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1 mutants has not been delineated for an anti-HIV antibody.
2 llomavirus (HPV), Neisseria gonorrhoeae, and HIV antibody.
3 nd intravenous immunoglobulin (IVIG) without HIV antibody.
4 um samples that were repeatedly reactive for HIV antibodies.
5 n more potent than broadly neutralizing anti-HIV antibodies.
6 ty than well-known broadly neutralizing anti-HIV antibodies.
7 mens that had previously tested positive for HIV antibodies.
8 e enterotoxin (shortest distance 31 A), anti-HIV antibody 2G12 (shortest distance 31 A), concanavalin
9 recognized by the broadly neutralizing anti-HIV antibody 2G12 are 3-fold more abundant on the native
10 hich were recognized by broadly neutralizing HIV antibody 2G12 with moderate affinities (150-500 nM K
11 proteins (Aspergillus niger phytase and anti-HIV antibody 2G12) produced in different plant species a
13 s) claim to detect both p24 antigen (Ag) and HIV antibodies (Ab) for early identification of acute in
16 he presence of maternal or administered anti-HIV antibody, alternative strategies may be required to
17 after the exposure, no participant developed HIV antibodies, although a second PEP course for a subse
18 o determine the frequency of vaccine-induced HIV antibody among uninfected HIV vaccine trial particip
19 These distinctive activities suggest that HIV antibodies and their derivatives may play an importa
20 was to determine the timing of clearance of HIV antibodies and to identify any associated biological
22 yearly follow-up visits included testing for HIV antibody and assessment of behavioural outcomes.
23 ot with HIV(IIIB), allowing for detection of HIV antibodies approximately 3 weeks earlier than by RT-
29 s of relatively inexpensive and quantitative HIV antibody assays may be useful indirect markers that
31 se the recognition of infected cells by anti-HIV antibodies at the cell surface, thereby reducing rec
32 tive for hepatitis B surface antigen or anti-HIV antibody at screening, or if they had previously bee
34 vectors encoding a secretory monoclonal anti-HIV antibody, b12 (IgG(1)), we were able to program huma
35 in the United States and tested positive for HIV antibodies between 1988 and 1997 while living in the
38 t a targeted conjugate consisting of an anti-HIV antibody bound to a toxic moiety could function to k
40 Passive transfer of broadly neutralizing HIV antibodies can prevent infection, which suggests tha
41 mbinations of polyclonal and monoclonal anti-HIV antibodies can produce additive or synergistic neutr
42 es aimed at stabilizing this region in other HIV antibodies could become an important approach to in
43 , (iii) potentiates the non-membrane-binding HIV antibody D5 10-100-fold (depending on the virus stra
45 mmunoassay for human immunodeficiency virus (HIV) antibody detection that localizes capture and detec
49 eveloped to exploit the titer and avidity of HIV antibody evolution following seroconversion for inci
50 s encoding a mixture of broadly neutralizing HIV antibodies for the treatment of HIV infection, parti
55 A series of potent, broadly neutralizing HIV antibodies have been isolated from B cells of HIV-in
61 we show that the polymeric IgA form of anti-HIV antibody inhibits HIV mucosal transmission more effe
63 studies, the time to seroconversion for anti-HIV antibodies is 1-9 months (mean, approximately 2-3 mo
64 nistration-approved enzyme immunoassay (EIA) HIV antibody kits were used to determine VISP, and a rou
65 Fourteen people with acute HIV infection (HIV antibody negative/NAT positive) were identified; the
68 inics in India, screening for p24 antigen in HIV antibody-negative persons was found to be a reliable
71 to HIV-infected mothers, 299 children became HIV-antibody-negative and 264 of these had been followed
72 gp160 enzyme immunoassay (EIA), detection of HIV antibodies occurred, on average, 33 days earlier tha
73 ASI reported having sexual partners who were HIV antibody positive (odds ratio = 1.36, 95% confidence
74 d those who knew that the source subject was HIV antibody positive were more likely to recruit their
78 mune response to HIV often occurs-serum anti-HIV antibodies reactive with live HIV-infected cells, te
79 n induced a distinct and characteristic anti-HIV antibody response that, in some animals, included ne
82 vaccine might consider eliciting protective HIV antibody responses selectively from alternative B-ce
86 e U.S. Food and Drug Administration-licensed HIV antibody screening, p24 antigen tests, HIV confirmat
88 ns with recent human immunodeficiency virus (HIV) antibody seroconversion is useful for treatment, re
89 pendently associated with p24 antigenemia in HIV antibody-seronegative persons included fever, which
92 spective living organ donors, and to conduct HIV antibody testing and NAT as close to the time of don
93 us self-administered questionnaires and oral HIV antibody testing in MSM recruited in gay social venu
98 pared the diagnostic performance of standard HIV antibody tests (i.e., enzyme immunoassay and Western
100 man immunodeficiency virus (HIV) to standard HIV antibody tests to detect persons with acute HIV infe
102 Thus far, however, few broadly neutralizing HIV antibodies that occur naturally have been characteri
104 with live HIV-infected cells, termed "early HIV antibodies." The specificities of these antibodies a
105 , however, are the unique properties of anti-HIV antibodies: the potential ability to opsonize viral
107 nability of most known specificities of anti-HIV antibodies to neutralise HIV primary isolates consis
108 ministered a macaque version of a human anti-HIV antibody to monkeys, after which the antibody persis
110 tion, the addition of one-time screening for HIV antibodies with an enzyme-linked immunosorbent assay
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