ライフサイエンスコーパス: HIV protein

1 P-gp and MRP-1 expressed similar amounts of HIV protein.

2 lade CTL activity is not limited to a single HIV protein.

3 ese cells showed antigen specificity against HIV proteins.

4 infected microglia and neuronal toxicity by HIV proteins.

5 sequences that encode inter-domain loops in HIV proteins.

6 ne; 218 of 354 (62%) recognised two to three HIV proteins.

7 consensus sequence peptide set spanning all HIV proteins.

8 ecombinant vaccinia virus vectors expressing HIV proteins.

9 mammalian cells in the absence of any other HIV proteins.

10 residues of Nef and was independent of other HIV proteins.

11 ignals from CD4+ T cells are affected by the HIV protein 3D structure; and thus the protectiveness of

12 icroscopy showed prominent colocalization of HIV protein and D47A, in agreement with the intracellula

13 in situ RNA hybridization with detection of HIV protein and flow cytometry, enabling detection of 0.

14 is associated with neuropathology involving HIV proteins and activation of proinflammatory cytokine

15 eduction in the number of cells positive for HIV proteins and by decreases in HIV proviral DNA and p2

16 es, we here predict five potential antisense HIV proteins and characterize common CTL responses again

17 -IgA immune complexes, the colocalization of HIV proteins and HIV-specific IgA was detected intracell

18 The direct effect of HIV proteins and illicit drugs was investigated on oxida

19 Here we focus on the effect of HIV proteins and inflammatory cytokines implicated in HA

20 information that is currently available for HIV proteins and reviews current structure-function and

21 accine is composed by five long fragments of HIV proteins and was recently shown to induce in seroneg

22 lated TGFbetaR1 and TGFbetaR2 on exposure to HIV-proteins and cocaine was confirmed in pulmonary smoo

23 pulmonary smooth muscle cells on exposure to HIV-proteins and/or cocaine due to severe down-modulatio

24 s the wild-type virus, suggesting that other HIV proteins are responsible for inducing apoptosis.

25 Given that HIV proteins can overcome host restriction factors and t

26 ultrastructural features represent bona fide HIV protein complexes.

27 small-molecule inhibitors against untargeted HIV proteins could be used to dissect key events in the

28 y driven by neuroinflammation and neurotoxic HIV proteins (e.g., envelope and Vpr).

29 ins a distinctive plasmid DNA expressing all HIV proteins except integrase to induce immune responses

30 The virus envelope (Env) is the only HIV protein expressed on the surfaces of virions and inf

31 mediated targeting of additional vaccinating HIV proteins fused to gp120(alphagal), thereby creating

32 ted the lower binding capacity for fungi and HIV protein gp-120 when the levels of miR-155 were highe

33 y, transgenic mice engineered to express the HIV protein gp120 exhibited increased brain levels of CD

34 The HIV protein gp120 reduced the length of neuronal process

35 posure of cultured neurons to the neurotoxic HIV proteins gp120 and Tat resulted in increased cellula

36 Peptide T20, which targets the HIV protein gp41, represents the first approved member o

37 e context of a polypeptide sequence from the HIV protein gp41, which represents an excellent testbed

38 as indicated by mimicry of the CHR domain of HIV protein gp41.

39 sease pathogenesis, particularly the role of HIV proteins in mediating renal tissue injury.

40 nded our approach to detect cells expressing HIV proteins in patients suppressed on ART.

41 rly, hCG treatment resulted in a decrease of HIV proteins in the skin of nonpregnant heterozygous tra

42 four different human immunodeficiency virus (HIV) proteins in lysing primary HIV-infected targets.

43 ell as renal cellular responses, mediated by HIV proteins (including an immune-activated microenviron

44 he viral DNA as well as several cellular and HIV proteins, including the integrase.

45 ation by functional ontology and known human-HIV protein interactions, we observed the enrichment for

46 otypes of natural or artificial mutations in HIV proteins--interpretation of mutation phenotypes is a

47 Importantly, we identified areas of HIV proteins leading to presentation of noncanonical pep

48 ti-HIV treatments targeting only 4 of the 15 HIV proteins, many potential viral vulnerabilities remai

49 ble-negative alpha/beta T cells that express HIV protein may be a component of the long-lived reservo

50 monocytogenes vaccines engineered to secrete HIV proteins may be ideal vectors for boosting cellular

51 romote p53 activation, suggest that distinct HIV proteins may converge on the p53 pathway to promote

52 thesized that hepatocytes exposed to HCV and HIV proteins might be susceptible to injury via an "inno

53 ates erecta led to isolation of a novel anti-HIV protein, named niphatevirin.

54 This, however, does not occur, as the HIV protein Nef acts as an antiautophagic maturation fac

55 HIV protein Nef causes dyslipidemia and formation of foa

56 The HIV protein Nef is thought to mediate immune evasion and

57 AR) and NF-kappaB activity and interact with HIV proteins nef and matrix.

58 The human immunodeficiency virus (HIV) protein Nef has been shown to increase the infectiv

59 Two HIV proteins, Nef and Tat, increase T cell activity, but

60 a viruses that stably express a chimeric B5R-HIV protein or a control HIV envelope protein with the o

61 TGF-beta(1) in response to stimulation with HIV proteins or peptides.

62 rus expressing human immunodeficiency virus (HIV) proteins or soluble MN rgp120.

63 The HIV protein p24 was detected by fluorescence confocal mi

64 Mouse fibrocytes pulsed in vitro with the HIV-proteins p24 or gp120 and delivered to a site of cut

65 t the three-dimensional (3D) structure of an HIV protein partially determines which epitopes are domi

66 Antibody levels to these HIV proteins persisted at high and stable levels in most

67 ines should elicit CD8+ T cells specific for HIV proteins presented on MHC class I products, because

68 Human immunodeficiency virus (HIV) protein R (Vpr) induces G2 arrest, and prolonged G2

69 ts suggest that OLs can be direct targets of HIV proteins released from infected cells.

70 e investigated the effect of Nef, a secreted HIV protein responsible for the impairment of cholestero

71 As a consequence, the levels of HEXIM1 and HIV proteins rose.

72 Our findings suggest that SIV/HIV protein(s) and morphine interact to cause the prolif

73 y transplanted the HIV 4E10 epitope onto non-HIV protein scaffolds for structural stabilization and i

74 tion structure of the potent 95 residue anti-HIV protein scytovirin has been determined and two carbo

75 A new anti-HIV protein, scytovirin, was isolated from aqueous extra

76 active Gq alpha subunit tagged with the TAT HIV protein sequence was introduced into an immortalized

77 s no clear consensus as to which of the nine HIV proteins should be used for vaccination.

78 support the concept that PI3K activation (by HIV proteins such as Nef) may contribute to reduced TLR4

79 Specific HIV proteins, such as Tat protein, can contribute to the

80 The HIV protein Tat is both neurotoxic and proinflammatory;

81 The HIV protein Tat, a viral regulator required for efficien

82 d in the presence of other de novo-expressed HIV proteins that may have had additive proapoptotic eff

83 In a cell culture model, we show that the HIV protein transactivator of transcription (Tat) initia

84 ld-type p38 protein (PTD-p38WT) in which the HIV protein transduction domain (PTD) was fused to the N

85 e system restriction factor APOBEC3G and the HIV protein Vif is a key host-retrovirus interaction.

86 indings (de Noronha et al.) showing that the HIV protein Vpr is crucial for causing transient herniat

87 The HIV protein Vpu antagonizes this host defense.

88 The accessory HIV protein Vpu inhibits a number of cellular pathways t

89 Griffithsin (GRFT), a novel anti-HIV protein, was isolated from an aqueous extract of the

90 was flowed through the chip and the released HIV proteins were assayed off-chip.

91 vivo CTL frequencies specific for different HIV proteins were consistently lower than responses spec

92 synaptic changes observed after exposure to HIV proteins, which may underlie cognitive impairment in

93 yzing two commercially available recombinant HIV proteins with affinity tags at the N-terminus, and h

94 dentification of potent (sub-nanomolar) anti-HIV proteins with significantly improved pharmaceutical