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1 he aetiopathogenesis of Kawasaki disease and Henoch-Schonlein purpura.
2 information about long-term consequences of Henoch-Schonlein purpura.
3 f the most common vasculitides of childhood: Henoch-Schonlein purpura.
4 ficant percentage of patients afflicted with Henoch-Schonlein purpura.
5 g predisposing factors in the development of Henoch-Schonlein purpura.
6 netic susceptibility and the pathogenesis of Henoch-Schonlein purpura.
7 of childhood, including Kawasaki disease and Henoch-Schonlein purpura.
8 usceptibility, pathogenesis and treatment of Henoch-Schonlein purpura.
9 f nephritis, or alter the natural history of Henoch-Schonlein purpura.
11 hers have described several polymorphisms in Henoch-Schonlein purpura and Kawasaki Disease as well as
13 ssive diseases: MEFV in Behcet's disease and Henoch-Schonlein purpura, and A1AT in Wegener's granulom
14 ation, such as IgA nephropathy, Tn syndrome, Henoch-Schonlein purpura, and malignant transformation,
15 e genetic susceptibility and pathogenesis of Henoch-Schonlein purpura, but there are still significan
16 l be able to elucidate the manifestations of Henoch-Schonlein purpura, determine appropriate treatmen
20 of a man with a 5-year history of relapsing Henoch-Schonlein purpura (HSP) and macroscopic polyarter
22 tions and molecular changes occurring during Henoch-Schonlein purpura, including cytokines, and endot
28 e in the pathogenesis of IgA nephropathy and Henoch-Schonlein purpura nephritis has evolved over the
30 sive agents in pediatric IgA nephropathy and Henoch-Schonlein purpura nephritis, recent data indicate
31 to determine the most effective treatment of Henoch-Schonlein purpura, particularly for patients with
32 and existing literature base, The Alder Hey Henoch Schonlein Purpura Pathway was developed, a revise
34 ition is the hallmark of IgA nephropathy and Henoch-Schonlein purpura, the onset of which often follo
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