ライフサイエンスコーパス: Helicobacter pylori

1 ., amino acids) and bacterial stimuli (e.g., Helicobacter pylori).

2 bacteria such as the human gastric pathogen Helicobacter pylori.

3 dy the epithelial response to infection with Helicobacter pylori.

4 main protein from the human gastric pathogen Helicobacter pylori.

5 uman papilloma virus, hepatitis C virus, and Helicobacter pylori.

6 was described as a chemotaxis attractant for Helicobacter pylori.

7 ence factor secreted by the gastric pathogen Helicobacter pylori.

8 uman papilloma virus, hepatitis C virus, and Helicobacter pylori.

9 se is infection by a gram-negative bacterium Helicobacter pylori.

10 s associated with tetracycline resistance in Helicobacter pylori.

11 tion also occurs in a homologous operon from Helicobacter pylori.

12 infection with the human bacterial pathogen Helicobacter pylori.

13 demic and epidemic typhus, trench fever, and Helicobacter pylori.

14 cytotoxin-associated gene A (CagA)-positive Helicobacter pylori.

15 el (Ni) is critical for the pathogenicity of Helicobacter pylori.

16 t ranges, such as the human stomach pathogen Helicobacter pylori.

17 i, Shigella sonnei, Campylobacter jejuni and Helicobacter pylori.

18 at infections caused by the gastric pathogen Helicobacter pylori.

19 nfection with the gastric bacterial pathogen Helicobacter pylori.

20 rgy and infection with the gastric bacterium Helicobacter pylori.

21 ainst SK from Mycobacterium tuberculosis and Helicobacter pylori.

22 s has been questioned since the discovery of helicobacter pylori.

23 hout rejection (group D: acute enteritis, 9; Helicobacter pylori, 1; Streptococcal pharyngitis 1; and

24 rophage EGFR signaling during infection with Helicobacter pylori, a bacterial pathogen that causes pe

25 The genome of the gastric pathogen Helicobacter pylori, a Gram-negative epsilonproteobacter

26 Although all individuals were infected with Helicobacter pylori, abnormal expression of alpha(1,2)fu

27 The Helicobacter pylori adhesin BabA binds mucosal ABO/Le(b)

28 l. (2016) perform structural analyses of the Helicobacter pylori adhesin BabA to determine how the ba

29 crophages mediate the epithelial response to Helicobacter pylori and are involved in the development

30 ely related to QFRs from the human pathogens Helicobacter pylori and Campylobacter jejuni.

31 ator of M1 macrophage activation during both Helicobacter pylori and Citrobacter rodentium infection.

32 ce were infected with Helicobacter felis and Helicobacter pylori and compared with noninfected animal

33 One example is Helicobacter pylori and gastric cancer.

34 Colonization of the human stomach by Helicobacter pylori and its role in causing gastric canc

35 ttachment factors for gastric pathogens like Helicobacter pylori and noroviruses.

36 model explicitly incorporated the impact of Helicobacter pylori and smoking on disease natural histo

37 -d-cholesteryl glucopyranoside (alphaCAG) of Helicobacter pylori and the corresponding galactose anal

38 Although Helicobacter pylori and use of non-steroidal anti-inflam

39 cquired infections, clarithromycin-resistant Helicobacter pylori, and fluoroquinolone-resistant Campy

40 ng for celiac disease, autoimmune gastritis, Helicobacter pylori, and hereditary forms of IDA is reco

41 forces do, however, appear to play a role in Helicobacter pylori, and some individual genes in all gr

42 enocarcinoma and seropositivity to different Helicobacter pylori antigens using multiplex serology ha

43 Helicobacter pylori arginase, a bimetallic enzyme, is cr

44 We explore the role of aconitase (AcnB) in Helicobacter pylori as a posttranscriptional regulator o

45 Previous characterization of the analogous Helicobacter pylori Asn-transamidosome revealed that it

46 as being involved in the pathophysiology of Helicobacter pylori-associated diseases, the role of oth

47 n chronic inflammatory conditions, including Helicobacter pylori-associated gastritis, where its prod

48 that shares similarities to the etiology of Helicobacter pylori-associated intestinal-type gastric a

49 Expression of the Helicobacter pylori blood group antigen binding adhesin

50 ce of this coincides with the eradication of Helicobacter pylori, both of which might alter the oesop

51 tors for the gram negative gastric bacterium Helicobacter pylori but only for a few this has unequivo

52 e, ghrelin, adiponectin and the influence of Helicobacter pylori, but also brings into light new proc

53 e in the evolution of the bacterial pathogen Helicobacter pylori, but its dynamics remain incompletel

54 tantly related species Bacillus subtilis and Helicobacter pylori, but its role in bacterial chemotaxi

55 mach biopsies, and validated the presence of Helicobacter pylori by quantitative PCR.

56 Heterogeneity at the Helicobacter pylori cagA gene promoter region has been l

57 ation of particular polymorphisms within the Helicobacter pylori CagL hypervariable motif (CagLHM) in

58 (Mycoplasma pneumoniae, Treponema pallidum, Helicobacter pylori, Campylobacter jejuni, Synechocystis

59 ave recently found that the gastric pathogen Helicobacter pylori can activate gastric stem cells and

60 : Despite inducing an inflammatory response, Helicobacter pylori can persist in the gastric mucosa fo

61 tly associated with sex, breast-feeding, and Helicobacter pylori carriership.

62 We report on the protonation state of Helicobacter pylori catalase compound II.

63 nization with the gastric bacterial pathogen Helicobacter pylori causes gastritis and predisposes inf

64 Helicobacter pylori causes numerous alterations in gastr

65 ld's population is chronically infected with Helicobacter pylori, causing gastritis, gastric ulcers a

66 ution crystal structure of dCACHE LBD of the Helicobacter pylori chemoreceptor TlpC.

67 Helicobacter pylori chronic infection is associated with

68 monas vaginalis, and the bacterial pathogens Helicobacter pylori, Clostridium difficile, and Bacteroi

69 Helicobacter pylori coinfection in human immunodeficienc

70 Helicobacter pylori colonizes the human stomach and conf

71 Helicobacter pylori colonizes the human stomach and incr

72 Helicobacter pylori confers protection against the anaph

73 We surveyed national Helicobacter pylori diagnostic testing practices and dia

74 We found that Helicobacter pylori DNA can be detected in human stool s

75 The human pathogen Helicobacter pylori employs a diverse collection of oute

76 The human pathogen Helicobacter pylori encodes two GluRS enzymes, with GluR

77 The Helicobacter pylori energy sensor TlpD determines tactic

78 The long-term effectiveness of Helicobacter pylori eradication programs for preventing

79 Helicobacter pylori eradication rates in Portugal are de

80 use and this can be treated empirically with Helicobacter pylori eradication therapy in those that ar

81 Helicobacter pylori establishes lifelong infections of t

82 to the HobA-interacting surface of DnaA from Helicobacter pylori even though HobA is an activator of

83 Helicobacter pylori exhibits a high level of intraspecie

84 Interestingly, Helicobacter pylori flagellin triggered robust Nlrc4 pho

85 s homologs from Agrobacterium, Brucella, and Helicobacter pylori form heterodimers.

86 antified the expression of a large number of Helicobacter pylori genes and found high expression of g

87 More than 50 Helicobacter pylori genes are predicted to encode outer

88 Helicobacter pylori genetic diversity is known to be inf

89 reened, for the first time to our knowledge, Helicobacter pylori GML-associated strains for their cap

90 Helicobacter pylori GroES (HpGroES), a potent immunogen,

91 Empiric triple treatments for Helicobacter pylori (H. pylori) are increasingly unsucce

92 We investigated whether Helicobacter pylori (H. pylori) CagA contributes to the

93 Helicobacter pylori (H. pylori) infection and excessive

94 er mortality due to therapy resistance, with Helicobacter pylori (H. pylori) infection being a major

95 Helicobacter pylori (H. pylori) infection is strongly as

96 The role Helicobacter Pylori (H. pylori) infection plays in the a

97 known to have an extremely low prevalence of Helicobacter pylori (H. pylori) infection, with the hypo

98 rictions to endoscopy and high prevalence of Helicobacter pylori (H. pylori) infection.

99 Helicobacter pylori (H. pylori) is a species of bacteria

100 The treatment for the eradication of Helicobacter pylori (H. pylori) is complex; full effecti

101 Helicobacter pylori (H. pylori) is the strongest identif

102 Helicobacter pylori (H. pylori) is the strongest known r

103 cal studies indicated that colonization with Helicobacter pylori (H. pylori) may affect body mass ind

104 -proliferation was reported in patients with Helicobacter pylori (H. pylori)-infected gastric mucosa

105 ve rapid urease test may be reused to detect Helicobacter pylori (H. pylori).

106 r's disease, primary open-angle glaucoma and Helicobacter pylori (H.pylori) infection in all possible

107 f of the world's population is infected with Helicobacter pylori (H.pylori), a bacterium shown to be

108 helical shape of the human stomach pathogen Helicobacter pylori has been suggested to provide mechan

109 The naturally competent bacterium Helicobacter pylori has highly diverse strain-specific t

110 Helicobacter pylori have become resistant to antimicrobi

111 lpha-D-glucopyranosides (alphaCPG) unique to Helicobacter pylori have been achieved.

112 ges that occur during chronic infection with Helicobacter pylori have been analysed, but little is kn

113 Blood was collected for Helicobacter pylori, HIV serology, gastrin-17, and pepsi

114 s like HIV, respiratory syncytial virus, and Helicobacter pylori; however, none of these products has

115 Helicobacter pylori (HP) and Epstein-Barr virus (EBV) ha

116 explorative study evaluates the efficacy of Helicobacter pylori (HP) eradication (HPE) therapy on ea

117 long-term complete remission after frontline Helicobacter pylori (HP) eradication (HPE).

118 ver, whether they have a higher incidence of Helicobacter pylori (HP) eradication is not known.

119 We evaluated the effect of Helicobacter pylori (HP) eradication on p53, cyclin D1 e

120 Helicobacter pylori (Hp) infection affects one-half of t

121 Helicobacter pylori (HP) infection is present in about 5

122 Helicobacter pylori (Hp) strains that carry the cag type

123 bination of serum pepsinogens(PGs), IgG anti-Helicobacter pylori (HpAb), and osteopontin (OPN) can be

124 alpha-Carbonic anhydrase of Helicobacter pylori (HpalphaCA) plays an important role

125 acterized the binding of parS and Spo0J from Helicobacter pylori (HpSpo0J) and solved the crystal str

126 , in some cases such as the gastric pathogen Helicobacter pylori, HtrA is secreted where it cleaves t

127 o investigate the frequency of cagA-positive Helicobacter pylori in Mexican patients with gastric pat

128 evalence of primary antibiotic resistance in Helicobacter pylori in the Asia-Pacific region.

129 ions, particularly infections resulting from Helicobacter pylori in the gastric tract.

130 Helicobacter pylori incites a futile inflammatory respon

131 he polymorphic CagA toxin is associated with Helicobacter pylori-induced disease.

132 , we investigated the BAFF/Th17 responses in Helicobacter pylori-induced gastritis in humans.

133 Helicobacter pylori induces the antiapoptotic protein my

134 A total of 200 Helicobacter pylori infected patients were retrospective

135 , gastric immunopathology was accelerated in Helicobacter pylori-infected Gkn2 knockout mice and was

136 proposed that the accompanying microbiota in Helicobacter pylori-infected individuals might affect di

137 wheat allergy (WA), coeliac disease (CD) and Helicobacter pylori infection (HP).

138 y, small intestinal bacterial overgrowth and Helicobacter pylori infection affect motor fluctuations

139 Current guidelines recommend testing for Helicobacter pylori infection among users of low-dose as

140 An inverse relation between Helicobacter pylori infection and allergic disease has b

141 We aimed to determine whether Helicobacter pylori infection and CD were associated amo

142 icting data regarding an association between Helicobacter pylori infection and iron deficiency anemia

143 lial cells (GECs) are the primary target for Helicobacter pylori infection and may act as APCs regula

144 We examined the association between Helicobacter pylori infection and the immune response fo

145 We examined the association between Helicobacter pylori infection and the incidence of diarr

146 Both the prevalence of Helicobacter pylori infection and the incidence of gastr

147 ing clarithromycin as a model antibiotic and Helicobacter pylori infection as a model disease.

148 Helicobacter pylori infection causes gastric cancer, the

149 cing identified more IM patients with active Helicobacter pylori infection compared with histopatholo

150 Helicobacter pylori infection contributes to the develop

151 Different disease outcomes of Helicobacter pylori infection correlate with distinct in

152 ence from developed countries indicates that Helicobacter pylori infection correlates with a reduced

153 Strategies to eradicate Helicobacter pylori infection could be improved by suppr

154 Eradication of Helicobacter pylori infection has been reported to reduc

155 After Helicobacter pylori infection in humans, gastric epithel

156 5 Ser-536 phosphorylation also occurs during Helicobacter pylori infection in macrophages and gastric

157 h may indicate an ethiopathogenesis role for Helicobacter pylori infection in this disease.

158 Helicobacter pylori infection induces chronic gastric in

159 Persistent Helicobacter pylori infection induces chronic inflammati

160 Helicobacter pylori infection is a risk factor for the d

161 Helicobacter pylori infection is characterized by chroni

162 Among other risk factors, Helicobacter pylori infection is considered the main dri

163 Helicobacter pylori infection is implicated in the aetio

164 Helicobacter pylori infection is increasingly difficult

165 Helicobacter pylori infection is marked by a vast preval

166 Helicobacter pylori infection is the leading cause for p

167 Helicobacter pylori infection is the main risk factor fo

168 Helicobacter pylori infection is the major cause of gast

169 Helicobacter pylori infection is the most important risk

170 Helicobacter pylori infection not only induces gastric i

171 Helicobacter pylori infection of the stomach is related

172 Helicobacter pylori infection triggers chronic inflammat

173 Chronic Helicobacter pylori infection triggers neoplastic transf

174 Helicobacter pylori infection was associated with decrea

175 of gastric epithelial cells is a hallmark of Helicobacter pylori infection, and altered epithelial ce

176 ce of peptic ulcers, strategies to eradicate Helicobacter pylori infection, and prophylaxis against u

177 egulation of iNOS has been observed in human Helicobacter pylori infection, but the cellular sources

178 with Helicobacter felis to create a model of Helicobacter pylori infection--the most common human chr

179 's population and are largely due to chronic Helicobacter pylori infection.

180 ecimens show evidence of gastric atrophy and Helicobacter pylori infection.

181 tric submucosal vessels in a murine model of Helicobacter pylori infection.

182 c adenocarcinoma is strongly associated with Helicobacter pylori infection; however, most infected pe

183 ication rates of standard triple therapy for Helicobacter pylori infections have decreased in recent

184 Helicobacter pylori infections were more common in T2D p

185 Helicobacter pylori infects over 3 billion people worldw

186 While Helicobacter pylori infects over 50% of the world's popu

187 Helicobacter pylori infects the human stomach and causes

188 Helicobacter pylori infects the stomachs of one in two h

189 Helicobacter pylori inhabits the gastric mucosa where it

190 CagA is a virulence factor that Helicobacter pylori inject into gastric epithelial cells

191 The gastric pathogen Helicobacter pylori interacts intimately with the gastri

192 Furthermore, we quantify the invasion of Helicobacter pylori into the glands of the mouse stomach

193 Flagellar biogenesis in Helicobacter pylori involves the coordinated expression

194 In Helicobacter pylori, iron balance is controlled by the F

195 Helicobacter pylori is a bacterial pathogen associated w

196 Helicobacter pylori is a bacterial pathogen that establi

197 The bacterial pathogen Helicobacter pylori is a constituent of the normal gastr

198 The ferric uptake regulator (Fur) of Helicobacter pylori is a global regulator that is import

199 Helicobacter pylori is a Gram-negative bacterium that co

200 Helicobacter pylori is a Gram-negative bacterium that co

201 Helicobacter pylori is a human-specific pathogen that ch

202 The plasticity region of Helicobacter pylori is a large chromosomal segment inclu

203 The gastric pathogen Helicobacter pylori is a major cause of acute chronic ga

204 Helicobacter pylori is a major cause of gastritis and ga

205 c infection with the Gram-negative bacterium Helicobacter pylori is a major risk factor for the devel

206 stent colonization of the human stomach with Helicobacter pylori is a risk factor for gastric adenoca

207 Colonization of the human stomach with Helicobacter pylori is a risk factor for peptic ulcerati

208 Helicobacter pylori is a spiral-shaped Gram-negative bac

209 Helicobacter pylori is a successful pathogen of the huma

210 Helicobacter pylori is an important human pathogen assoc

211 ytotoxin associated gene A (CagA) protein of Helicobacter pylori is associated with increased virulen

212 Infection with Helicobacter pylori is associated with severe digestive

213 emotaxis receptor mRNA of the human pathogen Helicobacter pylori is directly targeted by a small RNA

214 The helical cell shape of Helicobacter pylori is highly conserved and contributes

215 Helicobacter pylori is one of the most common bacterial

216 Helicobacter pylori is one of the most controversial bac

217 The stomach bacterium Helicobacter pylori is one of the most prevalent human p

218 of diseases elicited by the gastric pathogen Helicobacter pylori is partially determined by the effec

219 BACKGROUND & AIMS: Helicobacter pylori is remarkable for its genetic variat

220 The genome of Helicobacter pylori is remarkable for its large number o

221 Recently, we discovered that HtrA from Helicobacter pylori is secreted and cleaves E-cadherin t

222 Helicobacter pylori is the etiologic agent of a series o

223 Infection with the gram-negative bacterium Helicobacter pylori is the most prevalent chronic bacter

224 Helicobacter pylori is the principal cause of gastric ca

225 Helicobacter pylori is the strongest risk factor for gas

226 Helicobacter pylori is the strongest risk factor for gas

227 Helicobacter pylori is the strongest risk factor for the

228 nfection with the gastric bacterial pathogen Helicobacter pylori is typically contracted in early chi

229 nic atrophic gastritis due to infection with Helicobacter pylori; it might be a precursor to intestin

230 Helicobacter pylori lacks a gene encoding a homologue of

231 % of the world's population is infected with Helicobacter pylori, leading to chronic inflammation, wh

232 tructure of a soluble variant of full-length Helicobacter pylori MotB in which the plug helix was eng

233 In a Helicobacter pylori mouse infection model, PMN infiltrat

234 The Helicobacter pylori MTAN (HpMTAN) hydrolyzes 6-amino-6-d

235 The gastric pathogen Helicobacter pylori must combat chronic acid and oxidati

236 We performed a study of 24 healthy, Helicobacter pylori-negative volunteers with a small WC

237 Helicobacter pylori neutrophil-activating protein (HP-NA

238 was induced by infection of mice with either Helicobacter pylori or Helicobacter felis.

239 The Helicobacter pylori phase variable gene modH, typified b

240 omavirus, herpes virus) and bacterial (e.g., Helicobacter pylori, pneumonia) diseases, and autoimmune

241 link is provided by the association between Helicobacter pylori-positive gastritis and gastric MALT

242 re commonly used as a first-line therapy for Helicobacter pylori-positive patients; however, resistan

243 The gastric pathogen Helicobacter pylori possesses a highly active urease to

244 PCs) frequently occurs during infection with Helicobacter pylori, predisposing patients to cancer.

245 X-ray crystal structures of the Helicobacter pylori protein CagL revealed that RGD motif

246 panel to assess whether seropositivity to 15 Helicobacter pylori proteins was associated with subsequ

247 mologs from Campylobacter coli (R.CcoLI) and Helicobacter pylori (R.HpyAXII) and demonstrated their D

248 The pathogen Helicobacter pylori requires two nickel-containing enzym

249 Early studies of Helicobacter pylori's nutritional requirements alluded t

250 s, including norovirus, Campylobacter fetus, Helicobacter pylori, Salmonella enterica, and Giardia la

251 Helicobacter pylori secretes a pore-forming VacA toxin t

252 , P = .13); 20.0% of the cohort had positive Helicobacter pylori serology (16 vs 2 in the CG, P = .00

253 Helicobacter pylori seropositivity was defined as those

254 Helicobacter pylori seropositivity, especially H. pylori

255 Helicobacter pylori seroprevalence levels in US adults p

256 Helicobacter pylori specifically colonizes the human gas

257 Helicobacter pylori status was tested using a stool anti

258 ogical and molecular features including age, Helicobacter pylori status, tumor site, patient survival

259 Helicobacter pylori stimulates or inhibits acid secretio

260 Helicobacter pylori stimulates or inhibits depending upo

261 Pathogenic Helicobacter pylori strains can selectively activate epi

262 Most Helicobacter pylori strains express the BabA adhesin, wh

263 Chronic infection with Helicobacter pylori strains expressing the bacterial onc

264 Carriage of Helicobacter pylori strains producing more active (s1/i1

265 and gastric cancer are caused most often by Helicobacter pylori strains that harbor the cag pathogen

266 Helicobacter pylori strains that harbor the oncoprotein

267 Virulent Helicobacter pylori strains that specifically activate s

268 usly represented in the genomes of different Helicobacter pylori strains.

269 nd showed that it plays an important role in Helicobacter pylori stress tolerance and survival in the

270 Helicobacter pylori survival in acidic environments reli

271 Helicobacter pylori, the causative agent of gastric canc

272 Helicobacter pylori, the dominant member of the human ga

273 In Helicobacter pylori, the ferric uptake regulator (Fur) h

274 Helicobacter pylori, the main cause of peptic ulcer dise

275 cronutrients modulate gene expression within Helicobacter pylori, the strongest identified risk facto

276 tic distribution of human pathogens, such as Helicobacter pylori, thereby demonstrating the potential

277 Chemotaxis is important for Helicobacter pylori to colonize the stomach.

278 bA adhesin mediates high-affinity binding of Helicobacter pylori to the ABO blood group antigen-glyco

279 Adherence of Helicobacter pylori to the gastric epithelial cell line

280 Adherence of Helicobacter pylori to the gastric epithelial cell line

281 Adhesion of Helicobacter pylori to the gastric mucosa is a necessary

282 The Helicobacter pylori toxin vacuolating cytotoxin (VacA) p

283 Helicobacter pylori type IV secretion system injects the

284 The gastric pathogen Helicobacter pylori undergoes host-mediated oxidant stre

285 The gastric cancer-causing pathogen Helicobacter pylori up-regulates spermine oxidase (SMOX)

286 as inhibitors of Sporosarcina pasteurii and Helicobacter pylori ureases.

287 Helicobacter pylori use host cell integrins to provide a

288 Helicobacter pylori uses natural competence and homologo

289 The human pathogen Helicobacter pylori uses the host receptor alpha5beta1 i

290 The Epsilonproteobacterium Helicobacter pylori uses the Raetz pathway to synthesize

291 The gastric pathogen Helicobacter pylori uses the thioredoxin system to maint

292 Disease due to the gastric pathogen Helicobacter pylori varies in severity from asymptomatic

293 The last pathway makes MTAN essential for Helicobacter pylori viability.

294 The Helicobacter pylori virulence gene, cagA, and active for

295 of galectin-3 (Gal3) in gastric infection by Helicobacter pylori We first demonstrated that Gal3 was

296 been associated with chronic infection with Helicobacter pylori; we investigated the bacteria's pote

297 ory p53 isoforms by the pathogenic bacterium Helicobacter pylori, which is known for its strong assoc

298 oteins in the bacterial chemotaxis system of Helicobacter pylori, which requires two nonredundant cou

299 s at the interface of the bacterial pathogen Helicobacter pylori with its host.

300 we present two high-resolution structures of Helicobacter pylori XerH with its recombination site DNA