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1 e membrane potential created by the vacuolar H+ pump.
2 tion of an electrogenic ion pump such as the H+ pump.
3 nsporter could function as either a Na+ or a H+ pump.
4 rabidopsis genome encodes 11 plasma membrane H+ pumps.
5 al rates of ATP hydrolysis and ATP-dependent H+ pumping.
6 de the counterions required for electrogenic H+ pumping.
7 omplex is fully functional in ATPase-coupled H(+) pumping.
8 ins and are not involved directly in proton (H(+)) pumping.
10 , caused a 38 +/- 5% decrease in the initial H(+) pump activity in the wild type, while no change was
15 ia, and one archaeon additionally exhibit an H(+)-pumping activity in inverted membrane vesicles prep
16 pling ratio of the nitrate-insensitive fruit H+ pumping activity is lower than that of nitrate-sensit
17 old inactivation treatment failed to inhibit H+ pumping activity of the fruit membranes, even though
19 few transporters, including plasma membrane H+ pump AHA3, Ca2+ pump ACA9, and K+ channel SPIK, furth
20 ransporter along the proximal tubule and the H+-pump along the collecting duct), the model yields seg
21 intercalated cells, in which the polarity of H(+) pumps and Cl(-)/HCO(3)(-) exchangers is reversed, o
22 s were found in many transporters, including H(+) pumps and H(+):cation antiporters, often at residue
25 lar loop of subunit c restored ATP-dependent H+ pumping and transition state thermodynamic parameters
26 el establish a polarized distribution of Na+/H+ pumps and aquaporins in the cell membrane, which crea
29 genous yeast H+ ATPase activity, we analyzed H+ pumping at pH >/= 8.0 in detail in order to selective
30 0 complex, which is thought to determine the H+-pumping/ATP stoichiometry, was previously not determi
32 ton gradient generated through the action of H+ pumps belonging to the P-type ATPase superfamily.
33 6.82 +/- 0.06 and inhibition of the V-ATPase H(+) pump by Cl(-) removal or via the selective inhibito
35 This feature results in an optimization of H(+) pumping by the V-ATPase according to existing H(+)
38 ssay conditions, rates of NTP hydrolysis and H+ pumping by the H,K-ATPase for CTP are about 10% of th
45 in D178N, D303A, and D400A mutants where the H(+) pumping efficiency had already been significantly d
49 ents was investigated by assaying ATP driven H(+) pumping function before and after cross-linking pai
52 rate-sensitive and nitrate-insensitive fruit H+-pumps have identical Km values for MgATP, and show si
55 cter of E427Q mutants and the enhancement of H+ pumping in E427D mutants by comparison with wild type
58 acidic, calcium storage compartments with a H(+) pump located in their membrane that have been descr
61 tion that results in a constitutively active H(+) pump partially suppressed SAUR-immune pp2c.d5 pheno
62 is apical H+ secretion via Na-H exchange and H+ pumping, processes that can be studied using the NH4+
64 ive subunit of the H+-V-ATPase or an ectopic H+ pump, randomized embryonic situs without causing any
65 r cation channel SlTPC1 and the two vacuolar H(+)-pumps, SlAVP1 and SlVHA-A1, which in turn are revea
66 t represses the transcription of a lysosomal H+ pump subunit that is critical for lipid degradation a
67 oocytes indicates that the capacity of this H(+) pump to recharge the membrane potential is rooted i
69 al PP(i)-induced outward currents related to H(+) pumping, whereas they were significantly larger in