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1 NA was detected an average of 31 days before HCV antibody.
2 HIV-1-positive women who tested negative for HCV antibody.
3 with significantly different efficiency with HCV antibodies.
4 investigation who were seropositive for anti-HCV antibodies.
5 ved between viremia and the presence of anti-HCV antibodies.
6 ides mimicking this structure to elicit anti-HCV antibodies.
7 care workers tested negative for HCV RNA and HCV antibodies.
8 creases the efficacy of broadly neutralizing HCV antibodies.
9 differed by age (50 vs. 42 years), positive HCV antibody (11 vs. 2%) and death from stroke (51 vs. 4
11 nts transplanted during 1996 to 2001 who had HCV antibodies (Ab) measured before transplantation.
13 ibe early experience with integrated opt-out HCV antibody (Ab) screening of medically stable baby boo
14 through the HCV continuum of care (CoC) from HCV antibody (Ab) screening, HCV-RNA confirmation, engag
15 screening with liver function tests (LFTs), HCV antibody (Ab) screening, or HCV RNA screening in var
17 prevalence of IDU, HIV antibody, HBsAg, and HCV antibody among PWID were selected and, where multipl
20 n at Cairo University were screened for anti-HCV antibodies and HCV RNA, and viremic women were teste
22 ents had HCV RNA (chronic infection), 91 had HCV antibodies and no HCV RNA (cleared infection), and 1
26 rom injection drug users (IDUs) persistently HCV antibody and RNA negative despite high-risk behavior
29 s among women who tested positive for HIV-1, HCV antibody, and HCV RNA, compared with HIV-1-positive
30 lood samples were tested for the presence of HCV antibodies (anti-HCV), HBV surface antigen (HBsAg),
31 er, Women and Infants Transmission Study had HCV antibody (anti-HCV by second-generation ELISA) and H
33 rs, Chembio, OraSure, and MedMira, submitted HCV antibody (anti-HCV) rapid screening assays to the CD
34 se Control and Prevention in 1998 recommends HCV antibody (anti-HCV) testing for persons with specifi
35 y assigned to receive a one-time offering of HCV antibody (anti-HCV) testing via one of three indepen
36 ase, we identified all subjects initiated on HCV antibody (anti-HCV) therapy from 2001 to 2014, and a
37 ed with incident HCV in MSM since 1984, 5310 HCV antibody (anti-HCV)-negative MSM in the Multicenter
38 2-1993, the prevalence of hepatitis C virus (HCV) antibodies (anti-HCV) among US blood donors was 0.3
39 t EIA were 97.78, 93.54, and 97.66% for anti-HCV antibodies, anti-HBsAg antibodies, and HBsAg, respec
41 Seronegative participants were tested for HCV antibodies at baseline, at 6 months, and at 12 month
44 strates the principle that neutralizing anti-HCV antibodies can be induced by epitope-based, engineer
45 viruses, likely due to synergy between anti-HCV antibodies derived from different plasma donors, and
48 t hepatitis B surface antigen in three (6%), HCV antibody (enzyme-linked immunosorbent assay II suppl
51 C virus (HCV) viremia, involving testing for HCV antibody (HCVAb) followed by a nucleic acid test (NA
52 (RVF) immunotechnology for detection of anti-HCV antibodies in an effort to extend the capabilities o
53 ised 7,005 and 21,729 persons diagnosed with HCV antibodies in Denmark and Scotland, respectively.
55 alence was estimated by the presence of anti-HCV antibodies incorporating respondent-driven sampling
56 These results suggest that profiling anti-HCV antibody is useful for monitoring HCV therapy, espec
57 IV infection on B cell function, we compared HCV antibody levels and specificities in 29 HCV-infected
59 ds that simultaneously estimated the time to HCV-antibody loss in uninfected infants and the diagnost
65 ic blood donors from 1991 to 2002 and 10,259 HCV antibody-negative (HCV-) donors matched for year of
67 with a history of illicit drug use who were HCV antibody-negative in 1988 were followed semiannually
71 /HTLV-II(+), and HIV(-)/HTLV-II(-)), 376 had HCV antibodies, of whom 305 had detectable HCV load.
72 We performed both G2 and G3 EIAs for anti-HCV antibodies on 1,134 serum samples collected during t
76 ated with screening for HCV infection (using HCV antibody or RNA), performed temporal analyses to ass
77 ia whose HLA-identical donor was found to be HCV antibody positive and HCV RNA positive by polymerase
78 who were HCV antibody negative, 34 who were HCV antibody positive but RNA negative, and 112 who were
79 egative recipients who received kidneys from HCV antibody positive donors (D-HCV) (n=48); and (3) HCV
80 ups of adult renal allograft recipients: (1) HCV antibody positive recipients (R-HCV) (n=32); (2) HCV
82 f the 5777 people who inject drugs that were HCV antibody positive, 440 (5.5%) were aware of their st
84 nty-six of the 1000 (7.6%) participants were HCV antibody positive; none were confirmed by detection
88 D are living with HIV, 52.3% (42.4-62.1) are HCV-antibody positive, and 9.0% (5.1-13.2) are HBV surfa
89 tes between those who were transplanted from HCV antibody-positive (HCV+) vs. HCV antibody-negative d
90 ,259 recombinant immunoblot assay-confirmed, HCV antibody-positive (HCV+), allogeneic blood donors fr
93 isk of vertical HCV infection to children of HCV antibody-positive and RNA-positive women was 5.8% (9
94 of risk were generated for children born to HCV antibody-positive and viremic women, aged >/=18 mont
103 Sixty-two percent of respondents would refer HCV antibody-positive patients with abnormal transaminas
104 approach to the management of 2 hypothetical HCV antibody-positive patients, 1 with elevated and the
105 HCV-specific CD4+ T-helper-cell response in HCV antibody-positive persons who lack detectable plasma
113 est that HCV infection could cause some anti-HCV-antibody-producing hybridoma B cells to make less-pr
114 stern blot analyses with anti-c-Myc and anti-HCV antibodies provided positive identification of both
118 ent of respondents would retest patients for HCV antibody, regardless of risk factors and transaminas
119 ne the effect of HIV coinfection on the anti-HCV antibody response, we measured anti-HCV envelope bin
120 ends in HCV RNA testing following a positive HCV antibody result among persons in 4 large healthcare
121 rus (HCV) infection requires both a positive HCV antibody screen and confirmatory nucleic acid testin
122 cted to full clinical evaluation, ELISA anti-HCV antibodies screening, parasitological examination fo
123 between 2000 and 2011, 14 534 (85%) received HCV antibody screening within 3 months of enrolling in c
124 ntibody-positive at enrolment and those with HCV antibody seroconversion during follow-up (1996 to 20
125 atabase of 13,664 MHD patients who underwent HCV antibody serology testing at least once during a 3-y
127 o three genotype 1a-derived HCV antigens and HCV antibody serotype were examined in chronically HCV i
131 ements and detectable HCV RNA, or a positive HCV antibody test result if HCV RNA measurements were no
132 ements and detectable HCV RNA, or a positive HCV antibody test result if HCV RNA measurements were no
133 e adjusted risk ratio (aRR) for receiving an HCV antibody test, and costs were estimated using activi
137 sly undiagnosed HCV disease, as suggested by HCV antibody testing and HCV polymerase chain reaction a
138 months), saliva was collected for anonymous HCV antibody testing and risk behavior data were obtaine
141 solicitation), evaluated hepatitis C virus (HCV) antibody testing, diagnosis, and costs for each of
145 he combined anti-core, anti-E1, and anti-NS4 HCV antibody titers in those with SVRs but not in those
148 ce with either gradual or rapid loss of anti-HCV antibody was observed in four animals within 5 month
152 Using the HCV-LP ELISA, high-titer anti-HCV antibodies were detected in individuals infected wit
154 egative for human immunodeficiency virus and HCV antibodies were recruited into a prospective study i
156 t the proportion of patients with a positive HCV antibody who had a positive HCV RNA was 0.5 (95% con
158 Fully implemented birth cohort screening for HCV antibody would have missed 36 of 128 (28%) of cases
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