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1 nts for the diminished rate of activation of Hageman factor and therefore a diminished rate of activa
5 n factor; this feedback is necessary for the Hageman factor-dependent pathways to proceed at a normal
7 tic, and chemotactic defects and addition of Hageman factor fragments corrected the fibrinolytic defe
9 XII (FXIIa) is selectively inhibited by corn Hageman factor inhibitor (CHFI) among other plasma prote
11 inhibits mammalian trypsin and Factor XIIa (Hageman Factor) of the contact pathway of coagulation as
12 ear to be initiated by contact activation of Hageman factor, the kallikrein generated activates more
13 tor, the kallikrein generated activates more Hageman factor; this feedback is necessary for the Hagem
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