ライフサイエンスコーパス: HMW

1 HMW adiponectin and the ratio of HMW to total adiponecti

2 HMW adiponectin dose-dependently suppressed PDGF-induced

3 HMW concentration increases the fluid elasticity, thereb

4 HMW fibroblast growth factor-2 (FGF-2) inhibits cell mig

5 HMW fraction had the highest total phenolics, condensed

6 HMW multimers and the closure time with adenosine diphos

7 HMW peaks contained PDE3A1 and PDE3A2, whereas LMW peaks

8 HMW tau derived from CSF of AD patients was seed compete

9 HMW, highly aromatic, alkylated compounds decreased in r

10 HMW-HA binding to CD44 selectively inhibits the GTP load

11 HMW-HA inhibits the mitogen-dependent induction of cycli

12 HMW-HA triggers hypersensitivity of naked mole rat cells

13 HMW-MAA-specific CARs containing an antigen recognition

14 00, Siluron 5000, 7% HMW + Siluron 1000, 10% HMW + Siluron 1000, and 15% HMW + Siluron 1000; Fluoron

15 Fluid elasticity was optimum using 10% HMW.

16 iluron 1000, 10% HMW + Siluron 1000, and 15% HMW + Siluron 1000; Fluoron GmbH, Ulm, Germany) were mea

17 ation was at a minimum when using 10% or 15% HMW blends.

18 Siluron 1000, Siluron 2000, Siluron 5000, 7% HMW + Siluron 1000, 10% HMW + Siluron 1000, and 15% HMW

19 , 95% CI -0.38 to -0.30, P = 2.0 x 10(-70)), HMW adiponectin (beta = -0.40, 95% CI -0.43 to -0.36, P

20 mimics has been previously shown to induce a HMW-MAA-specific, T cell-dependent Ab response associate

21 Body composition (by absorptiometry), HMW adiponectin, and IGF-I were assessed at birth and 4

22 analyzed the reassembly of the most abundant HMW adiponectin species, the octadecamer, following its

23 ntly, under certain conditions, the abundant HMW oAbeta can dissociate into low molecular weight spec

24 erobic microbial metabolism for accelerating HMW-PAHs removal occurred within sediments after combini

25 In addition, HMW adiponectin could be separated into three distinct o

26 hese conditions, total arterial adiponectin, HMW, and the ratio of HMW to total adiponectin all were

27 uated the relationship of total adiponectin, HMW adiponectin, and the HMW-to-total adiponectin ratio

28 ovel insight into the mechanisms that affect HMW adiponectin homeostasis.

29 In conclusion, a Lm-based vaccine against HMW-MAA can trigger cell-mediated immune responses to th

30 combinant EtpA (anti-rEtpA.6H) recognized an HMW protein in culture supernatants of ETEC strains H104

31 onstituted <2% of the sum of the 15 analyzed HMW PAHs.

32 vely, inhibited the effects of LMW-FGF-2 and HMW-FGF-23 to stimulate FGF-23 promoter activity.

33 se TGFbeta1 and TGFbeta2 as well as FGF2 and HMW-HA.

34 lators of epicardial cell behavior, FGF2 and HMW-HA.

35 invasion in response to TGFbeta2, FGF2, and HMW-HA.

36 ry demonstrated co-localization of FGF23 and HMW FGF2 protein in osteoblasts and osteocytes from Hyp

37 IAIP and HMW-HA colocalized with histones in necrotic tissues and

38 plasmon resonance showed that both IAIP and HMW-HA directly bind to recombinant histone H4.

39 ivo neutralization of histones with IAIP and HMW-HA prevented histone-induced thrombocytopenia, bleed

40 lammatory response after exposure to LMW and HMW agents by specific inhalation challenge test (SIC).

41 suggest that IAIP, chondroitin sulfate, and HMW-HA are potential therapeutic agents to protect again

42 Total and HMW adiponectin are inversely associated with incident P

43 variants strongly influence plasma total and HMW adiponectin levels in East Asian populations but app

44 Total and HMW adiponectin plasma concentrations were measured befo

45 Both total and HMW-adiponectin decreased, and IL-6 increased with incre

46 t number affects both hmwA transcription and HMW-adhesin production such that as the number of repeat

47 links were discovered in monoclonal antibody HMW species.

48 olecular weight melanoma-associated antigen (HMW-MAA), also known as melanoma chondroitin sulfate pro

49 olecular weight melanoma-associated antigen (HMW-MAA), which is highly expressed on more than 90% of

50 olecular variant of rat DSP, referred to as "HMW-DSP", has been speculated to be a proteoglycan form

51 Unphosphorylated DUE-B HMW complex formation is decreased by PP2A inhibition or

52 the tumor growth of early established B16F10-HMW-MAA tumors in mice and both CD4(+) and CD8(+) T cell

53 se inhibition [% decrease from the baseline (HMW vs. LMW) was 36.9 vs. 74.1% (Abeta40, P<0.05) and 25

54 Because HMW is absent from mature flagella, we propose that HMW

55 m an AD brain contains potentially bioactive HMW tau species, giving new insights into the role of CS

56 nd the transient memory impairment caused by HMW oligomers, but did not prevent the persistent cognit

57 On the other hand, memory deficit induced by HMW AbetaOs (10 pmol) was found to be reversible.

58 g/m(2)]), which was accounted for largely by HMW polymers.

59 and gelatin was effectively precipitated by HMW fraction.

60 ip1) messenger RNA levels were unaffected by HMW-HA, but the expression of Skp2, the rate-limiting co

61 attachment site(s), we further characterized HMW-DSP.

62 Conversely, in children, HMW phthalate metabolites were inversely associated with

63 Low levels of circulating HMW adiponectin appear to increase the risk for insulin

64 may be involved in regulation of circulating HMW adiponectin levels and provide novel insight into th

65 on factor and inflammatory mediator, cleaves HMW FGF-2 into an LMW FGF-2-like form that stimulates en

66 ular weight RNA-induced silencing complexes (HMW-RISC) associated with target mRNA.

67 a- and omega-types whereas glutenins contain HMW- and LMW-types.

68 In contrast, HMW-FGF-2 stimulated FGF-23 promoter activity in osteobl

69 The PA appear to preferentially crosslink HMW-GS via hydrophobic interactions and hydrogen bonding

70 uently, mechanisms responsible for decreased HMW adiponectin in insulin resistance are not well under

71 anbaalenii PYR-1 mutant (6G11) that degrades HMW PAHs but not LMW PAHs.

72 We conclude that conditions that destabilize HMW oAbeta or retard the sequestration of smaller, more

73 We conclude that conditions that destabilize HMW oligomers or retard the sequestration of their small

74 components might be involved in determining HMW and total adiponectin levels.

75 omain inhibits the ability of Tax to disrupt HMW P-TEFb complexes.

76 Western blotting showed that endogenous HMW PDE3A1 was the principal PKA-phosphorylated isoform.

77 ributes to pioglitazone's ability to enhance HMW adiponectin levels, but additional factors likely af

78 tumors, which were not engineered to express HMW-MAA.

79 , 0.66 (0.39-1.13), and 0.40 (0.22-0.74) for HMW and 1.0, 0.74 (0.43-1.25), and 0.35 (0.18-0.65) for

80 After adjustment for HMW adiponectin levels, the minor allele was associated

81 ith insulin resistance before adjustment for HMW adiponectin levels.

82 d odds ratio and 95% confidence interval for HMW: 1.0, 0.62 [0.29-1.34], 0.30 [0.12-0.74]; total: 1.0

83 n vascular smooth muscle cells, as it is for HMW-HA, and that the opposing cell cycle effects of thes

84 nders, associations were not significant for HMW or DEHP metabolites, and results did not change subs

85 OC size classes was +/-0.25 per thousand for HMW fraction, +/- 0.54 per thousand for LMW fraction, an

86 variable analyses showed that the values for HMW multimers and CT-ADP at the end of TAVR were each as

87 trations, vesicular Abeta aggregates to form HMW species which are capable of seeding amyloid fibril

88 ative) form and lower molecular weight form (HMW- and LMW-HA, respectively).

89 omatography, high-molecular weight fraction (HMW) using high-performance size-exclusion chromatograph

90 ping PAD than in those remaining event free (HMW: 3.3 versus 3.8 mug/mL, P=0.0005; total: 5.6 versus

91 two independent IgE-reactive fragments from HMW Bx7 contained repetitive IgE epitopes.

92 <0.05) with temperature and time from LMW>WM>HMW.

93 On the other hand, HMW oligomers, but not LMW oligomers, induced oxidative

94 The HfaD HMW form is dependent on HfaA but not on holdfast polysa

95 o PU which could be attributed to its higher HMW PAH concentration.

96 ulfate and high-molecular-weight hyaluronan (HMW-HA) associated with IAIP.

97 lar weight-polycyclic aromatic hydrocarbons (HMW-PAHs) in sediments.

98 This identifies HMW as a novel, evolutionarily conserved component neces

99 To determine if HMW-DSP is the proteoglycan form of DSP and to identify

100 After the initial implantation, HMW multimers normalized in patients without aortic regu

101 sible for low-molecular-weight (LMW) PAHs in HMW PAH-metabolic networks remain poorly understood.

102 way increased the assembly of microRNAs into HMW-RISC, enhanced expression of the glycine-tryptophan

103 d predicted structural homologies with known HMW adhesins produced by other two-partner secretion loc

104 breast tumor model, immunization with Lm-LLO-HMW-MAA-C caused CD8(+) T-cell infiltration in the tumor

105 Immunization with Lm-LLO-HMW-MAA-C was able to impede the tumor growth of early e

106 a recombinant Listeria monocytogenes (Lm-LLO-HMW-MAA-C) that expresses and secretes a fragment of HMW

107 2/K(b) transgenic mice immunized with Lm-LLO-HMW-MAA-C.

108 phy into low- and high-molecular-weight (LMW/HMW) fractions.

109 erature and time from FL<FG<RL<RG and LMW<WM<HMW for all pHs.

110 The mAb HMW fractions were collected using preparative size-excl

111 n a dose-dependent manner, while maintaining HMW adiponectin.

112 dian total adiponectin and 32% higher median HMW adiponectin concentrations, as well as 16% lower res

113 PFA-CADP profiles mimicked HMW multimers recovery both in transcatheter aortic valv

114 s in vitro reconstituted (rcHC*HA) by mixing HMW HA, serum IalphaI, and recombinant TSG-6.

115 n of low MW molecules (LMW) to form high MW (HMW) molecules.

116 x was 0.63 for CRP, IL-6, C-peptide, and non-HMW adiponectin and 0.46 for GLDH, indicating good predi

117 centrations of CRP, IL-6, C-peptide, and non-HMW adiponectin were associated with higher risk of HCC

118 AGA-BRF infants, SGA-BRF infants had normal HMW adiponectin and IGF-I levels at 4 months, whereas SG

119 Notably, HMW Abeta decreased more slowly than other forms of Abet

120 morphism explained 4.1% of total and 6.5% of HMW adiponectin levels.

121 ciated form L166P resulted in the absence of HMW DJ-1 complexes.

122 can play a role in regulating the amount of HMW complex present in the cell by decreasing the bindin

123 Cells expressing large amounts of HMW adhesins may be critical for the establishment and m

124 e found that the major IgE-reactive areas of HMW glutenins are located in the repetitive regions of t

125 vascular smooth muscle cells; the binding of HMW-HA to CD44 inhibits cell cycle progression, whereas

126 rotein of 182 kDa, an essential component of HMW-RISC, and improved the ability of microRNAs to repre

127 Plasma concentrations of HMW adiponectin did not change significantly during the

128 synthesis or through the depolymerization of HMW products by the action of two specific endoglycanase

129 striking example is the opposing effects of HMW- and LMW-HA on the proliferation of vascular smooth

130 ittle is known about the combined effects of HMW-GS and PINs on dough functional properties.

131 nd CD44-null mice showed that the effects of HMW-HA/CD44 on cyclin D1 and Skp2 gene expression are de

132 Details are included for the expression of HMW TpsA glycoproteins as polyhistidine-tagged molecules

133 rostructure rather than through formation of HMW aggregates with resultant light scattering.

134 C) that expresses and secretes a fragment of HMW-MAA (residues 2,160-2,258) fused to the first 441 re

135 hology was not detected in the hippocampi of HMW oligomer-injected mice.

136 However, incubation of HMW oAbeta in mildly alkaline buffer led to their quanti

137 ereas SGA-FOF infants had elevated levels of HMW adiponectin (particularly SGA-FOF1) and IGF-I (parti

138 Baseline median levels of HMW and total adiponectin were significantly lower in wo

139 dealkylation trends and the overall loss of HMW species observed by FT-ICR MS has not previously bee

140 The results suggest that overexpression of HMW FGF2 increases FGF23/FGFR/KLOTHO signaling to down-r

141 The presence of HMW-multimer defects and a high value for a point-of-car

142 arterial adiponectin, HMW, and the ratio of HMW to total adiponectin all were lower (P < 0.01) in th

143 HMW adiponectin and the ratio of HMW to total adiponectin are lower in individuals with d

144 We further investigated the recovery of HMW multimers and monitored these changes with PFA-CADP

145 the time course of the induction/recovery of HMW multimers defects under instantaneous changes in she

146 ents demonstrated that induction/recovery of HMW multimers occurs within 5 minutes.

147 esidual aortic regurgitation, no recovery of HMW multimers was observed.

148 e decrease in shear stress and a recovery of HMW multimers within minutes of implantation which was s

149 nscriptional and translational regulation of HMW and LMW FGF-2 has been extensively investigated, lit

150 -, thrombin-, or collagen-induced release of HMW-EGF.

151 all population of adherent cells in spite of HMW adhesin specific antibody-mediated immunity.

152 dentified cyclin D1 as the primary target of HMW-HA.

153 take do not significantly increase plasma or HMW adiponectin concentrations in overweight-to-moderate

154 ns high-molecular-weight polyethylene oxide (HMW PEO).

155 ve highly toxic, high-molecular weight PAHs (HMW-PAHs) in coal-tar samples.

156 irreversibly shuffle HCs from pathological, HMW HC-HA to HA oligosaccharides, thereby restoring HC-H

157 hat a rare species of soluble phosphorylated HMW tau is the endogenous form of tau involved in propag

158 ins a high-molecular-weight S-layer protein (HMW SLP) and its low-molecular-weight partner protein (L

159 icular CSF from AD patients contained a rare HMW tau species that exerted a higher seeding activity.

160 duced the migration rate for portions of rat HMW-DSP to the level of DSP.

161 Disaccharide analysis showed that rat HMW-DSP contains glycosaminoglycan chains made of chondr

162 Recombinant HMW Bx7 may be included into the panel of allergens for

163 ve deterioration coincidentally with reduced HMW soluble oligomeric Abeta in the brain.

164 adenine DI-phosphate [PFA-CADP]), reflecting HMW multimers changes, could be used to monitor in real-

165 xyl phthalate metabolites (both representing HMW phthalate exposures) were positively associated with

166 of thrombin on these cell functions requires HMW FGF-2 cleavage.

167 lysis of a nationally representative sample, HMW phthalate metabolites, particularly MBzP, were posit

168 ere found in the potentially metal-sensitive HMW (Ag and Ni) and LMW (Tl) pools, whereas the MMW pool

169 ilar magnitude as TE, while increasing serum HMW adiponectin above SHAM and GX animals (p<0.05).

170 enome-wide association study (GWAS) of serum HMW adiponectin levels in individuals of European ancest

171 ng ADIPOQ locus (3q27) were related to serum HMW adiponectin levels.

172 equence reads have barcodes linked to single HMW DNA molecules.

173 pe neuropathology and the content of soluble HMW extracellular oligomeric Abeta peptides in the brain

174 ble amyloid plaques likely sequester soluble HMW oligomers, limiting their potential to dissociate.

175 DAMDEC1, which hydrolyzes pro-EGF to soluble HMW-EGF, that HMW-EGF is active, that proteolytic cleava

176 ctions available: high MW glutenin subunits (HMW-GS) over low MW-GS, and omega-gliadins over alpha- a

177 ns, high molecular weight glutenin subunits (HMW-GS), plays an important role in dough functional pro

178 We conclude that HMW adiponectin is downregulated in hyperinsulinemia and

179 These observations confirmed that HMW-DSP is the proteoglycan form of DSP (renamed "DSP-PG

180 hydrolyzes pro-EGF to soluble HMW-EGF, that HMW-EGF is active, that proteolytic cleavage of pro-EGF

181 absent from mature flagella, we propose that HMW is not a structural component of the motile axoneme

182 We previously reported that HMW-HA binding to CD44 antagonizes mitogen-induced S-pha

183 se digestion or NaOH treatment revealed that HMW HA was covalently linked with the heavy chains (HCs)

184 We show that HMW is conserved in species with motile cilia and that,

185 s. 88.0% (Abeta42, P<0.01)], suggesting that HMW Abeta oligomers clear more slowly than other forms f

186 The present study suggests that HMW fraction could be utilised as a source of polyphenol

187 The HMW DNA is digested using an appropriate restriction enz

188 The HMW fractions were IdeS digested, reduced, and analyzed

189 The HMW phthalate metabolite monobenzyl phthalate (MBzP) was

190 nanthrene, anthracene, and fluorene, and the HMW PAHs pyrene, fluoranthene, and benzo[a]pyrene, with

191 -0.43 to -0.36, P = 1.1 x 10(-117)), and the HMW-to-total adiponectin ratio (beta = -0.44, 95% CI -0.

192 total adiponectin, HMW adiponectin, and the HMW-to-total adiponectin ratio with incident symptomatic

193 his study was undertaken to characterize the HMW MMP activity in OA SF.

194 hmwA, which encodes the HMW adhesin, undergoes phase variation mediated by 7-bas

195 terized using recombinant fragments from the HMW Bx7 and synthetic peptides thereof for testing of al

196 same antibodies also immunoprecipitated the HMW gelatinase activity from OA SF.

197 HPSEC showed differences in the HMW fraction for different degrees of ripeness and both

198 evealed that among the proteins found in the HMW fraction is VPS35, a latent cytosolic component of t

199 ric reducing antioxidant power (FRAP) in the HMW fractions of 3.5-100 kDa and/or >100 kDa from the co

200 as many as seven covalent cross-links in the HMW fractions, where oxidized histidines react with inta

201 Cwp13 cleaves SlpA in the HMW SLP domain, which we suggest may reflect a role in c

202 yosin complex compared to 11 residues in the HMW striated muscle overlap complex.

203 ses to a known HLA-A2 epitope present in the HMW-MAA(2160-2258) fragment was detected in the HLA-A2/K

204 face hydrophobic residues of CD1 mediate the HMW complex assembly and affect the catalytic activity,

205 Notably, the HMW tau species was also detected in lumbar CSF from AD

206 were identified by mass spectrometry of the HMW complexes.

207 dropped, primarily due to a reduction of the HMW form, whereas LMW forms were not significantly affec

208 fective in BIR contain reduced levels of the HMW form.

209 Following optimization of the HMW-MAA-specific CAR for expression and function in huma

210 ecognition domain based on variations of the HMW-MAA-specific monoclonal antibody 225.28S and a T-cel

211 bioactive on synapses and microglia than the HMW species from which they are derived.

212 Our findings demonstrate that the HMW gelatinase activity in OA SF represents a complex of

213 Zymographic analysis showed that the HMW gelatinase in OA SF comigrated with a purified NGAL-

214 ts of Western immunoblotting showed that the HMW gelatinase was also recognized by antibodies specifi

215 bserved in the LMW fractions relative to the HMW fractions were substantially enhanced following a re

216 Furthermore, within the HMW-GS, PA bound more of the larger x-type than the smal

217 Therefore, HMW HA can serve as both an HC acceptor and an HC donor.

218 tly encountered with the expression of these HMW proteins, namely plasmid instability and protein deg

219 To evaluate this HMW-MAA-specific CAR in patients with metastatic melanom

220 nts lacking IgE to omega(5) -gliadin, and to HMW glutenin in 59%.

221 howed ADAMDEC1 hydrolyzed surface pro-EGF to HMW-EGF that stimulated HeLa EGF receptor (EGFR) reporte

222 samples from 20 subjects who were exposed to HMW (n = 10, Group I) and LMW (n = 10, Group II) at thei

223 Also, subjects exposed to HMW agents showed a significant increase in NL levels of

224 Exposure to HMW and LMW agents by SIC induced a differential nasal a

225 , correlated with shift of PDE3A from LMW to HMW peaks, and increased co-immunoprecipitation of SERCA

226 Viscosity increased proportionally to HMW concentrations.

227 e cytolytic, and proliferated in response to HMW-MAA-expressing cell lines.

228 ee high molecular weight glutenin sub units (HMW-GS) were decreased at e[CO2].

229 ease in intracellular high molecular weight (HMW) (>200 kDa) aggregates, which were absent in cells g

230 MW) (<3.5 kDa) and of high molecular weight (HMW) (3.5-100 kDa and >100 kDa) from cold water, hot wat

231 hesins, including the high molecular weight (HMW) adhesins that mediate attachment to the respiratory

232 High molecular weight (HMW) adiponectin is a predominant isoform of circulating

233 No differences in high molecular weight (HMW) adiponectin were observed between sexes or treatmen

234 AICAR), metformin, or high molecular weight (HMW) adiponectin.

235 recovered in distinct high molecular weight (HMW) and low molecular weight (LMW) peaks.

236 ples and measured the high-molecular weight (HMW) and low-molecular weight (LMW) fractions of adipone

237 racts of AD brain are high molecular weight (HMW) and relatively inactive.

238 is associated with a high molecular weight (HMW) complex.

239 1 predominantly forms high molecular weight (HMW) complexes that included RNA metabolism proteins hnR

240 DUE-B forms multiple high molecular weight (HMW) complexes.

241 causative agent was a high molecular weight (HMW) compound and in four cases it was a low molecular w

242 ct nuclei, containing high molecular weight (HMW) DNA, are isolated and embedded in agarose plugs.

243 he human genome using high molecular weight (HMW) DNA.

244 MW, 18 kDa) FGF-2 and high molecular weight (HMW) FGF-2 isoforms, which, respectively, activate cell

245 and assembles into a high molecular weight (HMW) form requiring HfaD, but not holdfast polysaccharid

246 nd characterized from high-molecular weight (HMW) fractions of an IgG1 monoclonal antibody (mAb).

247 ular weight (LMW) and high-molecular weight (HMW) fractions.

248 ed into low (LMW) and high molecular weight (HMW) fractions.

249 y 125-130-kd band for high molecular weight (HMW) gelatinase, which has not been characterized.

250 oding for portions of high molecular weight (HMW) glutenin subunits were identified by sequence analy

251 ophoresis showed that high molecular weight (HMW) HA (an average of approximately 3000 kDa) was predo

252 he transfer of HCs to high molecular weight (HMW) HA is a reversible event whereby TSG-6 can shuffle

253 High molecular weight (HMW) hyaluronan (HA) is widely distributed in the extrac

254 rowth factor 2 (FGF2) high molecular weight (HMW) isoforms in osteoblastic lineage cells in mice resu

255 induction/recovery of high molecular weight (HMW) multimers of von Willebrand factor defect could be

256 confirmed AD elute at high molecular weight (HMW) on nondenaturing size-exclusion chromatography.

257 ugh the conversion of high molecular weight (HMW) physiological alpha-syn conformers into compact, as

258 were analyzed for 15 high molecular weight (HMW) polycyclic aromatic hydrocarbons (PAH), using press

259 lasma adiponectin and high-molecular weight (HMW) polymeric adiponectin are strongly positively corre

260 mers, hexamers, and higher molecular weight (HMW) species, which are not fully characterized.

261 mers, hexamers, and higher molecular weight (HMW) species.

262 tion of a recombinant high molecular weight (HMW) two-partner secretion exoprotein (generically refer

263 transformed cells, or high molecular weight (HMW), found in muscle and nonmuscle cells.

264 Baseline total and high molecular weight (HMW)-adiponectin and interleukin (IL)-6 levels were meas

265 ght (LMW, 18 kDa) and high molecular weight (HMW, 22-24 kDa) forms that originate from alternative tr

266 importantly also the high molecular weight (HMW, 4-6 ring) PAHs that are considerably more toxic tha

267 nt hyaluronan of very high molecular weight (HMW-HA).

268 detecting polar and higher molecular weight (HMW; > 400 Da) components abundant in crude and heavy fu

269 DOC into 3 fractions: high molecular weight (HMW; 0.4-10 kDa), low molecular weight (LMW; 50-400 Da),

270 produced in high- and low-molecular-weight (HMW and LMW, respectively) fractions; however, only the

271 ular-weight (LMW) and high-molecular-weight (HMW) Abeta oligomers differentially impact synapses and

272 cumulation of soluble high-molecular-weight (HMW) Abeta oligomers has been proposed to be largely res

273 gher plasma total and high-molecular-weight (HMW) adiponectin concentrations and lower concentrations

274 ntrations of total or high-molecular-weight (HMW) adiponectin in healthy overweight-to-moderately obe

275 relation to total and high-molecular-weight (HMW) adiponectin using data from a genome-wide associati

276 diponectin, including high-molecular-weight (HMW) adiponectin, and incident peripheral artery disease

277 -6), C-peptide, total high-molecular-weight (HMW) adiponectin, leptin, fetuin-a, and glutamatdehydrog

278 ular-weight (LMW) and high-molecular-weight (HMW) agents have been recognized as causes of occupation

279 We could detect high-molecular-weight (HMW) and low-molecular-weight (LMW) Abeta oligomers in t

280 exists in hypoactive high-molecular-weight (HMW) complexes, which can be activated without apparent

281 mation of an aberrant high-molecular-weight (HMW) form of 2 microm.

282 form or an inactive, high-molecular-weight (HMW) form.

283 a/beta/gamma-gliadin, high-molecular-weight (HMW) glutenin, alpha-amylase inhibitor (AAI) dimer, and

284 essment of defects in high-molecular-weight (HMW) multimers of von Willebrand factor or point-of-care

285 f Abeta peptides into high-molecular-weight (HMW) oligomeric peptides, known to be involved in cognit

286 halates, particularly high-molecular-weight (HMW) phthalates, are suspected to contribute to allergy.

287 nowledge of bacterial high-molecular-weight (HMW) polycyclic aromatic hydrocarbon (PAH) metabolism, t

288 tly described soluble high-molecular-weight (HMW) species that is found in the postmortem AD brain an

289 oluble phosphorylated high-molecular-weight (HMW) tau, though very low in abundance, is taken up, axo

290 lecular-weight (LMW), high-molecular-weight (HMW), and di-2-ethylhexylphthalate (DEHP) metabolites, c

291 ed platelets released high-molecular-weight (HMW)-EGF, produced by a single cleavage between the EGF

292 th low- (LMW-BNP) and high-molecular-weight (HMW-BNP) forms.

293 e state (as judged by high-molecular-weight [HMW] adiponectin and IGF-I) of infants born small for ge

294 Zn among pools of various molecular weights (HMW: high molecular weight, >670-40 kDa; MMW: medium mol

295 rly to a 32-amino acid BNP standard, whereas HMW-BNP, when deglycosylated, was similar to deglycosyla

296 We assessed whether HMW FGF2 expression was altered in the Hyp mouse, a mous

297 While HMW agents act mainly through IgE-mediated mechanisms, L

298 outcomes were observed following dosing with HMW PEO alone.

299 Active immunization with HMW-MAA mimics has been previously shown to induce a HMW

300 Majority of varieties with HMW-GS combinations of 91kDa+80kDa+78kDa+74kDa PPs showe