ライフサイエンスコーパス: HT

1 se of mortality after heart transplantation (HT).

2 mplated hollow spheres (HS) or hollow tubes (HT).

3 ia-based glass-ceramics (e.g., IPS e.max CAD HT).

4 aphy for CAV diagnosis (median 5 years since HT).

5 confirmed using high-throughput sequencing (HTS).

6 entified out of a high-throughput screening (HTS).

7 whole-cell-based high-throughput screening (HTS).

8 wheat cultivars to high temperature stress (HTS).

9 in vitro chemosensitivity and Dox uptake at HT.

10 erived regions and the dorsal closure of the HT.

11 of CAV but have not been well studied after HT.

12 onergic signaling by increasing endogenous 5-HT.

13 g but only 3-DG was significantly reduced by HT.

14 s associated with abnormal FS (FSS</=8) post-HT.

15 iated with improved long-term survival after HT.

16 the second decade are associated with adults HT.

17 -fold lower than the biscuit formulated with HT.

18 d differential modulation by glutamate and 5-HT.

19 mipril on the development of CAV early after HT.

20 escending coronary artery, within 8 weeks of HT.

21 tunities in the prevention and management of HTS.

22 ion between the active and inactive state of hTS.

23 Combined HTS (11000 compounds) and in-house screening of a limite

24 d neural cells and applied to Illumina Human HT-12 v4 Expression BeadChips.

25 can in humans, we have recently found that 5-HT 1A receptor (5-HT1AR) function is modified after intr

26 t positive trends were observed for ALM, ALM/ht(2) (P < 0.001), and QS (P = 0.0028).

27 dicular lean mass normalized for height (ALM/ht(2)), and QS (2008-2011) and to 2) calculate adjusted

28 quartile 1) had significantly lower ALM, ALM/ht(2), and QS than did those in the higher quartiles of

29 Despite of the increased T-2/HT-2 concentrations found in some of the samples, the ob

30 As for final products, the highest T-2/HT-2 concentrations were determined in oat flakes (89.4m

31 udy was to investigate into the level of T-2/HT-2 toxins in different unprocessed cereals (n=201), as

32 , zearalenone (ZEA), four enniatins, T-2 and HT-2 toxins).

33 alenone, deoxynivalenol, fumonisins, T-2 and HT-2 toxins, fusarenone X, diacetoxyscirpenol, and 3- an

34 N), fumonisins (FB1+FB2+FB3), the sum of (T2+HT-2), NIV and ZEA, respectively.

35 signaling in the mouse hippocampal cell line HT-22 leading to an increase in CREB phosphorylation.

36 on inhibition fifty (IC50) for BL, SH, RG on HT-29 and HCT-116 cell proliferation ranged 0.9-2.0mg/mL

37 creases the efficacy of DOX-induced death in HT-29 and SW480 cells and in nude mice xenografts.

38 exerted cytotoxic activity against HeLa and HT-29 cell lines, but were inactive towards MRC-5 and MC

39 emonstrated using proteome-wide reactions of HT-29 cell lysates with a model probe of threonine beta-

40 rting measurable toxic effects on HEK293 and HT-29 cells.

41 HT-29 colon adenocarcinoma cells were formed into pellet

42 for their antiproliferative activity against HT-29 colon cancer cell lines.

43 shRNA to decrease NOX1 expression stably in HT-29 human colon cancer cells.

44 tes were also measured using a 70% Caco2/30% HT-29 human intestinal co-culture layer.

45 delphinidin-3-O-glucoside concentration with HT-29 inhibition (r=0.69).

46 HCT-116 and HT-29 inhibition correlated with total phenolics (r=0.87

47 nodrug, as well as animal survival rate in a HT-29 xenograft mouse model.

48 ble factor 1alpha (HIF-1alpha) expression in HT-29 xenografts initiated from NOX1 knockdown cells.

49 eaflet of the plasma membrane in intestinal (HT-29) cells and thereby reduce CT uptake into the cells

50 on of human colorectal adenocarcinoma cells (HT-29) under the control of visible light and the extent

51 rescence, and immunoblot analyses in Caco-2, HT-29, and T-84 cells human IECs cultured in 2 or 3 dime

52 ver and expression of 5-hydroxytryptamine (5-HT) 2A (5-HT2A) and 5-HT2C serotonin receptors in the hy

53 ese compounds include selective serotonin (5-HT)2C receptor antagonists, a 5-HT4 receptor agonist, a

54 ents were found in yellow-grained tritordeum HT 439 (12.16mug/gDW), followed by blue-grained wheat V1

55 Components of the 5-HT signalling system (5-HT, 5-HIAA, SERT) and TNF-alpha expression were examined

56 press TH2 and TPH1 and contain both DA and 5-HT, a dual neurotransmitter phenotype hitherto undescrib

57 f sour (acidic) tastants and this released 5-HT activates 5-HT3 receptors on the gustatory nerves.

58 ndings, we propose a regulatory mechanism of hTS activity that involves allosteric regulation of inte

59 Our results demonstrate that 5-HT acts as a potent antioxidant and binds with a superio

60 Because few women in the NHS initiated HT after age 60 years, we did not examine associations i

61 seeking during initial abstinence involves 5-HT and beta-adrenergic signaling in female DH, but only

62 s, we investigated the effects of blocking 5-HT and beta-adrenoceptor transmission in DH on drug seek

63 ype-specific control over BA circuitry via 5-HT and glutamate co-release to inhibit the BA output.SIG

64 We found that 5-HT neurons co-release 5-HT and glutamate onto BA neurons in a cell-type-specific

65 ationic viologen linker and, in the cases of HT and HS, permanent porosity.

66 subtype with the potential to release both 5-HT and substance P is necessary for normal respiratory d

67 HTS and FC showed similar 5-year EFS and OS for MRD-posi

68 We investigated the inactive conformation of hTS and found that the two insert regions are also invol

69 in both screens were confirmed in secondary HTS and low-throughput assays.

70 HTS and phylogenetic analysis of paired specimens confir

71 Serotonin (5-hydroxytryptamine, 5-HT) and brain-derived neurotrophic factor (BDNF) are two

72 rolled trials of menopausal hormone therapy (HT) and chronic disease risk appear to have divergent re

73 ctive and contain low levels of serotonin (5-HT) and high levels of the 5-HT metabolite 5-hydroxyindo

74 High-throughput sequencing (HTS) and bioinformatics have expanded our understanding

75 blood 5-HT levels, linking integrin beta3, 5-HT, and ASD risk.

76 The high-throughput (HT) approach we present can be readily adapted to target

77 Taken together, our results identify 5-HT as a potent inhibitor of lipid peroxidation and offer

78 Limitations: The HTEs as a function of the number of BP agents only were

79 yces Torulaspora delbrueckii, can synthesise HT, as this compound was identified in the intracellular

80 may be easily developed for certain enzymes, HTS assays designed to identify ligands that block prote

81 These biases are particularly evident in HTS assays for identifying regulatory regions in DNA (DN

82 centrations and their activities reported in HTS assays, we developed a probabilistic model for estim

83 The 11000 compounds were selected for the HTS based on the known phenothiazine Ndh inhibitors, tri

84 available tests, high-throughput sequencing (HTS)-based approaches are increasingly attractive for no

85 th a focus on the current and future role of HTS-based assays.

86 HTS-based methods can simultaneously identify multiple g

87 rate tool for analysis of sequences from our HTS-based protocol for CSR junctions, thereby facilitati

88 These effects were mediated by 5-HT because they were blocked by antagonists of 5-HT2A an

89 ing consecutive adult patients who underwent HT between January 2009 and December 2014 at our institu

90 The HT-BST may facilitate the rapid screening of 100000 chem

91 gh throughput biodegradation screening test (HT-BST).

92 ivation and sensitization of high-threshold (HT) but not wide-dynamic range trigeminovascular neurons

93 To compare the effectiveness of HT by means of home-based self-exercises using a CD with

94 BMI at birth was inversely associated with HT; c-BMI from school age to adulthood and c-height from

95 An HTS campaign using an RNA-dependent ATPase assay and ini

96 g patients (heterogeneous treatment effects [HTEs]) can be hidden in, and explain discordant results

97 Thus, the high-throughput (HT)-CBS technology was used for the concomitant quantita

98 -Danlos syndrome hypermobility type (JHS/EDS-HT), characterized by idiopathic chronic itch with predo

99 to enable more complete characterizations of HTS chemical matter.

100 resent study demonstrates that enzymatically HT-coated surface modification is an effective strategy

101 tudy examines the potency of neoadjuvant TSL HT combination therapy in two orthotopic mouse models of

102 ed analysis of chemical occurrence data with HTS data offers new opportunities to prioritize chemical

103 lyses translating high-throughput screening (HTS) data to human relevance have been limited.

104 When loading MK2i, 1) the HT depot surface gained high MK2i-loading efficiency thr

105 d poor (<5%) statistical power to detect the HTEs, despite large samples (n > 20 000), and produced b

106 Changes in heart transplantation (HT) donor and recipient demographics may influence the i

107 Conversely, e[CO2] + HT during flowering and early grain filling significantl

108 Large high-throughput screening (HTS) efforts are widely used in drug development and che

109 In the MOB, 5-HT elicited three types of responses in 93% of 180 cells

110 In comparison, 5-HT-evoked inhibitory responses in the AOB arose due to a

111 ith starch metabolism enzymes under e[CO2] + HT exposure.

112 In contrast, suppressing HT expression in SC.

113 a high-throughput enhanced yeast two-hybrid (HT-eY2H) assay and a mammalian-cell-based Gaussia prince

114 be the complete pattern of distribution of 5-HT fibers to the amygdala (proper) and to the extended a

115 As is well recognized, serotonergic (5-HT) fibers distribute widely throughout the forebrain, i

116 quinoline hit via high throughput screening (HTS) followed by optimization provided a 4-phenyl-3-aryl

117 psin, pepsin and a bacterial protease called HT for 1, 2, 4 and 24h.

118 dely distributed CNS modulator, serotonin (5-HT), for its ability to modulate the biophysical propert

119 soderm sliding over the endoderm, results in HT formation.

120 ing effect of age, time since menopause, and HT formulation.

121 rain QA23 was the most efficient producer of HT from tested yeasts.

122 n to maturity) and a combination of e[CO2] + HT (from heading to maturity) using field based open top

123 d important heterogeneous treatment effects (HTEs) from intensive weight loss interventions.

124 S children who survive at least 1 year after HT, FS is excellent for the majority of patients.

125 Total gene diversity (Ht), gene diversity within population (Hs), coefficient

126 he extracted proteins with either trypsin or HT generated more antioxidant activity than hydrolysis w

127 d be revised to incorporate the concept of 5-HT/glutamate cotransmission.

128 association with abnormal FS at 1 year post-HT: >/=18 years of age (odds ratio [OR], 1.8; 95% confid

129 The management of HTS has been challenging for clinicians, since current t

130 Serotonin or 5-hydroxytryptamine (5-HT) has been shown to be essential in lots of physiologi

131 osensitive liposomes (TSL) and hyperthermia (HT) has shown preclinically to achieve high local drug c

132 Inhibition of human TS (hTS) has been extensively investigated for cancer chemot

133 High-throughput screening (HTS) has enabled millions of compounds to be assessed fo

134 s (n = 65) and biopsies (n = 47) (Affymetrix HT HG-U133+ PM).

135 l optimization of high-throughput screening (HTS) hit 1 led to the identification of 3, which demonst

136 ng of biochemical high-throughput screening (HTS) hits led to the discovery of a series of ligands th

137 r studies identify a complex regulation of 5-HT homeostasis and behaviors by integrin alphavbeta3, re

138 All three materials-HT, HS, and COGF-serve as efficient adsorbents of iodine

139 s stabilized by hydrogen bonds between the 5-HT hydroxyl group and lipid headgroups and allows 5-HT t

140 wever, there was a high discordant rate with HTS identifying 55 (38.7%) more patients MRD positive at

141 exercises using a CD with that of individual HT (iHT) performed by qualified therapists.

142 sensitivity and lower false-negative rate of HTS improves upon FC for MRD detection in pediatric B-AL

143 primary monoamine oxidase that metabolizes 5-HT in C. elegans, and we also found that grk-2 loss-of-f

144 sor, and it is well known that it releases 5-HT in response to mechanical forces.

145 The EC cell model released 5-HT in response to stretch, and had Piezo2 mRNA and prote

146 -carboxyethyl-lysine, although the amount of HT in the biscuit was 100-fold lower than the biscuit fo

147 es dense 5-HT innervation, whether and how 5-HT in the dBNST normally modulates anxiety remains uncle

148 Low 5-HT in the placenta persisted, but forebrain levels norma

149 Combination treatment with TSLDox and HT in vivo was significantly more effective against MDA-

150 ations in brain extracellular dopamine and 5-HT in vivo.

151 cells, Type III cells, release serotonin (5-HT) in response to the presence of sour (acidic) tastant

152 tudy, we utilized high-throughput screening (HTS) in vitro data of PAHs to predict health risks assoc

153 nd antioxidants impinge on the complexity of HTS-induced responses over different genetic backgrounds

154 Among women aged 50-59 years at HT initiation, associations of CEE alone or CEE+MPA with

155 al part of the BNST (dBNST) receives dense 5-HT innervation, whether and how 5-HT in the dBNST normal

156 5-HT is an important pro-kinetic agent in the colon.

157 HT is associated with substantial improvement in FS for

158 Furthermore, the maximum concentration of HT is reached between the fourth and sixth day of fermen

159 HTS is the result of dysregulated wound healing, where e

160 by chemical signaling, of which serotonin (5-HT) is a key transmitter.

161 Hydroxytyrosol (HT) is a phenolic compound of recognized bioactivity tha

162 Serotonin (5-hydroxytryptamine, 5-HT) is a well-known neurotransmitter that is involved in

163 Serotonin (5-HT) is associated with mood and motivation but the funct

164 e modulation of the amygdala by serotonin (5-HT) is important for emotional regulation and is implica

165 Release of serotonin (5-HT) is thought to have an important role in the increase

166 rallel partitions of the olfactory system, 5-HT largely elicited MC excitation in the MOB while it ev

167 with e[CO2], Pusa 1121 exposure to e[CO2] + HT led to significant reduction in seed-set and sink sta

168 ter promoter region polymorphism and serum 5-HT level.

169 beta3 receptors that influence whole-blood 5-HT levels and may influence the risk for autism spectrum

170 decreased placenta and embryonic forebrain 5-HT levels at E14.5.

171 l SERT Ala56 genotype effects on forebrain 5-HT levels were accompanied by a broadening of 5-HT-sensi

172 offspring placental 5-HT levels, forebrain 5-HT levels, and neurodevelopment.

173 SERT function impacts offspring placental 5-HT levels, forebrain 5-HT levels, and neurodevelopment.

174 a subgroup of patients with elevated blood 5-HT levels, linking integrin beta3, 5-HT, and ASD risk.

175 tion, as well as elevated peripheral blood 5-HT levels.

176 is associated with an alteration in serum 5-HT levels.

177 We show that 5-HT locates at the hydrophobic-hydrophilic interface, bel

178 High serum 5-HT may serve as a potential forensic biomarker in autops

179 e results demonstrate that GRK-2 modulates 5-HT metabolism by regulating AMX-2 function and that 5-HI

180 of serotonin (5-HT) and high levels of the 5-HT metabolite 5-hydroxyindole acetic acid (5-HIAA).

181 laboratory-based high-throughput sequencing (HTS) methods for species level identification and phylog

182 eloping effective high-throughput screening (HTS) methods is of paramount importance in the early sta

183 urrent cell-based high-throughput screening (HTS) models to identify molecules affecting ECM accumula

184 B was sensitive to physiological levels of 5-HT modification, and that OT does not act directly on th

185 women and explored whether hormone therapy (HT) modified this association in 614 cases and 743 contr

186 cifically, the treatment of hormone therapy (HT)-naive cells or HT-treated metabolically dormant popu

187 Differential 5-HT neuromodulation of MCs across the MOB and AOB could c

188 Using genetic tools, we characterize a 5-HT neuron subtype defined by expression of Tachykinin1 a

189 Here, we characterize in mice a 5-HT neuron subtype identified by expression of Tac1 and t

190 etion disturbs the migration of serotonin (5-HT) neuronal precursors, leading to altered global serot

191 lective for Adelta-fibers (peripherally) and HT neurons (centrally).

192 We found that 5-HT neurons co-release 5-HT and glutamate onto BA neurons

193 Collectively, these findings suggest that 5-HT neurons exert a frequency-dependent, cell-type-specif

194 the physiological mechanisms through which 5-HT neurons in the dorsal raphe nuclei modulate amygdala

195 ological mechanisms through which midbrain 5-HT neurons modulate amygdala circuits could be pivotal i

196 ggest that theories on the contribution of 5-HT neurons to amygdala function should be revised to inc

197 ecture and increased spontaneous firing of 5-HT neurons.

198 h channelrhodopsin genetically targeted to 5-HT neurons.

199 H receives strong inputs from LC-NE and DR 5-HT neurons.

200 Serotonin (5-HT) neurons project from the raphe nuclei throughout the

201 fremanezumab inhibited naive high-threshold (HT) neurons, but not wide-dynamic range trigeminovascula

202 te to drug seeking via beta-adrenergic and 5-HT neurotransmission in DH.

203 nsight into the genetic network regulating 5-HT neurotransmission in the CNS that is also associated

204 mines such as dopamine (DA) and serotonin (5-HT) occur in the periventricular zones of the hypothalam

205 According to results here presented, HT of excess sludge with moderate (13%) PHA content can

206 Herein, hydrothermal treatment (HT) of PHA-containing sludge at 300 and 375 degrees C wa

207 In a high throughput screen (HTS) of 230000 small molecules designed to identify bioa

208 rtional hazards models to test the potential HTEs on the remaining half of the data (the testing set)

209 A mini-HTS on 4000 compounds selected using 2D fragment-based s

210 95% CI, 1.2-2.5), hospitalization status at HT (OR, 1.5; 95% CI, 1.0-2.19), chronic steroid use at H

211 5; 95% CI, 1.0-2.19), chronic steroid use at HT (OR, 1.5; 95% CI, 1.0-2.2), and treatment for early r

212 % CI, 1.1-2.0), support with >/=inotropes at HT (OR, 1.7; 95% CI, 1.2-2.5), hospitalization status at

213 A dopaminergic marker (TH), serotonin (5-HT) or GABA do not co-localize with Galphat-S-ir neurons

214 lized to conduct multisite measurements of 5-HT overflow from the entire colon.

215 xhibited higher ACE-inhibitory activity than HT (P<0.05).

216 iated with peripheral abnormalities in the 5-HT pathway.

217 ition, the increased analytic sensitivity of HTS permitted identification of 19.9% of SR patients wit

218 We found that 5-HT predominantly hyperpolarizes dBNST neurons, reducing

219 onic day E14.5, when peripheral sources of 5-HT predominate, and E18.5, when midbrain 5-HT projection

220 We show that 5-HT produced by the ADF neurons acts via the SER-5 recept

221 promote roaming in fasting worms, whereas 5-HT produced by the NSM neurons acts on the MOD-1 recepto

222 Early in gestation, before midbrain 5-HT projections have reached the cortex, peripheral sourc

223 5-HT predominate, and E18.5, when midbrain 5-HT projections have reached the forebrain.

224 tra and by DFT computations we show that the HT properties are independent of bridge substituents for

225 example, the pistil SI proteins S-RNase and HT protein function in a pistil-side IRB that causes rej

226 Thus, HT proteins are implicated in both S-RNase-dependent and

227 tially improved upon a genome assembled from HTS reads alone.

228 ermore, hybrid genome assembly with RTnS and HTS reads substantially improved upon a genome assembled

229 it egg laying in a manner dependent on the 5-HT receptor SER-1 and the G protein GOA-1.

230 ptor, OCTR-1, and a 5-HT1A-like serotonin (5-HT) receptor, SER-4, that involves a complex interaction

231 A10), a multifunctional protein binding to 5-HT receptors, in layer II/III neurons of the prelimbic c

232 cytotoxic activity of peripheral NK cells in HT recipients were evaluated using a noninvasive NK-cell

233 , double-blind, placebo-controlled trial, 96 HT recipients were randomized to undergo ramipril or pla

234 However, the mechanism by which 5-HT regulates BDNF signaling is unknown.

235 Piezo2 by force leads to inward currents, 5-HT release and an increase in mucosal secretion.

236 Jejunum mucosal pressure increased 5-HT release and short-circuit current via submucosal 5-HT

237 , but it was also important for regulating 5-HT release by these cells.

238 These data reveal that 5-HT release in the dBNST modulates anxiety-like behavior

239 Both inward currents and 5-HT release were inhibited by Piezo2 small interfering RN

240 motivation but the function of endogenous 5-HT remains controversial.

241 r, the underlying mechanism between OT and 5-HT remains unclear.

242 Within eukaryotes, the majority of HTs reported so far are transfers of transposable elemen

243 both murine and human CAFs promoted de novo HT resistance via the generation of CD133(hi) CSCs that

244 cal synapses, compared with its effects on 5-HT responses, a measure of cortical-cortical responses m

245 of Slc6a4 or treatment with Fluoxetine, a 5-HT reuptake inhibitor, restored normal breathing.

246 he published motifs estimated using the same HT-SELEX data, we demonstrate that BEESEM provides signi

247 ithms for estimating binding motifs based on HT-SELEX data.

248 In this study, we use HT-SELEX derived populations to study the landscape of R

249 microarrays (PBM) and high-throughput SELEX (HT-SELEX), have enabled rapid measurements of the specif

250 Conversely, in models of CD133(neg), HT-sensitive cancer cells, both murine and human CAFs pr

251 levels were accompanied by a broadening of 5-HT-sensitive thalamocortical axon projections.

252 rrect position-specific nucleotide biases in HTS short read data.

253 drenergic signaling in female DH, but only 5-HT signaling in male DH.

254 Components of the 5-HT signalling system (5-HT, 5-HIAA, SERT) and TNF-alpha

255 We examined whether alterations in colonic 5-HT signalling underlie age-related changes in faecal out

256 High temperature (HT) stress tolerant NL-44 and high yielding basmati Pusa

257 studies revealed a significant decrease in 5-HT synapses in the midbrain, accompanied by decreases in

258 ggesting that altered maternal or placenta 5-HT system function could impact the developing embryo.

259 This new in vivo neuroimaging atlas of the 5-HT system not only provides insight in the human brain's

260 56 variant have altered central serotonin (5-HT) system function, as well as elevated peripheral bloo

261 integrin beta3 subunit with the serotonin (5-HT) system, likely via its modulation of the 5-HT transp

262 map of the serotonin (5-hydroxytryptamine, 5-HT) system.

263 ine (LC-NE) and dorsal raphe serotonin (DR 5-HT) systems.

264 mes showed a high correlation for the five 5-HT targets and this enabled us to transform the atlas to

265 High throughput DNA sequencing (HTS) technologies generate an excessive number of small

266 Optical stimulation of 5-HT terminals at low frequencies (</=1 Hz) evoked a short

267 Furthermore, optical stimulation of 5-HT terminals did not evoke glutamate release onto BA pri

268 enetics, we demonstrate that activation of 5-HT terminals in the dBNST reduces anxiety in a highly an

269 hat show high-temperature superconductivity (HTS), the critical temperature (Tc) has a dome-shaped do

270 ed and nolatrexed, the two insert regions in hTS, the functions of which are unclear, undergo positio

271 se of late mortality after heart transplant (HT), there is a need to identify markers that reflect in

272 oxyl group and lipid headgroups and allows 5-HT to intercept reactive oxygen species, preventing memb

273 , which is implicated in transamidation of 5-HT to Rac1) are observed in the mouse prefrontal cortex

274 ched the cortex, peripheral sources supply 5-HT to the forebrain, suggesting that altered maternal or

275 Several lines of evidence suggest that 5-HT transmission in the amygdala is implicated in the sus

276 ) system, likely via its modulation of the 5-HT transporter (SERT).

277 tment of hormone therapy (HT)-naive cells or HT-treated metabolically dormant populations with CAF-de

278 In the present study, we found that 5-HT treatment increases BDNF receptor, TrkB (tropomyosin

279 l small optic lobe (SOL) calpain 2 d after 5-HT treatment or paired stimuli did not disrupt the maint

280 Soon after 5-HT treatment or paired stimuli, however, blocking classi

281 Although in vitro results of the TSLDox and HT treatment were favorable for both cell types, the the

282 antly enhancing tumor response to TSLDox and HT treatment.

283 atiety-signaling peptide YY 3-36 increased 5-HT turnover in the LH and ameliorated the ppDIO-induced

284 ls, we carried out a high throughput screen (HTS) using an ApoC-III homogenous time resolved fluoresc

285 ed causal forest modelling, which identifies HTEs, using a random half of the trial data (the trainin

286 To do so, 5-HT was added to red blood cells and lipid membranes bear

287 nificantly upregulated, and the content of 5-HT was decreased which negatively correlated with the ga

288 uman plasma, although release of the drug at HT was incomplete for the latter two.

289 In parallel, high-throughput screening (HTS) was conducted using the fluorescent probe substrate

290 A high-throughput screen (HTS) was undertaken against the respiratory chain dehydr

291 By high throughput sequencing (HTS), we obtained the first de novo transcriptome of mah

292 ands predicted in silico as well as found by HTS, were identified.

293 l systemic serotonin (5-hydroxytryptamine; 5-HT), which is an important neurotransmitter and endocrin

294 tioxidant activities compared to trypsin and HT, while trypsin and pepsin hydrolysates exhibited high

295 en were randomized (1:1 ratio) to home-based HT with a CD (CD group) or iHT performed by qualified th

296 Long-term effectiveness of home-based HT with a CD is noninferior to iHT performed by therapis

297 ves allosteric regulation of interactions of hTS with its own mRNA depending on cellular demands for

298 longitudinal subdivisions of the heart tube (HT), with the FHF contributing the left ventricle and pa

299 murine CAFs from PDX restored sensitivity to HT, with a concurrent reduction of CD133(hi) CSCs.

300 We describe an MS-based HTS workflow that addresses these challenges.