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1                                              HBIG and lamivudine combination therapy was administered
2                                              HBIG mono-therapy was administered to one patient.
3                                              HBIG was given perioperatively and continued thereafter;
4                                        Among HBIG recipients, the HBsAg-positive rate was significant
5 nts of HBsAg-positive women receive HepB and HBIG </=12 hours of birth.
6                                     HepB and HBIG are administered at birth to infants of HBsAg-posit
7                               Lamivudine and HBIG combination decreases HBV recurrence 4-fold.
8  combination prophylaxis with lamivudine and HBIG prevented hepatitis B recurrence following liver tr
9 n liver transplantation (LT) recipients, but HBIG is costly and inconvenient to administer, prompting
10 ed or selected by immune pressure exerted by HBIG.
11  who developed recurrent hepatitis B despite HBIG prophylaxis were compared.
12  who developed recurrent hepatitis B despite HBIG prophylaxis, and to determine if these mutations ca
13 nsplantation), it is possible to discontinue HBIG and maintain only long-term nucleos(t)ide analogue(
14  The combination of lamivudine and high-dose HBIG can protect against reinfection of the hepatic allo
15 ion with the use of lamivudine and high-dose HBIG.
16 ificant problem with monotherapy with either HBIG or lamivudine.
17 -negative mothers who were or were not given HBIG.
18 HbsAg- received hepatitis B immune globulin (HBIG) 10,000 IU i.v. daily for 7 days and monthly for 6
19 al and low-dose hepatitis B Immune globulin (HBIG) can effectively prevent HBV recurrence in greater
20                 Hepatitis B immune globulin (HBIG) dosing regimens have been poorly defined, utilize
21                 Hepatitis B immune globulin (HBIG) has been an integral component of prophylaxis agai
22       Long-term hepatitis B immune globulin (HBIG) has been shown to reduce hepatitis B virus (HBV) r
23 ose intravenous hepatitis B immune globulin (HBIG) treatment with continued lamivudine treatment.
24 unotherapy with hepatitis B immune globulin (HBIG).
25                               In this group, HBIG-free prophylaxis cannot be recommended.
26 the second case, hepatitis B immunoglobulin (HBIG) immunoprophylaxis was administered in an attempt t
27  rate, even with hepatitis B immunoglobulin (HBIG) prophylaxis.
28 d lamivudine and hepatitis B immunoglobulin (HBIG) significantly reduced the reinfection rate (P<0.03
29 nd administering hepatitis B immunoglobulin (HBIG) to newborns.
30 ovir, lamivudine+hepatitis B immunoglobulin (HBIG), or lamivudine+entecavir on direct sequencing were
31  infants receive hepatitis B immunoglobulin (HBIG).
32 -positive rate was 15% in HB immunoglobulin (HBIG) recipients (adjusted odds ratio [OR]: 15.63; 95% c
33 eceived hepatitis B-specific immunoglobulin (HBIG) at birth.
34       The efficacy of combination lamivudine/HBIG prophylaxis has not been reported.
35 .796; P=0.020) and combination of lamivudine/HBIG (RR, 0.249; P=0.021) are independently associated w
36 igen (HBeAG)-positive patients required more HBIG to achieve these goals than HBeAG-negative individu
37                            Administration of HBIG to infants born to HBeAg-negative mothers did not a
38 s B DNA levels did not predict the amount of HBIG required.
39      We conclude that a very short course of HBIG combined with long-term antiviral therapy is highly
40 e 'a' determinant region and the duration of HBIG therapy.
41                   After 6 months, 1000 IU of HBIG was given IM. every 2 weeks for 18 months.
42 ese mutations can revert after withdrawal of HBIG.
43 had a de novo change after the withdrawal of HBIG.
44 bined with a short course (in hospital only) HBIG in liver transplant recipients with HBV DNA less th
45 ed in 1 of 1050 children who did not receive HBIG (.095%).
46     Two control patients who did not receive HBIG had no change in the 'a' determinant in their postt
47           Among students who did not receive HBIG, there was a significantly negative association bet
48 ho did not were more likely to have received HBIG at birth, suggesting that passive antibody may inte
49 V DNA undetectable to 100 IU/mL who received HBIG 5000 IU in anhepatic phase and daily for 5 days tog
50              The single patient who received HBIG monotherapy became HbsAg+ at 6 months.
51  antigen (HBeAg) positivity and who received HBIG off-schedule.
52  in liver transplant recipients who received HBIG prophylaxis.
53                 In addition to HBV vaccines, HBIG was given to all 583 children with hepatitis B e an
54 antigen (HBsAg)-positive mothers and whether HBIG administration reduces transmission.
55  patients receiving combination therapy with HBIG and lamivudine have remained HbsAg-.
56                     Combination therapy with HBIG and lamivudine may prevent de novo HBV infection in
57  the nucleoside analogue lamivudine, without HBIG, in patients undergoing liver transplantation.

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