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1 tric submucosal vessels in a murine model of Helicobacter pylori infection.
2 en validated, with subsequent eradication of Helicobacter pylori infection.
3 oid tissue type is closely linked to chronic Helicobacter pylori infection.
4 incided with a decrease in the prevalence of Helicobacter pylori infection.
5 in the inflammatory response associated with Helicobacter pylori infection.
6 genetic and environmental factors, including Helicobacter pylori infection.
7 phoid tissue (MALT) lymphoma is dependent on Helicobacter pylori infection.
8 attention as a cause of tissue damage due to Helicobacter pylori infection.
9 a consequence of chronic gastritis caused by Helicobacter pylori infection.
10 nicopathologic entity, often associated with Helicobacter pylori infection.
11 ial serologic assays accurately detect adult Helicobacter pylori infection.
12 in urease vaccine-induced protection against Helicobacter pylori infection.
13 ptic ulcer disease pathogenesis secondary to Helicobacter pylori infection.
14 the expression of which is increased during Helicobacter pylori infection.
15 's population and are largely due to chronic Helicobacter pylori infection.
16 ecimens show evidence of gastric atrophy and Helicobacter pylori infection.
17 2), a gene associated with susceptibility to Helicobacter pylori infection.
18 mation and gastritis but leads to persistent Helicobacter pylori infection.
19 sia (GIN) with 80% prevalence 6 months after Helicobacter pylori infection.
20 istories reflect the overriding influence of Helicobacter pylori infection.
21 ies, and new approaches to the management of Helicobacter pylori infection.
22 d applied to rhesus monkeys with and without Helicobacter pylori infection.
23 by nonsteroidal anti-inflammatory drugs and Helicobacter pylori infection.
24 ve recently been shown to be associated with Helicobacter pylori infections.
26 y, small intestinal bacterial overgrowth and Helicobacter pylori infection affect motor fluctuations
27 role of serologic testing to confirm cure of Helicobacter pylori infection after antimicrobial therap
28 ially housed rhesus monkeys rapidly acquired Helicobacter pylori infection, although the organism was
29 Current guidelines recommend testing for Helicobacter pylori infection among users of low-dose as
33 a new immunoblot assay (Helicoblot 2.1) for Helicobacter pylori infection and CagA and VacA status b
38 e made significant progress in understanding Helicobacter pylori infection and gastric motility in th
39 are needed to clarify the natural history of Helicobacter pylori infection and identify predictors of
40 lass I- and class II-restricted functions in Helicobacter pylori infection and immunity upon oral imm
41 icting data regarding an association between Helicobacter pylori infection and iron deficiency anemia
43 ding the nature of cell-mediated immunity in Helicobacter pylori infection and its role in pathogenes
44 lial cells (GECs) are the primary target for Helicobacter pylori infection and may act as APCs regula
45 ociated lymphoid tissue (MALT) is related to Helicobacter pylori infection and may depend on this inf
46 l and infant plasma vitamin A, reduce infant Helicobacter pylori infection and nasopharyngeal pneumoc
48 ested a link between a high-salt diet during Helicobacter pylori infection and the development of hyp
54 of gastric epithelial cells is a hallmark of Helicobacter pylori infection, and altered epithelial ce
55 eradication, NOD1 contributes to controlling Helicobacter pylori infection, and NOD2 is involved in m
57 ce of peptic ulcers, strategies to eradicate Helicobacter pylori infection, and prophylaxis against u
58 gastric mucosa as a result of long standing Helicobacter pylori infection, and this acquired MALT ma
61 valence rates in developed nations, most new Helicobacter pylori infections are primarily acquired du
63 egulation of iNOS has been observed in human Helicobacter pylori infection, but the cellular sources
73 cing identified more IM patients with active Helicobacter pylori infection compared with histopatholo
76 ence from developed countries indicates that Helicobacter pylori infection correlates with a reduced
79 dard proton-pump inhibitor-based therapy for Helicobacter pylori infection fails in up to one quarter
83 stigated for centuries, the association with Helicobacter pylori infection has been recognized for on
86 of the humoral immune responses of people to Helicobacter pylori infection has facilitated the invest
87 une response of gastric T cells during acute Helicobacter pylori infection has not been previously ch
88 l therapies for the effective eradication of Helicobacter pylori infection have been identified and a
90 ication rates of standard triple therapy for Helicobacter pylori infections have decreased in recent
91 c adenocarcinoma is strongly associated with Helicobacter pylori infection; however, most infected pe
94 en made in understanding the epidemiology of Helicobacter pylori infection in developing countries an
95 acute antibody and T-cell immune response to Helicobacter pylori infection in humans has not been stu
96 lymphoid tissue (MALT) lymphoma linked with Helicobacter pylori infection in humans was observed in
99 5 Ser-536 phosphorylation also occurs during Helicobacter pylori infection in macrophages and gastric
101 level I evidence supporting the treatment of Helicobacter pylori infection in patients with duodenal
102 between water sources and the prevalence of Helicobacter pylori infection in Peruvian children was s
103 ter pylori infection, screening and treating Helicobacter pylori infection in selected transplant pat
105 duced immune mechanisms that protect against Helicobacter pylori infection in the mouse model have no
131 ey (1999-2000), the authors assessed whether Helicobacter pylori infection is associated with iron de
155 gastric inflammatory and immune response in Helicobacter pylori infection may be due to the effect o
168 easured a strong transcriptional response to Helicobacter pylori infection only in this cell type.
170 mHSP65 antibody titers were correlated with Helicobacter pylori infection (P=0.004), which maintaine
172 10 responses and increased susceptibility to Helicobacter pylori infection, phenotypes observed in NO
173 plastic cell lineages in response to chronic Helicobacter pylori infection predisposes to gastric neo
180 immunosuppressed transplant recipients with Helicobacter pylori infection, screening and treating He
182 histologic subtype and anatomic subsite, by Helicobacter pylori infection status, by geographic loca
184 nificantly less effective for eradication of Helicobacter pylori infection than are 5-day concomitant
185 umor tissues were more likely to have active Helicobacter pylori infection than those with stable tum
186 andomised controlled trial of eradication of Helicobacter pylori infection, the association between b
187 ns of household and environmental factors to Helicobacter pylori infection, the authors examined H. p
189 mal inductive sites for immunization against Helicobacter pylori infection, the protective efficacy o
190 with Helicobacter felis to create a model of Helicobacter pylori infection--the most common human chr
194 e presence of chronic atrophic gastritis and Helicobacter pylori infection was determined by measurem
199 (49%) of the 100 patients had biopsy-proved Helicobacter pylori infection with chronic active gastri
200 iological evidence links high-salt diets and Helicobacter pylori infection with increased risk of dev
201 We examined the longitudinal changes of Helicobacter pylori infection within 46 families with ch
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