ライフサイエンスコーパス: Heligmosomoides polygyrus

1 with the gastrointestinal helminth parasite Heligmosomoides polygyrus.

2 onically infected with the helminth parasite Heligmosomoides polygyrus.

3 culated with Nippostrongylus brasiliensis or Heligmosomoides polygyrus.

4 n mice infected with the intestinal helminth Heligmosomoides polygyrus.

5 2 responses required for protection against Heligmosomoides polygyrus.

6 s during infection with the enteric nematode Heligmosomoides polygyrus.

7 those observed in a secondary infection with Heligmosomoides polygyrus.

8 response to the intestinal nematode parasite Heligmosomoides polygyrus.

9 ction with the intestinal nematode parasite, Heligmosomoides polygyrus.

10 onse by infection with the nematode parasite Heligmosomoides polygyrus.

11 nfection of mice with the nematode parasite, Heligmosomoides polygyrus.

12 oral inoculation with the nematode parasite, Heligmosomoides polygyrus.

13 culation of mice with the nematode parasite, Heligmosomoides polygyrus.

14 was evaluated in mice infected earlier with Heligmosomoides polygyrus, a gastrointestinal worm known

15 e 2 immune response following infection with Heligmosomoides polygyrus, a natural murine parasitic ne

16 We colonized mice with Heligmosomoides polygyrus, an intestinal helminth, and a

17 IL-10(-/-) T cell transfer model of colitis, Heligmosomoides polygyrus, an intestinal helminth, preve

18 omologs belonging to the parasitic nematodes Heligmosomoides polygyrus and Heterodera glycines.

19 IL-4/IL-13 during challenge infections with Heligmosomoides polygyrus and Nippostrongylus brasiliens

20 to intestinal nematode parasites, including Heligmosomoides polygyrus and Nippostrongylus brasiliens

21 established by using the intestinal nematode Heligmosomoides polygyrus and S. Typhimurium.

22 oinfection with two natural mouse pathogens, Heligmosomoides polygyrus and Toxoplasma gondii, to inve

23 t chronic infection with the murine helminth Heligmosomoides polygyrus bakeri (Hpb) altered the intes

24 g infection with gastrointestinal helminths (Heligmosomoides polygyrus bakeri and Trichuris muris).

25 Helminths like Heligmosomoides polygyrus bakeri can induce regulatory T

26 leukin-25 (IL-25) in host protection against Heligmosomoides polygyrus bakeri infection in mice.

27 igated the effect of the intestinal nematode Heligmosomoides polygyrus bakeri on Th1 responses to Myc

28 production following infection of mice with Heligmosomoides polygyrus bakeri or Nippostrongylus bras

29 In a murine colitis model, the helminth Heligmosomoides polygyrus bakeri prevents colitis via in

30 In IBD murine models, the helminth Heligmosomoides polygyrus bakeri prevents colitis.

31 ssue-migrating larvae of the murine parasite Heligmosomoides polygyrus bakeri.

32 trointestinal nematodes is commonly known as Heligmosomoides polygyrus bakeri.

33 l nematodes Nippostrongylus brasiliensis and Heligmosomoides polygyrus bakeri.

34 The mouse parasite Heligmosomoides polygyrus can expand the host Treg popul

35 road activation of an antimicrobial program; Heligmosomoides polygyrus caused an increase in the abun

36 Heligmosomoides polygyrus colonization inhibits Th1 and

37 gut, we studied the influence of intestinal Heligmosomoides polygyrus colonization on LPS-induced la

38 Heligmosomoides polygyrus did not change the normal micr

39 Mice infected with the helminth Heligmosomoides polygyrus displayed increased levels of

40 Heligmosomoides polygyrus elevated Th2 cytokine expressi

41 ts from the model mouse intestinal parasite, Heligmosomoides polygyrus (equivalent to 7 mug of protei

42 ermined if exposure to the duodenal helminth Heligmosomoides polygyrus establishes cytokine pathways

43 and shown that coinfection with the helminth Heligmosomoides polygyrus exacerbates colitis induced by

44 hallenge with the strictly enteric helminth, Heligmosomoides polygyrus, GFP-positive innate and adapt

45 Oral infection with the nematode parasite Heligmosomoides polygyrus H. polygyrus is entirely restr

46 ostrongylus brasiliensis (N brasiliensis) or Heligmosomoides polygyrus (H polygyrus) or injected intr

47 ion of intestinal epithelial function during Heligmosomoides polygyrus (Hp) infection was investigate

48 C57BL/6 mice to infection with the helminth Heligmosomoides polygyrus, including TGF-beta signaling,

49 Acute GVHD was induced in helminth (Heligmosomoides polygyrus)-infected or uninfected BALB/c

50 f Th2 cells derive from Foxp3(+) cells after Heligmosomoides polygyrus infection and airway allergy.

51 rasiliensis or were drug-cured of an initial Heligmosomoides polygyrus infection and later reinfected

52 tode infection, we compared the responses to Heligmosomoides polygyrus infection between 2 mouse stra

53 Using influenza virus and Heligmosomoides polygyrus infections, we show that these

54 a normally chronic intestinal infection with Heligmosomoides polygyrus into an infection that was rap

55 Heligmosomoides polygyrus is a strictly murine enteric n

56 ng infection with the gut-dwelling roundworm Heligmosomoides polygyrus is critical for protective imm

57 rodents infected with Trichinella spiralis, Heligmosomoides polygyrus, Nippostronglyus brasiliensis,

58 of strictly enteric helminth infection with Heligmosomoides polygyrus on respiratory syncytial virus

59 fects of infection with a helminth parasite, Heligmosomoides polygyrus, on type 1 diabetes (T1D) in n

60 ice failed to expel the intestinal helminths Heligmosomoides polygyrus or Nippostrongylus brasiliensi

61 fected with the intestinal nematode parasite Heligmosomoides polygyrus prior to infection with S. man

62 tization to OVA or intestinal infection with Heligmosomoides polygyrus Specific Igs and plasmablasts

63 Products secreted by the mouse parasite Heligmosomoides polygyrus suppress type 2 (allergic) imm

64 BALB/c mice received an oral infection of Heligmosomoides polygyrus third-stage larvae, were given

65 Th2 cell-inducing gastrointestinal nematode Heligmosomoides polygyrus to influence experimentally in

66 tion with three distinct helminth parasites, Heligmosomoides polygyrus, Trichuris muris, and Schistos

67 xpulsion of Nippostrongylus brasiliensis and Heligmosomoides polygyrus, which both live in the intest

68 monstrate that the gastrointestinal nematode Heligmosomoides polygyrus, which infects mice, secretes

69 infection with the murine nematode parasite Heligmosomoides polygyrus, which resides in the duodenum