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1 transcription through HIF-1 association with HIV-1 long terminal repeat.
2 mplexes and histone-modifying enzymes to the HIV-1 long terminal repeat.
3 ing the binding activity of NF-kappaB to the HIV-1 long terminal repeat.
4 re the assembly of a specific complex on the HIV-1 long terminal repeat.
5 paB interaction with the viral promoter, the HIV-1 long terminal repeat.
6 Tat protein activates transcription from the HIV-1 long terminal repeat.
7 PKR under the transcriptional control of the HIV-1 long terminal repeat.
8 ntisense tat moiety under the control of the HIV-1 long terminal repeat.
9 ich single-stranded RNA40 (ssRNA40) from the HIV-1 long terminal repeat.
10 t protein (humanized S65T GFP) driven by the HIV-1 long terminal repeat.
11 or Sp1, and activated transcription from the HIV-1 long-terminal-repeat.
14 Tat transactivated gene expression from the HIV-1 long terminal repeat and COT Nef mediated downregu
15 provirus under the control of the endogenous HIV-1 long terminal repeat and demonstrated that human c
16 nstrated by electromobility shift assays and HIV-1 long terminal repeat and NF-kappa B-dependent repo
17 te HIV-1(JR-CSF) regulated by the endogenous HIV-1 long terminal repeat and the hu-cycT1 gene under t
19 mediate gene silencing, is recruited to the HIV-1 long terminal repeat by the host transcription fac
21 quired Skp2 since its transactivation of the HIV-1 long terminal repeat decreased in primary mouse em
22 cells transiently transfected by a panel of HIV-1 long terminal repeat deletion mutants linked to th
23 ells stably expressing CCR5 and carrying the HIV-1 long terminal repeat-driven secretory alkaline pho
24 mined the effects of different plasmid-based HIV-1 long-terminal-repeat-driven constructs expressing
25 not TRAF1, induced NF-kappaB activation and HIV-1-long terminal repeat-driven transcription in the T
26 HDAC2 and HDAC3 contribute to repression of HIV-1 long terminal repeat expression in the HeLa P4/R5
27 slocation of NF-kappaB and activation of the HIV-1 long terminal repeat in a kappaB enhancer-dependen
28 y of other cellular factors that bind to the HIV-1 long terminal repeat, including NF-kappaB, SP1, LB
29 idermidis releases factors that activate the HIV-1 long terminal repeat, induce cytokine release, and
30 in the human immunodeficiency virus, type 1 (HIV-1), long terminal repeat inhibited transcription in
31 The human immunodeficiency virus type 1 (HIV-1) long terminal repeat is present on both ends of t
33 -stranded RNA structure transcribed from the HIV-1 long terminal repeat known as TAR is critical for
34 n activate the human immunodeficiency virus (HIV)-1 long terminal repeat (LTR) in the Jurkat T lympho
40 cation was associated with inhibition of the HIV-1 long terminal repeat (LTR) and induction of ISGF-3
41 recruit histone deacetylase 1 (HDAC1) to the HIV-1 long terminal repeat (LTR) and inhibit transcripti
42 Tat-Tax fusion was able to activate both the HIV-1 long terminal repeat (LTR) and the HTVL-1 LTR at t
43 protein (C/EBP) beta and C/EBP sites in the HIV-1 long terminal repeat (LTR) are crucial for HIV-1 r
44 nduction of p53 and NF-kappaB binding to the HIV-1 long terminal repeat (LTR) by the T cell receptor
45 cillating pattern of RelA recruitment to the HIV-1 long terminal repeat (LTR) during continuous tumor
46 IC cDNA modulates Tat transactivation of the HIV-1 long terminal repeat (LTR) in a cell type-specific
47 s not significantly increase the activity of HIV-1 long terminal repeat (LTR) in T lymphocytes, but i
48 pression of wild-type WRN transactivates the HIV-1 long terminal repeat (LTR) in the absence of Tat,
49 ion factor LSF binds to sequences within the HIV-1 long terminal repeat (LTR) initiation region and r
50 y suggested, but rather transcription of the HIV-1 long terminal repeat (LTR) is increased in IL-4-pr
51 has the ability to modulate transcription of HIV-1 long terminal repeat (LTR) promoter activity and a
53 c and HDAC1 are coordinately resident at the HIV-1 long terminal repeat (LTR) promoter and absent fro
55 zation of the G-quadruplex structures in the HIV-1 long terminal repeat (LTR) promoter suppresses vir
56 GF-beta can stimulate transcription from the HIV-1 long terminal repeat (LTR) promoter through NF-kap
57 200 bp that represent the intact ends of the HIV-1 long terminal repeat (LTR) sequences (mini-HIV).
58 tor that up-regulates transcription from the HIV-1 long terminal repeat (LTR) through binding to a na
59 the p160 proteins (GRIP1) is tethered to the HIV-1 long terminal repeat (LTR) through kappaB-responsi
60 mutants demonstrated that MAPK activates the HIV-1 long terminal repeat (LTR) through the NF-kappaB s
61 an transcription factor, is known to inhibit HIV-1 long terminal repeat (LTR) transcription and virus
62 roteins mapped to the NF-kappaB sites in the HIV-1 long terminal repeat (LTR) U3 and could be transfe
63 pression and replication, transactivates the HIV-1 long terminal repeat (LTR) via its binding to the
64 C/EBPbeta expression was maintained and the HIV-1 long terminal repeat (LTR) was not maximally stimu
66 show that Tat transactivates the integrated HIV-1 long terminal repeat (LTR), even in the absence of
67 acid and 9-cis retinoic acid, also repressed HIV-1 long terminal repeat (LTR)-directed expression up
68 combinant ProTalpha protein potently inhibit HIV-1 long terminal repeat (LTR)-driven gene expression
69 d cyclin T1 and contained a transactivatable HIV-1 long terminal repeat (LTR)-green fluorescent prote
70 scriptional elongation complex essential for HIV-1 long terminal repeat (LTR)-mediated and general ce
78 rom the human immunodeficiency virus type 1 (HIV-1) long terminal repeat (LTR) by enhancing the elong
79 ation of histone proteins at the HIV type 1 (HIV-1) long terminal repeat (LTR) by histone deactylases
80 rom the human immunodeficiency virus type 1 (HIV-1) long terminal repeat (LTR) by increasing the proc
81 The human immunodeficiency virus type 1 (HIV-1) long terminal repeat (LTR) controls the expressio
82 The human immunodeficiency virus type-1 (HIV-1) long terminal repeat (LTR) initiates transcriptio
83 The human immunodeficiency virus type 1 (HIV-1) long terminal repeat (LTR) introduced in associat
84 NRE) of human immunodeficiency virus type-1 (HIV-1) long terminal repeat (LTR) is a defined region th
85 of the human immunodeficiency virus type 1 (HIV-1) long terminal repeat (LTR) promoter and increased
86 of the human immunodeficiency virus type 1 (HIV-1) long terminal repeat (LTR) promoter element by th
87 ranscriptional activation of the HIV type 1 (HIV-1) long terminal repeat (LTR) promoter element by th
88 of the human immunodeficiency virus type 1 (HIV-1) long terminal repeat (LTR) promoter element is re
89 egrated human immunodeficiency virus type 1 (HIV-1) long terminal repeat (LTR), suggesting that PITAL
93 ls, nuclear translocation and binding to the HIV-1 long terminal repeat of p65 and p50 proteins from
94 tion or cycling status were observed and Alu-HIV-1 long terminal repeat polymerase chain reaction (LT
95 virus (MMTV) promoter and of the Tat-induced HIV-1 long terminal repeat promoter by directly binding
98 role of uridine-rich ssRNA derived from the HIV-1 long terminal repeat (ssRNA40) on activation of NK
100 that E2F-1 may regulate the activity of the HIV-1 long terminal repeat through its ability to bind s
101 (from the 5' human immunodeficiency virus-1 [HIV-1] long terminal repeat) to examine the role of NF-k
103 and USF2 up-regulate gene expression (i.e. , HIV-1 long terminal repeats) via interaction with an E b
104 of the human immunodeficiency virus type 1 (HIV-1) long terminal repeat, we demonstrate that a full-
105 er and when Gag expression was driven by the HIV-1 long terminal repeat within pHXB2DeltaBalD25S, a n
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