ライフサイエンスコーパス: Gram

1 Gram for gram, MOFs can absorb as much energy as a high

2 Gram positive bacteria are the major contributor of bact

3 Gram-negative bacilli (GNB) bacteremia is typically tran

4 Gram-negative bacilli, Staphylococcus aureus, Chlamydia,

5 Gram-negative bacteremia (GNB) is a major cause of illne

6 Gram-negative bacteria (GNBs) are common pathogens causi

7 Gram-negative bacteria express a diverse array of lipopr

8 Gram-negative bacteria remodel their surfaces to interac

9 Gram-negative bacteria secrete proteins using a type III

10 Gram-negative bacteria such as Escherichia coli are prot

11 Gram-negative bacterial endotoxin lipopolysaccharide (LP

12 Gram-negative bacterial pathogens utilize virulence-asso

13 Gram-positive bacteria were responsible for a high propo

14 Gram-positive S. aureus lacks an RMF homolog and the str

15 A total of 78 clinical isolates of 13 Gram-negative species collected between April 2013 and N

16 y urinary (29.2%), gastrointestinal (20.4%), Gram negative (29.9%), Gram positive (16.8%), and cultur

17 teriaceae isolates, 5 Gram-positive cocci, 5 Gram-negative nonfermenting species, 9 Mycobacterium tub

18 comprising 10 Enterobacteriaceae isolates, 5 Gram-positive cocci, 5 Gram-negative nonfermenting speci

19 rointestinal (20.4%), Gram negative (29.9%), Gram positive (16.8%), and culture negative (30.7%).

20 Enterococcus faecalis, a Gram-positive bacterium, and Candida albicans, a fungus,

21 Pseudomonas aeruginosa is a Gram-negative bacterial pathogen associated with acute a

22 Aggregatibacter actinomycetemcomitans is a Gram-negative commensal bacterium of the oral cavity whi

23 Neisseria meningitidis (Nm) is a Gram-negative diplococcus that normally colonizes the na

24 Vibrio cholerae is a Gram-negative pathogen that can use its T6SS during anta

25 Pseudomonas aeruginosa is a Gram-negative, opportunistic pathogen that infects immun

26 competent opportunistic human pathogen, is a Gram-positive workhorse for genomics.

27 the gastric pathogen Helicobacter pylori, a Gram-negative epsilonproteobacterium, encodes two protei

28 This method was successfully applied to a Gram-negative bacterium; it has yet to be implemented in

29 light that, although BAM is conserved across Gram-negative bacteria, structural and functional differ

30 blood of asthmatic children with additional Gram + bacteria in the nasopharynx (Gr+/-).

31 isms of action of Ctn and Ctn(15-34) against Gram-negative bacteria.

32 They are more active against Gram positive bacteria than Gram negative bacteria; howe

33 emonstrate that promysalin is active against Gram-positive and Gram-negative bacteria using a microdi

34 cterial activity, including activity against Gram-negative pathogens.

35 n shows broad antibacterial activity against Gram-positive bacteria, but is also hemolytic and cytoto

36 somerase IV) display potent activity against Gram-positive pathogens and no target-mediated cross-res

37 overy and development of antibiotics against Gram-negative bacteria.

38 t activity and antibacterial effects against Gram-positive bacteria, namely methicillin-susceptible S

39 xhibits potent antimicrobial effects against Gram-positive bacteria.

40 natural product that is active only against Gram-positive organisms, into an antibiotic with activit

41 All Gram-positive organisms were susceptible to VAN, with th

42 ss regulator Spx is ubiquitously found among Gram-positive bacteria.

43 CDI systems are distributed widely among Gram-negative pathogens and are thought to mediate inter

44 The T6SS is widespread among Gram-negative bacteria, mostly within the Proteobacteriu

45 nisms, uncultivable asaccharolytic anaerobic Gram-positive rods and other uncultivable Gram-negative

46 growth of model Gram-negative (E. coli) and Gram-positive (S. aureus) bacteria to a great extent.

47 il microbial biomass, primarily by fungi and Gram-negative bacteria.

48 ell as PG isolated from various Gram (+) and Gram (-) bacterial species.

49 her MDR pathogens, such as malaria, HIV, and Gram-negative bacteria.

50 ial activity against model Gram negative and Gram positive bacteria is reported for selected compound

51 bility in a wider range of Gram negative and Gram positive bacteria.

52 n a synthetic community of Gram-negative and Gram-positive bacteria and fungi.

53 al strains, including both Gram-negative and Gram-positive bacteria, showing great potential for appl

54 secretion systems in many Gram-negative and Gram-positive bacteria.

55 biofilm resistance to both Gram-positive and Gram-negative bacteria and fungi: it remains almost "zer

56 lass of molecules found in Gram-positive and Gram-negative bacteria and most archaea(1-5).

57 eactivity to antigens from Gram-positive and Gram-negative bacteria is common in patients suffering f

58 ctivity against a panel of Gram-positive and Gram-negative bacteria revealed structure-activity relat

59 omysalin is active against Gram-positive and Gram-negative bacteria using a microdilution assay.

60 sidues that occurs in most Gram-positive and Gram-negative bacteria.

61 idal activity against both Gram-positive and Gram-negative bacteria.

62 t commensal and pathogenic Gram-positive and Gram-negative bacteria.

63 locks into the backbone of Gram-positive and Gram-negative bacterial PG utilizing metabolic cell wall

64 ibility profiling for both Gram-positive and Gram-negative bacterial species requires at least 48-72

65 e- and multidrug-resistant Gram-positive and Gram-negative pathogens.

66 at are active against both Gram-positive and Gram-negative pathogens.

67 multidrug resistant (MDR) Gram-positive and Gram-negative species.

68 rom both Gram-positive Bacillus subtilis and Gram-negative Pseudomonas aeruginosa.

69 against multidrug-resistant tuberculosis and Gram-negative bacteria.

70 eriovorus and Micavibrio aeruginosavorus are Gram-negative proteobacteria that are obligate predators

71 f the Burkholderia cepacia complex (Bcc) are Gram-negative opportunisitic bacteria that are capable o

72 bapenemase-producing organisms, or CPOs, are Gram-negative pathogens that produce a transmissible car

73 iens, Viridiplantae, Gram-positive Bacteria, Gram-negative Bacteria and Virus .

74 ericidal protein that limits contact between Gram-negative bacteria and the colonic epithelial surfac

75 s a rapid bactericidal activity against both Gram-positive and Gram-negative bacteria.

76 mical scaffolds that are active against both Gram-positive and Gram-negative pathogens.

77 cation and susceptibility profiling for both Gram-positive and Gram-negative bacterial species requir

78 structures for flagellar filaments from both Gram-positive Bacillus subtilis and Gram-negative Pseudo

79 ed on four bacterial strains, including both Gram-negative and Gram-positive bacteria, showing great

80 eported long-term biofilm resistance to both Gram-positive and Gram-negative bacteria and fungi: it r

81 acteria, while remaining insensitive to both Gram-positive and viral challenges.

82 g ventilator-associated pneumonia) caused by Gram-negative pathogens.

83 ent infections without organisms detected by Gram staining.

84 Activation of TLR4 by Gram-negative bacteria or lipopolysaccharide accelerates

85 , III, IV, and VI), which are widely used by Gram-negative bacteria for pathogenesis.

86 are the samples of choice for point-of-care Gram stain testing to diagnose Neisseria gonorrhoeae inf

87 se data suggest that, in asthmatic children, Gram- bacteria, which persist after antibiotic therapy,

88 pime-tazobactam when tested against clinical Gram-negative bacteria during clinical studies and routi

89 and fluoroquinolone resistance among common Gram-negative pathogens, and the emergence of MRSA, high

90 By contrast, more than 92.0% of common Gram-positive pathogens remain susceptible to either pen

91 tic cell-cell interactions between competing Gram-negative bacteria.

92 are also relevant for other T3SS-containing Gram-negative bacteria.

93 A total of 210 Bactec bottles demonstrating Gram-negative bacilli were prospectively enrolled for th

94 A total of 765 Bactec bottles demonstrating Gram-positive cocci in singles or clusters were tested d

95 In Drosophila the Imd pathway detects Gram-negative bacterial infections through recognition o

96 acter baumannii is one of the most difficult Gram-negative bacteria to treat and eradicate.

97 de to understand morphology in the dimorphic Gram-negative bacterium Caulobacter crescentus.

98 The zauPzapA operon is present in diverse Gram-negative bacteria, indicating a common mechanism fo

99 ival of microbiota members from the dominant Gram-negative phylum Bacteroidetes depends on their abil

100 pposed to protective, role for IL-17A during Gram-positive bacterial infections.

101 Eighty Gram-negative bacilli (54 Enterobacteriaceae and 26 nonf

102 vided into two groups by their hosts, either Gram-negative or Gram-positive bacteria.

103 tion being significantly shorter for enteric Gram-negative bacilli and enterococci (means, 3.6 h and

104 otein in the extracellular matrix of enteric Gram-negative bacteria.

105 bat multidrug resistant bacteria, especially Gram-negative bacteria for which the situation is partic

106 HFM and showed that HFM increases rat fecal Gram-negative bacteria, elevates lipopolysaccharides (LP

107 itory concentration (MIC) was much lower for Gram positive bacteria (Enterococcus spp. and Staphyloco

108 nflammasome-based surveillance machinery for Gram-negative bacterial infections has been recently dis

109 to collect sufficient cellular material for Gram stain testing remains unknown.

110 complexes constitute a primary mechanism for Gram-negative bacteria to expel toxic molecules for surv

111 owth much better than monobenzimidazoles for Gram-positive strains.

112 The TTOT for Gram-positive infection (GPI) was improved (64.04 versus

113 omplementing the method's proven utility for Gram-positive bacteria.

114 IgE reactivity to antigens from Gram-positive and Gram-negative bacteria is common in pa

115 or of coproporphyrinogen oxidase (CgoX) from Gram-positive bacteria, an enzyme essential for heme bio

116 urpose, we chose the pilus protein FimG from Gram-negative bacteria and a disulfide-bonded variant of

117 ression as a key modulator of mortality from Gram-negative sepsis.

118 The DnaB primosomal protein from Gram-positive bacteria plays a key role in DNA replicati

119 machines used for injection of proteins from Gram-negative bacteria into eukaryotic cells.

120 and TonB-dependent transporters (TBDT) from Gram-negative bacteria.

121 n E. faecalis and other clinically-important Gram-positive species.

122 e a number of clinical isolates of important Gram-negative species-Enterobacter cloacae, Escherichia

123 ases in virulence for a variety of important Gram-positive pathogens and concludes with a discussion

124 In Gram-negative bacteria, efflux pumps are able to prevent

125 In Gram-negative bacteria, lipid modification of proteins i

126 In Gram-negative bacteria, outer membrane phospholipase A (

127 In Gram-negative bacteria, some of these pumps form multi-p

128 In Gram-positive bacteria, CPS linkage is to either the cyt

129 LPS biosynthesis, transport, and assembly in Gram-negative bacteria.

130 basis for formation of the 100S complexes in Gram-positive bacteria, shedding light on the mechanism

131 e bacterial cell surface was demonstrated in Gram-negative bacteria.

132 However, the mechanisms of MV formation in Gram-positive bacteria are unclear, as these cells posse

133 ns are a diverse class of molecules found in Gram-positive and Gram-negative bacteria and most archae

134 e potentially useful biological functions in Gram negative bacteria.

135 multitude of essential cellular functions in Gram-negative bacteria, mitochondria and chloroplasts.

136 of any known membrane-embedded insertase in Gram-negative bacteria fold into a prepore before membra

137 eta-barrel outer membrane proteins (OMPs) in Gram-negative bacteria.

138 id development of resistance particularly in Gram-negative bacteria, illustrates the urgent need for

139 of antimicrobial resistance, particularly in Gram-negative hospital pathogens, which has led to renew

140 (PatB) catalyzes the O-acetylation of PG in Gram (-) bacteria, which aids in bacterial survival, as

141 e role of Type Vd secreted phospholipases in Gram-negative bacteria.

142 the formation of competence-induced pili in Gram-positive bacteria and corroborate the remarkable st

143 iaminopimelic-type peptidoglycans present in Gram-negative bacteria.

144 ed protein H-NS is a key global regulator in Gram-negative bacteria and is believed to be a crucial p

145 represent a major mechanism of resistance in Gram-negative bacteria showing multi-drug or extensively

146 similar to the HJ migration helicase RuvB in Gram-negative bacteria.

147 The Cu tolerance system in Gram-negative bacteria is composed minimally of a Cu sen

148 a six-component system that is widespread in Gram-negative bacteria and is thought to mediate retrogr

149 n across the cell envelope are widespread in Gram-negative bacteria, NBs are found exclusively in gam

150 anding the biology of tectiviruses infecting Gram-positive bacteria.

151 Brucella spp. are facultative intracellular Gram-negative bacteria that cause the zoonotic disease b

152 icle considers the cases of the non-invasive Gram-negative pathogen Vibrio cholerae and the invasive

153 Mouse and human RELMbeta selectively killed Gram-negative bacteria by forming size-selective pores t

154 at cyclic-di-adenosine monophosphate in live Gram-positive bacteria is a vita-PAMP, engaging the inna

155 an effective permeability barrier that makes Gram-negative bacteria inherently resistant to many anti

156 pment of novel therapeutic options to manage Gram-negative infections.

157 Many Gram-negative bacteria use type 2 secretion systems (T2S

158 ogous pore-forming proteins secreted by many Gram-positive bacterial pathogens.

159 re associated with secretion systems in many Gram-negative and Gram-positive bacteria.

160 In many Gram-negative bacteria, including Rhodobacter capsulatus

161 In many Gram-negative bacteria, the peptidoglycan synthase PBP1A

162 and utilization of enterobactin permits many Gram-negative bacteria to thrive in environments where l

163 beta-lactam activity in a broad range of MDR Gram-negative pathogens.

164 13 showed moderate activity against the MDR Gram-negative strains, with MICs in the range of 16-32 m

165 fections caused by multidrug-resistant (MDR) Gram-negative bacteria.

166 l activity against multidrug resistant (MDR) Gram-positive and Gram-negative species.

167 Mixed Gram-negative flora, suggesting fecal contamination was,

168 Antibacterial activity against model Gram negative and Gram positive bacteria is reported for

169 CD/linalool-IC-NFs inhibited growth of model Gram-negative (E. coli) and Gram-positive (S. aureus) ba

170 ation of MurNAc residues that occurs in most Gram-positive and Gram-negative bacteria.

171 The cell surface of most Gram-negative bacteria contains lipopolysaccharide that

172 imics the structural moieties of its natural Gram negative bacterial pathogen-associated molecular pa

173 The nefarious Gram-negative pathogen Pseudomonas aeruginosa encodes el

174 Among neonates, Gram-negative organisms were the predominant cause of ea

175 (54 Enterobacteriaceae and 26 nonfermenting Gram-negative bacilli) obtained from multiple institutio

176 arbapenemase-producing glucose-nonfermenting Gram-negative bacilli (CPNFs), including Pseudomonas aer

177 an discriminate between viable and nonviable Gram-negative bacteria to tune the immune response, ther

178 Numerous Gram-negative pathogens infect eukaryotes and use the ty

179 uld be used for the quantitative analysis of Gram-positive bacteria and might be applied potentially

180 f these building blocks into the backbone of Gram-positive and Gram-negative bacterial PG utilizing m

181 00 cells per run in a synthetic community of Gram-negative and Gram-positive bacteria and fungi.

182 Lipopolysaccharide (LPS) is the component of Gram-negative bacteria that activates Toll-like receptor

183 lipopolysaccharide, a cell wall component of Gram-negative Proteobacteria and known inducer of lupus

184 Depletion of Gram-negative microbiota, hematopoietic cell deletion of

185 mbrane has long defined the cell envelope of Gram-negative bacteria.

186 espread antibiotic resistance, especially of Gram-negative bacteria, has become a severe concern for

187 ce characteristics for the identification of Gram-negative bacilli commonly isolated from blood cultu

188 ., Northfield, IL) for the identification of Gram-negative bacilli.

189 at recurrent nonlethal gastric infections of Gram-negative Salmonella enterica Typhimurium (ST), a ma

190 needle-tip invasin proteins SipD and IpaD of Gram-negative bacterial type-3 secretion systems that br

191 evalence of MCRPE infection from isolates of Gram-negative bacteria collected at the hospitals from 2

192 ride (LPS) constituting the outer leaflet of Gram-negative bacteria.

193 ted excellent sensitivity to trace levels of Gram-negative bacteria, while remaining insensitive to b

194 le for detecting lipopolysaccharide (LPS) of Gram-negative bacteria, was immobilized on both a large

195 rains 0.3-8 mug/mL) than for the majority of Gram negative bacteria (Pseudomonas aeruginosa, 16-32 mu

196 le to rapidly traverse the outer membrane of Gram-negative bacteria and accumulate inside these cells

197 he periplasmic side of the inner membrane of Gram-negative bacteria and are then extracted by the Lpt

198 imeric channels across the outer membrane of Gram-negative bacteria that mediate the import or export

199 upies the space between the two membranes of Gram-negative bacteria.

200 Although a number of Gram-negative bacteria are known to catabolize quinate a

201 The outer membrane (OM) of Gram-negative bacteria is composed of lipopolysaccharide

202 rel proteins into the outer membrane (OM) of Gram-negative bacteria.

203 The outer membrane (OM) of Gram-negative is a unique lipid bilayer containing LPS i

204 of antibacterial activity against a panel of Gram-positive and Gram-negative bacteria revealed struct

205 tudies support a uniquely nuanced pathway of Gram-negative CAMPs resistance and provide a more detail

206 io aeruginosavorus are obligate predators of Gram-negative bacteria, and have been proposed to be use

207 The proportion of Gram positive bacterial pathogens was (88%), and Staphyl

208 nes encoding fibronectin-binding proteins of Gram-positive pathogens.

209 microbial susceptibility in a wider range of Gram negative and Gram positive bacteria.

210 d (SACCT) and determined the failure rate of Gram stain smears (GSS) due to insufficient cellular mat

211 enable gene exchange between five species of Gram-negative bacteria, and that the identity of the gen

212 communication within and between species of Gram-negative bacteria.

213 is markedly induced by avirulent strains of Gram-negative bacteria, Yersinia and Klebsiella, and les

214 line for pilus production at the surface of Gram-negative bacteria and the archetypical protein-poly

215 of complement by FH6-7/Fc on the surface of Gram-positive bacteria such as S. pyogenes will enable p

216 timal antibiotic choice for the treatment of Gram-positive endophthalmitis.

217 dable in vitro activity against a variety of Gram-positive bacteria.

218 The cell wall of Gram-positive bacteria contains abundant surface-exposed

219 g, modifying and finally destroying walls of Gram-negative prey bacteria, modifying their own PG as t

220 On Gram stain of urethral exudates, Nm can be misidentified

221 Promysalin acts on Gram-positive bacteria with a mechanism of action involv

222 rio bacteriovorus bacteria naturally prey on Gram-negative pathogens, including antibiotic-resistant

223 ased on self-assembly of vancomycin (Van) on Gram-positive bacteria for imaging bacterial infection.

224 oups by their hosts, either Gram-negative or Gram-positive bacteria.

225 here enhances our understanding of how other Gram-positive bacteria produce essential components of t

226 acteria that are obligate predators of other Gram-negative bacteria and are considered potential alte

227 ring elongation in rod-shaped and ovococcoid Gram-positive bacteria.

228 iviridae can infect commensal and pathogenic Gram-positive and Gram-negative bacteria.

229 for developing countermeasures in pathogenic Gram-negative bacteria.

230 Many pathogenic Gram-negative bacteria use the type III secretion system

231 LyeTxI/betaCD was determined for planktonic Gram-negative periodontopathogens.

232 ococcus aureus and Streptococcus pneumoniae, Gram-positive bacterial pathogens of significant clinica

233 Predominant Gram-negative baseline pathogens in the microbiologicall

234 The highest ranked Gram-positive bacteria (high priority) were vancomycin-r

235 Notably, sTLR2 treatment markedly reduced Gram-positive and -negative bacteria-induced fibrosis in

236 whose hosts are Bacillus cereus and related Gram-positive bacteria.

237 shes it from other staphylococci and related Gram-positive cocci.

238 sceptibility profiles of clinically relevant Gram-negative bacteria within two hours of antibiotic in

239 only associated with antimicrobial-resistant Gram-negative pathogens.

240 inst a diverse panel of multi-drug-resistant Gram-negative pathogens.

241 ms predominate, whereas later more resistant Gram-negative organisms are found.

242 els of tetracycline- and multidrug-resistant Gram-positive and Gram-negative pathogens.

243 enterica serovar Typhi is a human-restricted Gram-negative bacterial pathogen responsible for causing

244 ling and export of amyloid protein sequences.Gram-negative bacteria assemble biofilms from amyloid fi

245 cal development for the treatment of serious Gram-negative infections.

246 lated inhibitor of DsbB enzymes from several Gram-negative bacteria.

247 he biosensor construct was tested in several Gram-negative bacteria including Pseudomonas, Shewanella

248 with the virulence of medically significant Gram-negative bacteria.

249 e III and type IV effector proteins from six Gram-negative bacterial species to interact with the euk

250 Twenty-one small Gram-negative motile coccobacilli were isolated from 15

251 trategy can also extend activity of specific Gram-positive antibiotics to Gram-negative bacteria.

252 was considered pneumococcal if either sputum Gram stain, sputum culture, blood culture, or the immuno

253 We evaluated induced sputum Gram stain smears and cultures from hospitalized childre

254 postburn hospitalization, more susceptible, Gram-positive organisms predominate, whereas later more

255 domains are widespread in toxins that target Gram-negative bacteria.

256 e active against Gram positive bacteria than Gram negative bacteria; however zerumbone showed highest

257 al functions of specific compounds, and that Gram-positive bacteria considered to be obligate aerobes

258 Recently, it was discovered that Gram-positive pathogens use a unique heme biosynthesis p

259 Given that Gram-positive bacteria genomes encode a variety of sorta

260 The Gram-negative bacterial outer membrane (OM) is a unique

261 The Gram-negative bacterium Bordetella pertussis is the caus

262 The Gram-positive pathogen Staphylococcus aureus uses one pr

263 TTOT (48.21 versus 11.75 h; P < 0.001), the Gram-negative infection (GNI) TTOT (71.83 versus 35.98 h

264 TTOT (75.17 versus 43.06 h; P < 0.001), the Gram-positive contaminant TTOT (48.21 versus 11.75 h; P

265 s and Enterococcus faecalis, and against the Gram-negative bacteria Escherichia coli, Escherichia col

266 not only the Gram-negative T6SS but also the Gram-positive type VII secretion system, a pathway recen

267 ions with model biological membranes and the Gram-negative bacterium Shewanella oneidensis MR-1.

268 h TLR4, as well as through activation by the Gram-negative bacteria E. coli, results in reduced NET p

269 Tularemia is caused by the Gram-negative bacterial pathogen Francisella tularensis

270 is a potent phospholipase A2 secreted by the Gram-negative opportunistic pathogen, Pseudomonas aerugi

271 Biofilms formed by the Gram-positive bacterium Bacillus subtilis depend on the

272 first physiological barrier breached by the Gram-positive facultative pathogen Listeria monocytogene

273 By contrast, the Gram-positive Staphylococcus aureus cells appeared to be

274 nce with previously identified DSDs from the Gram-negative genus, Acinetobacter, but instead shows li

275 Here, we have addressed this question in the Gram-negative model bacterium Burkholderia thailandensis

276 ids as a positive determinant of size in the Gram-positive bacterium Bacillus subtilis and the single

277 coli dnaK mutants, rather than those in the Gram-positive model organism Bacillus subtilis.

278 component of LPS in the outer leaflet of the Gram-negative bacterial outer membrane.

279 The mission of the Gram-Negative Committee is to advance our knowledge of t

280 bacterial clearance in, animal models of the Gram-negative pathogens Haemophilus influenzae and Neiss

281 polymers are omnipresent constituents of the Gram-positive bacterial cell wall where they fulfill a v

282 lethanolamine halve during elongation of the Gram-positive bacterium Listeria innocua.

283 The mission of the Gram-Positive Committee of the Antibacterial Resistance

284 Research on the Gram-positive human-restricted pathogen Streptococcus py

285 al NADases predicted to transit not only the Gram-negative T6SS but also the Gram-positive type VII s

286 of broad-spectrum compounds transcending the Gram negative-positive borderline.

287 These Gram-negative bacteria, including the species Vibrio vul

288 ity of specific Gram-positive antibiotics to Gram-negative bacteria.

289 endent Ags, and they are more susceptible to Gram-negative sepsis.

290 vel E3 ligase (NEL) domain that is unique to Gram-negative pathogens and whose activity is repressed

291 nder a low light dose (0.6 J cm(-2) ) toward Gram-negative bacteria E. coli, making it a remarkably e

292 iscuous plasmids and their preference toward Gram-positive bacteria.

293 class of molecules toward difficult-to-treat Gram-negative pathogens.

294 ic Gram-positive rods and other uncultivable Gram-negative rods, and, rarely, opportunistic microorga

295 G mimic, as well as PG isolated from various Gram (+) and Gram (-) bacterial species.

296 Investigating the susceptibility of various Gram-positive pathogens to histones, we found high-level

297 ding Eukaryota, Homo sapiens, Viridiplantae, Gram-positive Bacteria, Gram-negative Bacteria and Virus

298 ype II secretion (T2S) is one means by which Gram-negative pathogens secrete proteins into the extrac

299 purposing candidate to treat infections with Gram-negative bacteria.

300 t clinical needs in treating infections with Gram-negative bacteria.

301 NAs in monocytes isolated from patients with Gram-negative sepsis compared with healthy control subje