ライフサイエンスコーパス: HeLa cell

1 in 8.5 +/- 3.4% Synt5 expression after 24h (HeLa cells).

2 pol II pausing index on a subset of genes in HeLa cells.

3 red blood cell lysis or cytotoxicity against HeLa cells.

4 ts (pH 5.0-5.5) after 5 h of incubation with HeLa cells.

5 to human glioblastoma A172 and synchronized HeLa cells.

6 during and reactivation from persistence in HeLa cells.

7 abling the formation of gel fibers on living HeLa cells.

8 miRNAs in total RNA extracted from 293T and HeLa cells.

9 nretrovirus Gag localizing to the nucleus of HeLa cells.

10 es as well as all six Atg8 family members in HeLa cells.

11 is enriched at distal Golgi compartments in HeLa cells.

12 us fluorescent protein using gene editing in HeLa cells.

13 and up-regulation of chaperone expression in HeLa cells.

14 a-deficient murine embryonic fibroblasts and HeLa cells.

15 autophagosome recruitment to mitochondria in HeLa cells.

16 o screen for proteins regulated by NHERF1 in HeLa cells.

17 10(5) molecules per cell) nuclear protein in HeLa cells.

18 maging of intracellular thiols within living HeLa cells.

19 by alternative splicing in both HEK293T and HeLa cells.

20 1 CTD, which binds PP1 in vitro and in human HeLa cells.

21 ed loss of IFI16 in HFFs and NOKs but not in HeLa cells.

22 r long-term monitoring of local pH values in HeLa cells.

23 measure drug-stimulated PKA from lysates of HeLa cells.

24 n in human bronchial epithelial cells and H1-HeLa cells.

25 s recovered from the cytoplasm of individual HeLa cells.

26 30 significantly reduced CVB3 replication in HeLa cells.

27 ffect on attachment to CAR or replication in HeLa cells.

28 of the phosphatidylcholine pool in infected HeLa cells.

29 displace lactobacilli previously adhered to HeLa cells.

30 cient in EGF-mediated ERM phosphorylation in HeLa cells.

31 e intercellular bridge during cytokinesis in HeLa cells.

32 ells showing better permeability compared to HeLa cells.

33 nts investigating the heat-shock response in HeLa cells.

34 tioned nucleosome within the FOS promoter in HeLa cells.

35 sed BMP4 expression was demonstrated only in HeLa cells.

36 re validated in vitro in HEK293T, HepG2, and HeLa cells.

37 ells carrying episomal HPV18 minicircles and HeLa cells.

38 s with active R-Ras in transiently expressed HeLa cells.

39 d the integrated portion of HPV18 present in HeLa cells.

40 dynein recruitment in neural stem cells and HeLa cells.

41 y was 100-fold greater in Vero cells than in HeLa cells.

42 port GATA-3 transactivation in human HEK and HeLa cells.

43 ria induced a G2/M phase transition delay in HeLa cells.

44 the newly formed virions are able to infect HeLa cells.

45 ependent selective mitophagy in CCCP-treated HeLa cells.

46 artial aqueous medium and intracellularly in HeLa cells.

47 s are due to nucleic acid contamination from HeLa cells.

48 opy in neurons, human fibroblasts, U2OS, and HeLa cells.

49 by selective labeling of lipid and hCAII in Hela cells.

50 teracting with TCTP in human cervical cancer HeLa cells.

51 f-organization of the actin cortex in living HeLa cells.

52 pproximately 120 nm x approximately 80 nm in HeLa cells.

53 n is induced upon siRNA knock-down of SMN in HeLa cells.

54 aurosporine-induced (intrinsic) apoptosis in HeLa cells.

55 cing of untreated, apoptotic, and recovering HeLa cells.

56 y tryptic digestion of protein extracts from HeLa cells.

57 viability-based genome-wide siRNA screen in HeLa cells.

58 e that OA induces mature miR-7 production in HeLa cells.

59 EGF-mediated activations of both kinases in HeLa cells.

60 ed to HAE cells but also includes HEK293 and HeLa cells.

61 en protein-coding and lincRNA genes in human HeLa cells.

62 unoglobulin M (micros), is well-tolerated in HeLa cells.

63 them to have generally low cytotoxicities in Hela cells.

64 ective variant reduces cell proliferation in HeLa cells.

65 In vitro cell-studies showed enhanced HeLa cell adhesion and proliferation on the MWCNT-algina

66 ed mechanism because Dynasore also protected HeLa cells against streptolysin O.

67 mARC2 was shown to protect HeLa cells against the apoptotic effects of the base ana

68 conserved, as depletion of Snx1 and Snx2 in HeLa cells also led to greater overlap of Rme-8 and Hrs

69 comet) Now we have isolated from extracts of HeLa cells an ATP-dependent factor that releases Cdc20 f

70 ielded an unprecedented quantitative view of HeLa cell anatomy and organellar composition, at the pro

71 3.6 times higher than branched PEI 25 kDa in HeLa cells and 7.4 times higher than Lipofectamine(R) 20

72 ls are recapitulated by knockdown of APE1 in Hela cells and are rescued by overexpression of NRF1/2.

73 of telomeres and leads to cell senescence in HeLa cells and cell death in HeLa, U2OS and IMR90 cells,

74 nd sseL), S. enterica subsp. salamae invades HeLa cells and contains homologues of S. bongori sboK an

75 Green tag into the endogenous MBNL1 locus in HeLa cells and established a flow cytometry-based screen

76 NM2A associates with the plasma membrane of HeLa cells and fibrosarcoma cells independently of F-act

77 es, pit formation, and endocytosis in COS-7, HeLa cells and hippocampal neurons.

78 oteins in WT and CRISPR-mediated GNPTAB(-/-) HeLa cells and identified changes in numerous glycoprote

79 lex as a potential ER Ca(2+) leak channel in HeLa cells and identified ER lumenal molecular chaperone

80 re also tested for their cytotoxicity toward HeLa cells and in vivo in a Plasmodium berghei malaria m

81 ficient to activate the NF-kappaB pathway in HeLa cells and induce apoptotic cell death in both HeLa

82 rReal2, leads to tubulin hyperacetylation in HeLa cells and induces destabilization of the checkpoint

83 oreactive cross-linkers were internalized by HeLa cells and irradiated at set time intervals, then is

84 exhibits a half-life of approximately 6 h in HeLa cells and is targeted for ubiquitylation through ac

85 red using RPAD from human cervical carcinoma HeLa cells and mouse C2C12 myoblasts led to two surprisi

86 on two parental cell lines (e.g., wild-type HeLa cells and MRP1-overexpressing HeLa-R cells).

87 using p22(phox) short-hairpin RNA-engineered HeLa cells and Nox2(-/-) coronary microvascular endothel

88 ty and LD sizes during glucose starvation of HeLa cells and transforming growth factor beta-induced e

89 achieved an effective depletion of eIF5A in HeLa cells and undertook in vivo comprehensive proteomic

90 the cytotoxicity of Pdots were evaluated on HeLa cells and zebrafish embryos to demonstrate their gr

91 mixture of gold nanoparticles and DBA-tagged HeLa cell, and subsequently propagating laser light thro

92 199 localized with ERGIC and COPI markers in HeLa cells, and electron microscopy of a liver biopsy sh

93 than 99 % reduction of mitochondrial rRNA in HeLa cells, and enrichment of pathogen sequences in pati

94 iated endogenous gene loci in HEK293T cells, HeLa cells, and human induced pluripotent stem cells.

95 sensors in the cytosol compared to the ER of HeLa cells, and identify the formation of oxidative olig

96 transfecting mutant and wild-type cDNA into HeLa cells, and transfection showed ligand-independent c

97 model proteins was demonstrated on cultured HeLa cells, and two clinically-relevant markers of demen

98 , exhibited substantial cytotoxicity towards HeLa cells, and was a highly sensitive substrate of the

99 er, the two techniques did not agree for the HeLa cells, and we postulate potential reasons for this.

100 mber information for over 8700 proteins from HeLa cells, approaching comprehensive coverage.

101 microtubules, metaphase spindles in MDCK and HeLa cells are not randomly positioned along their x-z d

102 NF764 on the glucocorticoid receptor (GR) in HeLa cells as a model system.

103 specifically accumulate in the ER of living HeLa cells, as imaged by confocal laser scanning microsc

104 Using cell extracts from HeLa cells, as well as transfection of luciferase replic

105 All strains adhered to HeLa cells at low densities, and cytotoxic effects were

106 In HeLa cells, at 200 pmol ASO (with PA:LFN-GAL4), 5.4 +/-

107 treatment had similar effects on irradiated HeLa cells, blocking the formation of RPA and Rad51 foci

108 n E, as lipid peroxidation was suppressed in HeLa cells both under basal conditions and in the presen

109 Furthermore, in HeLa cells, both mutations caused a depletion of hGle1 a

110 uicide gene effectively induces apoptosis of HeLa cells but does not activate fibroblasts to secrete

111 d fatty acids are powerful CPP activators in HeLa cells but not in model membranes.

112 HFFs), normal oral keratinocytes (NOKs), and HeLa cells but not in U2OS cells.

113 expected, SRSF2 and SRSF5 shuttle poorly in HeLa cells but surprisingly display considerable shuttli

114 d for the productive infection of HEK293 and HeLa cells by AAV2, whereas NS4 is sufficient for viral

115 on the microtubule network was visualized in HeLa cells by immunofluorescence microscopy using Bimole

116 f transient modulation of TFEB expression in HeLa cells by measuring the cytosolic Ca(2+) response af

117 mechanistically the ICWs elicited in single HeLa cells by the tandem bubble-induced jetting flow in

118 Furthermore, knockdown of IQGAP1 in THP1 and HeLa cells causes significantly more IFN-beta production

119 In the late stage, HeLa cells change from proliferating to migratory.

120 We found that HeLa cell chromatin-associated nascent pre-mRNA (CA-RNA)

121 ty-purified MRP ribonucleoprotein (RNP) from HeLa cells cleaves the human pre-rRNA in vitro at at lea

122 We generated NONO-silenced HeLa cell clones and found that lack of NONO decreased c

123 FLIM images of F2N12S-labeled live HeLa cells confirmed that the plasma membrane was mainly

124 The confocal laser scanning micrographs of HeLa cells confirmed the cell permeability of the PA-1 a

125 d ATP synthase subunit 6 in the cytoplasm of HeLa cells confirms export to the endoplasmic reticulum.

126 an altered expression in RALY-down-regulated HeLa cells, consequently causing impairments in transcri

127 ospitable to implanted cells and showed that HeLa cells could survive for up to a week using this met

128 This phenotype can be replicated in a HeLa cell culture model by siRNA knockdown of AP-5 zeta.

129 s did not elongate lauric acid, but infected HeLa cell cultures elongated laurate to myristate and pa

130 holipids significantly increased in infected HeLa cell cultures.

131 duced the secretion of effector proteins and HeLa cell cytotoxicity.

132 osphatidylcholine (PC) lipids extracted from HeLa cells demonstrated greater sensitivity via precurso

133 Knockdown of CD98hc expression in HeLa cells demonstrated that it is essential for Brucell

134 e anchorage-independent growth capability of HeLa cells depleted of Cat-1 expression.

135 In HeLa cells depleted of clathrin by siRNA, activated PAR4

136 on method, and antitumor efficacy toward the HeLa cell-derived tumor spheroids in vitro.

137 HeLa cells did not elongate lauric acid, but infected He

138 In HeLa cells, each pore localised to the ER and caused Ca(

139 de of action, peroxide treatment of parental HeLa cells elevated phospho-Met levels whereas antioxida

140 al tubular epithelial cells, 293T cells, and HeLa cells enabled the infection of these cells; exposur

141 In addition, LIMCH1-depleted HeLa cells exhibited a decrease in the number of actin s

142 DNA fiber track assays with HeLa cells exposed to hydroxyurea demonstrated that Tim

143 COS-7 and HeLa cells express ArgBP2 (by Western analysis), but exp

144 emonstrated that when gap junction-deficient HeLa cells expressed the N14K and D50N mutants, they und

145 illustrate the approach using the example of HeLa cells expressing paxillin-EGFP to visualize focal a

146 Confocal microscopy of HeLa cells expressing recombinant UPF5086 proteins revea

147 Using membranes from HeLa cells expressing SERT and intact rat basophilic leu

148 Similar results were obtained in HeLa cells expressing the ALS-causing FUS R495X NLS muta

149 ion of phosphopeptides from serum digest and HeLa cell extract.

150 ants, nonfat milk, egg yolk, human serum and HeLa cell extract.

151 ing electrophoretic mobility shift assays in HeLa cell extracts, we show that OA treatment disrupts p

152 ive supercoils in ICL-containing plasmids in HeLa cell extracts.

153 pplied this methodology to RNA isolated from HeLa cells for the quantification of alternatively splic

154 Although knocking down Cat-1 prevents HeLa cells from forming colonies in soft agar, when paxi

155 the ability to label differentially necrotic HeLa cells from living cells.

156 ontinuous isolation of fluorescently labeled HeLa cells from unlabeled cells at a throughput of appro

157 ty, as well as lower rates of cell growth in HeLa cells, further supporting a role for cell prolifera

158 When testisin and PAR-2 are co-expressed in HeLa cells, GPI-anchored testisin specifically releases

159 rotein in Vero cell-grown virions but not in HeLa cell-grown virions, suggesting a difference in G-pr

160 ree (SF)-MRP1] expressed in either HEK293 or HeLa cells had low Km and Vmax values for As(GS)3, simil

161 ting to knockout five autophagy receptors in HeLa cells, here we show that two receptors previously l

162 n can be produced at the leading edge of the HeLa cell in standoff distance Sd </= 30 mum, driven by

163 age, drug release (via heat), and killing of HeLa cells in culture is investigated.

164 r 1 (N-CoR) that accumulated in synchronized HeLa cells in late G2 phase and mitosis.

165 At 20-50 nM, 33 and 44 arrested >80% of HeLa cells in the G2/M phase of the cell cycle, with sta

166 ins as single fluorescent spots in the ER of HeLa cells in which expression of endogenous SMSr was ab

167 eabilization studies of SINC-GFP-transfected HeLa cells indicate that SINC targets the inner nuclear

168 r changes upon photodynamic treatment of the HeLa cells indicated that the studied photosensitizers i

169 ant in WI-38 diploid fibroblasts and weak in HeLa cells, indicating profound differences in the regul

170 (IC50) values that were less than 20 muM in HeLa cells, indicating that these compounds represent a

171 t that acute expression of oncogenic KRAS in HeLa cells induces mitotic delay and defects in chromoso

172 encoding a proteolytically resistant form in HeLa cells inhibited activation of the NF-kappaB pathway

173 erexpressing SK2 causes a 2-fold increase in HeLa cell invasion.

174 in AOA1-mutant fibroblasts and APTX-depleted Hela cells is caused by decreased expression of SDHA and

175 g this construct, detecting Zn(2+) in living HeLa cells is demonstrated.

176 w that the ICP0-independent loss of IFI16 in HeLa cells is dependent in part on the activity of the v

177 polymerized actin cytoskeleton (F-actin) in HeLa cells is disorganized by NHERF1, whereas actin prot

178 demonstrate that DOCK5 recruitment to FAs in Hela cells is restricted by GIT2, an established regulat

179 Using a chemically-fixed HeLa cell labeled with fluorescent core-shell nanopartic

180 show that knocking down Cat-1 expression in HeLa cells leads to a reduction in Akt activation, which

181 Overexpression of SET-Nup214 in HeLa cells leads to the formation of similar nuclear bod

182 Expression of this mutant motor in HeLa cells led to a dramatic reorganization of cortical

183 We developed a transduced HeLa cell line (HeLa-E8) stably expressing CDHR3-Y529 th

184 exerted cytotoxic activity only against the HeLa cell line among several others (HeLa, MCF7, HT-29 a

185 Using a knockout HeLa cell line generated by CRISPR/Cas9, we provide func

186 Using an engineered HeLa cell line that expresses a green fluorescent protei

187 e molecular function of A6, we established a HeLa cell line that inducibly expressed VACV-A6, which a

188 tool to study these mechanisms, we generated HeLa cell lines that express a fusion protein termed NLS

189 To test this idea, transformed HeLa cell lines were created with fluorescent cargos (mC

190 ts were able to inhibit the proliferation of HeLa cell lines.

191 in hamster (CHO), monkey (COS7), and human (HeLa) cell lines.

192 aging and quantification are demonstrated in HeLa cells loaded with nanosensors and their responsiven

193 In HeLa cells, LpdA generated PA at vesicles and the plasma

194 e) and TSLP receptor-knockout mice with sham HeLa cell lysate or RV.

195 ome-wide protein interactions in E. coli and HeLa cell lysates, respectively, identifying 1,158 and 3

196 ase HPLC fractionation of Chlamydia-infected HeLa cell lysates.

197 Similarly, PDGFRalpha overexpression in HeLa cells markedly increased the levels of HCMV gene ex

198 l human fibroblast and nitrous oxide-treated HeLa cell models.

199 5-7 % of the telomeres at a time in S-phase HeLa cells; no nucleolar localization is detected.

200 s studies, we reported that fractionation of HeLa cell nuclear extracts on glycerol gradients reveale

201 o produce three-dimensional snapshots of the HeLa cell nuclear periphery.

202 ere measured for the methylation of isolated HeLa cell nucleosomes by NSD2.

203 Here, we systematically depleted HeLa cells of the BiP co-chaperones by siRNA-mediated ge

204 tored by PDGFRalpha overexpression in either HeLa cells or TAg-immortalized fibroblasts, suggesting a

205 In HeLa cells, our SirReal-based PROTAC induced isotype-sel

206 DiI-SiR, we visualized filopodia dynamics in HeLa cells over 25 min at 0.5 s temporal resolution, and

207 h-resolution 3D images showing MOF uptake by HeLa cells over a 24 h period.

208 HRV replication was significantly reduced in HeLa cells overexpressing wild-type and mutant forms of

209 chip) analysis showed that DHTS treatment of HeLa cells paradoxically enriched HuR binding to mRNAs w

210 tion, and subsequent interaction with single HeLa cells patterned on fibronectin-coated islands (32 x

211 y were strongly suppressed in L6 myotubes or HeLa cells preincubated with LOA.

212 Moreover, we found that untransfected HeLa cells produce and release sufficient amounts of NAR

213 ow that knocking down paxillin expression in HeLa cells promotes their ability to form colonies in so

214 treatment had similar effects on irradiated HeLa cells, promoting loss of previously assembled Rad51

215 hesion and Wnt-signaling pathways, was among HeLa cell proteins coimmunoprecipitated by antibodies ag

216 In the HeLa cell proteome data set, MAGIC processed over a thou

217 Applied to HeLa cells, RBDmap uncovered 1,174 RNA-binding sites in

218 We identified 1,174 binding sites within 529 HeLa cell RBPs, discovering numerous RNA-binding domains

219 for biological investigation by imaging live HeLa cells, red blood cells, and neurons.

220 Knocking out KAT7 in HeLa cells reduced centromeric CENP-A assembly.

221 In addition, although HeLa cells regulate cyclin C in a manner similar to MEF

222 s eliminated while cell uptake in HEK293 and HeLa cells remained high, which improved the overall cha

223 populations ( approximately 10(7) cells) of HeLa cells require Kif15 to survive K5I treatment.

224 TBK1 activation in HeLa cells requires OPTN and NDP52 and OPTN ubiquitin ch

225 ters during mitosis in C. elegans zygotes or HeLa cells, respectively.

226 aluate thousands of TSG-drug combinations in HeLa cells, resulting in networks of conserved synthetic

227 Functional studies of Cx26 in HeLa cells revealed co-expression of Cx26-Asp50Asn and w

228 expressing the two MTMR2 protein isoforms in HeLa cells revealed that both localize to nuclear puncta

229 method for expressing uptake transporters in HeLa cells revealed that OATP1A2, but not OATP1B1 or OAT

230 alysis and mapping of nascent transcripts in HeLa cells revealed that sequences at reverse start site

231 escence microscopy of LC3-GFP-overexpressing HeLa cells, revealed lower autophagic activity in cells

232 proteotoxic challenges in C. elegans and in HeLa cells revealing conservation of redox homeostasis.

233 N-terminal fragment of NleE (NleE(34-52)) in HeLa cells showed competitive inhibition of wild type Nl

234 multicolor live cell imaging experiments in HeLa cells showed high selectivity of probe 1 for mitoch

235 HDAC10 overexpression in HeLa cells stimulates cellular DNA MMR activity, whereas

236 In single HeLa cells, stronger adhesion to the substrate directed

237 ficantly impacted CRD-BP binding to mRNAs in HeLa cells, suggesting that the dynamics of the CRD-BP-m

238 that MOP 7 is 10-fold more cytotoxic toward HeLa cells than equimolar quantities of DOX prodrug 2.

239 o be readily reversible, they demonstrate in HeLa cells that once RNA is released from chromatin, the

240 When expressed in HEK293 or stable HeLa cells, the 3 mutated netrin-1 proteins were almost

241 induced apoptosis upon ectopic expression in HeLa cells, the percentage of infected macrophages under

242 In human cervical carcinoma HeLa cells, the RNA-binding protein HuR, which enhances

243 resistant viruses can be easily selected in HeLa cells, they do not emerge in Vero cells, in spite t

244 We also loaded these nanoprobes into live HeLa cells through endocytosis, and then monitored chang

245 TORC1, increased ER-mitochondria coupling in HeLa cells to a similar extent as did tunicamycin.

246 AURKB, NEK1, TTK, and WEE1 causes simulated HeLa cells to accumulate in the M phase.

247 to modulate Ca(2+) binding and to sensitize HeLa cells to ATP-induced Ca(2+) release.

248 Exposure of HeLa cells to Cu(PyBD).SO4 (IC50 = 10 muM) results in a

249 Simultaneous live cell imaging using HeLa cells to investigate the intracellular concentratio

250 In addition, the response of HeLa cells to OX-induced cytotoxicity proved to be slowe

251 super-resolution method was applied to live HeLa cells to resolve NPs and provided remarkable sub-di

252 The proteome of HeLa cells transduced with eIF5A shRNA was compared with

253 s supported by experimental data obtained in HeLa cells transfected with connexin45, which is among c

254 Further studies in HeLa cells transfected with PAR1 constructs revealed tha

255 HeLa cell transfection with c-Jun and c-Fos increased CD

256 In HeLa cells transiently expressing C2GnT-M-GFP, knockdown

257 compared, with both peptide standards and a HeLa cell tryptic digest.

258 yperthermia efforts to kill cancer cells and hela cells under 800 nm laser irradiation.

259 ced photodynamic inhibition efficacy against Hela cells under a broad spectrum of light illuminations

260 he probe efficiently images Au(3+) in living HeLa cells under a fluorescence microscope.

261 e of production of lipid peroxyl radicals in HeLa cells under basal conditions is 33 nM/h within the

262 Results in HeLa cells under purine-depleted conditions demonstrated

263 QD-based FRET sensor microinjected into live HeLa cells upon extracellular exposure to bisulfide.

264 using sucrose gradient sedimentation and in HeLa cells using fluorescence correlation spectroscopy.

265 Gag and the transmembrane proteins in T and HeLa cells using quantitative two-color superresolution

266 luorinated gamma-CF2-apg PNAs in NIH 3T3 and HeLa cells was 2-3-fold higher compared to that of nonfl

267 uptake of fluorinated homooligomeric PNAs by HeLa cells was as facile as that of nonfluorinated PNA.

268 rk, further research using H9e hydrogel with HeLa cells was carried out considering H9e hydrogel's in

269 The uptake of the GNPs in HeLa cells was either achieved via incubation or transfe

270 The time-dependent effect of DOX on HeLa cells was monitored and found to have a delayed ons

271 tingly, ectopically expressed nuclear AID in HeLa cells was preferentially found in the early S phase

272 By shRNA-mediated NDUFB11 knockdown in HeLa cells, we demonstrate that NDUFB11 is essential for

273 Using siRNA in HeLa cells, we found that reducing endogenous mOGT expre

274 Using this method in HeLa cells, we have observed polyethylenimine/siRNA poly

275 g through all cortically expressed miRNAs in HeLa cells, we show that several members of the miR-34/4

276 Using single cell analysis in HeLa cells, we show that the CDC20-MAD2 complex is cell

277 In a previous study with HeLa cells, we showed that ER-to-mitochondria Ca(2+) tra

278 Here, we show that depletion of Naa50 in HeLa cells weakens the interaction between cohesin and i

279 n virus-cell junction of HPV18 integrated in HeLa cells were also present in some samples.

280 t surface and surface-associated proteins in HeLa cells were identified; of these, 27 were significan

281 The proliferations of Caco-2, MCF-7 and HELA cells were more inhibited when treated with the WSE

282 d with GFP and two other fluorescent dyes in HeLa cells were resolved with an image quality that is c

283 In the cell culture experiment, HeLa cells were simultaneously embedded in the H9e hydro

284 trate the high accumulation of nanorods into HeLa cells whereas viability analysis supports their low

285 BST-2-positive HepG2 hepatoma cells but not HeLa cells, whereas Vpu failed to efficiently inhibit BS

286 ed and almost exclusively cytoplasmic in the HeLa cells; whereas the WT-MIP was stable dispersed thro

287 and triggers caspase-dependent apoptosis in HeLa cells, which are more sensitive to inhibition by 1

288 tory light chain (MRLC) diphosphorylation in HeLa cells, which was restored by reexpression of small

289 Both mutant fibroblasts and HeLa cells with 60% PiC loss showed a less interconnecte

290 Furthermore, pre-treatment of HeLa cells with [Ru(dppz)2(PIP)](2+) before external bea

291 However, following infection of HEK293 and HeLa cells with AAV2 virions, HBoV1 NS2 (but not NS4), N

292 In HeLa cells with cBIN1 overexpression, knockdown of CHMP4

293 Treatment of HeLa cells with fluorescently labeled PMO chimeras demon

294 egrating image-based phenotypic screening in HeLa cells with high-resolution untargeted metabolomics

295 First, infection of HeLa cells with human rhinovirus (HRV) induced the phosp

296 Treatment of HeLa cells with MLN4924 or expression of a dominant nega

297 ChIP and reporter assays in HeLa cells with monoallelic CD177 expression showed that

298 proteins in the abortive infection of human HeLa cells with the poxvirus strain NYVAC, for which an

299 an individual expressing these variants and HeLa cells with varying degrees of PiC depletion, PiC lo

300 xpression studies, it was determined that in HeLa cells ZBTB33 directly occupies the promoters of cyc