                             Planococcus is a Gram-positive halotolerant bacterial genus in the phylum
220 hes reveal occurrence of SPDIR events in the gram-positive human pathogen Streptococcus pneumoniae an
221 ptococcus (GAS, Streptococcus pyogenes) is a Gram-positive human pathogen that must adapt to unique h
222                              Research on the Gram-positive human-restricted pathogen Streptococcus py
223                                 The TTOT for Gram-positive infection (GPI) was improved (64.04 versus
224  to discover non-antibiotic therapeutics for gram positive infections.
225 y lead to new therapeutic strategies against Gram-positive infections.
226                    During infection with the Gram-positive intracellular pathogen Listeria monocytoge
227                  Listeria monocytogenes is a Gram-positive intracellular pathogen that causes spontan
228              By contrast, the role of Hfq in Gram-positive is less established and varies among speci
229                                  None of the gram-positive isolates were vancomycin resistant.
230  we compared the immunomodulatory effects of Gram-positive (Lactobacillus rhamnosus strain GG [LGG])
231 nges in resistance patterns of the commensal Gram-positive microbiota.
232             Also, increased mass addition to Gram-positive mimetic membranes from AMP disruption corr
233 f the isoleucine biosynthesis pathway in the Gram-positive model bacterium Bacillus subtilis.
234       During phosphate-limited growth of the Gram-positive model organism Bacillus subtilis 168, WTA
235  coli dnaK mutants, rather than those in the Gram-positive model organism Bacillus subtilis.
236 xonuclease RNase J1 are not essential in the Gram-positive model organism,Bacillus subtilis, facilita
237 es in mortality risk were observed with both gram-positive (OR, 0.73; 95% CI, .55-.97) and gram-negat
238  inciting bacterial stimulus (gram-negative, gram-positive, or bacterial products).
239 st a variety of pathogenic Gram-negative and Gram-positive organisms is outlined.
240  postburn hospitalization, more susceptible, Gram-positive organisms predominate, whereas later more
241 gonorrheae and improved activity against the Gram-positive organisms Staphylococcus aureus and Entero
242                                          All Gram-positive organisms were susceptible to VAN, with th
243  natural product that is active only against Gram-positive organisms, into an antibiotic with activit
244 llenges for research of infections caused by gram-positive organisms.
245 oved antibacterial potency across a panel of Gram-positive organisms.
246 sponse pathway in enterococci and many other Gram-positive organisms.
247 ), Staphylococcus aureus (SA) (10.2%), other Gram-positive (other-GP) bacteria (7.4%) and Gram-negati
248 mall alarmone synthetase (SAS) RelQ from the Gram-positive pathogen Enterococcus faecalis is a sequen
249                                          The Gram-positive pathogen Staphylococcus aureus uses one pr
250 The cause might be allergic reactions to the gram-positive pathogen Staphylococcus aureus, a frequent
251                                       In the Gram-positive pathogen Staphylococcus aureus, the membra
252 up B Streptococcus (GBS) is an encapsulated, gram-positive pathogen that is an important cause of neo
253                   Staphylococcus aureus is a Gram-positive pathogen that resists many facets of innat
254 phylococcus aureus, a metabolically flexible gram-positive pathogen, causes infections in a variety o
255 ctivity also increased significantly against gram positive pathogens.
256  selective bactericidal activity against key Gram-positive pathogens (including Staphylococcus aureus
257 ases in virulence for a variety of important Gram-positive pathogens and concludes with a discussion
258 somerase IV) display potent activity against Gram-positive pathogens and no target-mediated cross-res
259             Approved ARLG projects involving gram-positive pathogens include (1) a pharmacokinetics/p
260       By contrast, more than 92.0% of common Gram-positive pathogens remain susceptible to either pen
261  Investigating the susceptibility of various Gram-positive pathogens to histones, we found high-level
262             Recently, it was discovered that Gram-positive pathogens use a unique heme biosynthesis p
263  interacts with the cytoplasmic membranes of Gram-positive pathogens, causing membrane permeabilizati
264                                     For many gram-positive pathogens, conjugative plasmid transfer is
265 in (near-iron transporter [NEAT]), common in Gram-positive pathogens, elicits protection in a murine
266                   Although effective against Gram-positive pathogens, novobiocin has limited activity
267 pton, which exhibits potent activity against Gram-positive pathogens.
268 moxazole; 68.3% of Gram-negative and 6.6% of Gram-positive pathogens.
269 hyrin III and leads to photosensitization of Gram-positive pathogens.
270 ry platform and class of materials to target Gram-positive pathogens.
271 own to enhance biofilm formation in multiple Gram-positive pathogens.
272  of CcpA targets in other medically relevant gram-positive pathogens.
273 ormation of biofilms by clinically important Gram-positive pathogens.
274 s exhibiting bactericidal activities against Gram-positive pathogens.
275 nes encoding fibronectin-binding proteins of Gram-positive pathogens.
276 cine candidate against enterococci and other gram-positive pathogens.
277                                   To uncover Gram-positive PBP regulatory factors, we used transposon
278    Here, we demonstrate a mechanism by which Gram-positive pili are able to dissipate mechanical ener
279 wever, little is known about the behavior of Gram-positive pili under force.
280 6, 27%), streptococci (32/126, 25.4%), and a Gram-positive rod (1/126, 0.8%).
281 zed by uncultivable asaccharolytic anaerobic Gram-positive rods and anaerobic Gram-negative rods, whi
282 nisms, uncultivable asaccharolytic anaerobic Gram-positive rods and other uncultivable Gram-negative
283 rity of these isolates are catalase-positive Gram-positive rods from multiple genera routinely classi
284 re non-spore forming, lactic acid producing, gram-positive rods.
285                                              Gram-positive S. aureus lacks an RMF homolog and the str
286  growth of model Gram-negative (E. coli) and Gram-positive (S. aureus) bacteria to a great extent.
287 n E. faecalis and other clinically-important Gram-positive species.
288  of the conjugate was determined against two gram positive (Staphyllococcus aureus and Enterococcus)
289 esponsible for hospital acquired infections: gram-positive Staphylococcus aureus and gram-negative Ps
290                             By contrast, the Gram-positive Staphylococcus aureus cells appeared to be
291                             In opportunistic Gram-positive Staphylococcus aureus, a small protein cal
292 inimum inhibitory concentrations against the Gram-positive Staphylococcus aureus.
293 d to successfully monitor the growth of both gram-positive (Staphylococcus aureus and Streptococcus p
294 synergistic antimicrobial activities against Gram-positive strains including methicillin-resistant St
295 owth much better than monobenzimidazoles for Gram-positive strains.
296  an important host defense mechanism against Gram-positive Streptococci.
297                                       In the gram-positive Streptococcus gordonii, the ability to for
298 ever, the signalling molecules recognized by Gram-positive stressosomes have yet to be identified, hi
299 not only the Gram-negative T6SS but also the Gram-positive type VII secretion system, a pathway recen
300 competent opportunistic human pathogen, is a Gram-positive workhorse for genomics.

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