1 ssified Comamonadaceae, Cloacibacterium, and
Helicobacter.
2 ease in the abundance of the bacterial genus
Helicobacter.
3 sociations among infection with liver fluke,
Helicobacter and hepatobiliary fibrosis.
4 sthorchis viverrini serves as a reservoir of
Helicobacter and implicate Helicobacter in pathogenesis
5 was enriched in oxygen-tolerant taxa (e.g.,
Helicobacter and Treponema), while the lumenal microbiot
6 colitis and colon cancer when infected with
Helicobacter bilis (H. bilis).
7 In this model system, the pathobiont
Helicobacter bilis instigates disease following sub-path
8 She was found to have
Helicobacter canis bacteremia.
9 ial as a supportive therapy during and after
Helicobacter eradication therapy.
10 Additionally,
Helicobacter,
Faecalibacterium, and Lactobacillus levels
11 pe (WT) and APRIL Tg mice were infected with
Helicobacter felis and Helicobacter pylori and compared
12 (-/-) and c-Rel(-/-) mice were infected with
Helicobacter felis for 6 weeks or 12 months.
13 n of mice with either Helicobacter pylori or
Helicobacter felis.
14 xpress gastrin and IL-8Tg mice infected with
Helicobacter felis.
15 ight for the first time that arginase of all
Helicobacter gastric pathogens utilizes a unique non-cat
16 wo residues exclusively in arginase of other
Helicobacter gastric pathogens, which may have similar f
17 th critical residues in the homolog of other
Helicobacter gastric pathogens.
18 Helicobacter (
H.) suis causes gastric pathologies in bot
19 Helicobacter have been detected in human bile and hepato
20 In the absence of IL-10,
Helicobacter hepaticus (Hh) induces colitis.
21 Helicobacter hepaticus is a member of the mouse intestin
22 is an emerging human foodborne pathogen, and
Helicobacter hepaticus is a mouse pathogen; both species
23 c, pathology in C57BL/6 mice inoculated with
Helicobacter hepaticus plus anti-IL-10 receptor (IL-10R)
24 The introduction of
Helicobacter hepaticus to the gut of nonsusceptible mice
25 In this study, we show using
Helicobacter hepaticus-induced intestinal inflammation t
26 Helicobacter hepaticus-infected Rag2(-/-) mice emulate m
27 on-dependent manner following infection with
Helicobacter hepaticus.
28 We show that
Helicobacter htrA is an essential bifunctional gene with
29 as a reservoir of Helicobacter and implicate
Helicobacter in pathogenesis of opisthorchiasis-associat
30 The progression of
Helicobacter-
induced disease to precancerous and cancero
31 Using a mouse model of
Helicobacter-
induced gastric cancer, we assessed the rol
32 The role of MyD88 in
Helicobacter-
induced gastric malignancy is unknown.
33 Lineage tracing in
Helicobacter-
infected Slfn4 reporter mice revealed that
34 on may help reducing tissue damage caused by
Helicobacter infection in both humans and pigs, highligh
35 development and progression to cancer during
Helicobacter infection.
36 r development of drugs to specifically treat
Helicobacter infections.
37 A hypothesis that a "
Helicobacter-
like" process causes PC justified this pilo
38 Two
Helicobacter mutant strains (katA(H56A) and katA(Y339A))
39 xis BAFF/Th17 exists in chronic gastritis of
Helicobacter(+)
patients and that its presence strictly
40 Our results show that the mucosa from
Helicobacter(+)
patients with chronic gastritis is enric
41 as the two cytokines are weakly expressed in
Helicobacter(-)
patients with chronic gastritis; moreove
42 oborated using two different PGTs; PglC from
Helicobacter pullorum and WecA from Thermatoga maritima.
43 Helicobacter pullorum is an emerging human foodborne pat
44 Helicobacter pullorum, a bacterium initially isolated fr
45 Empiric triple treatments for
Helicobacter pylori (H. pylori) are increasingly unsucce
46 We investigated whether
Helicobacter pylori (H. pylori) CagA contributes to the
47 Helicobacter pylori (H. pylori) infection and excessive
48 er mortality due to therapy resistance, with
Helicobacter pylori (H. pylori) infection being a major
49 Helicobacter pylori (H. pylori) infection is strongly as
50 The role
Helicobacter Pylori (H. pylori) infection plays in the a
51 rictions to endoscopy and high prevalence of
Helicobacter pylori (H. pylori) infection.
52 Helicobacter pylori (H. pylori) is a species of bacteria
53 Helicobacter pylori (H. pylori) is the strongest identif
54 Helicobacter pylori (H. pylori) is the strongest known r
55 cal studies indicated that colonization with
Helicobacter pylori (H. pylori) may affect body mass ind
56 -proliferation was reported in patients with
Helicobacter pylori (H. pylori)-infected gastric mucosa
57 ve rapid urease test may be reused to detect
Helicobacter pylori (H. pylori).
58 r's disease, primary open-angle glaucoma and
Helicobacter pylori (H.pylori) infection in all possible
59 f of the world's population is infected with
Helicobacter pylori (H.pylori), a bacterium shown to be
60 Helicobacter pylori (HP) and Epstein-Barr virus (EBV) ha
61 long-term complete remission after frontline
Helicobacter pylori (HP) eradication (HPE).
62 We evaluated the effect of
Helicobacter pylori (HP) eradication on p53, cyclin D1 e
63 Helicobacter pylori (HP) infection is present in about 5
64 Helicobacter pylori (Hp) strains that carry the cag type
65 bination of serum pepsinogens(PGs), IgG anti-
Helicobacter pylori (HpAb), and osteopontin (OPN) can be
66 alpha-Carbonic anhydrase of
Helicobacter pylori (HpalphaCA) plays an important role
67 acterized the binding of parS and Spo0J from
Helicobacter pylori (HpSpo0J) and solved the crystal str
68 mologs from Campylobacter coli (R.CcoLI) and
Helicobacter pylori (R.HpyAXII) and demonstrated their D
69 The
Helicobacter pylori adhesin BabA binds mucosal ABO/Le(b)
70 l. (2016) perform structural analyses of the
Helicobacter pylori adhesin BabA to determine how the ba
71 ator of M1 macrophage activation during both
Helicobacter pylori and Citrobacter rodentium infection.
72 ce were infected with Helicobacter felis and
Helicobacter pylori and compared with noninfected animal
73 One example is
Helicobacter pylori and gastric cancer.
74 Colonization of the human stomach by
Helicobacter pylori and its role in causing gastric canc
75 -d-cholesteryl glucopyranoside (alphaCAG) of
Helicobacter pylori and the corresponding galactose anal
76 Although
Helicobacter pylori and use of non-steroidal anti-inflam
77 enocarcinoma and seropositivity to different
Helicobacter pylori antigens using multiplex serology ha
78 Helicobacter pylori arginase, a bimetallic enzyme, is cr
79 Previous characterization of the analogous
Helicobacter pylori Asn-transamidosome revealed that it
80 Expression of the
Helicobacter pylori blood group antigen binding adhesin
81 tors for the gram negative gastric bacterium
Helicobacter pylori but only for a few this has unequivo
82 mach biopsies, and validated the presence of
Helicobacter pylori by quantitative PCR.
83 Heterogeneity at the
Helicobacter pylori cagA gene promoter region has been l
84 ation of particular polymorphisms within the
Helicobacter pylori CagL hypervariable motif (CagLHM) in
85 ave recently found that the gastric pathogen
Helicobacter pylori can activate gastric stem cells and
86 : Despite inducing an inflammatory response,
Helicobacter pylori can persist in the gastric mucosa fo
87 tly associated with sex, breast-feeding, and
Helicobacter pylori carriership.
88 We report on the protonation state of
Helicobacter pylori catalase compound II.
89 Helicobacter pylori causes numerous alterations in gastr
90 ution crystal structure of dCACHE LBD of the
Helicobacter pylori chemoreceptor TlpC.
91 Helicobacter pylori chronic infection is associated with
92 Helicobacter pylori coinfection in human immunodeficienc
93 Helicobacter pylori colonizes the human stomach and conf
94 Helicobacter pylori colonizes the human stomach and incr
95 Helicobacter pylori confers protection against the anaph
96 We surveyed national
Helicobacter pylori diagnostic testing practices and dia
97 We found that
Helicobacter pylori DNA can be detected in human stool s
98 The
Helicobacter pylori energy sensor TlpD determines tactic
99 Helicobacter pylori eradication rates in Portugal are de
100 to the HobA-interacting surface of DnaA from
Helicobacter pylori even though HobA is an activator of
101 Helicobacter pylori exhibits a high level of intraspecie
102 Interestingly,
Helicobacter pylori flagellin triggered robust Nlrc4 pho
103 s homologs from Agrobacterium, Brucella, and
Helicobacter pylori form heterodimers.
104 antified the expression of a large number of
Helicobacter pylori genes and found high expression of g
105 More than 50
Helicobacter pylori genes are predicted to encode outer
106 Helicobacter pylori genetic diversity is known to be inf
107 reened, for the first time to our knowledge,
Helicobacter pylori GML-associated strains for their cap
108 Helicobacter pylori GroES (HpGroES), a potent immunogen,
109 helical shape of the human stomach pathogen
Helicobacter pylori has been suggested to provide mechan
110 lpha-D-glucopyranosides (alphaCPG) unique to
Helicobacter pylori have been achieved.
111 ges that occur during chronic infection with
Helicobacter pylori have been analysed, but little is kn
112 o investigate the frequency of cagA-positive
Helicobacter pylori in Mexican patients with gastric pat
113 evalence of primary antibiotic resistance in
Helicobacter pylori in the Asia-Pacific region.
114 ions, particularly infections resulting from
Helicobacter pylori in the gastric tract.
115 Helicobacter pylori incites a futile inflammatory respon
116 Helicobacter pylori induces the antiapoptotic protein my
117 A total of 200
Helicobacter pylori infected patients were retrospective
118 wheat allergy (WA), coeliac disease (CD) and
Helicobacter pylori infection (HP).
119 y, small intestinal bacterial overgrowth and
Helicobacter pylori infection affect motor fluctuations
120 Current guidelines recommend testing for
Helicobacter pylori infection among users of low-dose as
121 An inverse relation between
Helicobacter pylori infection and allergic disease has b
122 icting data regarding an association between
Helicobacter pylori infection and iron deficiency anemia
123 We examined the association between
Helicobacter pylori infection and the immune response fo
124 Both the prevalence of
Helicobacter pylori infection and the incidence of gastr
125 ing clarithromycin as a model antibiotic and
Helicobacter pylori infection as a model disease.
126 Helicobacter pylori infection causes gastric cancer, the
127 cing identified more IM patients with active
Helicobacter pylori infection compared with histopatholo
128 Helicobacter pylori infection contributes to the develop
129 ence from developed countries indicates that
Helicobacter pylori infection correlates with a reduced
130 Eradication of
Helicobacter pylori infection has been reported to reduc
131 After
Helicobacter pylori infection in humans, gastric epithel
132 5 Ser-536 phosphorylation also occurs during
Helicobacter pylori infection in macrophages and gastric
133 h may indicate an ethiopathogenesis role for
Helicobacter pylori infection in this disease.
134 Helicobacter pylori infection induces chronic gastric in
135 Persistent
Helicobacter pylori infection induces chronic inflammati
136 Helicobacter pylori infection is a risk factor for the d
137 Helicobacter pylori infection is characterized by chroni
138 Among other risk factors,
Helicobacter pylori infection is considered the main dri
139 Helicobacter pylori infection is implicated in the aetio
140 Helicobacter pylori infection is increasingly difficult
141 Helicobacter pylori infection is marked by a vast preval
142 Helicobacter pylori infection is the leading cause for p
143 Helicobacter pylori infection is the main risk factor fo
144 Helicobacter pylori infection is the major cause of gast
145 Helicobacter pylori infection is the most important risk
146 Helicobacter pylori infection not only induces gastric i
147 Helicobacter pylori infection of the stomach is related
148 Helicobacter pylori infection triggers chronic inflammat
149 Chronic
Helicobacter pylori infection triggers neoplastic transf
150 Helicobacter pylori infection was associated with decrea
151 ce of peptic ulcers, strategies to eradicate
Helicobacter pylori infection, and prophylaxis against u
152 egulation of iNOS has been observed in human
Helicobacter pylori infection, but the cellular sources
153 tric submucosal vessels in a murine model of
Helicobacter pylori infection.
154 's population and are largely due to chronic
Helicobacter pylori infection.
155 c adenocarcinoma is strongly associated with
Helicobacter pylori infection; however, most infected pe
156 ication rates of standard triple therapy for
Helicobacter pylori infections have decreased in recent
157 While
Helicobacter pylori infects over 50% of the world's popu
158 Helicobacter pylori infects the human stomach and causes
159 Helicobacter pylori infects the stomachs of one in two h
160 Helicobacter pylori inhabits the gastric mucosa where it
161 CagA is a virulence factor that
Helicobacter pylori inject into gastric epithelial cells
162 The gastric pathogen
Helicobacter pylori interacts intimately with the gastri
163 Furthermore, we quantify the invasion of
Helicobacter pylori into the glands of the mouse stomach
164 Flagellar biogenesis in
Helicobacter pylori involves the coordinated expression
165 Helicobacter pylori is a bacterial pathogen associated w
166 The bacterial pathogen
Helicobacter pylori is a constituent of the normal gastr
167 Helicobacter pylori is a Gram-negative bacterium that co
168 Helicobacter pylori is a Gram-negative bacterium that co
169 Helicobacter pylori is a human-specific pathogen that ch
170 The gastric pathogen
Helicobacter pylori is a major cause of acute chronic ga
171 Helicobacter pylori is a spiral-shaped Gram-negative bac
172 Helicobacter pylori is a successful pathogen of the huma
173 Helicobacter pylori is an important human pathogen assoc
174 ytotoxin associated gene A (CagA) protein of
Helicobacter pylori is associated with increased virulen
175 Infection with
Helicobacter pylori is associated with severe digestive
176 emotaxis receptor mRNA of the human pathogen
Helicobacter pylori is directly targeted by a small RNA
177 Helicobacter pylori is one of the most common bacterial
178 Helicobacter pylori is one of the most controversial bac
179 The stomach bacterium
Helicobacter pylori is one of the most prevalent human p
180 of diseases elicited by the gastric pathogen
Helicobacter pylori is partially determined by the effec
181 BACKGROUND & AIMS:
Helicobacter pylori is remarkable for its genetic variat
182 The genome of
Helicobacter pylori is remarkable for its large number o
183 Helicobacter pylori is the etiologic agent of a series o
184 Infection with the gram-negative bacterium
Helicobacter pylori is the most prevalent chronic bacter
185 Helicobacter pylori is the principal cause of gastric ca
186 Helicobacter pylori is the strongest risk factor for gas
187 Helicobacter pylori is the strongest risk factor for gas
188 Helicobacter pylori is the strongest risk factor for the
189 Helicobacter pylori lacks a gene encoding a homologue of
190 tructure of a soluble variant of full-length
Helicobacter pylori MotB in which the plug helix was eng
191 In a
Helicobacter pylori mouse infection model, PMN infiltrat
192 The
Helicobacter pylori MTAN (HpMTAN) hydrolyzes 6-amino-6-d
193 The gastric pathogen
Helicobacter pylori must combat chronic acid and oxidati
194 Helicobacter pylori neutrophil-activating protein (HP-NA
195 was induced by infection of mice with either
Helicobacter pylori or Helicobacter felis.
196 The
Helicobacter pylori phase variable gene modH, typified b
197 The gastric pathogen
Helicobacter pylori possesses a highly active urease to
198 X-ray crystal structures of the
Helicobacter pylori protein CagL revealed that RGD motif
199 panel to assess whether seropositivity to 15
Helicobacter pylori proteins was associated with subsequ
200 The pathogen
Helicobacter pylori requires two nickel-containing enzym
201 Helicobacter pylori secretes a pore-forming VacA toxin t
202 , P = .13); 20.0% of the cohort had positive
Helicobacter pylori serology (16 vs 2 in the CG, P = .00
203 Helicobacter pylori seropositivity was defined as those
204 Helicobacter pylori specifically colonizes the human gas
205 Helicobacter pylori status was tested using a stool anti
206 ogical and molecular features including age,
Helicobacter pylori status, tumor site, patient survival
207 Helicobacter pylori stimulates or inhibits acid secretio
208 Helicobacter pylori stimulates or inhibits depending upo
209 Most
Helicobacter pylori strains express the BabA adhesin, wh
210 Carriage of
Helicobacter pylori strains producing more active (s1/i1
211 and gastric cancer are caused most often by
Helicobacter pylori strains that harbor the cag pathogen
212 Helicobacter pylori strains that harbor the oncoprotein
213 nd showed that it plays an important role in
Helicobacter pylori stress tolerance and survival in the
214 Chemotaxis is important for
Helicobacter pylori to colonize the stomach.
215 bA adhesin mediates high-affinity binding of
Helicobacter pylori to the ABO blood group antigen-glyco
216 Adherence of
Helicobacter pylori to the gastric epithelial cell line
217 Adherence of
Helicobacter pylori to the gastric epithelial cell line
218 Adhesion of
Helicobacter pylori to the gastric mucosa is a necessary
219 Helicobacter pylori type IV secretion system injects the
220 The gastric pathogen
Helicobacter pylori undergoes host-mediated oxidant stre
221 The gastric cancer-causing pathogen
Helicobacter pylori up-regulates spermine oxidase (SMOX)
222 as inhibitors of Sporosarcina pasteurii and
Helicobacter pylori ureases.
223 Helicobacter pylori uses natural competence and homologo
224 The human pathogen
Helicobacter pylori uses the host receptor alpha5beta1 i
225 The Epsilonproteobacterium
Helicobacter pylori uses the Raetz pathway to synthesize
226 The gastric pathogen
Helicobacter pylori uses the thioredoxin system to maint
227 The
Helicobacter pylori virulence gene, cagA, and active for
228 of galectin-3 (Gal3) in gastric infection by
Helicobacter pylori We first demonstrated that Gal3 was
229 s at the interface of the bacterial pathogen
Helicobacter pylori with its host.
230 we present two high-resolution structures of
Helicobacter pylori XerH with its recombination site DNA
231 Early studies of
Helicobacter pylori's nutritional requirements alluded t
232 rophage EGFR signaling during infection with
Helicobacter pylori, a bacterial pathogen that causes pe
233 The genome of the gastric pathogen
Helicobacter pylori, a Gram-negative epsilonproteobacter
234 Although all individuals were infected with
Helicobacter pylori, abnormal expression of alpha(1,2)fu
235 cquired infections, clarithromycin-resistant
Helicobacter pylori, and fluoroquinolone-resistant Campy
236 ng for celiac disease, autoimmune gastritis,
Helicobacter pylori, and hereditary forms of IDA is reco
237 forces do, however, appear to play a role in
Helicobacter pylori, and some individual genes in all gr
238 ce of this coincides with the eradication of
Helicobacter pylori, both of which might alter the oesop
239 e in the evolution of the bacterial pathogen
Helicobacter pylori, but its dynamics remain incompletel
240 tantly related species Bacillus subtilis and
Helicobacter pylori, but its role in bacterial chemotaxi
241 (Mycoplasma pneumoniae, Treponema pallidum,
Helicobacter pylori, Campylobacter jejuni, Synechocystis
242 ld's population is chronically infected with
Helicobacter pylori, causing gastritis, gastric ulcers a
243 monas vaginalis, and the bacterial pathogens
Helicobacter pylori, Clostridium difficile, and Bacteroi
244 Blood was collected for
Helicobacter pylori, HIV serology, gastrin-17, and pepsi
245 , in some cases such as the gastric pathogen
Helicobacter pylori, HtrA is secreted where it cleaves t
246 % of the world's population is infected with
Helicobacter pylori, leading to chronic inflammation, wh
247 omavirus, herpes virus) and bacterial (e.g.,
Helicobacter pylori, pneumonia) diseases, and autoimmune
248 s, including norovirus, Campylobacter fetus,
Helicobacter pylori, Salmonella enterica, and Giardia la
249 Helicobacter pylori, the causative agent of gastric canc
250 Helicobacter pylori, the dominant member of the human ga
251 Helicobacter pylori, the main cause of peptic ulcer dise
252 cronutrients modulate gene expression within
Helicobacter pylori, the strongest identified risk facto
253 tic distribution of human pathogens, such as
Helicobacter pylori, thereby demonstrating the potential
254 oteins in the bacterial chemotaxis system of
Helicobacter pylori, which requires two nonredundant cou
255 as being involved in the pathophysiology of
Helicobacter pylori-associated diseases, the role of oth
256 n chronic inflammatory conditions, including
Helicobacter pylori-associated gastritis, where its prod
257 that shares similarities to the etiology of
Helicobacter pylori-associated intestinal-type gastric a
258 he polymorphic CagA toxin is associated with
Helicobacter pylori-induced disease.
259 , we investigated the BAFF/Th17 responses in
Helicobacter pylori-induced gastritis in humans.
260 , gastric immunopathology was accelerated in
Helicobacter pylori-infected Gkn2 knockout mice and was
261 proposed that the accompanying microbiota in
Helicobacter pylori-infected individuals might affect di
262 We performed a study of 24 healthy,
Helicobacter pylori-negative volunteers with a small WC
263 link is provided by the association between
Helicobacter pylori-positive gastritis and gastric MALT
264 re commonly used as a first-line therapy for
Helicobacter pylori-positive patients; however, resistan
265 s has been questioned since the discovery of
helicobacter pylori.
266 bacteria such as the human gastric pathogen
Helicobacter pylori.
267 dy the epithelial response to infection with
Helicobacter pylori.
268 main protein from the human gastric pathogen
Helicobacter pylori.
269 uman papilloma virus, hepatitis C virus, and
Helicobacter pylori.
270 was described as a chemotaxis attractant for
Helicobacter pylori.
271 ence factor secreted by the gastric pathogen
Helicobacter pylori.
272 uman papilloma virus, hepatitis C virus, and
Helicobacter pylori.
273 se is infection by a gram-negative bacterium
Helicobacter pylori.
274 tion also occurs in a homologous operon from
Helicobacter pylori.
275 infection with the human bacterial pathogen
Helicobacter pylori.
276 s associated with tetracycline resistance in
Helicobacter pylori.
277 demic and epidemic typhus, trench fever, and
Helicobacter pylori.
278 cytotoxin-associated gene A (CagA)-positive
Helicobacter pylori.
279 rgy and infection with the gastric bacterium
Helicobacter pylori.
280 ainst SK from Mycobacterium tuberculosis and
Helicobacter pylori.
281 nic atrophic gastritis due to infection with
Helicobacter pylori; it might be a precursor to intestin
282 ined in this study are of great interest for
Helicobacter-
related diseases and the development of nov
283 on of MyD88-deficient (Myd88(-/-)) mice with
Helicobacter resulted in early and rapid advancement to
284 own, whereby oxidant-stressed (HOCl-exposed)
Helicobacter retained viability even upon extracellular
285 se-negative, fusiform, novel bacterium named
Helicobacter saguini was isolated from the intestines an
286 This model using a nonhuman primate
Helicobacter sp. can be used to study the pathogenic pot
287 Helicobacter sp. was detected in 69% of feces or intesti
288 rulence factors found in other enterohepatic
helicobacter species (EHS) and H. pylori These include f
289 oyl-ACP methyl ester esterase present in the
Helicobacter species and their occurrence only in H. pyl
290 Enterohepatic
Helicobacter species are associated with several digesti
291 boratory studies, it remains unclear whether
Helicobacter species contribute to these cancers in huma
292 t of gastric lymphoid infiltrates induced by
Helicobacter species in vivo.
293 The CdtB of these
Helicobacter species induced nuclear factor kappaB nucle
294 FP-based phylogenetic trees of seven gastric
Helicobacter species matched those obtained by analysis
295 We believe that APRIL Tg mice infected by
Helicobacter species may represent a novel animal model
296 We present a study of the infection by
Helicobacter species of transgenic (Tg) C57BL6 mice, ect
297 Despite evidence that
Helicobacter species promote gallstone formation and hep
298 spensable for Mycobacterium, Salmonella, and
Helicobacter species.
299 ated bacteria, such as Dysgonomonas spp. and
Helicobacter spp., was profoundly lower in Rb3/Rd-treate
300 monocyte-derived macrophages in vitro, after
Helicobacter stimulation.