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1 e guinea pig model, a human isolate of novel H7N9 influenza virus, A/Anhui/1/2013 (An/13), is highly
3 upon infection of humans with avian H5N1 or H7N9 influenza viruses, adapting the viral polymerase fo
4 the host transcriptomic response to emerging H7N9 influenza virus and compare it with the responses t
5 ertain poultry species are reservoirs of the H7N9 influenza virus and that the virus is highly tropic
6 Prevention reported human infections with an H7N9 influenza virus, and by 20 July 2013, the numbers o
10 ion of genetically related poultry and human H7N9 influenza viruses differing by four amino acids, in
13 nfect humans, but the recently emerged avian H7N9 influenza viruses have caused sporadic infections i
16 Since emerging in 2013, the avian-origin H7N9 influenza viruses have resulted in over 400 human i
19 urrence of human infections by a novel avian H7N9 influenza virus in China has demonstrated the conti
20 Severe human disease caused by the emerging H7N9 influenza virus in China warrants a rapid response.
23 gence and seasonal persistence of pathogenic H7N9 influenza viruses in China have raised concerns abo
24 132 people known to have been infected with H7N9 influenza viruses in China, 37 died, and many were
26 highly pathogenic H5N1 and the newly emerged H7N9 influenza viruses in humans, the mechanism(s) by wh
27 alizing antibodies specific for the novel A (H7N9) influenza virus indicates a lack of prior human ex
31 tagonist in human and avian cells, the novel H7N9 influenza virus NS1 protein is defective at inhibit
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