ライフサイエンスコーパス: HFRS

1 HFRS has prominent systemic manifestations.

2 HFRS is primarily a Eurasian disease, whereas HPS appear

3 HFRS patients have increased thrombopoiesis and platelet

4 ociated with HPS [New York-1 virus (NY-1V)], HFRS [Hantaan virus (HTNV)], or by a hantavirus not asso

5 ate ratio of AMI/stroke in the 21 days after HFRS against 2 different control periods either excludin

6 xamined the risk of cancer development after HFRS in the Swedish population between 1997 and 2011 (n

7 nd stroke in the immediate time period after HFRS.

8 roke events occurred in the first week after HFRS.

9 ection by HPS causing Andes virus (ANDV) and HFRS causing Hantaan virus (HTNV) are inhibited by alpha

10 e findings indicate that pathogenic HPS- and HFRS-causing hantaviruses enter cells via beta3 integrin

11 a to humans or from human to human and cause HFRS and HCPS, with mortalities of 15% and 50%, respecti

12 us is an Old World hantavirus known to cause HFRS.

13 We collected HFRS patients' information and captured field and reside

14 reased intravascular coagulation risk during HFRS, which could be due to platelet activation.

15 all indicate increased thrombopoiesis during HFRS.

16 et fraction, and mean platelet volume during HFRS.

17 enome sequences of Hantaan virus (HTNV) from HFRS patients and rodent hosts in endemic areas.

18 responses in 13 Finnish individuals who had HFRS between 1984 and 1995.

19 tails of pathogenic Andes (HPS) and Hantaan (HFRS) viruses are also degraded by the proteasome and th

20 ain findings of this study are that the high HFRS incidence (19.3/100,000) is correlated to the captu

21 tly increased in vivo platelet activation in HFRS patients with intravascular coagulation (DIC and th

22 that may contribute to immune components of HFRS disease.

23 his study is to determine the correlation of HFRS incidence with capture rate and hantavirus infectio

24 The correlations of HFRS incidence to rodent capture rate and hantavirus inf

25 t, we demonstrate that the cellular entry of HFRS-associated hantaviruses is facilitated by specific

26 HTNV causes the most severe form of HFRS (5 to 15% case-fatality rate) and afflicts tens of

27 tect humans against the most severe forms of HFRS.

28 easomal degradation of the G1 tail in HPS or HFRS is unclear, these findings link G1 tail degradation

29 onal identification numbers from the Swedish HFRS patient database (1997-2012; n=6643) were cross-lin

30 mans: hemorrhagic fever with renal syndrome (HFRS) and hantavirus cardiopulmonary syndrome (HCPS).

31 cause hemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome (HPS) in humans.

32 lt in hemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome (HPS).

33 ects, hemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome (HPS).

34 ases, hemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome (HPS).

35 ases: hemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome (HPS).

36 sult: hemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome (HPS).

37 ases, hemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome (HPS).

38 omes: hemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome.

39 ) and hemorrhagic fever with renal syndrome (HFRS) are severe diseases associated with hantavirus inf

40 cause hemorrhagic fever with renal syndrome (HFRS) in Europe and Asia.

41 Hemorrhagic fever with renal syndrome (HFRS) is a severe acute disease.

42 ither hemorrhagic fever with renal syndrome (HFRS) or hantavirus cardiopulmonary syndrome (HCPS) in h

43 Hemorrhagic fever with renal syndrome (HFRS) was considered to be transmitted by Apodemus agrar

44 auses hemorrhagic fever with renal syndrome (HFRS), which is an important public health problem in la

45 with hemorrhagic fever with renal syndrome (HFRS), which is caused by hantaviruses.

46 ludes hemorrhagic fever with renal syndrome (HFRS).

47 ) and hemorrhagic fever with renal syndrome (HFRS).

48 n endothelial cells and Vero E6 cells by the HFRS-causing hantaviruses Hantaan (HTN), Seoul (SEO), an

49 Therefore, HFRS patients should be carefully monitored during the a

50 nd nonpathogenic hantaviruses, contribute to HFRS and HPS pathogenesis, and provide insight into dise

51 ccines or specific drugs to prevent or treat HFRS and HCPS and the requirement for conducting experim

52 However, it is not known whether HFRS is a risk factor for the acute cardiovascular event