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1 enes, Staphylococcus aureus, and potentially Haemophilus influenzae).
2 hylococcus and Gram-negative bacteria and to Haemophilus influenzae.
3 t line of defense against the human pathogen Haemophilus influenzae.
4 ococcus pneumoniae, and 54% for non-typeable Haemophilus influenzae.
5 were previously misidentified as nontypeable Haemophilus influenzae.
6 ainst efflux-negative strains of E. coli and Haemophilus influenzae.
7 s successful in creating unmarked mutants in Haemophilus influenzae.
8 crobial peptides (sap operon) in nontypeable Haemophilus influenzae.
9 luding the phylogenetically related pathogen Haemophilus influenzae.
10 eria gonorrheae, Neisseria meningitidis, and Haemophilus influenzae.
11 dA from Yersinia enterocolitica and Hia from Haemophilus influenzae.
12 on between influenza virus and the bacterium Haemophilus influenzae.
13 Bordetella pertussis, Escherichia coli, and Haemophilus influenzae.
14 of both typeable and nontypeable strains of Haemophilus influenzae.
15 fastidious gram-negative pathogens including Haemophilus influenzae.
16 y bacterial pathogens, including nontypeable Haemophilus influenzae.
17 d is absent from the closely related species Haemophilus influenzae.
18 ccus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae.
19 ng Streptococcus pneumoniae and non-typeable Haemophilus influenzae.
20 nd in some regions, for all pathogens except Haemophilus influenzae.
21 ssue, which included known pathogens such as Haemophilus influenzae.
22 representing a reduced relative abundance of Haemophilus influenzae (35.3% [5.5-91.6] vs 6.7% [0.8-74
25 ibrocytes) are able to recognize nontypeable Haemophilus influenzae, a major pathogen of middle ear i
27 colonization by Streptococcus pneumoniae and Haemophilus influenzae among children has been noted in
29 sensitivity and specificity for identifying Haemophilus influenzae and differentiating it from H. ha
30 activity has been achieved against wild-type Haemophilus influenzae and efflux-deficient mutants of E
32 ator to the respiratory pathogen nontypeable Haemophilus influenzae and identify the Haemophilus surf
35 nization with Gram-negative bacteria such as Haemophilus influenzae and Moraxella catarrhalis was fou
36 animal models of the Gram-negative pathogens Haemophilus influenzae and Neisseria meningitidis We hyp
37 reveal their striking genetic similarity to Haemophilus influenzae and other members of the Pasteure
40 vaccination with conjugate vaccines against Haemophilus influenzae and Streptococcus pneumoniae has
42 piratory syncytial virus; RSV) and bacteria (Haemophilus influenzae and Streptococcus pneumoniae) in
43 ion of EVI1 itself is induced by nontypeable Haemophilus influenzae and TNF-alpha in an NF-kappaB-dep
45 ccus aureus, 10 Streptococcus pneumoniae, 10 Haemophilus influenzae, and 5 Escherichia coli isolates
46 orins from Neisseriae, Shigella, Salmonella, Haemophilus influenzae, and Fusobacterium nucleatum, whi
48 antitative PCR for Streptococcus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis were p
49 ovirus, bocavirus, Streptococcus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis were s
50 c airway bacteria (Streptococcus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis) were
51 ive conjugate vaccines against S pneumoniae, Haemophilus influenzae, and Neisseria meningitides to ch
53 phocholine, phosphocholine-modified LPS from Haemophilus influenzae, and phosphocholine-modified prot
56 thogens: Pseudomonas aeruginosa, nontypeable Haemophilus influenzae, and Salmonella enterica serovar
60 atory tract pathogens Moraxella catarrhalis, Haemophilus influenzae, and Streptococcus pneumoniae, bu
61 is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus pneumoniae, in
62 w that EVI1 negatively regulates nontypeable Haemophilus influenzae- and TNF-alpha-induced NF-kappaB-
64 ococcus aureus, Streptococcus pneumoniae and Haemophilus influenzae are the major causes of conjuncti
66 eudomonas aeruginosa, Staphylococcus aureus, Haemophilus influenzae, Aspergillus species, Streptococc
67 5922 (0.008 to 0.03 mug/ml and 30 to 36 mm), Haemophilus influenzae ATCC 49247 (0.002 to 0.015 mug/ml
69 ureus ATCC 29213 (MIC range, 1 to 4 mug/ml), Haemophilus influenzae ATCC 49247 (MIC and disk diffusio
70 and ATCC 25923, Escherichia coli ATCC 25922, Haemophilus influenzae ATCC 49247, and Streptococcus pne
71 23, Streptococcus pneumoniae ATCC 49619, and Haemophilus influenzae ATCC 4927 strains were evaluated.
72 in Southern England immunized with DTaP5/IPV/Haemophilus influenzae b (Hib-TT) vaccine at 2-3-4 month
77 with the emergence of the clonal variants of Haemophilus influenzae biogroup aegyptius causing Brazil
78 protection against PC-expressing nontypeable Haemophilus influenzae, but not PC-negative nontypeable
79 ccus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae, but the mechanism by which they
81 ve a significantly lower risk of nontypeable Haemophilus influenzae carriage in particular (relative
85 homodimeric periplasmic domain of TolR from Haemophilus influenzae, determined with conventional, NO
88 son of derivatives of a laboratory strain of Haemophilus influenzae expressing either surface-associa
89 , we turned our attention to bacteria, i.e., Haemophilus influenzae, expressing cell-surface adhesins
91 marker for differentiating nontypeable (NT) Haemophilus influenzae from Haemophilus haemolyticus in
92 terial burden and a significant outgrowth of Haemophilus influenzae from the existing microbiota of s
94 oach was applied to comprehensively identify Haemophilus influenzae genes required to delay bacterial
95 ed AIMIE to analyze the Escherichia coli and Haemophilus influenzae genomes in order to demonstrate t
99 y to inhibit catalytic activity of DapE from Haemophilus influenzae (HiDapE) and ArgE from Escherichi
106 ch data across various human pathogens (e.g. Haemophilus influenzae, human immunodeficiency virus (HI
107 colonization by Streptococcus pneumoniae and Haemophilus influenzae in a Toll-like receptor (TLR)-dep
108 ficiency further affected internalization of Haemophilus influenzae in bronchial epithelial cells.
109 d for TolR is of the periplasmic domain from Haemophilus influenzae in which N- and C-terminal residu
110 % vs 23%-31% of AOM isolates), while that of Haemophilus influenzae increased (41%-43% vs 56%-57%) pr
111 In this study, we found that nontypeable Haemophilus influenzae induces the association of Itch w
112 he survival rate after a lethal non-typeable Haemophilus influenzae infection in wild-type mice, but
113 tudy persistent Streptococcus pneumoniae and Haemophilus influenzae infections, to show that structur
125 lmonary inflammation induced by non-typeable Haemophilus influenzae is significantly attenuated in IR
126 a catarrhalis, Streptococcus pneumoniae, and Haemophilus influenzae, is associated with later develop
127 configurations were predicted in nontypeable Haemophilus influenzae isolates based on the presence of
128 e operon was significantly more prevalent in Haemophilus influenzae isolates causing otitis media and
129 -resolution X-ray structure determination of Haemophilus influenzae KDO8PP bound to KDO/VO3(-) and Ba
131 d antimicrobial activity against a strain of Haemophilus influenzae lacking its major efflux pump.
132 tly from CSF specimens: Escherichia coli K1, Haemophilus influenzae, Listeria monocytogenes, Neisseri
133 we have shown that the C-terminal domain of Haemophilus influenzae LpoA (HiLpoA) has a highly conser
136 ccines with/without protein D of nontypeable Haemophilus influenzae, M. catarrhalis has become a high
137 ed as having meningococcal, pneumococcal, or Haemophilus influenzae meningitis in the period 1977-200
139 were cultured for Streptococcus pneumoniae, Haemophilus influenzae, Moraxella catarrhalis, and Staph
140 piratory pathogens Streptococcus pneumoniae, Haemophilus influenzae, Moraxella catarrhalis, and Staph
141 irways with the pathogenic bacterial strains Haemophilus influenzae, Moraxella catarrhalis, and Strep
142 y associated with bacterial coinfection with Haemophilus influenzae, Moraxella catarrhalis, or Strept
144 pathogens were detected frequently, notably Haemophilus influenzae (mostly nontypeable) together wit
145 ium, Bacillus subtilis, Helicobacter pylori, Haemophilus influenzae, Mycobacterium tuberculosis, Pseu
146 re Pseudomonas aeruginosa (n = 10 patients), Haemophilus influenzae (n = 12), Prevotella (n = 18), an
147 s simplex virus [n = 5], adenovirus [n = 5], Haemophilus influenzae [n = 5], and Streptococcus pneumo
148 lude Escherichia coli, Campylobacter jejuni, Haemophilus influenzae, Neisseria meningitidis, and Past
150 cterial pathogens (Streptococcus pneumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplas
152 ta-lactamase-producing strain of nontypeable Haemophilus influenzae (NTHi 86-028NP) and an isogenic m
156 Haemophilus haemolyticus and nontypeable Haemophilus influenzae (NTHi) are closely related upper
157 by Streptococcus pneumoniae and nontypeable Haemophilus influenzae (NTHi) are frequently implicated
159 istic human respiratory pathogen nontypeable Haemophilus influenzae (NTHI) are required for type IV p
161 typically recovered quantity of nontypeable Haemophilus influenzae (NTHi) bacteria in an ex vivo mid
162 ocator function is necessary for nontypeable Haemophilus influenzae (NTHI) behaviors that mediate dis
163 ram-negative commensal bacterium nontypeable Haemophilus influenzae (NTHI) can cause respiratory trac
169 reptococcus pneumoniae (Spn) and nontypeable Haemophilus influenzae (NTHi) in stringently defined oti
170 xed Streptococcus pneumoniae and nontypeable Haemophilus influenzae (NTHi) infections (M-OM) and thos
186 am-negative pathogenic bacterium nontypeable Haemophilus influenzae (NTHi) is surface exposed and a l
191 vaccine against nonencapsulated isolates of Haemophilus influenzae (NTHi) lies in the genetic divers
192 ere we examine the impact of the nontypeable Haemophilus influenzae (NTHI) ModA2 phasevarion on patho
198 ce lipooligosaccharides (LOS) of nontypeable Haemophilus influenzae (NTHi), a human-specific commensa
201 with bacterial opportunists like nontypeable Haemophilus influenzae (NTHi), and a wealth of evidence
202 were calculated for pneumococcal, nontypable Haemophilus influenzae (NTHi), Moraxella catarrhalis, St
203 of several adhesins expressed by nontypeable Haemophilus influenzae (NTHI), the outer membrane protei
204 l collapse of biofilms formed by nontypeable Haemophilus influenzae (NTHI), those directed against a
205 ctions with pathogens, including nontypeable Haemophilus influenzae (NTHI), yet the reasons for this
216 tly colonized with bacteria [eg, nontypeable Haemophilus influenzae (NTHi)] that cause pulmonary infl
218 crystal structure of a bacterial homologue (Haemophilus influenzae) of SLAC1 at 1.20 A resolution, a
220 onfidence interval [CI], 1.90 and 10.19) and Haemophilus influenzae (OR, 2.04; 95% CI, 1.38 and 3.02)
221 During model murine nasal colonization, Haemophilus influenzae outcompetes another member of the
222 (p = 0.001), NFkB activation by nontypeable Haemophilus influenzae (p = 0.001), TLR4 (p = 0.008) and
223 nces, most notably in potentially pathogenic Haemophilus influenzae (P = 2.7 x 10(-20)), from a preex
224 used to vaccinate children globally against Haemophilus influenzae, pneumococcus, and meningococcus.
225 ts of the 10-valent pneumococcal nontypeable Haemophilus influenzae protein D-conjugate vaccine (PHiD
229 m phylogenetically distant Aquifex aeolicus, Haemophilus influenzae Rd, and Synechocystis sp. were fo
231 lls) to the respiratory pathogen nontypeable Haemophilus influenzae resulted in a marked increase in
232 us, while challenge of Trim29(-/-) mice with Haemophilus influenzae resulted in lethal lung inflammat
233 morphism in humans evades TbpA variants from Haemophilus influenzae, revealing a functional basis for
234 tococcus pneumoniae, Neisseria meningitidis, Haemophilus influenzae, S suis) and O tsutsugamushi, Ric
236 tis in a healthy adult patient, secondary to Haemophilus influenzae serotype f infection, and we revi
237 rologous genetic background of a nonadherent Haemophilus influenzae strain, and quantitative assays r
241 and 24 months of age were cultured to detect Haemophilus influenzae, Streptococcus pneumoniae, Moraxe
242 Chinchillas were infected with nontypeable Haemophilus influenzae, Streptococcus pneumoniae, or a c
243 usters characterized by enrichment of either Haemophilus influenzae, Streptococcus, Corynebacterium,
244 e show that glucocorticoids and non-typeable Haemophilus influenzae synergistically upregulate IRAK-M
245 abs positive for Streptococcus pneumoniae or Haemophilus influenzae than were males (OR 9.09; 95% CI
248 of a collection of compound fragments using Haemophilus influenzae TrmD identified inhibitory, fused
249 n the first detection of 2 cases of invasive Haemophilus influenzae type a (Hia) disease in Italy.
250 t diphtheria, tetanus, pertussis, polio, and Haemophilus influenzae type b (DTaP-IPV-Hib) administere
251 s-acellular pertussis-inactivated poliovirus-Haemophilus influenzae type b (DTaP-IPV-Hib) vaccine sin
253 treptococcus pneumoniae (S. pneumoniae), and Haemophilus influenzae type b (Hib) are three most commo
254 ed (DTaP), inactivated poliovirus (IPV), and Haemophilus influenzae type b (Hib) conjugate vaccine (D
255 e United States in the early 1990s, when the Haemophilus influenzae type b (Hib) conjugate vaccine fo
260 ng hospital in Malawi during introduction of Haemophilus influenzae type b (Hib) vaccination and the
261 rst country in Africa to introduce conjugate Haemophilus influenzae type b (Hib) vaccine, which, as i
262 Streptococcus pneumoniae polysaccharide and Haemophilus influenzae type b (Hib) vaccines in ITP pati
264 ribitol (PRP) polysaccharides extracted from Haemophilus influenzae type b (Hib), and the correspondi
265 duction of vaccines against pneumococcus and Haemophilus influenzae type b (the most important causes
266 become the predominant invasive pathogen as Haemophilus influenzae type b and pneumococcal vaccine u
267 ca, the widespread use of vaccines targeting Haemophilus influenzae type b and Streptococcus pneumoni
268 and of occult bacteremia since the advent of Haemophilus influenzae type b and Streptococcus pneumoni
269 -tetanus-acellular pertussis-inactived polio-Haemophilus influenzae type b combined vaccine (DTaP-IPV
270 C, W, Y polysaccharide vaccine (PsACWY); or Haemophilus influenzae type b conjugate vaccine (Hib-TT)
272 clinical significance and characteristics of Haemophilus influenzae type b genogroup strains isolated
274 a, tetanus, pertussis, measles, rubella, and Haemophilus influenzae type b vaccine antigens were comp
275 s-acellular pertussis-inactivated poliovirus/Haemophilus influenzae type b vaccine; age 6/10/ 14 week
276 that pediatric providers conserve available Haemophilus influenzae type B vaccines by delaying the a
278 r pertussis, inactivated polio, hepatitis B, Haemophilus influenzae type b vaccines); (2) 4CMenB at 2
280 , and whole-cell pertussis; hepatitis B; and Haemophilus influenzae type b) and pneumococcal vaccine.
281 , tetanus, pertussis, hepatitis B virus, and Haemophilus influenzae type b), yellow fever, measles, a
282 ent vaccine (diphtheria, tetanus, pertussis, Haemophilus influenzae type b, and hepatitis B) at 6, 10
283 include pneumococcus, group B Streptococcus, Haemophilus influenzae type b, and meningococcus vaccine
284 against some types of bacterial meningitis (Haemophilus influenzae type b, Neisseria meningitidis gr
285 to characterise disease syndromes caused by Haemophilus influenzae type b, pneumococcus, rotavirus,
286 cine priming, before a booster of a combined Haemophilus influenzae type b-MenC conjugate vaccine and
292 the sialic acid-specific SBP, SiaP, from the Haemophilus influenzae virulence-related SiaPQM TRAP tra
293 ctivity against an efflux-negative strain of Haemophilus influenzae was 4- to 8-fold higher, the comb
296 caused by either Streptococcus pneumoniae or Haemophilus influenzae were compared for pathogen-specif
297 phtericum and Corynebacterium propinquum and Haemophilus influenzae were significantly more abundant
298 the sialic acid TRAP transporter SiaPQM from Haemophilus influenzae, where the membrane proteins are
299 een H. parainfluenzae and its close relative Haemophilus influenzae, which is also commonly carried w
300 on X-ray crystal structures of the DapE from Haemophilus influenzae with one and two zinc ions bound
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