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1 ssified Comamonadaceae, Cloacibacterium, and Helicobacter.
2 ease in the abundance of the bacterial genus Helicobacter.
3 sociations among infection with liver fluke, Helicobacter and hepatobiliary fibrosis.
4 sthorchis viverrini serves as a reservoir of Helicobacter and implicate Helicobacter in pathogenesis
5  was enriched in oxygen-tolerant taxa (e.g., Helicobacter and Treponema), while the lumenal microbiot
6  colitis and colon cancer when infected with Helicobacter bilis (H. bilis).
7         In this model system, the pathobiont Helicobacter bilis instigates disease following sub-path
8                        She was found to have Helicobacter canis bacteremia.
9 ial as a supportive therapy during and after Helicobacter eradication therapy.
10                                Additionally, Helicobacter, Faecalibacterium, and Lactobacillus levels
11 pe (WT) and APRIL Tg mice were infected with Helicobacter felis and Helicobacter pylori and compared
12 (-/-) and c-Rel(-/-) mice were infected with Helicobacter felis for 6 weeks or 12 months.
13 n of mice with either Helicobacter pylori or Helicobacter felis.
14 xpress gastrin and IL-8Tg mice infected with Helicobacter felis.
15 ight for the first time that arginase of all Helicobacter gastric pathogens utilizes a unique non-cat
16 wo residues exclusively in arginase of other Helicobacter gastric pathogens, which may have similar f
17 th critical residues in the homolog of other Helicobacter gastric pathogens.
18                                              Helicobacter (H.) suis causes gastric pathologies in bot
19                                              Helicobacter have been detected in human bile and hepato
20                     In the absence of IL-10, Helicobacter hepaticus (Hh) induces colitis.
21                                              Helicobacter hepaticus is a member of the mouse intestin
22 is an emerging human foodborne pathogen, and Helicobacter hepaticus is a mouse pathogen; both species
23 c, pathology in C57BL/6 mice inoculated with Helicobacter hepaticus plus anti-IL-10 receptor (IL-10R)
24                          The introduction of Helicobacter hepaticus to the gut of nonsusceptible mice
25                 In this study, we show using Helicobacter hepaticus-induced intestinal inflammation t
26                                              Helicobacter hepaticus-infected Rag2(-/-) mice emulate m
27 on-dependent manner following infection with Helicobacter hepaticus.
28                                 We show that Helicobacter htrA is an essential bifunctional gene with
29 as a reservoir of Helicobacter and implicate Helicobacter in pathogenesis of opisthorchiasis-associat
30                           The progression of Helicobacter-induced disease to precancerous and cancero
31                       Using a mouse model of Helicobacter-induced gastric cancer, we assessed the rol
32                         The role of MyD88 in Helicobacter-induced gastric malignancy is unknown.
33                           Lineage tracing in Helicobacter-infected Slfn4 reporter mice revealed that
34 on may help reducing tissue damage caused by Helicobacter infection in both humans and pigs, highligh
35 development and progression to cancer during Helicobacter infection.
36 r development of drugs to specifically treat Helicobacter infections.
37                         A hypothesis that a "Helicobacter-like" process causes PC justified this pilo
38                                          Two Helicobacter mutant strains (katA(H56A) and katA(Y339A))
39 xis BAFF/Th17 exists in chronic gastritis of Helicobacter(+) patients and that its presence strictly
40        Our results show that the mucosa from Helicobacter(+) patients with chronic gastritis is enric
41 as the two cytokines are weakly expressed in Helicobacter(-) patients with chronic gastritis; moreove
42 oborated using two different PGTs; PglC from Helicobacter pullorum and WecA from Thermatoga maritima.
43                                              Helicobacter pullorum is an emerging human foodborne pat
44                                              Helicobacter pullorum, a bacterium initially isolated fr
45                Empiric triple treatments for Helicobacter pylori (H. pylori) are increasingly unsucce
46                      We investigated whether Helicobacter pylori (H. pylori) CagA contributes to the
47                                              Helicobacter pylori (H. pylori) infection and excessive
48 er mortality due to therapy resistance, with Helicobacter pylori (H. pylori) infection being a major
49                                              Helicobacter pylori (H. pylori) infection is strongly as
50                                     The role Helicobacter Pylori (H. pylori) infection plays in the a
51 rictions to endoscopy and high prevalence of Helicobacter pylori (H. pylori) infection.
52                                              Helicobacter pylori (H. pylori) is a species of bacteria
53                                              Helicobacter pylori (H. pylori) is the strongest identif
54                                              Helicobacter pylori (H. pylori) is the strongest known r
55 cal studies indicated that colonization with Helicobacter pylori (H. pylori) may affect body mass ind
56 -proliferation was reported in patients with Helicobacter pylori (H. pylori)-infected gastric mucosa
57 ve rapid urease test may be reused to detect Helicobacter pylori (H. pylori).
58 r's disease, primary open-angle glaucoma and Helicobacter pylori (H.pylori) infection in all possible
59 f of the world's population is infected with Helicobacter pylori (H.pylori), a bacterium shown to be
60                                              Helicobacter pylori (HP) and Epstein-Barr virus (EBV) ha
61 long-term complete remission after frontline Helicobacter pylori (HP) eradication (HPE).
62                   We evaluated the effect of Helicobacter pylori (HP) eradication on p53, cyclin D1 e
63                                              Helicobacter pylori (HP) infection is present in about 5
64                                              Helicobacter pylori (Hp) strains that carry the cag type
65 bination of serum pepsinogens(PGs), IgG anti-Helicobacter pylori (HpAb), and osteopontin (OPN) can be
66                  alpha-Carbonic anhydrase of Helicobacter pylori (HpalphaCA) plays an important role
67 acterized the binding of parS and Spo0J from Helicobacter pylori (HpSpo0J) and solved the crystal str
68 mologs from Campylobacter coli (R.CcoLI) and Helicobacter pylori (R.HpyAXII) and demonstrated their D
69                                          The Helicobacter pylori adhesin BabA binds mucosal ABO/Le(b)
70 l. (2016) perform structural analyses of the Helicobacter pylori adhesin BabA to determine how the ba
71 ator of M1 macrophage activation during both Helicobacter pylori and Citrobacter rodentium infection.
72 ce were infected with Helicobacter felis and Helicobacter pylori and compared with noninfected animal
73                               One example is Helicobacter pylori and gastric cancer.
74         Colonization of the human stomach by Helicobacter pylori and its role in causing gastric canc
75 -d-cholesteryl glucopyranoside (alphaCAG) of Helicobacter pylori and the corresponding galactose anal
76                                     Although Helicobacter pylori and use of non-steroidal anti-inflam
77 enocarcinoma and seropositivity to different Helicobacter pylori antigens using multiplex serology ha
78                                              Helicobacter pylori arginase, a bimetallic enzyme, is cr
79   Previous characterization of the analogous Helicobacter pylori Asn-transamidosome revealed that it
80                            Expression of the Helicobacter pylori blood group antigen binding adhesin
81 tors for the gram negative gastric bacterium Helicobacter pylori but only for a few this has unequivo
82 mach biopsies, and validated the presence of Helicobacter pylori by quantitative PCR.
83                         Heterogeneity at the Helicobacter pylori cagA gene promoter region has been l
84 ation of particular polymorphisms within the Helicobacter pylori CagL hypervariable motif (CagLHM) in
85 ave recently found that the gastric pathogen Helicobacter pylori can activate gastric stem cells and
86 : Despite inducing an inflammatory response, Helicobacter pylori can persist in the gastric mucosa fo
87 tly associated with sex, breast-feeding, and Helicobacter pylori carriership.
88        We report on the protonation state of Helicobacter pylori catalase compound II.
89                                              Helicobacter pylori causes numerous alterations in gastr
90 ution crystal structure of dCACHE LBD of the Helicobacter pylori chemoreceptor TlpC.
91                                              Helicobacter pylori chronic infection is associated with
92                                              Helicobacter pylori coinfection in human immunodeficienc
93                                              Helicobacter pylori colonizes the human stomach and conf
94                                              Helicobacter pylori colonizes the human stomach and incr
95                                              Helicobacter pylori confers protection against the anaph
96                         We surveyed national Helicobacter pylori diagnostic testing practices and dia
97                                We found that Helicobacter pylori DNA can be detected in human stool s
98                                          The Helicobacter pylori energy sensor TlpD determines tactic
99                                              Helicobacter pylori eradication rates in Portugal are de
100 to the HobA-interacting surface of DnaA from Helicobacter pylori even though HobA is an activator of
101                                              Helicobacter pylori exhibits a high level of intraspecie
102                               Interestingly, Helicobacter pylori flagellin triggered robust Nlrc4 pho
103 s homologs from Agrobacterium, Brucella, and Helicobacter pylori form heterodimers.
104 antified the expression of a large number of Helicobacter pylori genes and found high expression of g
105                                 More than 50 Helicobacter pylori genes are predicted to encode outer
106                                              Helicobacter pylori genetic diversity is known to be inf
107 reened, for the first time to our knowledge, Helicobacter pylori GML-associated strains for their cap
108                                              Helicobacter pylori GroES (HpGroES), a potent immunogen,
109  helical shape of the human stomach pathogen Helicobacter pylori has been suggested to provide mechan
110 lpha-D-glucopyranosides (alphaCPG) unique to Helicobacter pylori have been achieved.
111 ges that occur during chronic infection with Helicobacter pylori have been analysed, but little is kn
112 o investigate the frequency of cagA-positive Helicobacter pylori in Mexican patients with gastric pat
113 evalence of primary antibiotic resistance in Helicobacter pylori in the Asia-Pacific region.
114 ions, particularly infections resulting from Helicobacter pylori in the gastric tract.
115                                              Helicobacter pylori incites a futile inflammatory respon
116                                              Helicobacter pylori induces the antiapoptotic protein my
117                               A total of 200 Helicobacter pylori infected patients were retrospective
118 wheat allergy (WA), coeliac disease (CD) and Helicobacter pylori infection (HP).
119 y, small intestinal bacterial overgrowth and Helicobacter pylori infection affect motor fluctuations
120     Current guidelines recommend testing for Helicobacter pylori infection among users of low-dose as
121                  An inverse relation between Helicobacter pylori infection and allergic disease has b
122 icting data regarding an association between Helicobacter pylori infection and iron deficiency anemia
123          We examined the association between Helicobacter pylori infection and the immune response fo
124                       Both the prevalence of Helicobacter pylori infection and the incidence of gastr
125 ing clarithromycin as a model antibiotic and Helicobacter pylori infection as a model disease.
126                                              Helicobacter pylori infection causes gastric cancer, the
127 cing identified more IM patients with active Helicobacter pylori infection compared with histopatholo
128                                              Helicobacter pylori infection contributes to the develop
129 ence from developed countries indicates that Helicobacter pylori infection correlates with a reduced
130                               Eradication of Helicobacter pylori infection has been reported to reduc
131                                        After Helicobacter pylori infection in humans, gastric epithel
132 5 Ser-536 phosphorylation also occurs during Helicobacter pylori infection in macrophages and gastric
133 h may indicate an ethiopathogenesis role for Helicobacter pylori infection in this disease.
134                                              Helicobacter pylori infection induces chronic gastric in
135                                   Persistent Helicobacter pylori infection induces chronic inflammati
136                                              Helicobacter pylori infection is a risk factor for the d
137                                              Helicobacter pylori infection is characterized by chroni
138                    Among other risk factors, Helicobacter pylori infection is considered the main dri
139                                              Helicobacter pylori infection is implicated in the aetio
140                                              Helicobacter pylori infection is increasingly difficult
141                                              Helicobacter pylori infection is marked by a vast preval
142                                              Helicobacter pylori infection is the leading cause for p
143                                              Helicobacter pylori infection is the main risk factor fo
144                                              Helicobacter pylori infection is the major cause of gast
145                                              Helicobacter pylori infection is the most important risk
146                                              Helicobacter pylori infection not only induces gastric i
147                                              Helicobacter pylori infection of the stomach is related
148                                              Helicobacter pylori infection triggers chronic inflammat
149                                      Chronic Helicobacter pylori infection triggers neoplastic transf
150                                              Helicobacter pylori infection was associated with decrea
151 ce of peptic ulcers, strategies to eradicate Helicobacter pylori infection, and prophylaxis against u
152 egulation of iNOS has been observed in human Helicobacter pylori infection, but the cellular sources
153 tric submucosal vessels in a murine model of Helicobacter pylori infection.
154 's population and are largely due to chronic Helicobacter pylori infection.
155 c adenocarcinoma is strongly associated with Helicobacter pylori infection; however, most infected pe
156 ication rates of standard triple therapy for Helicobacter pylori infections have decreased in recent
157                                        While Helicobacter pylori infects over 50% of the world's popu
158                                              Helicobacter pylori infects the human stomach and causes
159                                              Helicobacter pylori infects the stomachs of one in two h
160                                              Helicobacter pylori inhabits the gastric mucosa where it
161              CagA is a virulence factor that Helicobacter pylori inject into gastric epithelial cells
162                         The gastric pathogen Helicobacter pylori interacts intimately with the gastri
163     Furthermore, we quantify the invasion of Helicobacter pylori into the glands of the mouse stomach
164                      Flagellar biogenesis in Helicobacter pylori involves the coordinated expression
165                                              Helicobacter pylori is a bacterial pathogen associated w
166                       The bacterial pathogen Helicobacter pylori is a constituent of the normal gastr
167                                              Helicobacter pylori is a Gram-negative bacterium that co
168                                              Helicobacter pylori is a Gram-negative bacterium that co
169                                              Helicobacter pylori is a human-specific pathogen that ch
170                         The gastric pathogen Helicobacter pylori is a major cause of acute chronic ga
171                                              Helicobacter pylori is a spiral-shaped Gram-negative bac
172                                              Helicobacter pylori is a successful pathogen of the huma
173                                              Helicobacter pylori is an important human pathogen assoc
174 ytotoxin associated gene A (CagA) protein of Helicobacter pylori is associated with increased virulen
175                               Infection with Helicobacter pylori is associated with severe digestive
176 emotaxis receptor mRNA of the human pathogen Helicobacter pylori is directly targeted by a small RNA
177                                              Helicobacter pylori is one of the most common bacterial
178                                              Helicobacter pylori is one of the most controversial bac
179                        The stomach bacterium Helicobacter pylori is one of the most prevalent human p
180 of diseases elicited by the gastric pathogen Helicobacter pylori is partially determined by the effec
181                           BACKGROUND & AIMS: Helicobacter pylori is remarkable for its genetic variat
182                                The genome of Helicobacter pylori is remarkable for its large number o
183                                              Helicobacter pylori is the etiologic agent of a series o
184   Infection with the gram-negative bacterium Helicobacter pylori is the most prevalent chronic bacter
185                                              Helicobacter pylori is the principal cause of gastric ca
186                                              Helicobacter pylori is the strongest risk factor for gas
187                                              Helicobacter pylori is the strongest risk factor for gas
188                                              Helicobacter pylori is the strongest risk factor for the
189                                              Helicobacter pylori lacks a gene encoding a homologue of
190 tructure of a soluble variant of full-length Helicobacter pylori MotB in which the plug helix was eng
191                                         In a Helicobacter pylori mouse infection model, PMN infiltrat
192                                          The Helicobacter pylori MTAN (HpMTAN) hydrolyzes 6-amino-6-d
193                         The gastric pathogen Helicobacter pylori must combat chronic acid and oxidati
194                                              Helicobacter pylori neutrophil-activating protein (HP-NA
195 was induced by infection of mice with either Helicobacter pylori or Helicobacter felis.
196                                          The Helicobacter pylori phase variable gene modH, typified b
197                         The gastric pathogen Helicobacter pylori possesses a highly active urease to
198              X-ray crystal structures of the Helicobacter pylori protein CagL revealed that RGD motif
199 panel to assess whether seropositivity to 15 Helicobacter pylori proteins was associated with subsequ
200                                 The pathogen Helicobacter pylori requires two nickel-containing enzym
201                                              Helicobacter pylori secretes a pore-forming VacA toxin t
202 , P = .13); 20.0% of the cohort had positive Helicobacter pylori serology (16 vs 2 in the CG, P = .00
203                                              Helicobacter pylori seropositivity was defined as those
204                                              Helicobacter pylori specifically colonizes the human gas
205                                              Helicobacter pylori status was tested using a stool anti
206 ogical and molecular features including age, Helicobacter pylori status, tumor site, patient survival
207                                              Helicobacter pylori stimulates or inhibits acid secretio
208                                              Helicobacter pylori stimulates or inhibits depending upo
209                                         Most Helicobacter pylori strains express the BabA adhesin, wh
210                                  Carriage of Helicobacter pylori strains producing more active (s1/i1
211  and gastric cancer are caused most often by Helicobacter pylori strains that harbor the cag pathogen
212                                              Helicobacter pylori strains that harbor the oncoprotein
213 nd showed that it plays an important role in Helicobacter pylori stress tolerance and survival in the
214                  Chemotaxis is important for Helicobacter pylori to colonize the stomach.
215 bA adhesin mediates high-affinity binding of Helicobacter pylori to the ABO blood group antigen-glyco
216                                 Adherence of Helicobacter pylori to the gastric epithelial cell line
217                                 Adherence of Helicobacter pylori to the gastric epithelial cell line
218                                  Adhesion of Helicobacter pylori to the gastric mucosa is a necessary
219                                              Helicobacter pylori type IV secretion system injects the
220                         The gastric pathogen Helicobacter pylori undergoes host-mediated oxidant stre
221          The gastric cancer-causing pathogen Helicobacter pylori up-regulates spermine oxidase (SMOX)
222  as inhibitors of Sporosarcina pasteurii and Helicobacter pylori ureases.
223                                              Helicobacter pylori uses natural competence and homologo
224                           The human pathogen Helicobacter pylori uses the host receptor alpha5beta1 i
225                   The Epsilonproteobacterium Helicobacter pylori uses the Raetz pathway to synthesize
226                         The gastric pathogen Helicobacter pylori uses the thioredoxin system to maint
227                                          The Helicobacter pylori virulence gene, cagA, and active for
228 of galectin-3 (Gal3) in gastric infection by Helicobacter pylori We first demonstrated that Gal3 was
229 s at the interface of the bacterial pathogen Helicobacter pylori with its host.
230 we present two high-resolution structures of Helicobacter pylori XerH with its recombination site DNA
231                             Early studies of Helicobacter pylori's nutritional requirements alluded t
232 rophage EGFR signaling during infection with Helicobacter pylori, a bacterial pathogen that causes pe
233           The genome of the gastric pathogen Helicobacter pylori, a Gram-negative epsilonproteobacter
234  Although all individuals were infected with Helicobacter pylori, abnormal expression of alpha(1,2)fu
235 cquired infections, clarithromycin-resistant Helicobacter pylori, and fluoroquinolone-resistant Campy
236 ng for celiac disease, autoimmune gastritis, Helicobacter pylori, and hereditary forms of IDA is reco
237 forces do, however, appear to play a role in Helicobacter pylori, and some individual genes in all gr
238 ce of this coincides with the eradication of Helicobacter pylori, both of which might alter the oesop
239 e in the evolution of the bacterial pathogen Helicobacter pylori, but its dynamics remain incompletel
240 tantly related species Bacillus subtilis and Helicobacter pylori, but its role in bacterial chemotaxi
241  (Mycoplasma pneumoniae, Treponema pallidum, Helicobacter pylori, Campylobacter jejuni, Synechocystis
242 ld's population is chronically infected with Helicobacter pylori, causing gastritis, gastric ulcers a
243 monas vaginalis, and the bacterial pathogens Helicobacter pylori, Clostridium difficile, and Bacteroi
244                      Blood was collected for Helicobacter pylori, HIV serology, gastrin-17, and pepsi
245 , in some cases such as the gastric pathogen Helicobacter pylori, HtrA is secreted where it cleaves t
246 % of the world's population is infected with Helicobacter pylori, leading to chronic inflammation, wh
247 omavirus, herpes virus) and bacterial (e.g., Helicobacter pylori, pneumonia) diseases, and autoimmune
248 s, including norovirus, Campylobacter fetus, Helicobacter pylori, Salmonella enterica, and Giardia la
249                                              Helicobacter pylori, the causative agent of gastric canc
250                                              Helicobacter pylori, the dominant member of the human ga
251                                              Helicobacter pylori, the main cause of peptic ulcer dise
252 cronutrients modulate gene expression within Helicobacter pylori, the strongest identified risk facto
253 tic distribution of human pathogens, such as Helicobacter pylori, thereby demonstrating the potential
254 oteins in the bacterial chemotaxis system of Helicobacter pylori, which requires two nonredundant cou
255  as being involved in the pathophysiology of Helicobacter pylori-associated diseases, the role of oth
256 n chronic inflammatory conditions, including Helicobacter pylori-associated gastritis, where its prod
257  that shares similarities to the etiology of Helicobacter pylori-associated intestinal-type gastric a
258 he polymorphic CagA toxin is associated with Helicobacter pylori-induced disease.
259 , we investigated the BAFF/Th17 responses in Helicobacter pylori-induced gastritis in humans.
260 , gastric immunopathology was accelerated in Helicobacter pylori-infected Gkn2 knockout mice and was
261 proposed that the accompanying microbiota in Helicobacter pylori-infected individuals might affect di
262          We performed a study of 24 healthy, Helicobacter pylori-negative volunteers with a small WC
263  link is provided by the association between Helicobacter pylori-positive gastritis and gastric MALT
264 re commonly used as a first-line therapy for Helicobacter pylori-positive patients; however, resistan
265 s has been questioned since the discovery of helicobacter pylori.
266  bacteria such as the human gastric pathogen Helicobacter pylori.
267 dy the epithelial response to infection with Helicobacter pylori.
268 main protein from the human gastric pathogen Helicobacter pylori.
269 uman papilloma virus, hepatitis C virus, and Helicobacter pylori.
270 was described as a chemotaxis attractant for Helicobacter pylori.
271 ence factor secreted by the gastric pathogen Helicobacter pylori.
272 uman papilloma virus, hepatitis C virus, and Helicobacter pylori.
273 se is infection by a gram-negative bacterium Helicobacter pylori.
274 tion also occurs in a homologous operon from Helicobacter pylori.
275  infection with the human bacterial pathogen Helicobacter pylori.
276 s associated with tetracycline resistance in Helicobacter pylori.
277 demic and epidemic typhus, trench fever, and Helicobacter pylori.
278  cytotoxin-associated gene A (CagA)-positive Helicobacter pylori.
279 rgy and infection with the gastric bacterium Helicobacter pylori.
280 ainst SK from Mycobacterium tuberculosis and Helicobacter pylori.
281 nic atrophic gastritis due to infection with Helicobacter pylori; it might be a precursor to intestin
282 ined in this study are of great interest for Helicobacter-related diseases and the development of nov
283 on of MyD88-deficient (Myd88(-/-)) mice with Helicobacter resulted in early and rapid advancement to
284 own, whereby oxidant-stressed (HOCl-exposed) Helicobacter retained viability even upon extracellular
285 se-negative, fusiform, novel bacterium named Helicobacter saguini was isolated from the intestines an
286          This model using a nonhuman primate Helicobacter sp. can be used to study the pathogenic pot
287                                              Helicobacter sp. was detected in 69% of feces or intesti
288 rulence factors found in other enterohepatic helicobacter species (EHS) and H. pylori These include f
289 oyl-ACP methyl ester esterase present in the Helicobacter species and their occurrence only in H. pyl
290                                Enterohepatic Helicobacter species are associated with several digesti
291 boratory studies, it remains unclear whether Helicobacter species contribute to these cancers in huma
292 t of gastric lymphoid infiltrates induced by Helicobacter species in vivo.
293                            The CdtB of these Helicobacter species induced nuclear factor kappaB nucle
294 FP-based phylogenetic trees of seven gastric Helicobacter species matched those obtained by analysis
295    We believe that APRIL Tg mice infected by Helicobacter species may represent a novel animal model
296       We present a study of the infection by Helicobacter species of transgenic (Tg) C57BL6 mice, ect
297                        Despite evidence that Helicobacter species promote gallstone formation and hep
298 spensable for Mycobacterium, Salmonella, and Helicobacter species.
299 ated bacteria, such as Dysgonomonas spp. and Helicobacter spp., was profoundly lower in Rb3/Rd-treate
300 monocyte-derived macrophages in vitro, after Helicobacter stimulation.

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