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1                                              HT biscuit was able to inhibit efficiently the formation
2                                              HT is associated with substantial improvement in FS for
3                                              HT, OLE, QE and GA were added in the range of 0.25-0.75%
4                                              HT-29 colon adenocarcinoma cells were formed into pellet
5 rescence, and immunoblot analyses in Caco-2, HT-29, and T-84 cells human IECs cultured in 2 or 3 dime
6 -10, and<5kDa caused cytotoxicity to Caco-2, HT-29, HCT-116 human colon cancer cells, and reduced inf
7                 Despite of the increased T-2/HT-2 concentrations found in some of the samples, the ob
8       As for final products, the highest T-2/HT-2 concentrations were determined in oat flakes (89.4m
9 udy was to investigate into the level of T-2/HT-2 toxins in different unprocessed cereals (n=201), as
10 tes were also measured using a 70% Caco2/30% HT-29 human intestinal co-culture layer.
11                                            5-HT is an important pro-kinetic agent in the colon.
12     Using genetic tools, we characterize a 5-HT neuron subtype defined by expression of Tachykinin1 a
13            Here, we characterize in mice a 5-HT neuron subtype identified by expression of Tac1 and t
14  of Slc6a4 or treatment with Fluoxetine, a 5-HT reuptake inhibitor, restored normal breathing.
15 l small optic lobe (SOL) calpain 2 d after 5-HT treatment or paired stimuli did not disrupt the maint
16                                 Soon after 5-HT treatment or paired stimuli, however, blocking classi
17 oxyl group and lipid headgroups and allows 5-HT to intercept reactive oxygen species, preventing memb
18  are likely subserved by differences among 5-HT neuron subtypes at the cellular and molecular levels,
19 ations in brain extracellular dopamine and 5-HT in vivo.
20 te to drug seeking via beta-adrenergic and 5-HT neurotransmission in DH.
21                   Both inward currents and 5-HT release were inhibited by Piezo2 small interfering RN
22 r, the underlying mechanism between OT and 5-HT remains unclear.
23             It is well known that 5-HT and 5-HT transporter (5-HTT) play important roles in the pulmo
24 press TH2 and TPH1 and contain both DA and 5-HT, a dual neurotransmitter phenotype hitherto undescrib
25 d differential modulation by glutamate and 5-HT.
26 blood 5-HT levels, linking integrin beta3, 5-HT, and ASD risk.
27 c acid [DTPA]-gadolinium or indium 111-bis-5-HT-DTPA, respectively).
28 s, we investigated the effects of blocking 5-HT and beta-adrenoceptor transmission in DH on drug seek
29 beta3 receptors that influence whole-blood 5-HT levels and may influence the risk for autism spectrum
30                                      Blood 5-HT levels were dynamic across pregnancy and were increas
31 a subgroup of patients with elevated blood 5-HT levels, linking integrin beta3, 5-HT, and ASD risk.
32 tion, as well as elevated peripheral blood 5-HT levels.
33 subtype with the potential to release both 5-HT and substance P is necessary for normal respiratory d
34             These effects were mediated by 5-HT because they were blocked by antagonists of 5-HT2A an
35 We examined whether alterations in colonic 5-HT signalling underlie age-related changes in faecal out
36                             In comparison, 5-HT-evoked inhibitory responses in the AOB arose due to a
37  Piezo2 by force leads to inward currents, 5-HT release and an increase in mucosal secretion.
38 al part of the BNST (dBNST) receives dense 5-HT innervation, whether and how 5-HT in the dBNST normal
39                               Differential 5-HT neuromodulation of MCs across the MOB and AOB could c
40 H receives strong inputs from LC-NE and DR 5-HT neurons.
41 ine (LC-NE) and dorsal raphe serotonin (DR 5-HT) systems.
42  motivation but the function of endogenous 5-HT remains controversial.
43 onergic signaling by increasing endogenous 5-HT.
44 mes showed a high correlation for the five 5-HT targets and this enabled us to transform the atlas to
45 decreased placenta and embryonic forebrain 5-HT levels at E14.5.
46 l SERT Ala56 genotype effects on forebrain 5-HT levels were accompanied by a broadening of 5-HT-sensi
47 offspring placental 5-HT levels, forebrain 5-HT levels, and neurodevelopment.
48 es dense 5-HT innervation, whether and how 5-HT in the dBNST normally modulates anxiety remains uncle
49 ver and expression of 5-hydroxytryptamine (5-HT) 2A (5-HT2A) and 5-HT2C serotonin receptors in the hy
50  re-uptakes excessive 5-hydroxytryptamine (5-HT) from effective location to terminate its physiologic
51          Serotonin or 5-hydroxytryptamine (5-HT) has been shown to be essential in lots of physiologi
52 sory neuron 2 d after 5-hydroxytryptamine (5-HT) treatment reversed persistent nonassociative LTF.
53            Serotonin (5-hydroxytryptamine, 5-HT) and brain-derived neurotrophic factor (BDNF) are two
54 alities in serotonin (5-hydroxytryptamine, 5-HT) in regions of the brainstem critical in homeostatic
55            Serotonin (5-hydroxytryptamine, 5-HT) is a well-known neurotransmitter that is involved in
56        The serotonin (5-hydroxytryptamine, 5-HT) system modulates many important brain functions and
57 map of the serotonin (5-hydroxytryptamine, 5-HT) system.
58 l systemic serotonin (5-hydroxytryptamine; 5-HT), which is an important neurotransmitter and endocrin
59       Taken together, our results identify 5-HT as a potent inhibitor of lipid peroxidation and offer
60 ivity of 5-HT Transporter (SERT/Slc6a4) in 5-HT neurons leading to an increase of 5-HT uptake.
61 studies revealed a significant decrease in 5-HT synapses in the midbrain, accompanied by decreases in
62         Jejunum mucosal pressure increased 5-HT release and short-circuit current via submucosal 5-HT
63 atiety-signaling peptide YY 3-36 increased 5-HT turnover in the LH and ameliorated the ppDIO-induced
64 seeking during initial abstinence involves 5-HT and beta-adrenergic signaling in female DH, but only
65                                        Low 5-HT in the placenta persisted, but forebrain levels norma
66 primary monoamine oxidase that metabolizes 5-HT in C. elegans, and we also found that grk-2 loss-of-f
67 ological mechanisms through which midbrain 5-HT neurons modulate amygdala circuits could be pivotal i
68        Early in gestation, before midbrain 5-HT projections have reached the cortex, peripheral sourc
69 5-HT predominate, and E18.5, when midbrain 5-HT projections have reached the forebrain.
70                                In the MOB, 5-HT elicited three types of responses in 93% of 180 cells
71 e results demonstrate that GRK-2 modulates 5-HT metabolism by regulating AMX-2 function and that 5-HI
72                      Age increased mucosal 5-HT availability and TNF-alpha expression and decreased m
73 be the complete pattern of distribution of 5-HT fibers to the amygdala (proper) and to the extended a
74 ole, the BST contained moderate numbers of 5-HT fibers, spread fairly uniformly throughout BST.
75 r studies identify a complex regulation of 5-HT homeostasis and behaviors by integrin alphavbeta3, re
76 is revealed that optogenetic inhibition of 5-HT inputs into the dBNST increases anxiety in a less anx
77 erved following optogenetic stimulation of 5-HT inputs into the dBNST.
78 have shown nonpharmacological functions of 5-HT linked to its chemical properties.
79      The addition of 5-HT or inhibition of 5-HT metabolism also rescued the egg laying defect.
80 B was sensitive to physiological levels of 5-HT modification, and that OT does not act directly on th
81 impact of phasic optogenetic activation of 5-HT neurons in mice over time scales from seconds to week
82 ggest that theories on the contribution of 5-HT neurons to amygdala function should be revised to inc
83 ecture and increased spontaneous firing of 5-HT neurons.
84                            The addition of 5-HT or inhibition of 5-HT metabolism also rescued the egg
85 lized to conduct multisite measurements of 5-HT overflow from the entire colon.
86 onic day E14.5, when peripheral sources of 5-HT predominate, and E18.5, when midbrain 5-HT projection
87                     Optical stimulation of 5-HT terminals at higher frequencies (10-20 Hz) evoked bot
88                     Optical stimulation of 5-HT terminals at low frequencies (</=1 Hz) evoked a short
89        Furthermore, optical stimulation of 5-HT terminals did not evoke glutamate release onto BA pri
90 enetics, we demonstrate that activation of 5-HT terminals in the dBNST reduces anxiety in a highly an
91 , which is implicated in transamidation of 5-HT to Rac1) are observed in the mouse prefrontal cortex
92 ow an increased expression and activity of 5-HT Transporter (SERT/Slc6a4) in 5-HT neurons leading to
93 n these mice, and that the localization of 5-HT transporters to specific compartments within the syna
94  in 5-HT neurons leading to an increase of 5-HT uptake.
95 nificantly upregulated, and the content of 5-HT was decreased which negatively correlated with the ga
96 levels were accompanied by a broadening of 5-HT-sensitive thalamocortical axon projections.
97 n, is critical for appropriate reuptake of 5-HT.
98 d be revised to incorporate the concept of 5-HT/glutamate cotransmission.
99 cal synapses, compared with its effects on 5-HT responses, a measure of cortical-cortical responses m
100 drenergic signaling in female DH, but only 5-HT signaling in male DH.
101 , no previous report has described overall 5-HT projections to the amygdala in the rat.
102 ggesting that altered maternal or placenta 5-HT system function could impact the developing embryo.
103  SERT function impacts offspring placental 5-HT levels, forebrain 5-HT levels, and neurodevelopment.
104 e-art techniques, we show that presynaptic 5-HT function is altered in these mice, and that the local
105 nsight into the genetic network regulating 5-HT neurotransmission in the CNS that is also associated
106 , but it was also important for regulating 5-HT release by these cells.
107      We found that 5-HT neurons co-release 5-HT and glutamate onto BA neurons in a cell-type-specific
108 f sour (acidic) tastants and this released 5-HT activates 5-HT3 receptors on the gustatory nerves.
109                 The EC cell model released 5-HT in response to stretch, and had Piezo2 mRNA and prote
110 sor, and it is well known that it releases 5-HT in response to mechanical forces.
111 in vivo atlas of four of the human brain's 5-HT receptors (5-HT1A, 5-HT1B, 5-HT2A, and 5-HT4) and the
112       As is well recognized, serotonergic (5-HT) fibers distribute widely throughout the forebrain, i
113 al evidence has implicated both serotonin (5-HT) 2C and 2A receptors as potential mechanisms for medi
114 ctive and contain low levels of serotonin (5-HT) and high levels of the 5-HT metabolite 5-hydroxyindo
115 is elegans, the biogenic amines serotonin (5-HT) and octopamine regulate a number of food-related beh
116  cells, Type III cells, release serotonin (5-HT) in response to the presence of sour (acidic) tastant
117 by chemical signaling, of which serotonin (5-HT) is a key transmitter.
118                                 Serotonin (5-HT) is associated with mood and motivation but the funct
119 e modulation of the amygdala by serotonin (5-HT) is important for emotional regulation and is implica
120                      Release of serotonin (5-HT) is thought to have an important role in the increase
121 etion disturbs the migration of serotonin (5-HT) neuronal precursors, leading to altered global serot
122                                 Serotonin (5-HT) neurons project from the raphe nuclei throughout the
123 mines such as dopamine (DA) and serotonin (5-HT) occur in the periventricular zones of the hypothalam
124     A dopaminergic marker (TH), serotonin (5-HT) or GABA do not co-localize with Galphat-S-ir neurons
125 ptor, OCTR-1, and a 5-HT1A-like serotonin (5-HT) receptor, SER-4, that involves a complex interaction
126                             The serotonin (5-HT) system and the amygdala are key regulators of emotio
127 56 variant have altered central serotonin (5-HT) system function, as well as elevated peripheral bloo
128 integrin beta3 subunit with the serotonin (5-HT) system, likely via its modulation of the 5-HT transp
129 dely distributed CNS modulator, serotonin (5-HT), for its ability to modulate the biophysical propert
130 dulated by the neurotransmitter serotonin (5-HT).
131 ese compounds include selective serotonin (5-HT)2C receptor antagonists, a 5-HT4 receptor agonist, a
132 ter promoter region polymorphism and serum 5-HT level.
133  is associated with an alteration in serum 5-HT levels.
134                                 High serum 5-HT may serve as a potential forensic biomarker in autops
135                                      Serum 5-HT, adjusted for postconceptional age, was significantly
136                                  To do so, 5-HT was added to red blood cells and lipid membranes bear
137 ched the cortex, peripheral sources supply 5-HT to the forebrain, suggesting that altered maternal or
138  Components of the 5-HT signalling system (5-HT, 5-HIAA, SERT) and TNF-alpha expression were examined
139 rallel partitions of the olfactory system, 5-HT largely elicited MC excitation in the MOB while it ev
140 can in humans, we have recently found that 5-HT 1A receptor (5-HT1AR) function is modified after intr
141               Our results demonstrate that 5-HT acts as a potent antioxidant and binds with a superio
142                      It is well known that 5-HT and 5-HT transporter (5-HTT) play important roles in
143 ntitative behavioral imaging, we show that 5-HT and octopamine jointly influence locomotor activity a
144                               We show that 5-HT locates at the hydrophobic-hydrophilic interface, bel
145                              We found that 5-HT neurons co-release 5-HT and glutamate onto BA neurons
146  Collectively, these findings suggest that 5-HT neurons exert a frequency-dependent, cell-type-specif
147                              We found that 5-HT predominantly hyperpolarizes dBNST neurons, reducing
148                               We show that 5-HT produced by the ADF neurons acts via the SER-5 recept
149                     These data reveal that 5-HT release in the dBNST modulates anxiety-like behavior
150                    These results show that 5-HT transients have strong and opposing short and long-te
151     Several lines of evidence suggest that 5-HT transmission in the amygdala is implicated in the sus
152        In the present study, we found that 5-HT treatment increases BDNF receptor, TrkB (tropomyosin
153 s stabilized by hydrogen bonds between the 5-HT hydroxyl group and lipid headgroups and allows 5-HT t
154   Although a few reports have examined the 5-HT innervation of select nuclei of the amygdala in the r
155 of serotonin (5-HT) and high levels of the 5-HT metabolite 5-hydroxyindole acetic acid (5-HIAA).
156 iated with peripheral abnormalities in the 5-HT pathway.
157 it egg laying in a manner dependent on the 5-HT receptor SER-1 and the G protein GOA-1.
158                          Components of the 5-HT signalling system (5-HT, 5-HIAA, SERT) and TNF-alpha
159 vely enhanced alphavbeta3 signaling to the 5-HT system in the brain.
160 This new in vivo neuroimaging atlas of the 5-HT system not only provides insight in the human brain's
161 5-HT1A, 5-HT1B, 5-HT2A, and 5-HT4) and the 5-HT transporter (5-HTT).
162 ) system, likely via its modulation of the 5-HT transporter (SERT).
163 h channelrhodopsin genetically targeted to 5-HT neurons.
164 A10), a multifunctional protein binding to 5-HT receptors, in layer II/III neurons of the prelimbic c
165 ype-specific control over BA circuitry via 5-HT and glutamate co-release to inhibit the BA output.SIG
166  promote roaming in fasting worms, whereas 5-HT produced by the NSM neurons acts on the MOD-1 recepto
167 the physiological mechanisms through which 5-HT neurons in the dorsal raphe nuclei modulate amygdala
168            However, the mechanism by which 5-HT regulates BDNF signaling is unknown.
169 , double-blind, placebo-controlled trial, 96 HT recipients were randomized to undergo ramipril or pla
170 nodrug, as well as animal survival rate in a HT-29 xenograft mouse model.
171 the second decade are associated with adults HT.
172 s (n = 65) and biopsies (n = 47) (Affymetrix HT HG-U133+ PM).
173 ients at higher risk of developing CAV after HT.
174 mipril on the development of CAV early after HT.
175  of CAV but have not been well studied after HT.
176 iated with improved long-term survival after HT.
177 S children who survive at least 1 year after HT, FS is excellent for the majority of patients.
178 for their antiproliferative activity against HT-29 colon cancer cell lines.
179                                  HCT-116 and HT-29 inhibition correlated with total phenolics (r=0.87
180 , zearalenone (ZEA), four enniatins, T-2 and HT-2 toxins).
181 alenone, deoxynivalenol, fumonisins, T-2 and HT-2 toxins, fusarenone X, diacetoxyscirpenol, and 3- an
182 rting measurable toxic effects on HEK293 and HT-29 cells.
183  exerted cytotoxic activity against HeLa and HT-29 cell lines, but were inactive towards MRC-5 and MC
184 ing effect of age, time since menopause, and HT formulation.
185 lective for Adelta-fibers (peripherally) and HT neurons (centrally).
186  example, the pistil SI proteins S-RNase and HT protein function in a pistil-side IRB that causes rej
187 tioxidant activities compared to trypsin and HT, while trypsin and pepsin hydrolysates exhibited high
188        Combination treatment with TSLDox and HT in vivo was significantly more effective against MDA-
189  Although in vitro results of the TSLDox and HT treatment were favorable for both cell types, the the
190 antly enhancing tumor response to TSLDox and HT treatment.
191  age structure of the populations and age at HT initiation.
192 uman plasma, although release of the drug at HT was incomplete for the latter two.
193 % CI, 1.1-2.0), support with >/=inotropes at HT (OR, 1.7; 95% CI, 1.2-2.5), hospitalization status at
194  95% CI, 1.2-2.5), hospitalization status at HT (OR, 1.5; 95% CI, 1.0-2.19), chronic steroid use at H
195  in vitro chemosensitivity and Dox uptake at HT.
196 5; 95% CI, 1.0-2.19), chronic steroid use at HT (OR, 1.5; 95% CI, 1.0-2.2), and treatment for early r
197              Among women aged 50-59 years at HT initiation, associations of CEE alone or CEE+MPA with
198 ng postmenopausal women, aged 50-79 years at HT initiation.
199 ins M1, B1, B2, G1, G2, ochratoxins A and B, HT-2 and T-2 toxins, deepoxy-deoxynivalenol, zearalenone
200 en were randomized (1:1 ratio) to home-based HT with a CD (CD group) or iHT performed by qualified th
201        Long-term effectiveness of home-based HT with a CD is noninferior to iHT performed by therapis
202 g but only 3-DG was significantly reduced by HT.
203 ia-based glass-ceramics (e.g., IPS e.max CAD HT).
204 psin, pepsin and a bacterial protease called HT for 1, 2, 4 and 24h.
205 on of human colorectal adenocarcinoma cells (HT-29) under the control of visible light and the extent
206 n to maturity) and a combination of e[CO2] + HT (from heading to maturity) using field based open top
207                         Conversely, e[CO2] + HT during flowering and early grain filling significantl
208 ith starch metabolism enzymes under e[CO2] + HT exposure.
209  with e[CO2], Pusa 1121 exposure to e[CO2] + HT led to significant reduction in seed-set and sink sta
210 een morphogenesis and differentiation during HT formation and provide a new framework to understand h
211 -Danlos syndrome hypermobility type (JHS/EDS-HT), characterized by idiopathic chronic itch with predo
212 resent study demonstrates that enzymatically HT-coated surface modification is an effective strategy
213   We found that all three accessions express HT proteins, which previously had been known to function
214 renal-risk variants, and used Illumina human HT-12 v4 arrays and Affymetrix HTA 2.0 arrays to generat
215 d neural cells and applied to Illumina Human HT-12 v4 Expression BeadChips.
216 a high-throughput enhanced yeast two-hybrid (HT-eY2H) assay and a mammalian-cell-based Gaussia prince
217                              Hydroxytyrosol (HT) is a phenolic compound of recognized bioactivity tha
218 gain have been associated with hypertension (HT), the association of linear growth, independently of
219 osensitive liposomes (TSL) and hyperthermia (HT) has shown preclinically to achieve high local drug c
220        Individual gut-directed hypnotherapy (HT) is effective in pediatric irritable bowel syndrome (
221 iated natural killer (NK) cell activation in HT biopsies; however, little is known about the role of
222 cytotoxic activity of peripheral NK cells in HT recipients were evaluated using a noninvasive NK-cell
223 creases the efficacy of DOX-induced death in HT-29 and SW480 cells and in nude mice xenografts.
224                      Focal-leaks detected in HT-29/B6 colonic monolayers were verified for native tis
225 ble factor 1alpha (HIF-1alpha) expression in HT-29 xenografts initiated from NOX1 knockdown cells.
226  Radil in neutrophil-like HL-60 cells, or in HT-1080 fibrosarcoma cells.
227 ubes to penetrate farther into the pistil in HT suppressed plants, but not to reach the ovary.
228 soderm sliding over the endoderm, results in HT formation.
229  shRNA to decrease NOX1 expression stably in HT-29 human colon cancer cells.
230 exercises using a CD with that of individual HT (iHT) performed by qualified therapists.
231 the other hand, the grape variety influences HT wine concentrations.
232       Because few women in the NHS initiated HT after age 60 years, we did not examine associations i
233 eaflet of the plasma membrane in intestinal (HT-29) cells and thereby reduce CT uptake into the cells
234 signaling in the mouse hippocampal cell line HT-22 leading to an increase in CREB phosphorylation.
235 tial modifier genes unlinked to the S-locus (HT-B, 120K, and NaStEP) unveils a higher degree of compl
236                          All three materials-HT, HS, and COGF-serve as efficient adsorbents of iodine
237         Conversely, in models of CD133(neg), HT-sensitive cancer cells, both murine and human CAFs pr
238  both murine and human CAFs promoted de novo HT resistance via the generation of CD133(hi) CSCs that
239 -carboxyethyl-lysine, although the amount of HT in the biscuit was 100-fold lower than the biscuit fo
240 ationic viologen linker and, in the cases of HT and HS, permanent porosity.
241    Furthermore, the maximum concentration of HT is reached between the fourth and sixth day of fermen
242              To compare the effectiveness of HT by means of home-based self-exercises using a CD with
243 body-dependent NK cell cytotoxic function of HT recipients were associated with the FCGR3A-VV genotyp
244 rain QA23 was the most efficient producer of HT from tested yeasts.
245 have a great potential for the production of HT.
246 of both the apparent barrier and the rate of HT for strong donor substituents in the para-cases.
247 emonstrated using proteome-wide reactions of HT-29 cell lysates with a model probe of threonine beta-
248 prevents the activation and sensitization of HT neurons by CSD.
249 escending coronary artery, within 8 weeks of HT.
250           Within eukaryotes, the majority of HTs reported so far are transfers of transposable elemen
251 ithms for estimating binding motifs based on HT-SELEX data.
252 on inhibition fifty (IC50) for BL, SH, RG on HT-29 and HCT-116 cell proliferation ranged 0.9-2.0mg/mL
253 tment of hormone therapy (HT)-naive cells or HT-treated metabolically dormant populations with CAF-de
254 he extracted proteins with either trypsin or HT generated more antioxidant activity than hydrolysis w
255 s associated with abnormal FS (FSS</=8) post-HT.
256 omparison with listing FS, FS at 1 year post-HT increased in 91% and declined/remained unchanged in 9
257  association with abnormal FS at 1 year post-HT: >/=18 years of age (odds ratio [OR], 1.8; 95% confid
258         According to results here presented, HT of excess sludge with moderate (13%) PHA content can
259 he published motifs estimated using the same HT-SELEX data, we demonstrate that BEESEM provides signi
260 microarrays (PBM) and high-throughput SELEX (HT-SELEX), have enabled rapid measurements of the specif
261 aphy for CAV diagnosis (median 5 years since HT).
262                                  Suppressing HT expression in SC S. pennellii LA0716 allows S. lycope
263                     In contrast, suppressing HT expression in SC.
264 yces Torulaspora delbrueckii, can synthesise HT, as this compound was identified in the intracellular
265 N), fumonisins (FB1+FB2+FB3), the sum of (T2+HT-2), NIV and ZEA, respectively.
266                            High temperature (HT) stress tolerant NL-44 and high yielding basmati Pusa
267 gh throughput biodegradation screening test (HT-BST).
268 s-5-hydroxytryptamide-tetraazacyclododecane [HT]-diethyneletriaminepentaacetic acid [DTPA]-gadolinium
269 xhibited higher ACE-inhibitory activity than HT (P<0.05).
270 Mechanistically, these findings suggest that HT neurons play a critical role in the initiation of the
271                                          The HT-BST may facilitate the rapid screening of 100000 chem
272                    When loading MK2i, 1) the HT depot surface gained high MK2i-loading efficiency thr
273 bital coefficients are large accelerates the HT rate for meta-conjugated compounds just as for the pa
274  trypsin and pepsin hydrolysates than in the HT hydrolysate.
275 erived regions and the dorsal closure of the HT.
276 tra and by DFT computations we show that the HT properties are independent of bridge substituents for
277 rolled trials of menopausal hormone therapy (HT) and chronic disease risk appear to have divergent re
278  women and explored whether hormone therapy (HT) modified this association in 614 cases and 743 contr
279 cifically, the treatment of hormone therapy (HT)-naive cells or HT-treated metabolically dormant popu
280 ivation and sensitization of high-threshold (HT) but not wide-dynamic range trigeminovascular neurons
281 fremanezumab inhibited naive high-threshold (HT) neurons, but not wide-dynamic range trigeminovascula
282                         The high-throughput (HT) approach we present can be readily adapted to target
283                   Thus, the high-throughput (HT)-CBS technology was used for the concomitant quantita
284                                        Thus, HT proteins are implicated in both S-RNase-dependent and
285 ered bacteria displayed induced adherence to HT-29 cells.
286 murine CAFs from PDX restored sensitivity to HT, with a concurrent reduction of CD133(hi) CSCs.
287 be utilized to accelerate the hole transfer (HT) between redox sites attached in para- or in meta-pos
288 se of late mortality after heart transplant (HT), there is a need to identify markers that reflect in
289            Changes in heart transplantation (HT) donor and recipient demographics may influence the i
290 se of mortality after heart transplantation (HT).
291              Herein, hydrothermal treatment (HT) of PHA-containing sludge at 300 and 375 degrees C wa
292 ents were found in yellow-grained tritordeum HT 439 (12.16mug/gDW), followed by blue-grained wheat V1
293 tudy examines the potency of neoadjuvant TSL HT combination therapy in two orthotopic mouse models of
294 longitudinal subdivisions of the heart tube (HT), with the FHF contributing the left ventricle and pa
295 mplated hollow spheres (HS) or hollow tubes (HT).
296 ing consecutive adult patients who underwent HT between January 2009 and December 2014 at our institu
297                        In this study, we use HT-SELEX derived populations to study the landscape of R
298   BMI at birth was inversely associated with HT; c-BMI from school age to adulthood and c-height from
299 delphinidin-3-O-glucoside concentration with HT-29 inhibition (r=0.69).
300 -fold lower than the biscuit formulated with HT.

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