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1 H2O2 fluctuations can be monitored in real time using fa
2 H2O2 has been implicated in several neurodegenerative di
3 H2O2 is an early danger cue required for innate immune c
4 H2O2 levels were consistent across subsamples of the sam
5 H2O2 oxidizes capping agent CSH, modulating the growth o
6 H2O2 production is blocked by stigmatellin, indicating i
7 H2O2-induced modifications commence with oxidation of Ty
8 H2O2/Zn(2+) induced concentration-dependent increases in
9 ncentrations of H2O2 in the range of 0-0.144 H2O2 to protein ratios (HTPR) by the addition of the req
10 degrees C) by chemical oxidation with 5-15% H2O2 and investigated the changes in surface chemistry a
11 L(OH)) was previously found to act as both a H2O2-responsive MRI contrast agent and a mimic of supero
13 nts for reactions containing O2 but no added H2O2 The kcat/Km for H2O2-driven degradation of chitin w
14 ctra of purified recombinant TcAPx-CcP after H2O2 reaction denote the formation of a compound I-like
15 erous bacterial and fungal pathogens against H2O2-induced oxidative damage from host immune responses
20 tected erythrocytes from oxidative AAPH- and H2O2-induced hemolysis, but at high concentrations a pro
21 ecrease in both glucose oxidase activity and H2O2 production as well as defensin-1 amount was observe
23 Formation of the stable adduct of CPA and H2O2 was also observed when the reaction mixture was eva
24 to stress, as stress signals doxorubicin and H2O2 each must free p53 from PEPD in order to achieve ro
26 s with oxo atom donors (PhIO, IBX-ester, and H2O2) to afford a rare example of a singly oxygenated su
27 and proanthocyanidins, in lignin-forming and H2O2-scavenging cultures and supported that monolignols
28 d digestion procedure using diluted HNO3 and H2O2 was developed for multi-element determination in gu
29 ition of 15mL of juice sample using HNO3 and H2O2 was performed in a digester block with reflux syste
30 f carbonyl compounds with hydroperoxides and H2O2 in acidic media, as such reactions involve alpha-ca
31 ion, Lon proteolytic activity induction, and H2O2 stress adaptation and produces the male-specific pa
33 he aptamer; the resulting ECL of luminol and H2O2 at the anodic poles is monitored using a photomulti
37 m resulting in increased levels of O2(-) and H2O2 are capable of disrupting intracellular iron metabo
38 for Citrate-Fe(II) mediated O2 to O2(-) and H2O2 to OH were 3.0 +/- 0.7 and (4.2 +/- 1.7) x 10(3) M(
44 lyses revealed that scavenging of apoplastic H2O2 resulted in remodulation of the transcriptome, with
47 ting Akt-FoxO1 signaling and also attenuates H2O2-induced Bim activation via inhibiting JNK phosphory
49 oreover, we identify that BiVO4 has the best H2O2 generation amount of those oxides and can achieve a
50 MtrC as a new benchmark in biotechnological H2O2 reduction with scope for applications in fuel cells
51 The phophorothioated DNA reacted to both H2O2 and hydroxyl radicals in vivo, and protected genomi
52 llulosic chromophores and its degradation by H2O2 is well-established, the second intermediate, 1,4,5
53 ese proteins prevented PTP1B inactivation by H2O2 Intriguingly, we discovered that TrxR1/NADPH direct
54 horn inhibited lipid peroxidation induced by H2O2, however, the non-polar fraction reduced more power
56 suggest that TRPA1 activation is mediated by H2O2 and reactive oxygen species, early markers of tissu
63 (7) M(-1)s(-1)) and catalytically decomposes H2O2 using Cc as the reducing substrate with higher effi
64 nd sdhaf2 showed lower rates of SA-dependent H2O2 production in vitro in line with their low SA-depen
65 n and streptococcal pyruvate oxidase-derived H2O2 production were required for cardiomyocyte killing.
67 ctrochemical sensor was used for determining H2O2 in apple juice, and the sensor electrode provided s
70 te and hydrous ferric oxide for EC-O2 and EC-H2O2, respectively), regardless of pH and Fe(II) product
72 in trapping, and MS analysis after equimolar H2O2 addition, supporting an alternative electron transf
74 ivity to shield the cytoplasm from exogenous H2O2 However, it receives electrons from the quinone poo
79 chieve a Faraday efficiency of about 98% for H2O2 production.Producing hydrogen peroxide via electroc
81 sensor using the GC/rGO-Nf@Ag6 electrode for H2O2 determination was calculated to be 5.35x10(-7)M wit
82 taining O2 but no added H2O2 The kcat/Km for H2O2-driven degradation of chitin was on the order of 10
85 S in an MPPC, and the energy requirement for H2O2 production was low ( approximately 0.87 kWh/kg H2O2
86 ensity functional theory predicted trend for H2O2 evolution is further confirmed by our experimental
89 ished by reactive oxygen species (ROS), e.g. H2O2, which oxidize the catalytically indispensable acti
91 yzes the reduction of endogenously generated H2O2 Prx1 is synthesized on cytosolic ribosomes as a pre
92 myeloperoxidase was incubated with glycine, H2O2, malondialdehyde, and a lysine analog in PBS at a p
94 of the observed CBP21 inactivation at higher H2O2 levels, we conclude that controlled generation of H
95 hat this is the first report that identifies H2O2-induced covalent modifications as an essential comp
97 derivatives, quinine and mefloquine impacted H2O2 levels in mitochondria, whereas chloroquine and a g
98 d repair after wounding, along with impaired H2O2 responses after exposure to the intestinal pathogen
100 ase and glutathione peroxidase activities in H2O2 treated CCD and Caco-2 cells compared to PEPS, EPS
101 gordonii, followed by a gradual decrease in H2O2 concentration (>30 min) to almost zero as lactate w
103 wn-regulated plants revealed an elevation in H2O2 production within the guard cells, increased sensit
104 crease catalase activity that is involved in H2O2 breakdown; and 4) result in DNA strand breaks.
105 active oxygen species and malondialdehyde in H2O2 treated CCD 841 CoN (CCD) and Caco-2 cells were sig
109 on reaction-diffusion principles that infers H2O2 degradation rates from intravital H2O2-biosensor im
110 ishmania mitochondrial SOD may also initiate H2O2-mediated redox signaling that regulates gene expres
111 oducts and SOA mass yields relative to input H2O2 concentrations, the second-generation dihydroxy hyd
114 oduction was low ( approximately 0.87 kWh/kg H2O2) compared to previous studies using real wastewater
115 nocarcinoma cells generated micromolar level H2O2 during just 1 min of direct CAP treatment on these
116 we observed an initial increase in the local H2O2 concentration of approximately 12 +/- 5 muM above S
117 n < 200 nmol/mg mito protein resulted in low H2O2 emission flux, increasing thereafter in Sham and T1
120 ignaling by increased rates of mitochondrial H2O2 production, leading to part of the SA-dependent tra
125 mum diameter, highly sensitive, nonenzymatic H2O2 sensor with a detection limit of 250 nM and a broad
126 photosynthesis inhibitor) attenuates nuclear H2O2 accumulation and high light-responsive gene express
127 nd S3QELs, suppressors of mitochondrial O2()/H2O2 generation that do not inhibit oxidative phosphoryl
128 H2O2 We discuss their contributions to O2()/H2O2 production under native conditions in mitochondria
130 ssed in oxyR2 mutants even in the absence of H2O2 Further genetic analyses suggest that OxyR2-activat
133 ated products were formed than the amount of H2O2 consumed, suggesting that the controlled breakdown
134 suggesting that the controlled breakdown of H2O2 activates methane, which subsequently incorporates
136 we show that low oxidative concentrations of H2O2 also impede chemokinesis and chemotaxis of previous
137 ntaining low intracellular concentrations of H2O2 Furthermore, we showed that DeltaoxyR2 and Deltaahp
138 were treated with varying concentrations of H2O2 in the range of 0-0.144 H2O2 to protein ratios (HTP
143 be used to ensure the selective detection of H2O2, enabling confident characterization of the role th
146 based biosensor towards the determination of H2O2 and glucose in the real samples have been demonstra
147 and interference study for determination of H2O2 in wastewater samples demonstrated the selectivity
148 d well for the amperometric determination of H2O2 over a linear range of 0.03-1mM with a detection li
150 tolysis conditions, no significant effect of H2O2 addition on CBDs abatement was observed due to an e
152 n this study, we investigated the effects of H2O2, a prototypical reactive oxygen species that is als
154 The MitoB method allows an evaluation of H2O2 levels in living organisms over a timescale from ho
155 Provision of exogenous ROS in the form of H2O2 reversed the necrotic phenotype and restored CD95 e
156 ical results clearly proved the formation of H2O2 in the leaves of plants 3h after the E. cloacae ino
159 kylhydroperoxide reductase, independently of H2O2 A conserved cysteine residue in OxyR2 is critical f
161 n LPMO catalysis, such as the involvement of H2O2 Our results show that residues on the substrate-bin
163 sgenic Arabidopsis maintained lower level of H2O2 than wild type plants did in response to the stress
165 leading to increased steady-state levels of H2O2; however, the mechanism(s) for cancer cell-selectiv
166 de sensor was used for in-situ monitoring of H2O2 produced in A. tequilana leaves after inoculation o
167 evel property resulting from nonlinearity of H2O2 scavenging by peroxiredoxins and our study reveals
168 eactions (NOx photo-oxidation, photolysis of H2O2, ozonolysis, or thermal decomposition of N2O5).
169 l death processes and shows the potential of H2O2 as a cellular damage biomarker, with a clear potenc
170 transcription requires both the presence of H2O2 and the absence of O2 Experiments show that Ccp lac
171 ) production rates, high pH, the presence of H2O2 instead of O2 as the initial Fe(II) oxidant, or a c
172 f the microelectrode sensor, the presence of H2O2 was detected in the root hairs by 3,3-diaminobenzid
174 selective electrochemical quantification of H2O2, because it is often enzymatically generated at bio
175 O is utilized herein for the quantitation of H2O2 in a wide concentration range, from 100nM to 100muM
177 opy results indicated rapid initial rates of H2O2 disproportionation slowing concomitantly with the a
183 olically controlled mitochondrial sources of H2O2 as well as glutathione- and thioredoxin-related pat
184 hat the improvement in the heat stability of H2O2 treated WPI solution was attributed to the signific
185 c model which explains the observed trend of H2O2, showing that FeS2 dissolution can act as a natural
186 immunoprecipitation experiments performed on H2O2-treated HCT116 cells, endogenous MLK3 associated wi
187 k electrons to oxygen to produce O2() and/or H2O2 We discuss their contributions to O2()/H2O2 product
188 ed sufficiently negative to drive the ORR or H2O2 reduction on the platinum surface, mainly using squ
191 -expression of stromal ascorbate peroxidase (H2O2 scavenger) or treatment with DCMU (photosynthesis i
193 tes superoxide (O2()) and hydrogen peroxide (H2O2) as bona fide products in reactions involving 1- or
196 on of cysteine (CSH) with hydrogen peroxide (H2O2) enzymatically generated by alcohol oxidase (AOx).
197 ceived that extracellular hydrogen peroxide (H2O2) generated by Duox diffuses through the tissue to d
198 noninvasively quantifying hydrogen peroxide (H2O2) in aqueous solutions based on chemical exchange sa
201 onradical species such as hydrogen peroxide (H2O2) or singlet molecular oxygen, rather than free-radi
202 One proposed signal is hydrogen peroxide (H2O2) produced by chloroplasts in a light-dependent mann
203 n quantified by measuring hydrogen peroxide (H2O2) reduction by chronoamperometry at -0.35V (vs pseud
205 oped for the detection of hydrogen peroxide (H2O2) using a reduced graphene oxide-nafion@silver6 (rGO
206 es the reduction of O2 to hydrogen peroxide (H2O2), has been implicated in the cardiac and lung myofi
208 xidant in the presence of hydrogen peroxide (H2O2), we demonstrated that the resulting methanol incor
209 e an apparent decrease of hydrogen peroxide (H2O2), which was previously shown to be a major player i
218 However, measuring ROS (hydrogen peroxide, H2O2) content in vivo is now possible using the MitoB pr
222 ) was found to be more effectively protected H2O2-induced IP3R1 dysfunction by reducing disulfide bon
225 Here, we stably expressed the ratiometric H2O2 redox sensor roGFP2-Orp1 in the cytosol and the mit
228 oreover, AMC provoked the production of ROS, H2O2, and NO, modulating the PI3K/Akt, MAPK, NFkappaB an
229 with C119S/C162S being incapable of sensing H2O2 Similarly, disulfide heterodimer formation was abol
231 owth inhibition induced by oxidative stress (H2O2 or menadione), significantly ameliorated the H2O2-d
234 of hypertension with a mitochondria-targeted H2O2 scavenger, mitochondria-targeted hydrogen peroxide
241 cy of the different catalysts and shows that H2O2 is formed with overpotentials as low as 90 mV.
242 e presence of aerobic bacteria suggests that H2O2 diffusion to the anode side caused inhibition of me
244 or menadione), significantly ameliorated the H2O2-dependent increase in matrix keratinocyte apoptosis
246 UO126, or ERK1/2 siRNA knockdown blocked the H2O2-induced shift of MLK3, while MLK3 inhibition with C
247 utrophil myeloperoxidase (MPO) catalyzes the H2O2-dependent oxidation of chloride anion to generate h
249 d RNA sequencing to uncover the scope of the H2O2 (peroxide)-stress regulon and to further explore th
250 mum above the biofilm in the presence of the H2O2-decomposing salivary lactoperoxidase and thiocyanat
251 roparticles by monitoring, in real time, the H2O2 and dissolved O2 concentration under oxic and anoxi
252 ygen species of two cancer cell lines to the H2O2-containing environments might result in the specifi
253 e catalytic activity of CoOxH-GO towards the H2O2-mediated oxidation of AR to form reddish resorufin
255 of the CoOxH-GO nanohybrid in detail via the H2O2-mediated oxidation of Amplex Red (AR) to form fluor
256 ll KIM-PTP family members to determine their H2O2 oxidation profiles and identify their reversible in
257 Our results indicate that at high tissue H2O2 levels the peroxiredoxin-thioredoxin antioxidant ch
258 ytes studied using the sensor combining TMB, H2O2, and GBR in phosphate buffer of pH 4.48, the S(2-)
260 of ribosomes did not appear to change due to H2O2 treatment, nor did posttranslational modifications,
261 ardiomyocytes and isolated hearts exposed to H2O2 This disulfide heterodimer was sensitive to reducti
263 phic bacteria, that converts methanethiol to H2O2, formaldehyde, and H2S, an activity not previously
264 ly selective two-electron reduction of O2 to H2O2 (93-99%) using decamethylferrocene (Fc*) as the red
265 posure of isolated aortic rings from rats to H2O2 for 6 hours dramatically impaired acetylcholine-ind
266 ahpC rendered V. cholerae more resistant to H2O2 RNA sequencing analyses indicated that OxyR1-activa
268 ends in activity for water oxidation towards H2O2 evolution on four different metal oxides, i.e., WO3
269 Melatonin treatment at 100mumol/L triggered H2O2 accumulation, which result from higher superoxide d
270 Increased ER glutathione import triggers H2O2-dependent Bip oxidation through Ero1 reductive acti
271 rate analog revealed a highly unconventional H2O2-activating distal environment with the reactive pro
277 in vitro exposure to oxidative stress using H2O2 induces miR-500a-5p overexpression and downregulati
278 r irradiation at 253.7 nm, ozonation, and UV-H2O2 advanced oxidation, transformation of the humic-lik
281 ter are interested in converting from the UV/H2O2 to the UV/free chlorine advanced oxidation process
282 for the oxygen reduction reaction (water vs H2O2) was evaluated in the presence and absence of Pt-Al
284 ritical need to identify mechanisms by which H2O2 modulates cellular processes in general and how it
286 t turnover number (TON) 3-fold compared with H2O2, highlighting the importance of oxidant choice as a
287 oxidation was significantly correlated with H2O2 generation (Pearson's r = 0.91), no correlation was
288 o sequential oxidative decarboxylations with H2O2 as the oxidant, coproheme III as substrate and cofa
289 s(pyridyl-2-methyl)amine) ligand family with H2O2/AcOH or AcOOH at -40 degrees C reveal the formation
290 re, we show that the enzyme reacts fast with H2O2 (k = 2.9 x 10(7) M(-1)s(-1)) and catalytically deco
291 (Fenton < (NH4)2S2O8 with H2SO4 < HNO3 with H2O2) the concentration of oxygen-containing surface gro
292 ilical vein endothelial cells incubated with H2O2 for 2 hours, accompanied with restoration of BH4.
295 2O4 up to -2.5 mA/cm(2) at 0.6 V vs RHE with H2O2 as an electron scavenger, and they show a charge se
296 ll viability compared to control stress with H2O2 (5mM/2h), recovering viability to values between 34
297 in the oxidation of the same substrates with H2O2 catalyzed by manganese complexes, supporting the hy
299 rectal carcinoma (HCT116) cells treated with H2O2, extracellular signal-regulated kinases 1 and 2 (ER
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