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1 rates that syndecan functions as an in trans HIV receptor.
2 sceptibility to HIV infection by rearranging HIV receptors.
3 infection of cells coexpressing the specific HIV receptors.
4 omotes, copatching of HLE with the canonical HIV receptors.
5 ependent of depression status, downregulates HIV receptor and coreceptor expression and may reduce su
6 ts the rapid, actin-dependent recruitment of HIV receptors and adhesion molecules to the interface, r
7 IV-1 cell-to-cell transmission by binding to HIV receptors and coreceptors and regulating their clust
8 o Th17 cells, including higher expression of HIV receptors and lack of autocrine CCR5 ligands, are as
9 s mode of protection suggested that the anti-HIV receptor antibody is efficacious for prophylaxis aft
10 proteins in pathways known to be impacted by HIV-receptor binding, changed significantly within a min
13 e absence of antigen-specific signaling, the HIV receptors CD4, CCR5, and CXCR4 on the T cell were re
14 by determinations of its ability to bind the HIV receptor, CD4, and its ability to bind a panel of we
15 ression of the human immunodeficiency virus (HIV) receptor cluster of differentiation 4 (CD4) and cor
16 CD4, an integral component of the functional HIV receptor complex, through accelerated endocytosis of
19 n the relationship between the expression of HIV receptor/coreceptors, susceptibility, and chemokine-
23 regulates the cell-surface expression of the HIV receptor glycoprotein CD4, but the significance of t
24 dition, intracellular signaling triggered by HIV receptors has been shown to play important roles in
26 as reagents containing portions of CD4, the HIV receptor, have demonstrated partial efficacy in supp
27 CD4 molecule, in addition to serving as the HIV receptor, may also be involved in postbinding events
30 d not affect the membrane disposition of the HIV receptor proteins CD4, CXCR4, and CCR5, as determine
31 both iNKT cells and DCs expressing CD1d and HIV receptors resided in the female genital mucosa, a si
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