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1 intracytoplasmic membrane heme transport in Haemophilus.
3 etry (MALDI-TOF MS) in the identification of Haemophilus, Aggregatibacter, Cardiobacterium, Eikenella
4 espiratory pathogens belonging to the genera Haemophilus and Pseudomonas, colonized the early biofilm
5 pathogens, including Pseudomonas aeruginosa, Haemophilus, Aspergillus fumigatus, and nontuberculous m
6 creased abundance of Psuedomonas, Mycoplana, Haemophilus, Blautia, and Dorea genera in MS patients, w
8 to the role of P4 as an important factor for Haemophilus colonization and subsequent respiratory trac
9 ficantly higher relative abundances, whereas Haemophilus, Corynebacterium, Cellulosimicrobium and Cam
11 Among infants with lower CCL5 levels, the Haemophilus-dominant microbiota profile was associated w
13 s of CDTs from Escherichia coli (Ec-CDT) and Haemophilus ducreyi (Hd-CDT), which share limited amino
23 y the role of macrophage polarization during Haemophilus ducreyi infection, we analyzed a panel of ma
32 cause skin infections in the Pacific islands-Haemophilus ducreyi-as causes of skin ulceration in a ya
34 r no changes in the prevalence of Neisseria, Haemophilus, Gemella, Leptotrichia, Solobacterium, Parvi
37 nontypeable (NT) Haemophilus influenzae from Haemophilus haemolyticus in respiratory-tract samples, b
39 e also investigated their prevalences in 148 Haemophilus haemolyticus strains, a closely related spec
42 ekly challenged with a lysate of nontypeable Haemophilus influenza (NTHi), which induces COPD-type in
43 (1% rabies-vaccine recipients), one case of Haemophilus influenza meningitis (1% rabies-vaccine reci
45 rway inflammation in response to nontypeable Haemophilus influenza, which was associated with elevate
47 representing a reduced relative abundance of Haemophilus influenzae (35.3% [5.5-91.6] vs 6.7% [0.8-74
49 y to inhibit catalytic activity of DapE from Haemophilus influenzae (HiDapE) and ArgE from Escherichi
51 pathogens were detected frequently, notably Haemophilus influenzae (mostly nontypeable) together wit
52 re Pseudomonas aeruginosa (n = 10 patients), Haemophilus influenzae (n = 12), Prevotella (n = 18), an
54 ta-lactamase-producing strain of nontypeable Haemophilus influenzae (NTHi 86-028NP) and an isogenic m
59 by Streptococcus pneumoniae and nontypeable Haemophilus influenzae (NTHi) are frequently implicated
61 istic human respiratory pathogen nontypeable Haemophilus influenzae (NTHI) are required for type IV p
62 ocator function is necessary for nontypeable Haemophilus influenzae (NTHI) behaviors that mediate dis
63 ram-negative commensal bacterium nontypeable Haemophilus influenzae (NTHI) can cause respiratory trac
68 reptococcus pneumoniae (Spn) and nontypeable Haemophilus influenzae (NTHi) in stringently defined oti
69 xed Streptococcus pneumoniae and nontypeable Haemophilus influenzae (NTHi) infections (M-OM) and thos
83 am-negative pathogenic bacterium nontypeable Haemophilus influenzae (NTHi) is surface exposed and a l
87 vaccine against nonencapsulated isolates of Haemophilus influenzae (NTHi) lies in the genetic divers
88 ere we examine the impact of the nontypeable Haemophilus influenzae (NTHI) ModA2 phasevarion on patho
93 were calculated for pneumococcal, nontypable Haemophilus influenzae (NTHi), Moraxella catarrhalis, St
94 l collapse of biofilms formed by nontypeable Haemophilus influenzae (NTHI), those directed against a
95 ctions with pathogens, including nontypeable Haemophilus influenzae (NTHI), yet the reasons for this
104 tly colonized with bacteria [eg, nontypeable Haemophilus influenzae (NTHi)] that cause pulmonary infl
105 onfidence interval [CI], 1.90 and 10.19) and Haemophilus influenzae (OR, 2.04; 95% CI, 1.38 and 3.02)
106 (p = 0.001), NFkB activation by nontypeable Haemophilus influenzae (p = 0.001), TLR4 (p = 0.008) and
107 nces, most notably in potentially pathogenic Haemophilus influenzae (P = 2.7 x 10(-20)), from a preex
109 s simplex virus [n = 5], adenovirus [n = 5], Haemophilus influenzae [n = 5], and Streptococcus pneumo
111 colonization by Streptococcus pneumoniae and Haemophilus influenzae among children has been noted in
113 sensitivity and specificity for identifying Haemophilus influenzae and differentiating it from H. ha
114 activity has been achieved against wild-type Haemophilus influenzae and efflux-deficient mutants of E
115 ator to the respiratory pathogen nontypeable Haemophilus influenzae and identify the Haemophilus surf
117 nization with Gram-negative bacteria such as Haemophilus influenzae and Moraxella catarrhalis was fou
118 animal models of the Gram-negative pathogens Haemophilus influenzae and Neisseria meningitidis We hyp
119 vaccination with conjugate vaccines against Haemophilus influenzae and Streptococcus pneumoniae has
121 piratory syncytial virus; RSV) and bacteria (Haemophilus influenzae and Streptococcus pneumoniae) in
122 ion of EVI1 itself is induced by nontypeable Haemophilus influenzae and TNF-alpha in an NF-kappaB-dep
125 ococcus aureus, Streptococcus pneumoniae and Haemophilus influenzae are the major causes of conjuncti
127 5922 (0.008 to 0.03 mug/ml and 30 to 36 mm), Haemophilus influenzae ATCC 49247 (0.002 to 0.015 mug/ml
129 ureus ATCC 29213 (MIC range, 1 to 4 mug/ml), Haemophilus influenzae ATCC 49247 (MIC and disk diffusio
130 and ATCC 25923, Escherichia coli ATCC 25922, Haemophilus influenzae ATCC 49247, and Streptococcus pne
131 23, Streptococcus pneumoniae ATCC 49619, and Haemophilus influenzae ATCC 4927 strains were evaluated.
132 in Southern England immunized with DTaP5/IPV/Haemophilus influenzae b (Hib-TT) vaccine at 2-3-4 month
135 ve a significantly lower risk of nontypeable Haemophilus influenzae carriage in particular (relative
140 son of derivatives of a laboratory strain of Haemophilus influenzae expressing either surface-associa
142 marker for differentiating nontypeable (NT) Haemophilus influenzae from Haemophilus haemolyticus in
143 terial burden and a significant outgrowth of Haemophilus influenzae from the existing microbiota of s
145 ficiency further affected internalization of Haemophilus influenzae in bronchial epithelial cells.
146 d for TolR is of the periplasmic domain from Haemophilus influenzae in which N- and C-terminal residu
147 In this study, we found that nontypeable Haemophilus influenzae induces the association of Itch w
148 he survival rate after a lethal non-typeable Haemophilus influenzae infection in wild-type mice, but
149 tudy persistent Streptococcus pneumoniae and Haemophilus influenzae infections, to show that structur
155 lmonary inflammation induced by non-typeable Haemophilus influenzae is significantly attenuated in IR
156 configurations were predicted in nontypeable Haemophilus influenzae isolates based on the presence of
157 e operon was significantly more prevalent in Haemophilus influenzae isolates causing otitis media and
158 -resolution X-ray structure determination of Haemophilus influenzae KDO8PP bound to KDO/VO3(-) and Ba
159 d antimicrobial activity against a strain of Haemophilus influenzae lacking its major efflux pump.
160 we have shown that the C-terminal domain of Haemophilus influenzae LpoA (HiLpoA) has a highly conser
162 ed as having meningococcal, pneumococcal, or Haemophilus influenzae meningitis in the period 1977-200
164 ts of the 10-valent pneumococcal nontypeable Haemophilus influenzae protein D-conjugate vaccine (PHiD
166 m phylogenetically distant Aquifex aeolicus, Haemophilus influenzae Rd, and Synechocystis sp. were fo
167 lls) to the respiratory pathogen nontypeable Haemophilus influenzae resulted in a marked increase in
168 us, while challenge of Trim29(-/-) mice with Haemophilus influenzae resulted in lethal lung inflammat
170 tis in a healthy adult patient, secondary to Haemophilus influenzae serotype f infection, and we revi
172 e show that glucocorticoids and non-typeable Haemophilus influenzae synergistically upregulate IRAK-M
173 abs positive for Streptococcus pneumoniae or Haemophilus influenzae than were males (OR 9.09; 95% CI
176 of a collection of compound fragments using Haemophilus influenzae TrmD identified inhibitory, fused
177 n the first detection of 2 cases of invasive Haemophilus influenzae type a (Hia) disease in Italy.
178 t diphtheria, tetanus, pertussis, polio, and Haemophilus influenzae type b (DTaP-IPV-Hib) administere
179 s-acellular pertussis-inactivated poliovirus-Haemophilus influenzae type b (DTaP-IPV-Hib) vaccine sin
181 treptococcus pneumoniae (S. pneumoniae), and Haemophilus influenzae type b (Hib) are three most commo
182 ed (DTaP), inactivated poliovirus (IPV), and Haemophilus influenzae type b (Hib) conjugate vaccine (D
183 e United States in the early 1990s, when the Haemophilus influenzae type b (Hib) conjugate vaccine fo
187 ng hospital in Malawi during introduction of Haemophilus influenzae type b (Hib) vaccination and the
188 rst country in Africa to introduce conjugate Haemophilus influenzae type b (Hib) vaccine, which, as i
189 Streptococcus pneumoniae polysaccharide and Haemophilus influenzae type b (Hib) vaccines in ITP pati
191 ribitol (PRP) polysaccharides extracted from Haemophilus influenzae type b (Hib), and the correspondi
192 duction of vaccines against pneumococcus and Haemophilus influenzae type b (the most important causes
193 become the predominant invasive pathogen as Haemophilus influenzae type b and pneumococcal vaccine u
194 ca, the widespread use of vaccines targeting Haemophilus influenzae type b and Streptococcus pneumoni
195 and of occult bacteremia since the advent of Haemophilus influenzae type b and Streptococcus pneumoni
196 -tetanus-acellular pertussis-inactived polio-Haemophilus influenzae type b combined vaccine (DTaP-IPV
197 C, W, Y polysaccharide vaccine (PsACWY); or Haemophilus influenzae type b conjugate vaccine (Hib-TT)
199 clinical significance and characteristics of Haemophilus influenzae type b genogroup strains isolated
200 a, tetanus, pertussis, measles, rubella, and Haemophilus influenzae type b vaccine antigens were comp
201 s-acellular pertussis-inactivated poliovirus/Haemophilus influenzae type b vaccine; age 6/10/ 14 week
202 r pertussis, inactivated polio, hepatitis B, Haemophilus influenzae type b vaccines); (2) 4CMenB at 2
204 , and whole-cell pertussis; hepatitis B; and Haemophilus influenzae type b) and pneumococcal vaccine.
205 , tetanus, pertussis, hepatitis B virus, and Haemophilus influenzae type b), yellow fever, measles, a
206 ent vaccine (diphtheria, tetanus, pertussis, Haemophilus influenzae type b, and hepatitis B) at 6, 10
207 include pneumococcus, group B Streptococcus, Haemophilus influenzae type b, and meningococcus vaccine
208 to characterise disease syndromes caused by Haemophilus influenzae type b, pneumococcus, rotavirus,
209 cine priming, before a booster of a combined Haemophilus influenzae type b-MenC conjugate vaccine and
211 the sialic acid-specific SBP, SiaP, from the Haemophilus influenzae virulence-related SiaPQM TRAP tra
212 ctivity against an efflux-negative strain of Haemophilus influenzae was 4- to 8-fold higher, the comb
214 caused by either Streptococcus pneumoniae or Haemophilus influenzae were compared for pathogen-specif
215 phtericum and Corynebacterium propinquum and Haemophilus influenzae were significantly more abundant
218 ibrocytes) are able to recognize nontypeable Haemophilus influenzae, a major pathogen of middle ear i
219 ccus aureus, 10 Streptococcus pneumoniae, 10 Haemophilus influenzae, and 5 Escherichia coli isolates
220 orins from Neisseriae, Shigella, Salmonella, Haemophilus influenzae, and Fusobacterium nucleatum, whi
222 antitative PCR for Streptococcus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis were p
223 ovirus, bocavirus, Streptococcus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis were s
224 c airway bacteria (Streptococcus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis) were
226 phocholine, phosphocholine-modified LPS from Haemophilus influenzae, and phosphocholine-modified prot
227 phylococci (CoNS), Streptococcus pneumoniae, Haemophilus influenzae, and Pseudomonas aeruginosa.
228 eumoniae, Staphylococcus aureus, Nontypeable Haemophilus influenzae, and Pseudomonas aeruginosa.
229 thogens: Pseudomonas aeruginosa, nontypeable Haemophilus influenzae, and Salmonella enterica serovar
232 atory tract pathogens Moraxella catarrhalis, Haemophilus influenzae, and Streptococcus pneumoniae, bu
233 is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus pneumoniae, in
234 eudomonas aeruginosa, Staphylococcus aureus, Haemophilus influenzae, Aspergillus species, Streptococc
235 protection against PC-expressing nontypeable Haemophilus influenzae, but not PC-negative nontypeable
236 , we turned our attention to bacteria, i.e., Haemophilus influenzae, expressing cell-surface adhesins
238 a catarrhalis, Streptococcus pneumoniae, and Haemophilus influenzae, is associated with later develop
239 tly from CSF specimens: Escherichia coli K1, Haemophilus influenzae, Listeria monocytogenes, Neisseri
240 ccines with/without protein D of nontypeable Haemophilus influenzae, M. catarrhalis has become a high
241 were cultured for Streptococcus pneumoniae, Haemophilus influenzae, Moraxella catarrhalis, and Staph
242 piratory pathogens Streptococcus pneumoniae, Haemophilus influenzae, Moraxella catarrhalis, and Staph
243 irways with the pathogenic bacterial strains Haemophilus influenzae, Moraxella catarrhalis, and Strep
244 y associated with bacterial coinfection with Haemophilus influenzae, Moraxella catarrhalis, or Strept
246 lude Escherichia coli, Campylobacter jejuni, Haemophilus influenzae, Neisseria meningitidis, and Past
248 cterial pathogens (Streptococcus pneumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplas
249 used to vaccinate children globally against Haemophilus influenzae, pneumococcus, and meningococcus.
252 morphism in humans evades TbpA variants from Haemophilus influenzae, revealing a functional basis for
253 tococcus pneumoniae, Neisseria meningitidis, Haemophilus influenzae, S suis) and O tsutsugamushi, Ric
254 and 24 months of age were cultured to detect Haemophilus influenzae, Streptococcus pneumoniae, Moraxe
255 usters characterized by enrichment of either Haemophilus influenzae, Streptococcus, Corynebacterium,
257 the sialic acid TRAP transporter SiaPQM from Haemophilus influenzae, where the membrane proteins are
258 een H. parainfluenzae and its close relative Haemophilus influenzae, which is also commonly carried w
259 w that EVI1 negatively regulates nontypeable Haemophilus influenzae- and TNF-alpha-induced NF-kappaB-
274 pecific bacteria such as Enterobacteriaceae, Haemophilus, Methylobacterium, and Ralstonia species wer
275 cts were uniquely enriched in members of the Haemophilus, Neisseria, Fusobacterium, and Porphyromonas
276 ond their ability to mediate DNA transfer in Haemophilus, OMV stimulation of host immunomodulatory cy
279 Streptococcus mitis, Rothia mucilaginosa and Haemophilus parainfluenzae were the most significantly a
280 ubation of asthmatic airway macrophages with Haemophilus parainfluenzae, a uniquely expanded potentia
281 his technology for the identification of 103 Haemophilus parainfluenzae, Aggregatibacter aphrophilus,
282 lobacter gracilis, Capnocytophaga granulosa, Haemophilus parainfluenzae, and Lautropia mirabilis were
283 ardiobacterium hominis, Gemella haemolysans, Haemophilus parainfluenzae, Kingella oralis, Lautropia m
289 y 56% identical amino acids, both FH-binding Haemophilus proteins similarly interacted with the compl
290 distinguish H. haemolyticus from its closest Haemophilus relatives and provide clues to the identity
291 We developed mouse models of Chlamydia and Haemophilus respiratory infection-mediated, ovalbumin-in
292 obiota diversity (P = .009) and dominance of Haemophilus species operational taxonomic units (P = .01
296 activity among staphylococci, streptococci, Haemophilus spp., and Moraxella catarrhalis were minimal
298 able Haemophilus influenzae and identify the Haemophilus surface protein E (PE) as a new plasminogen-
300 First characterized as transformasomes in Haemophilus, these membranous blebs facilitate transfer
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