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1 type frequencies deviated significantly from Hardy-Weinberg equilibrium.
2 All control samples were in Hardy-Weinberg equilibrium.
3 ously and did not deviate significantly from Hardy-Weinberg equilibrium.
4 e distribution of all 5 polymorphisms was in Hardy-Weinberg equilibrium.
5 H depletes heterozygotes, thereby disrupting Hardy-Weinberg equilibrium.
6 lex survey designs to assess deviations from Hardy-Weinberg equilibrium.
7 cus in a population, under the assumption of Hardy-Weinberg equilibrium.
8 All SNPs were in Hardy-Weinberg equilibrium.
9 The three polymorphisms were in Hardy-Weinberg equilibrium.
10 nonequilibrium population that deviates from Hardy-Weinberg equilibrium.
11 All SNPs were in Hardy-Weinberg equilibrium.
12 All SNPs were in Hardy-Weinberg equilibrium.
13 ere observed in both populations and were in Hardy-Weinberg equilibrium.
14 ism, perfectly matched the expectation under Hardy-Weinberg equilibrium.
15 es in the study population were assessed for Hardy-Weinberg equilibrium.
16 All observed genotype frequencies were in Hardy-Weinberg equilibrium.
17 in the general population than predicted by Hardy-Weinberg Equilibrium.
18 ns, and frequencies of genotypes were in the Hardy-Weinberg equilibrium.
19 re randomly sampled from a population not in Hardy-Weinberg equilibrium.
20 he allele distribution was inconsistent with Hardy-Weinberg equilibrium.
21 duplication were present in concordance with Hardy-Weinberg equilibrium (13% and 23%, respectively),
22 homogeneity is known to be valid only under Hardy-Weinberg equilibrium, a property that may not hold
23 ease be done on the basis of deviations from Hardy-Weinberg equilibrium among affected individuals.
24 States Caucasian random blood donors was in Hardy-Weinberg equilibrium and CCR5Delta32 homozygotes r
25 o STR locus was observed to deviate from the Hardy-Weinberg equilibrium and linkage disequilibriums a
26 met stringent criteria for concordance with Hardy-Weinberg equilibrium and low genotyping error rate
27 The PTPN2 variant rs2542151 deviated from Hardy-Weinberg equilibrium and was excluded from analyse
29 iteria for missing genotypes, departure from Hardy-Weinberg equilibrium, and low minor allele frequen
30 xcess of homozygotes, over that predicted by Hardy-Weinberg equilibrium; and (3) the model-predicted
31 ality control (QC) tests, including tests of Hardy-Weinberg equilibrium, are not sensitive enough to
33 res on population genetics to illustrate the Hardy-Weinberg equilibrium, calculate allele frequencies
35 dies have explored the use of departure from Hardy-Weinberg equilibrium (DHW) for fine mapping Mendel
39 Recently, frequentist statistical tests for Hardy-Weinberg equilibrium have been proposed specifical
41 es, e.g. STRUCTURE and L-POP, usually assume Hardy-Weinberg equilibrium (HWE) and linkage equilibrium
43 We explore the extent of deviations from Hardy-Weinberg equilibrium (HWE) at a marker locus and l
45 th newborns of first-time mothers outside of Hardy-Weinberg equilibrium (HWE) during peak PM season.
46 ypotheses that could explain deviations from Hardy-Weinberg equilibrium (HWE) expectations due to het
47 al and per subpopulation, and calculation of Hardy-Weinberg Equilibrium (HWE) for each subpopulation.
48 Genotype distributions for all SNPs were in Hardy-Weinberg equilibrium (HWE) in controls, but in cas
54 tween markers and disease were analyzed by a Hardy-Weinberg equilibrium (HWE) test, a conventional ca
55 -genome case-control association studies and Hardy-Weinberg equilibrium (HWE) testing for data qualit
61 APO, ESR1 and PV92 loci were found to be in Hardy-Weinberg equilibrium in all the ethnic groups, whi
64 gote deficiency in females but which were in Hardy-Weinberg equilibrium in males, consistent with deg
66 lid method that corrects for deviations from Hardy-Weinberg Equilibrium is presented, so that the cha
68 t-guided filters based on QC variables [e.g. Hardy-Weinberg equilibrium, missing proportion (MSP) and
70 ition, our method relaxes the requirement of Hardy-Weinberg equilibrium of haplotype frequencies in c
71 he likelihood-ratio statistic, which assumes Hardy-Weinberg equilibrium of the marker phenotype propo
72 IM, incorporating chosen assumptions such as Hardy-Weinberg equilibrium or exchangeability of parenta
75 allele distribution strongly deviating from Hardy-Weinberg equilibrium, possibly implying selection
76 g Vieland and Huang's proof is that of joint Hardy-Weinberg equilibrium proportions at two trait loci
77 otypes in the general population comply with Hardy-Weinberg Equilibrium proportions, which may not al
78 similar to those previously observed and in Hardy-Weinberg equilibrium: SS 61.1%, Ss 36.2%, and ss 2
79 to approximate the discrete distributions of Hardy-Weinberg equilibrium test statistics and P-values.
81 n this paper, the authors present a test for Hardy-Weinberg equilibrium that adjusts for the sample w
83 e show that it is robust to the violation of Hardy-Weinberg equilibrium, to the presence of missing d
84 After removing six loci that departed from Hardy-Weinberg equilibrium, we measured genetic variatio
85 homozygotes compared with that expected from Hardy-Weinberg equilibrium, whereas in males there is an
86 ives the first Bayesian approach for testing Hardy-Weinberg equilibrium with biallelic markers at the
88 , which could be partly explained by lack of Hardy-Weinberg equilibrium, with an excess of homozygote
89 ulation cohorts, this mutation deviated from Hardy-Weinberg equilibrium, with an overrepresentation o
90 ccomplished by eliminating the assumption of Hardy-Weinberg equilibrium within clusters and, instead,
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