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1                                              HA and M2 are strongly coclustered in the plasma membran
2                                              HA degradation requires either ERManI enzymatic activity
3                                              HA digestion in wild type NSC cultures or in the SGZ ind
4                                              HA dose-dependently inhibited TLR2-induced TNF-alpha pro
5                                              HA is known to swell, fitting a linear elastic model wit
6                                              HA is synthesized by NSCs and increases in the SGZ with
7                                              HA polymers are rationally designed from relatively low-
8                                              HA was determined by a competition between the coating H
9  at residues 156, 158, 189, and 193 of MN/10 HA to those in BJ/92 switched the MN/10 antigenic phenot
10          Compared to nHA-LC, Ostim((R)) (35% HA content) showed less and smaller particle agglomerate
11                              The nHA-LC (38% HA content) paste supported bone formation with a high d
12               We found that, when segment 4 (HA) of coinfecting parental viruses was modified, there
13 on The highly concentrated nHA-HC paste (48% HA content) formed oversized particle agglomerates which
14 ophilia A (AHA) and congenital hemophilia A (HA) are primarily directed to the A2 and C2 domains.
15 ixtures (200 mmol/L) high in either acetate (HA), propionate (HP), butyrate (HB) or placebo (PLA) wer
16  changing, for instance, the pKa of the acid HA versus the concentration or partial pressure of a rea
17 s using the common PCR inhibitor humic acid (HA) as a model.
18 nd Ag(+) in solution when adding humic acid (HA) to bacterial suspensions.
19 h is relatively enriched in hyaluronic acid (HA) and flexible.
20 ime-dependent deposition of hyaluronic acid (HA) and increased expression of markers for alternative
21 ing pathological amounts of hyaluronic acid (HA) or chondroitin sulfate (CS) did not directly increas
22 gional interactions between hyaluronic acid (HA), collagen and the spatial origins of mechanical stre
23 bolism and the signaling of hyaluronic acid (HA), the major component of the extracellular matrix (EC
24 on [D-dimer], and fibrosis (hyaluronic acid [HA]) were measured at baseline and through 96 weeks of A
25 id (SA), natural organic matter (humic acid, HA), and dissolved silicates.
26                 Hyaluronan (hyaluronic acid; HA) instillation to the bladder has been used to treat K
27 particles loaded with hydroxycinnamic acids (HA-NCs) have diameter of 224-253nm, encapsulation effici
28   TOP-PCR adopts homogeneous "half adaptor" (HA), generated by annealing P oligo (carrying a phosphat
29                                Additionally, HA and HC-HA/PTX3 inhibited migration but only HC-HA/PTX
30 ood supply to bile duct and lack of adequate HA flow is thought to be a risk factor for biliary compl
31 th its ability to induce homotypic adhesion (HA).
32  H3N2 strains, the acute Ab response against HA exhibited an inherent bias toward lambda L chain usag
33 othelial barrier-associated protein, altered HA production, and induced abnormal urothelial different
34 18 days of exposure to 3454 m high altitude (HA) without drug intervention (control, CONT) as well as
35                             Anti-CD44 Ab and HA treatments reduced NF-kappaB translocation, IL-1beta
36 yos, HYAL2 and HA were readily detected, and HA levels decreased with age.
37 RP, TNF, sIL-6R, I-FABP, sCD14, D-dimer, and HA levels were elevated in acute HIV infection.
38                      Conversely, CA, FA, and HA increased Pu sorption to goethite at pH 3, suggesting
39 t with this, in wild-type embryos, HYAL2 and HA were readily detected, and HA levels decreased with a
40         A single reassortant with the NP and HA gene segments from CIV-H3N2 was selected for characte
41 or (HR) expression were detected by qPCR and HA secretion by enzymatic immunoassay.
42 aman bands associated with the scaffolds and HA with the spatial offset depended on the depth at whic
43 lucidate the differences in trogocytosis and HA formation mediated by anti-CD20 mAbs RTX and GA101, a
44 antly increased accumulation of versican and HA, especially in the perivascular and peribronchial reg
45                                         Anti-HA antibodies bind to, but do not neutralize, the report
46                                         Anti-HA antibodies were cross-reactive against multiple subty
47 ricted, leukemia-associated minor H antigen, HA-1; (2) a CD8 coreceptor to promote function of the cl
48 A (HAAA) sections compared to normal aortae (HA).
49                             Hepatocyte area (HA) and lobule radius (LR) were also measured.
50 e effect of lower measured hepatic arterial (HA) flow (<400 mL/min) on biliary complications and graf
51 that, in several mouse tumour models such as HA-expressing 4T1 mammary carcinoma cells, OVA-expressin
52  NSC proliferation, and CD44-null as well as HA-disrupted wild type NSCs demonstrate delayed neuronal
53                           Surface-associated HA was required for eIF4E's oncogenic activities suggest
54            Specifically, HR was increased at HA from 64 +/- 10 to 74 +/- 12 beats min(-1) during the
55      Unexpectedly, the sympathoactivation at HA that was evidenced by increased circulating noradrena
56 ation of both Eley-Rideal (ER) and hot atom (HA) mechanisms when H impinges on adsorbed CO2, and we s
57         Given the strong correlation between HA neuron excitability and behavioral arousal, we invest
58 riable and no significant difference between HA and FA was found for major chemical groups, that is,
59  WTI and is promoted by a cross-talk between HA, CD73/adenosine signaling, and other profibrotic medi
60 rase domain of hemagglutinin (HA) and blocks HA-mediated membrane fusion.
61 tivity mediated by native TCR coexpressed by HA-1 TCR T cells.
62 the transient CD44 overexpression induced by HA.
63 f proinflammatory response in macrophages by HA was mediated by CD44.
64  the reduction in NF-kappaB translocation by HA.
65 and microcomputed tomography to characterize HA in mouse models of advanced breast cancer in relevant
66 rrets with viral vectors expressing chimeric HA, aimed at boosting Ab reactivity against the HA stem
67 ermined by a competition between the coating HA-Ag and the HRP labeled HA antibody (HRP-HA-Ab).
68                               The T-11 COBRA HA vaccine elicited antibodies with HAI and neutralizati
69 ike particle (VLP) vaccines expressing COBRA HA proteins elicited antibodies with hemagglutination in
70  characterization of 17 prototype H3N2 COBRA HA proteins were screened in mice and ferrets for the el
71                        The top leading COBRA HA candidates were tested against cocirculating variants
72 s with COBRA HA based viruses or using COBRA HA based vaccines to boost preexisting antibodies induce
73 ies induced by wild-type H1N1 viruses, COBRA HA antigens elicited sera with the broadest HAI reactivi
74    Overall, priming naive ferrets with COBRA HA based viruses or using COBRA HA based vaccines to boo
75 , while the stunting of fusion by deacylated HA acting in isolation may be balanced by other viral pr
76         Compared to wild-type HA, deacylated HA is correlated with released particles with flat envel
77 porphyrins in DMF as an example, decreasing [HA] 10-fold lowers etaeff by 59 mV and decreases the TOF
78  pegvorhyaluronidase alfa (PEGPH20) degrades HA, thereby increasing drug delivery.
79 RA technology was effectively used to design HA immunogens that elicited antibodies that neutralized
80 designed trimers can both capture and detect HA in a paper-based diagnostic format, neutralizes influ
81 rochemical immunosensor capable of detecting HA with high sensitivity and selectivity was developed.
82 hibitors, gefitinib or ibrutinib, diminishes HA formation and trogocytosis by GA101.
83  support for a model in which PEP87 disrupts HA function by displacing native interactions of the neu
84 by metastatic breast cancer feature distinct HA materials properties.
85                                The dual drug HA conjugate can inhibit 4T1 tumor growth in vivo during
86 vo than free DOX and GEM and the single drug HA conjugates.
87 o increase bioaccessibility, we encapsulated HAs in lipid-core nanocapsules (NCs) based on a biodegra
88      Four conditions including and excluding HA + ROCK and its effect on early attachment rates and p
89 the elongated 260-loop increases the exposed HA surface and can contribute to antigenic variation in
90                                          For HA immunoreactivity, we documented both consistently and
91              This increased accumulation for HA-treated MSC yielded a substantial reduction in inflam
92  that demannosylation is a critical step for HA degradation.
93 clade classifications, an automated tool for HA subtype numbering conversion, linkouts to disease eve
94 mice were exposed to ozone or short-fragment HA (sHA), and AHR was assayed via flexiVent.
95 l contribution to biliary complications from HA thrombosis.
96                                 Furthermore, HA and NP have been shown to be concentrated in choleste
97 oblasts isolated from control mice generated HA-enriched cable structures in the ECM, providing a sub
98                         We analyzed human H1 HA head and stalk domain sequences and found substantial
99  the conserved conformation as in H7 and H10 HAs.
100                       Since avian-origin H15 HA viruses have been shown to cause enhanced disease in
101                   Here, we show that the H15 HA has a high preference for avian receptor analogs by g
102             The extended 150-loop of the H15 HA retains the conserved conformation as in H7 and H10 H
103 can contribute to antigenic variation in H15 HAs.
104 overlaps with the pocket occupied by some H3 HA-specific inhibitors, indicating the high relevance of
105 nt formulations to broaden responses to H5N1 HA based antigens, and show that this broadening is func
106 T-cell epitopes and low immunogenicity of H7 HA remains unknown due to the lack of animal models repr
107 to recapitulate the low immunogenicity of H7 HA.
108 cantly improved the immunogenicity of the H7 HA in the humanized mouse model, leading to a greater th
109        ADCC-Abs titers directed against H7N9 HA or NA proteins.
110                           m826 binds to H7N9 HA with subnanomolar affinity at acidic pH and 10-fold l
111 rystal structure of m826 complexed with H7N9 HA indicates that m826 binds an epitope that may be full
112 higher toward H7N9 NP, as compared with H7N9 HA or NA proteins, and correlated strongly with ADCC-Abs
113       Heavy chain-Hyaluronan/Pentraxin 3 (HC-HA/PTX3) is a complex purified from human amniotic membr
114                      Additionally, HA and HC-HA/PTX3 inhibited migration but only HC-HA/PTX3 inhibite
115                              Furthermore, HC-HA/PTX3 significantly reduced the extent of infiltration
116 onjunctival and subcutaneous injection of HC-HA/PTX3 preserved tear secretion and conjunctival goblet
117                                      Only HC-HA/PTX3 dose-dependently inhibited proliferation and EMT
118 d HC-HA/PTX3 inhibited migration but only HC-HA/PTX3 inhibited collagen gel contraction.
119 a mouse model of cGVHD to examine whether HC-HA/PTX3 could attenuate dry eye disease elicited by cGVH
120                                           HC/HA volume surgeons had a significantly lower rate of sur
121 performed by high cumulative/high annual (HC/HA) surgeons increased from 38.3% to 58.4% (P < 0.01) wi
122                               Hemagglutinin (HA), a glycoprotein abundant on the virion surface, is i
123 roteins that bind influenza A hemagglutinin (HA) at its conserved receptor binding site.
124 and by ECL replacement with a hemagglutinin (HA) tag.
125  murine macrophages through a hemagglutinin (HA)-mediated mechanism.
126 predicted phenotypic effects, hemagglutinin (HA) clade classifications, an automated tool for HA subt
127  of the surface glycoproteins hemagglutinin (HA) and neuraminidase (NA) with the cell surface recepto
128 052, in combination with H5N1 hemagglutinin (HA) based antigens.
129 ntified T-cell epitopes in H7 hemagglutinin (HA) which potentially enhance regulatory T cell response
130 uished, based on variation in hemagglutinin (HA): B/Victoria/2/87-like (B/Vic) and B/Yamagata/16/88-l
131  tetanus toxoid and influenza hemagglutinin (HA) from H1N1 and newly emergent subtypes such as H5N1 a
132           Some proteins, like hemagglutinin (HA), NA, and M2, are integral membrane proteins.
133  vestigial esterase domain of hemagglutinin (HA) and blocks HA-mediated membrane fusion.
134 ely on the antigenic match of hemagglutinin (HA) for vaccine strain selection, and most vaccines rely
135 he influenza envelope protein hemagglutinin (HA), the low pH in the endosome triggers a transition fr
136 nfected cells and recombinant hemagglutinin (HA), neuraminidase (NA), and nucleoprotein (NP) proteins
137 s with relatively acid-stable hemagglutinin (HA) proteins are rendered incapable of pH-induced trigge
138 rotype 6 (Ad6) to express the hemagglutinin (HA) gene from influenza A/PR/8/34 virus.
139 usly acquire mutations in the hemagglutinin (HA) glycoprotein that abrogate binding of human antibodi
140 to influenza virus target the hemagglutinin (HA) glycoprotein, which is the major antigen on the surf
141 f the Ab response against the hemagglutinin (HA) protein elicited by influenza infection.
142 o acid at position 158 of the hemagglutinin (HA) protein substantially affected the systemic replicat
143 ntibody responses against the hemagglutinin (HA) surface glycoprotein; however, the diversity of HAs
144    Key mutations in the virus hemagglutinin (HA) protein or reassortment with other pandemic viruses
145 f RIV4 (45 mug of recombinant hemagglutinin [HA] per strain, 180 mug of protein per dose) versus stan
146 pe and mutant MN/10 or BJ/92 hemagglutinins (HAs) were constructed and probed for reactivity with fer
147                                   Histamine (HA) is a biogenic amine that can accumulate to high conc
148 uit is the posterior hypothalamic histamine (HA) system, implicated in supporting wakefulness and hig
149                               Histaminergic (HA) neurons, found in the posterior hypothalamic tuberom
150                                     However, HA instillation ameliorated bladder hyperactivity, lesse
151                                     However, HA-enriched matrices increased production of vascular en
152 g HA-Ag and the HRP labeled HA antibody (HRP-HA-Ab).
153                                  Hyaluronan (HA) is a major component of this structure.
154 ma is characterized by excessive hyaluronan (HA) accumulation in the tumor microenvironment, elevatin
155 ceptor for the glycosaminoglycan hyaluronan (HA) demonstrate a number of neurological disturbances in
156 llular matrix (ECM), enriched in hyaluronan (HA) and its binding partner versican, which promotes mon
157 absence of a CD44-specific Ab or hyaluronan (HA).
158 CE STATEMENT We demonstrate that hyaluronan (HA) with different molecular weights produces opposing n
159 um- and phosphate-containing hydroxyapatite (HA) mineral within a collagenous matrix.
160              Nanocrystalline hydroxyapatite (HA) has good biocompatibility and the potential to suppo
161                       Porous hydroxyapatite (HA) bone grafting material has been used to fill periodo
162                            We found that IAV HA glycoproteins induce ER stress, resulting in HA degra
163 ed upon pH-induced conformational changes in HA.
164 leading to a greater than 4-fold increase in HA-binding IgG responses.
165 glycoproteins induce ER stress, resulting in HA degradation via ERAD and consequent inhibition of IAV
166  harbouring the same mutation (Ser301Phe) in HA that abolishes 46B8 binding to HA at low pH.
167                  The T-cell product includes HA-1 TCR CD4(+) T cells to augment the persistence and f
168 and that primary tumors can further increase HA immaturity even before secondary tumor formation, mim
169                                    Increased HA levels and mesenchymal cells, but not vascular endoth
170 tabolism in Hyal2(-/-) mice causes increased HA, which may promote endothelial-to-mesenchymal transit
171 able HA content, we confirmed that increased HA leads to mechanically softer hydrogels, consistent wi
172 abnormalities were associated with increased HA, vimentin-positive cells, and fibrosis in Hyal2(-/-)
173 wn of these proteins substantially increases HA expression and IAV replication.
174                                Individually, HA, NA, M1, M2, and NP were shown to self-associate in o
175              The importance of IL-10-induced HA synthesis for regenerative wound healing is demonstra
176 f chimeric VLPs (cVLPs) containing influenza HA and GPI-anchored CCL28 as antigen and mucosal adjuvan
177 nI, which play a critical role in initiating HA degradation.
178                               Interestingly, HA-CS NPs differentially adsorbed two unique anti-inflam
179 n the case of humic acid, incorporation into HA aggregates.
180 etween the coating HA-Ag and the HRP labeled HA antibody (HRP-HA-Ab).
181  levels or engagement of CD44 by its ligand (HA) or a CD44-specific Ab reduced NF-kappaB translocatio
182 he dissolution of QDs, while with low light, HA alone did not change their dissolution.
183                       With sufficient light, HA increased the dissolution of QDs, while with low ligh
184 ers, comprising bundles of small needle-like HA crystals, formed on etched surfaces that were cut per
185 on of large, randomly distributed plate-like HA crystals that were weakly attached, regardless of rod
186                                        Lower HA flows were associated with decreased graft survival (
187  regulates the expression of CD44, the major HA cell membrane receptor.
188 tumor cell dissemination contain less-mature HA (i.e., smaller, less-perfect, and less-oriented cryst
189 odel of localized breast cancer, metaphyseal HA nanocrystals were also smaller and less perfect than
190 ion of 2 amino acid mutations and a modified HA tag at the C terminus of PB1, which is sufficient to
191  to control membrane curvature and to modify HA's interaction with M1 protein, while the stunting of
192 e data demonstrate that disruption of normal HA catabolism in Hyal2(-/-) mice causes increased HA, wh
193 rmed by hyaluronic acid-coated chitosan NPs (HA-CS NPs).
194 tally relevant ligands (e.g., 10 mg L(-1) of HA or 100 muM of silicates).
195 th AHA (n = 115) and in 52%, 57%, and 81% of HA inhibitor patients (n = 63).
196 ns is preceded by intraislet accumulation of HA, a highly hygroscopic polymer.
197 nes of evidence suggest that the activity of HA neurons is important in the regulation of vigilance d
198 ed glycosylation site in antigenic site B of HA emerged, and these viruses remain prevalent today.
199 zymes that synthesize the building blocks of HA, UDP-Glucuronic acid and UDP-N-Acetyl-Glucosamine, as
200 used to compare the molecular composition of HA and FA.
201 dol stabilizes the prefusion conformation of HA that inhibits the large conformational rearrangements
202                                The degree of HA varies between CLL patients and positively correlates
203 d both the electrophysiological diversity of HA neurons in brain slices and the effect of their acute
204                                The effect of HA on HR depended on the type of autonomic inhibition (P
205                                The effect of HA-coatings on murine MSC was functionally determined bo
206 rt because it facilitates rapid formation of HA-HC complexes.
207 th spatially-matched histochemical images of HA, collagen and vessel perfusion.
208 und healing is demonstrated by inhibition of HA synthesis in a murine wound model by administering 4-
209 umbelliferone, a small-molecule inhibitor of HA synthesis, reduced HA accumulation, diminished swelli
210 raditional vaccines induced robust levels of HA inhibition (HI) titers, but failed to protect against
211 r dose) versus standard-dose IIV4 (15 mug of HA per strain, 60 mug of protein per dose) to compare th
212  a fundamental stage in the precipitation of HA, with PHOSPHO1 being identified as the principal enzy
213  to the fluorescence and color properties of HA and that hydrogen peroxide might whiten HA-AAAs by ox
214                            While the role of HA in malignancy is well-defined, the mechanisms driving
215                   The 45 degrees rotation of HA providing full intensity modulation of transmitted th
216 t the acute, cell type-specific silencing of HA neurons during wakefulness is sufficient to not only
217 H3 numbering) in the major antigenic site of HA was reported to be responsible for the antigenic drif
218 , 165, or 166 in the major antigenic site of HA.
219                 The major antigenic sites of HA are located in the globular head subdomain, which is
220 little is known about the optimal spacing of HA ligands, which ought to bridge binding sites within o
221                       A crystal structure of HA with bound Fab6649 shows the conserved antibody footp
222 mL for grade 4 FLA (1 mL equals 1 syringe of HA filler).
223 This occurs despite the observed tendency of HA to inhibit colonization on bare glass surfaces when s
224                The total mean (SD) volume of HA used was 6.1 (3.1) mL for grade 2 FLA; 9.3 (4.2) mL f
225 face glycoprotein; however, the diversity of HAs across species and antigenic drift of circulating st
226 receptor-binding site and the stem region on HA is severely constrained by their functional roles in
227  strain selection, and most vaccines rely on HA inhibition titers to determine efficacy, despite the
228 ggesting that eIF4E potentiates an oncogenic HA program.
229  specific to the HA head and stalk, but only HA stalk-specific antibodies mediated ADCC efficiently a
230 on of hyaluronan-metabolizing enzymes and/or HA receptors in KIC.
231 t influence VLP shape, lipid composition, or HA lateral spacing, acylation significantly affected env
232 ngs did not exhibit increases in versican or HA in these regions and had strikingly reduced numbers o
233 l region contained smaller and less-oriented HA nanocrystals relative to ones that constitute the dia
234 ion that occurred in epidemic strains, other HA mutations can confer resistance to antibodies that re
235 solate with six gene segments (PB2, PB1, PA, HA, NA, and NS) sharing >99% sequence identity with thos
236      Here, we obtained two anti-A(H1N1)pdm09 HA monoclonal antibodies that failed to neutralize virus
237 s with bimodal listening experience (CI plus HA in contralateral ear) completed a questionnaire that
238  The majority of continuing bimodal (CI plus HA) participants reported adapting to using both devices
239 ound in the PRF (56.46% +/- 9.26%) and PRF + HA (63.39% +/- 16.52%) groups compared to controls (15.9
240 effectiveness of autologous PRF versus PRF + HA in treatment of IBDs in patients with chronic periodo
241 ity magnetic immunocapture to rapidly purify HA-tagged mitochondria from homogenized mammalian cells
242 12 (pdm2009); antibodies in CL6649 recognize HAs from the entire period.
243             Both enzymes cleaved recombinant HA from several strains of the H1 and/or H3 virus subtyp
244                    Electrochemically reduced HA enhanced NO2-to-HONO conversion by a factor of 2 rela
245 ion by a factor of 2 relative to non-reduced HA.
246 -molecule inhibitor of HA synthesis, reduced HA accumulation, diminished swelling, and restored basal
247 Strikingly, eIF4E inhibition alone repressed HA levels as effectively as directly targeting HA with h
248           A combination of virus resistance, HA interaction, and molecular dynamics simulation studie
249 s diseases associated with oxidative stress, HAs can be exploited for attractive nutraceutical applic
250  levels as effectively as directly targeting HA with hyaluronidase.
251 to incorporate ts mutations and a C-terminal HA tag.
252     Gene Ontology analysis demonstrates that HA-CS NPs were the least immunogenic nanocarriers.
253   This work was based on the hypothesis that HA treatment altered the bladder urothelial layer and th
254                        The results show that HA can be reliably detected at depths of 0-2.3 mm, which
255                                          The HA is composed of a globular head domain for receptor bi
256 appa-biased Ab response directed against the HA globular head and stem regions.
257 allenge, and antibodies directed against the HA head were the major contributor toward immune protect
258  aimed at boosting Ab reactivity against the HA stem region, also elicited an Igkappa-biased response
259   Using recombinant viruses, we identify the HA genomic segment as the mediator of cell death inhibit
260 h in the PB2 and NS1 proteins and two in the HA protein.
261 her, our results indicate that shifts in the HA receptor affinity are just an early adaptation step o
262 t viruses encoding amino acid changes in the HA stalk domain replicated well in vitro, and viruses in
263 Arbidol binds in a hydrophobic cavity in the HA trimer stem at the interface between two protomers.
264  position towards the N-terminus side of the HA PCS (P2 position) avoided hydrophobic residues, where
265 glycosylation at positions 158 to 160 of the HA protein and that this N-linked glycosylation enhanced
266  augment the persistence and function of the HA-1 TCR CD8(+) T cells and includes only memory T cells
267 9 safety switch to enable elimination of the HA-1 TCR T cells in case of toxicity; and (4) a CD34-CD2
268 an optokinetic zone spans a ZII+/- pair: the HA zones span the P5+/- and P7+/- ZII stripe pairs, wher
269 Moreover, our observations indicate that the HA prefusion structure (and perhaps the metastable state
270          These findings demonstrate that the HA protein stalk domain can undergo limited drift under
271 infection induced antibodies specific to the HA head and stalk, but only HA stalk-specific antibodies
272                                          The HAs of H15 viruses contain an insertion in the 150-loop
273 safety and efficacy and supports use of this HA filler for treatment of HIV-associated FLA with durab
274 approximately 40-fold more likely to bind to HA than nonrolling cells in shear flow.
275 301Phe) in HA that abolishes 46B8 binding to HA at low pH.
276  protein levels in HAAA sections compared to HA.
277 red cell product and tracking of transferred HA-1 TCR T cells.
278  binding sites within or across the trimeric HA molecules.
279                                        Tumor HA levels were measured retrospectively using a novel af
280   Among the four mutations detected, the two HA mutations were analyzed by generating recombinant vir
281 ating H3N2 strains compared to the wild-type HA antigens that were represented in commercial influenz
282 cted by antibodies elicited by the wild-type HA from viruses selected as the vaccine candidates.
283 panel than VLP vaccines expressing wild-type HA proteins.
284                        Compared to wild-type HA, deacylated HA is correlated with released particles
285                   In hydrogels with variable HA content, we confirmed that increased HA leads to mech
286                                  While viral HA acylation is crucial in virus replication, its physic
287  antibodies that recognize influenza A virus HA can be protective, but the mechanism is not completel
288 s of Arbidol in complex with influenza virus HA from pandemic 1968 H3N2 and recent 2013 H7N9 viruses.
289                   While low molecular weight HA increases sensitivity to mechanical stimulation, high
290 echanical stimulation, high molecular weight HA reduces sensitization, attenuating inflammatory and n
291 patial offset depended on the depth at which HA was located.
292                                        While HA acylation did not influence VLP shape, lipid composit
293 f HA and that hydrogen peroxide might whiten HA-AAAs by oxidizing the benzene ring in AAAs.
294 rrets for the elicitation of antibodies with HA inhibition (HAI) activity against human seasonal H3N2
295 d acute lung injury and, in association with HA, generates an ECM that promotes leukocyte infiltratio
296 fluence within 10-15 days when cultured with HA + ROCK.
297 ly cocluster, but the association of M1 with HA or NA is dependent upon the means of expression.
298 .53 to 1.00; P = .049) and for patients with HA-high tumors (HR, 0.51; 95% CI, 0.26 to 1.00; P = .048
299                             In patients with HA-high tumors (PAG v AG), the objective response rate w
300 s much HONO was formed when NO2 reacted with HA that was photoreduced by irradiation with UV-visible

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