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1 HT biscuit was able to inhibit efficiently the formation
2 HT is associated with substantial improvement in FS for
3 HT, OLE, QE and GA were added in the range of 0.25-0.75%
4 HT-29 colon adenocarcinoma cells were formed into pellet
5 rescence, and immunoblot analyses in Caco-2, HT-29, and T-84 cells human IECs cultured in 2 or 3 dime
6 -10, and<5kDa caused cytotoxicity to Caco-2, HT-29, HCT-116 human colon cancer cells, and reduced inf
9 udy was to investigate into the level of T-2/HT-2 toxins in different unprocessed cereals (n=201), as
12 Using genetic tools, we characterize a 5-HT neuron subtype defined by expression of Tachykinin1 a
15 l small optic lobe (SOL) calpain 2 d after 5-HT treatment or paired stimuli did not disrupt the maint
17 oxyl group and lipid headgroups and allows 5-HT to intercept reactive oxygen species, preventing memb
18 are likely subserved by differences among 5-HT neuron subtypes at the cellular and molecular levels,
24 press TH2 and TPH1 and contain both DA and 5-HT, a dual neurotransmitter phenotype hitherto undescrib
28 s, we investigated the effects of blocking 5-HT and beta-adrenoceptor transmission in DH on drug seek
29 beta3 receptors that influence whole-blood 5-HT levels and may influence the risk for autism spectrum
31 a subgroup of patients with elevated blood 5-HT levels, linking integrin beta3, 5-HT, and ASD risk.
33 subtype with the potential to release both 5-HT and substance P is necessary for normal respiratory d
35 We examined whether alterations in colonic 5-HT signalling underlie age-related changes in faecal out
38 al part of the BNST (dBNST) receives dense 5-HT innervation, whether and how 5-HT in the dBNST normal
44 mes showed a high correlation for the five 5-HT targets and this enabled us to transform the atlas to
46 l SERT Ala56 genotype effects on forebrain 5-HT levels were accompanied by a broadening of 5-HT-sensi
48 es dense 5-HT innervation, whether and how 5-HT in the dBNST normally modulates anxiety remains uncle
49 ver and expression of 5-hydroxytryptamine (5-HT) 2A (5-HT2A) and 5-HT2C serotonin receptors in the hy
50 re-uptakes excessive 5-hydroxytryptamine (5-HT) from effective location to terminate its physiologic
52 sory neuron 2 d after 5-hydroxytryptamine (5-HT) treatment reversed persistent nonassociative LTF.
54 alities in serotonin (5-hydroxytryptamine, 5-HT) in regions of the brainstem critical in homeostatic
58 l systemic serotonin (5-hydroxytryptamine; 5-HT), which is an important neurotransmitter and endocrin
61 studies revealed a significant decrease in 5-HT synapses in the midbrain, accompanied by decreases in
63 atiety-signaling peptide YY 3-36 increased 5-HT turnover in the LH and ameliorated the ppDIO-induced
64 seeking during initial abstinence involves 5-HT and beta-adrenergic signaling in female DH, but only
66 primary monoamine oxidase that metabolizes 5-HT in C. elegans, and we also found that grk-2 loss-of-f
67 ological mechanisms through which midbrain 5-HT neurons modulate amygdala circuits could be pivotal i
71 e results demonstrate that GRK-2 modulates 5-HT metabolism by regulating AMX-2 function and that 5-HI
73 be the complete pattern of distribution of 5-HT fibers to the amygdala (proper) and to the extended a
75 r studies identify a complex regulation of 5-HT homeostasis and behaviors by integrin alphavbeta3, re
76 is revealed that optogenetic inhibition of 5-HT inputs into the dBNST increases anxiety in a less anx
80 B was sensitive to physiological levels of 5-HT modification, and that OT does not act directly on th
81 impact of phasic optogenetic activation of 5-HT neurons in mice over time scales from seconds to week
82 ggest that theories on the contribution of 5-HT neurons to amygdala function should be revised to inc
86 onic day E14.5, when peripheral sources of 5-HT predominate, and E18.5, when midbrain 5-HT projection
90 enetics, we demonstrate that activation of 5-HT terminals in the dBNST reduces anxiety in a highly an
91 , which is implicated in transamidation of 5-HT to Rac1) are observed in the mouse prefrontal cortex
92 ow an increased expression and activity of 5-HT Transporter (SERT/Slc6a4) in 5-HT neurons leading to
93 n these mice, and that the localization of 5-HT transporters to specific compartments within the syna
95 nificantly upregulated, and the content of 5-HT was decreased which negatively correlated with the ga
99 cal synapses, compared with its effects on 5-HT responses, a measure of cortical-cortical responses m
102 ggesting that altered maternal or placenta 5-HT system function could impact the developing embryo.
103 SERT function impacts offspring placental 5-HT levels, forebrain 5-HT levels, and neurodevelopment.
104 e-art techniques, we show that presynaptic 5-HT function is altered in these mice, and that the local
105 nsight into the genetic network regulating 5-HT neurotransmission in the CNS that is also associated
107 We found that 5-HT neurons co-release 5-HT and glutamate onto BA neurons in a cell-type-specific
108 f sour (acidic) tastants and this released 5-HT activates 5-HT3 receptors on the gustatory nerves.
111 in vivo atlas of four of the human brain's 5-HT receptors (5-HT1A, 5-HT1B, 5-HT2A, and 5-HT4) and the
113 al evidence has implicated both serotonin (5-HT) 2C and 2A receptors as potential mechanisms for medi
114 ctive and contain low levels of serotonin (5-HT) and high levels of the 5-HT metabolite 5-hydroxyindo
115 is elegans, the biogenic amines serotonin (5-HT) and octopamine regulate a number of food-related beh
116 cells, Type III cells, release serotonin (5-HT) in response to the presence of sour (acidic) tastant
119 e modulation of the amygdala by serotonin (5-HT) is important for emotional regulation and is implica
121 etion disturbs the migration of serotonin (5-HT) neuronal precursors, leading to altered global serot
123 mines such as dopamine (DA) and serotonin (5-HT) occur in the periventricular zones of the hypothalam
124 A dopaminergic marker (TH), serotonin (5-HT) or GABA do not co-localize with Galphat-S-ir neurons
125 ptor, OCTR-1, and a 5-HT1A-like serotonin (5-HT) receptor, SER-4, that involves a complex interaction
127 56 variant have altered central serotonin (5-HT) system function, as well as elevated peripheral bloo
128 integrin beta3 subunit with the serotonin (5-HT) system, likely via its modulation of the 5-HT transp
129 dely distributed CNS modulator, serotonin (5-HT), for its ability to modulate the biophysical propert
131 ese compounds include selective serotonin (5-HT)2C receptor antagonists, a 5-HT4 receptor agonist, a
137 ched the cortex, peripheral sources supply 5-HT to the forebrain, suggesting that altered maternal or
138 Components of the 5-HT signalling system (5-HT, 5-HIAA, SERT) and TNF-alpha expression were examined
139 rallel partitions of the olfactory system, 5-HT largely elicited MC excitation in the MOB while it ev
140 can in humans, we have recently found that 5-HT 1A receptor (5-HT1AR) function is modified after intr
143 ntitative behavioral imaging, we show that 5-HT and octopamine jointly influence locomotor activity a
146 Collectively, these findings suggest that 5-HT neurons exert a frequency-dependent, cell-type-specif
151 Several lines of evidence suggest that 5-HT transmission in the amygdala is implicated in the sus
153 s stabilized by hydrogen bonds between the 5-HT hydroxyl group and lipid headgroups and allows 5-HT t
154 Although a few reports have examined the 5-HT innervation of select nuclei of the amygdala in the r
155 of serotonin (5-HT) and high levels of the 5-HT metabolite 5-hydroxyindole acetic acid (5-HIAA).
160 This new in vivo neuroimaging atlas of the 5-HT system not only provides insight in the human brain's
164 A10), a multifunctional protein binding to 5-HT receptors, in layer II/III neurons of the prelimbic c
165 ype-specific control over BA circuitry via 5-HT and glutamate co-release to inhibit the BA output.SIG
166 promote roaming in fasting worms, whereas 5-HT produced by the NSM neurons acts on the MOD-1 recepto
167 the physiological mechanisms through which 5-HT neurons in the dorsal raphe nuclei modulate amygdala
169 , double-blind, placebo-controlled trial, 96 HT recipients were randomized to undergo ramipril or pla
181 alenone, deoxynivalenol, fumonisins, T-2 and HT-2 toxins, fusarenone X, diacetoxyscirpenol, and 3- an
183 exerted cytotoxic activity against HeLa and HT-29 cell lines, but were inactive towards MRC-5 and MC
186 example, the pistil SI proteins S-RNase and HT protein function in a pistil-side IRB that causes rej
187 tioxidant activities compared to trypsin and HT, while trypsin and pepsin hydrolysates exhibited high
189 Although in vitro results of the TSLDox and HT treatment were favorable for both cell types, the the
193 % CI, 1.1-2.0), support with >/=inotropes at HT (OR, 1.7; 95% CI, 1.2-2.5), hospitalization status at
194 95% CI, 1.2-2.5), hospitalization status at HT (OR, 1.5; 95% CI, 1.0-2.19), chronic steroid use at H
196 5; 95% CI, 1.0-2.19), chronic steroid use at HT (OR, 1.5; 95% CI, 1.0-2.2), and treatment for early r
199 ins M1, B1, B2, G1, G2, ochratoxins A and B, HT-2 and T-2 toxins, deepoxy-deoxynivalenol, zearalenone
200 en were randomized (1:1 ratio) to home-based HT with a CD (CD group) or iHT performed by qualified th
205 on of human colorectal adenocarcinoma cells (HT-29) under the control of visible light and the extent
206 n to maturity) and a combination of e[CO2] + HT (from heading to maturity) using field based open top
209 with e[CO2], Pusa 1121 exposure to e[CO2] + HT led to significant reduction in seed-set and sink sta
210 een morphogenesis and differentiation during HT formation and provide a new framework to understand h
211 -Danlos syndrome hypermobility type (JHS/EDS-HT), characterized by idiopathic chronic itch with predo
212 resent study demonstrates that enzymatically HT-coated surface modification is an effective strategy
213 We found that all three accessions express HT proteins, which previously had been known to function
214 renal-risk variants, and used Illumina human HT-12 v4 arrays and Affymetrix HTA 2.0 arrays to generat
216 a high-throughput enhanced yeast two-hybrid (HT-eY2H) assay and a mammalian-cell-based Gaussia prince
218 gain have been associated with hypertension (HT), the association of linear growth, independently of
219 osensitive liposomes (TSL) and hyperthermia (HT) has shown preclinically to achieve high local drug c
221 iated natural killer (NK) cell activation in HT biopsies; however, little is known about the role of
222 cytotoxic activity of peripheral NK cells in HT recipients were evaluated using a noninvasive NK-cell
225 ble factor 1alpha (HIF-1alpha) expression in HT-29 xenografts initiated from NOX1 knockdown cells.
233 eaflet of the plasma membrane in intestinal (HT-29) cells and thereby reduce CT uptake into the cells
234 signaling in the mouse hippocampal cell line HT-22 leading to an increase in CREB phosphorylation.
235 tial modifier genes unlinked to the S-locus (HT-B, 120K, and NaStEP) unveils a higher degree of compl
238 both murine and human CAFs promoted de novo HT resistance via the generation of CD133(hi) CSCs that
239 -carboxyethyl-lysine, although the amount of HT in the biscuit was 100-fold lower than the biscuit fo
241 Furthermore, the maximum concentration of HT is reached between the fourth and sixth day of fermen
243 body-dependent NK cell cytotoxic function of HT recipients were associated with the FCGR3A-VV genotyp
246 of both the apparent barrier and the rate of HT for strong donor substituents in the para-cases.
247 emonstrated using proteome-wide reactions of HT-29 cell lysates with a model probe of threonine beta-
252 on inhibition fifty (IC50) for BL, SH, RG on HT-29 and HCT-116 cell proliferation ranged 0.9-2.0mg/mL
253 tment of hormone therapy (HT)-naive cells or HT-treated metabolically dormant populations with CAF-de
254 he extracted proteins with either trypsin or HT generated more antioxidant activity than hydrolysis w
256 omparison with listing FS, FS at 1 year post-HT increased in 91% and declined/remained unchanged in 9
257 association with abnormal FS at 1 year post-HT: >/=18 years of age (odds ratio [OR], 1.8; 95% confid
259 he published motifs estimated using the same HT-SELEX data, we demonstrate that BEESEM provides signi
260 microarrays (PBM) and high-throughput SELEX (HT-SELEX), have enabled rapid measurements of the specif
264 yces Torulaspora delbrueckii, can synthesise HT, as this compound was identified in the intracellular
268 s-5-hydroxytryptamide-tetraazacyclododecane [HT]-diethyneletriaminepentaacetic acid [DTPA]-gadolinium
270 Mechanistically, these findings suggest that HT neurons play a critical role in the initiation of the
273 bital coefficients are large accelerates the HT rate for meta-conjugated compounds just as for the pa
276 tra and by DFT computations we show that the HT properties are independent of bridge substituents for
277 rolled trials of menopausal hormone therapy (HT) and chronic disease risk appear to have divergent re
278 women and explored whether hormone therapy (HT) modified this association in 614 cases and 743 contr
279 cifically, the treatment of hormone therapy (HT)-naive cells or HT-treated metabolically dormant popu
280 ivation and sensitization of high-threshold (HT) but not wide-dynamic range trigeminovascular neurons
281 fremanezumab inhibited naive high-threshold (HT) neurons, but not wide-dynamic range trigeminovascula
287 be utilized to accelerate the hole transfer (HT) between redox sites attached in para- or in meta-pos
288 se of late mortality after heart transplant (HT), there is a need to identify markers that reflect in
292 ents were found in yellow-grained tritordeum HT 439 (12.16mug/gDW), followed by blue-grained wheat V1
293 tudy examines the potency of neoadjuvant TSL HT combination therapy in two orthotopic mouse models of
294 longitudinal subdivisions of the heart tube (HT), with the FHF contributing the left ventricle and pa
296 ing consecutive adult patients who underwent HT between January 2009 and December 2014 at our institu
298 BMI at birth was inversely associated with HT; c-BMI from school age to adulthood and c-height from
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