ライフサイエンスコーパス: Hg

1 Hg concentration and stable isotope data from an inland-

2 Hg-based probes allow the extension of SECM investigatio

3 Hg-based probes are capable of performing amalgamation r

4 CAC=0 and 24 for CAC>100, when SBP 120-139mm Hg).

5 The use of delta(202)Hg and Delta(199)Hg values in SIBER enabled us to estimate Hg isotopic ni

6 Delta(199)Hg was related to Hg levels of fish but we also suggest

7 d (delta(15)NPhe), and Hg isotope (Delta(199)Hg, Delta(201)Hg, delta(202)Hg) data for six species of

8 ent of heavy metals (Cd(2+), Co(2+), Cu(2+), Hg(2+), Ni(2+), and Pb(2+)) from aqueous solution with i

9 he), and Hg isotope (Delta(199)Hg, Delta(201)Hg, delta(202)Hg) data for six species of Hawaiian marin

10 The use of delta(202)Hg and Delta(199)Hg values in SIBER enabled us to estima

11 A consistent negative offset in delta(202)Hg values ( approximately 0.28 per thousand) was observe

12 otope (Delta(199)Hg, Delta(201)Hg, delta(202)Hg) data for six species of Hawaiian marine bottomfish.

13 consumption of 4 being 9:1 in terms of [4]:[Hg(2+)] molar ratio.

14 ous and non-ferrous metals could result in a Hg intake that exceeds the current statutory limits.

15 ir catalytically amplified collisions with a Hg-coated microelectrode used as the tip in the scanning

16 protection against amalgam saturation allows Hg disc-wells to operate safely in highly concentrated e

17 et absorbance (SUVA254)), and Hg(II)-DOM and Hg(II)-DOM-sulfide equilibration times (4-142 h).

18 Clear increases in Hg and Hg/TOC are observed at the end-Triassic extinction horiz

19 lular metabolism, cellular localization, and Hg(II) coordination in aerobically respiring Escherichia

20 C), nitrogen amino acid (delta(15)NPhe), and Hg isotope (Delta(199)Hg, Delta(201)Hg, delta(202)Hg) da

21 /Fs and 16 pesticides whereas Cd, As, Pb and Hg were assayed by ICP-MS.

22 onships between biogeochemical processes and Hg bioaccumulation.

23 cific ultraviolet absorbance (SUVA254)), and Hg(II)-DOM and Hg(II)-DOM-sulfide equilibration times (4

24 Hg) have substantially increased atmospheric Hg levels during the 20th century compared to preindustr

25 be driven by decreasing regional atmospheric Hg emissions although they may be partly counterbalanced

26 ent with the decline in regional atmospheric Hg emissions and water Hg concentrations.

27 transformation), fluorescence and SERS-based Hg(2+)sensor platform in the water.

28 tely 0.28 per thousand) was observed between Hg in the suspended particulate and dissolved phases, an

29 d the history of heavy metal (V, Cu, Zn, Cd, Hg, Tl, Pb, U) pollution in Lake Baikal seals over the p

30 Here we present a comprehensive Hg-deposition mass-balance study, and show that most of

31 Under optimal conditions, Hg(2+) can be detected in a wide linear range from 20aM

32 e (AgNP-GCE) in aqueous solutions containing Hg(2+).

33 sed on a continuous high-resolution ice-core Hg record from the Belukha glacier in the Siberian Altai

34 LDH ratio of 1:5 which can be used to detect Hg(2+) in water by naked eyes.

35 The aptasensor was used for detection Hg(2+) ions from samples of tap waters, carp and saltwat

36 Total dissolved Hg meltwater runoff of 14.3 (+/- 0.7) mg/ha in 2008 and

37 tic tundra is derived from gaseous elemental Hg (Hg(0)) deposition, with only minor contributions fro

38 and 74% of the air emissions were elemental Hg.

39 eral studies have shown that DOM can enhance Hg methylation, especially under sulfidic conditions, wh

40 ces that promote AMDEs, may provide enhanced Hg deposition, reduced Hg emission and, ultimately, an i

41 99)Hg values in SIBER enabled us to estimate Hg isotopic niches, successfully discriminating several

42 and selected substances (C, Cd, Cr, Cu, Fe, Hg, N, Ni, P, Pb, Zn) are developed to characterize this

43 forest soils have a strong sink capacity for Hg, and indicate that the sequestered Hg is bound in soi

44 , we report a novel fabrication protocol for Hg disc-well ultramicroelectrodes (UMEs), which retain a

45 d ECL aptasensor showed high selectivity for Hg(2+) determination compared to other environmentally r

46 importance of boreal forest humus soils for Hg storage and reveals that this pool is likely to persi

47 es of Hg to the Arctic in its oxidized form (Hg(ii)).

48 Hg concentrations consistently derived from Hg(0), suggesting that the Arctic tundra might be a glob

49 Tundra uptake of gaseous Hg(0) leads to high soil Hg concentrations, with Hg mass

50 Gaseous elemental mercury (GEM, Hg) emissions are transformed to divalent reactive Hg (R

51 onfirming that a volcanically induced global Hg cycle perturbation occurred at that time.

52 tundra is derived from gaseous elemental Hg (Hg(0)) deposition, with only minor contributions from th

53 Arctic tundra might be a globally important Hg sink.

54 Clear increases in Hg and Hg/TOC are observed at the end-Triassic extinctio

55 racterize the structural order of mercury in Hg(II)-DOM-sulfide systems for a range of sulfide concen

56 tions, whereas others show that DOM inhibits Hg methylation due to strong Hg-DOM complexation.

57 Inorganic Hg(II) and methylmercury ([CH3Hg(II)](+)) are commonly c

58 A systematic interconversion of EtHg into Hg(II) was obtained for all matrixes reaching values up

59 Divalent mercury ion (Hg(2+)) is one of the most common pollutants in water wi

60 onic processes, but the risk of irreversible Hg amalgam saturation limits their operation to rapid ti

61 Of this liberated Hg, 470 Gg were emitted directly into the atmosphere, an

62 he maximum admissible level in this matrix ([Hg]=1mg/kgwet weight,ww).

63 We measured Hg, major ions, and stable water isotopes from the snowp

64 Mercury (Hg) bioavailability to bacteria in marine systems is the

65 solved organic matter (DOM) affects mercury (Hg) redox reactions and anaerobic microbial methylation

66 o the heavy metals cadmium (Cd) and mercury (Hg) is known to increase the risk of chronic diseases.

67 onments in which inorganic divalent mercury (Hg(II)) is transformed to methylmercury (MeHg) by anaero

68 emissions of the toxic heavy metal mercury (Hg) have substantially increased atmospheric Hg levels d

69 Current understanding of mercury (Hg) dynamics in the Arctic is hampered by a lack of data

70 Environmental regulations on mercury (Hg) emissions and associated ecosystem restoration are c

71 Our study reports the first data on mercury (Hg) isotope composition in marine European fish, for sev

72 activities have led to large-scale mercury (Hg) pollution in the Arctic.

73 51%) for systolic blood pressure and -4.0 mm Hg (95% CI, -5.6 to -2.5; 6 studies; I2 = 17%) for diast

74 g in the treatment group versus 0.5+/-5.0 mm Hg in the control group (P=0.14).

75 n DCT and GAT measurements was -3.3 (2.0) mm Hg (95% CI, 2.9-3.6).

76 /GA percentile was associated with a 1.04-mm Hg decrement in adult systolic BP (95% confidence interv

77 t </=700 cells/mm(3) contributed to a 2.1 mm Hg decrease in IOP.

78 y weight) and oxygenation (273.4 +/- 72.1 mm Hg).

79 h ranges of 12.5-15.5 mm Hg and 11.7-15.1 mm Hg, respectively.

80 ulture negative), 9 with IOP more than 10 mm Hg greater than baseline, 2 with IOP higher than 35 mm H

81 h intraocular pressure (IOP) more than 10 mm Hg greater than baseline; ocular adverse events in the b

82 1.09-1.86), and blood pressure (HR per 10 mm Hg, 0.87; 95% CI, 0.78-0.98), but not prior stroke.

83 +/- 3.2 kg/m(2) ; Child A 92%; HVPG >/=10 mm Hg, 72%).

84 7 times more likely to have an IOP </=10 mm Hg, and patients with a CD4 count </=700 cells/mm(3) wer

85 s in both mPAP (36 +/- 7 versus 47 +/- 10 mm Hg, P < 0.0001) and pulmonary vascular resistance (3.0 +

86 13 times more likely to have an IOP </=10 mm Hg.

87 ed further under bolus resuscitation (-10 mm Hg; p < 0.001) and was lower under bolus resuscitation t

88 ulmonary valve implantation (39 versus 10 mm Hg; P<0.001).

89 atients who died (3.0+/-8 versus 1.7+/-10 mm Hg; P=0.003).

90 patients with a V-wave decrease of >/=11 mm Hg were 3.8x more likely to improve their 6MWT (P=0.05).

91 92] at systolic blood pressure 160 vs 110 mm Hg) but not for diastolic blood pressure or lipid measur

92 ital systolic pressure between 40 and 119 mm Hg were included.

93 in portal pressure gradient (PPG) to <12 mm Hg after placement of a transjugular intrahepatic portos

94 In considering the 12 mm Hg threshold, concordance between immediate PPG and earl

95 nts with early PPG vs late PPG values <12 mm Hg threshold.

96 ccess criterion was defined as IOP </= 12 mm Hg without and with antiglaucoma medications (absolute s

97 systolic blood pressure of less than 120 mm Hg (intensive treatment) than among those who were assig

98 on without diabetes, targeting an SBP<120 mm Hg compared with <140 mm Hg reduced rates of major cardi

99 to intensive BP lowering (target SBP <120 mm Hg) and 4078 assigned to standard BP lowering (target SB

100 ) with intensive (systolic BP target <120 mm Hg) BP treatment and data from the National Health and N

101 sive BP lowering (target systolic BP <120 mm Hg) compared with standard BP lowering (target systolic

102 o intensive (goal systolic pressure < 120 mm Hg) versus standard (<140 mm Hg) treatment.

103 um (19-31 mm), severe basal LVOTO (70-120 mm Hg), and left atrial dilatation (44-57 mm).

104 systolic blood pressure of less than 120 mm Hg, were similar to those among participants who receive

105 may benefit from a SBP target goal of 120 mm Hg.

106 h higher mortality (20.0% for SBP 100-129 mm Hg versus 12.0% for SBP 130-170 mm Hg; P<0.001).

107 ffice blood pressure was reduced by 22/13 mm Hg (p<0.0001).

108 systolic BP lowering to a target of <130 mm Hg may reduce the risk of several important outcomes inc

109 ubgroups categorized by both SBP (120-139 mm Hg, 140-159 mm Hg, and 160-179 mm Hg) and estimated 10-y

110 ere assigned to a target of less than 140 mm Hg (standard treatment).

111 er, and a mean 24-h ambulatory SBP of 140 mm Hg or greater and less than 170 mm Hg at second screenin

112 In vitro, islets cultured under 140 mm Hg oxygen showed reduced central necrosis and increased

113 eting an SBP<120 mm Hg compared with <140 mm Hg reduced rates of major cardiovascular events and all-

114 in therapy), blood pressure control (<140 mm Hg systolic, <90 mm Hg diastolic), angiotensin-convertin

115 systolic blood pressure of less than 140 mm Hg to reduce the risk for recurrent stroke.

116 systolic blood pressure of less than 140 mm Hg to reduce the risk for stroke or cardiac events.

117 s, intensive BP control (systolic BP <140 mm Hg) decreased MACE, including cardiovascular mortality a

118 dard BP lowering (target systolic BP <140 mm Hg) resulted in lower rates of developing new LVH in tho

119 essure < 120 mm Hg) versus standard (<140 mm Hg) treatment.

120 mparing standard (systolic BP target <140 mm Hg) with intensive (systolic BP target <120 mm Hg) BP tr

121 to standard BP lowering (target SBP <140 mm Hg).

122 s systolic blood pressure of at least 140 mm Hg, or diastolic blood pressure of at least 90 mm Hg, or

123 ntensive or standard SBP goal (120 or 140 mm Hg, respectively).

124 (</=132 versus >132 to <145 versus >/=145 mm Hg).

125 /=25 mm Hg and mean wedged PAP (PAWP) >15 mm Hg.

126 fice systolic blood pressure (SBP) of 150 mm Hg or greater and less than 180 mm Hg, office diastolic

127 ood pressure persistently at or above 150 mm Hg to achieve a target systolic blood pressure of less t

128 systolic blood pressure of less than 150 mm Hg to reduce the risk for mortality, stroke, and cardiac

129 patients with a PaO2/FiO2 lower than 150 mm Hg.

130 rized by both SBP (120-139 mm Hg, 140-159 mm Hg, and 160-179 mm Hg) and estimated 10-year ASCVD risk

131 systolic pressure were 2+/-3 and 15+/-16 mm Hg, respectively.

132 on (office systolic blood pressure >/=160 mm Hg despite taking at least three antihypertensive agents

133 of 140 mm Hg or greater and less than 170 mm Hg at second screening underwent renal angiography and w

134 00-129 mm Hg versus 12.0% for SBP 130-170 mm Hg; P<0.001).

135 120-139 mm Hg, 140-159 mm Hg, and 160-179 mm Hg) and estimated 10-year ASCVD risk (using the American

136 en CAC and events when SBP was 160 to 179 mm Hg, irrespective of ASCVD risk level.

137 rosis (MESA) with SBP between 120 and 179 mm Hg.

138 of 150 mm Hg or greater and less than 180 mm Hg, office diastolic blood pressure (DBP) of 90 mm Hg or

139 h blood pressure with the quadpill was 19 mm Hg (95% CI 14-23), and office blood pressure was reduced

140 +/-0.1 cm(2)/m(2); peak gradient, 53+/-19 mm Hg) were randomized to placebo or metoprolol treatment f

141 e Dresdner correction formula (17.6 [4.1] mm Hg) was closer to the DCT measurement than the original

142 Diastolic BP decreased by 12.2 mm Hg (95% CI, 11.2-13.2 mm Hg) in the intervention group a

143 d iron supplementation in infancy was 2.2 mm Hg (95% CI: 0.3, 4.2 mm Hg) lower than in those who were

144 blood pressure-lowering effects were 5/2 mm Hg and 7/5 mm Hg, respectively (both p<0.0001), and ther

145 Overall IOP was 18.0 +/- 6.2 mm Hg before surgery and 15.7 +/- 4.8 mm Hg 6 months after

146 ean arterial pressure at 6 hours was 72.2 mm Hg in the renin-angiotensin-aldosterone system inhibitor

147 ecreased by 12.2 mm Hg (95% CI, 11.2-13.2 mm Hg) in the intervention group and 6.9 mm Hg (95% CI, 5.9

148 n infancy was 2.2 mm Hg (95% CI: 0.3, 4.2 mm Hg) lower than in those who were unsupplemented (P = 0.0

149 20 minutes (bolus resuscitation: 57 +/- 2 mm Hg, closed loop: 69 +/- 4 mm Hg; p = 0.036).

150 pressure (bolus resuscitation: 19.3 +/- 2 mm Hg, decision assist, closed loop: 24 +/- 0.4 mm Hg; p <

151 ect intracranial hypertension (ICP >/= 20 mm Hg) was highest for ONSD (area under the curve [AUC] 0.9

152 severe hyperoxia as PaO2 greater than 200 mm Hg.

153 a was defined as PaO2 between 120 and 200 mm Hg; severe hyperoxia as PaO2 greater than 200 mm Hg.

154 Success required IOP </=22 mm Hg and 20% reduction without additional glaucoma surgery

155 ting glaucoma (n = 39) or high IOP (>/=22 mm Hg) with suspected glaucoma (n = 23), of whom several re

156 ean pulmonary artery pressure (PAP) >/=25 mm Hg and mean wedged PAP (PAWP) >15 mm Hg.

157 d as mean pulmonary arterial pressure >25 mm Hg and pulmonary vascular resistance [PVR] >/=240 dynes.

158 al blood CO2 tension when increased by 25 mm Hg can induce MBF to the same level as a standard dose o

159 hese data demonstrate that mean DBP >/=25 mm Hg during CPR in infants and >/=30 mm Hg in children >/=

160 population with maximum baseline IOP < 25 mm Hg in both studies (ROCKET-2, primary outcome measure an

161 Maintaining mean DBP >/=25 mm Hg in infants and >/=30 mm Hg in children >/=1 year old

162 e hypercapnic stimulus ( approximately 25 mm Hg increase in PaCO2) can increase MBF to that observed

163 LT, 39% of recipients had PH (mPAP >/= 25 mm Hg) and 10.3% had mPAP >/= 35 mm Hg.

164 on (mean pulmonary artery pressure, >/=25 mm Hg) was present in 82 patients (51%), including 29 (18%)

165 rval [CI], 2.64-6.51) and diastolic (2.25 mm Hg; 95% CI, 0.83-3.67) blood pressures.

166 rences for systolic blood pressure (-1.26 mm Hg [95% CI, -1.77 to -0.75]; 22 trials [n = 57953]), dia

167 were similar between groups (6.4 +/- 2.3 mm Hg vs. 5.8 +/- 2.7 mm Hg; p = 0.17), whereas the ViR gro

168 PPG values (8.5 +/- 2.5 mm Hg vs 8 +/- 3 mm Hg), or between proportions of patients with early PPG v

169 Mean arterial pressure was reduced to 30 mm Hg for 90 minutes, followed by resuscitation.

170 mean DBP >/=25 mm Hg in infants and >/=30 mm Hg in children >/=1 year old occurred in 101 of 164 chil

171 =25 mm Hg during CPR in infants and >/=30 mm Hg in children >/=1 year old was associated with greater

172 th PaO2/setFiO2 less than or equal to 300 mm Hg admitted to the intensive care unit.

173 d exercise PcCO2 less than or equal to 35 mm Hg (hypocapnia).

174 ortality was 0% for those with mPAP of 35 mm Hg or greater (vs 2.2% if mPAP < 35 mm Hg, P = 1.0).

175 However, mPAP of 35 mm Hg or greater can also occur in the setting of normal pu

176 re (mean pulmonary artery pressure, >/=35 mm Hg) and 28 (34%) also had increased pulmonary vascular r

177 than baseline, 2 with IOP higher than 35 mm Hg, and 1 with angle-closure glaucoma not attributed to

178 35 mm Hg or greater (vs 2.2% if mPAP < 35 mm Hg, P = 1.0).

179 P >/= 25 mm Hg) and 10.3% had mPAP >/= 35 mm Hg.

180 relative wall thickness (0.41 versus 0.35 mm Hg; P=0.009), and lower incidence of eccentric remodelin

181 PD values (3 [-1 to 6] versus 0 [-4 to 3] mm Hg; P<0.01) and a greater proportion of Cpc-PH (24% vers

182 eak -7 mm Hg (-13, 0; P=0.05) and mean -4 mm Hg (-7, -1; P=0.03) gradients, without affecting stroke

183 blood pressure than control groups: -6.4 mm Hg (95% CI, -8.6 to -4.2; 6 studies; I2 = 51%) for systo

184 ; the difference in the reduction was 5.4 mm Hg (95% CI, 4.0-6.8 mm Hg; P < .001).

185 the Repositioning group and -3.8 +/- 6.4 mm Hg (P < .001) in the Exchange group (group difference: P

186 an difference between PVP and CVP was 0.4 mm Hg and between PVP and pulmonary capillary wedge pressur

187 Peak PCWP decreased by 3.5+/-6.4 mm Hg in the treatment group versus 0.5+/-5.0 mm Hg in the

188 trengths of implant, respectively, vs 8.4 mm Hg in topical bimatoprost-treated pooled fellow eyes (da

189 Hg; P = .015) or deep sedation (12 +/- 4 mm Hg vs 10.5 +/- 4 mm Hg; P <.001).

190 decision assist, closed loop: 24 +/- 0.4 mm Hg; p < 0.05) and hemoglobin concentration were signific

191 ep sedation (12 +/- 4 mm Hg vs 10.5 +/- 4 mm Hg; P <.001).

192 on: 57 +/- 2 mm Hg, closed loop: 69 +/- 4 mm Hg; p = 0.036).

193 8 cm(2), mean aortic valve gradient >/=40 mm Hg, and dimensionless index <0.25.

194 +/- 0.8 (mean +/- SD) mm Hg (range, 35-43 mm Hg).

195 howed an increase in SBP of 0.94 +/- 0.44 mm Hg (pcom = 0.01) per risk variant copy.

196 lease, compared to those maintained in 45 mm Hg oxygen.

197 57953]), diastolic blood pressure (-0.49 mm Hg [95% CI, -0.82 to -0.16]; 23 trials [n = 58022]), low

198 ths was similar, with ranges of 12.5-15.5 mm Hg and 11.7-15.1 mm Hg, respectively.

199 re, and standardized this exposure to a 5 mm Hg reduction in mean arterial pressure.

200 ced using general anesthesia (8.5 +/- 3.5 mm Hg vs 10 +/- 3.5 mm Hg; P = .015) or deep sedation (12 +

201 arly PPG and late PPG values (8.5 +/- 2.5 mm Hg vs 8 +/- 3 mm Hg), or between proportions of patients

202 in study eyes was 7.2, 7.4, 8.1, and 9.5 mm Hg with the 6-mug, 10-mug, 15-mug, and 20-mug dose stren

203 e-lowering effects were 5/2 mm Hg and 7/5 mm Hg, respectively (both p<0.0001), and there were no side

204 diastolic BP: -12.3/-8.2 versus -6.8/-3.5 mm Hg, respectively, Delta systolic BP P=3x10(-4), Delta di

205 ulmonary capillary wedge pressure was 7.5 mm Hg.

206 76) effect sizes approximately 1.7 to 2.5 mm Hg.

207 esthesia (8.5 +/- 3.5 mm Hg vs 10 +/- 3.5 mm Hg; P = .015) or deep sedation (12 +/- 4 mm Hg vs 10.5 +

208 s directly associated with systolic (4.58 mm Hg; 95% confidence interval [CI], 2.64-6.51) and diastol

209 le lifestyle score) had 3.6, 3.5, and 3.6 mm Hg lower systolic BP in low, middle, and high genetic ri

210 a maximum of 17.9 (95% CI -28.3 to -7.6) mm Hg 3.75 hours later.

211 ean of 30.4 (+/- 10.3) to 24.9 (+/- 10.6) mm Hg at 6 months postoperatively.

212 08 mL/min/g; P = 0.002) at a PETco2 of 60 mm Hg.

213 55, and 60 mm Hg; repeat of PETco2 at 60 mm Hg; and repeat of baseline).

214 teaus (baseline; PETco2 at 50, 55, and 60 mm Hg; repeat of PETco2 at 60 mm Hg; and repeat of baseline

215 ce interval (CI): -2.14, 0.06) and a 0.63-mm Hg decrement in diastolic BP (95% CI: -1.35, 0.09), cont

216 01) and distensibility (0.47 versus 0.64%/mm Hg; P=0.02).

217 =90 mm Hg or mean arterial pressure </=65 mm Hg) presenting to the emergency department at a 1500-bed

218 ge, IOP by the rebound tonometer was 2.66 mm Hg lower than Goldmann applanation tonometry (95% limits

219 tile], 4.0 [3.1-5.1] versus 2.9 [2.4-3.6] mm Hg and -1.3 [-1.6 to -1.1] versus -1.2 [-1.6 to -1.1)] s

220 , metoprolol reduced aortic valve peak -7 mm Hg (-13, 0; P=0.05) and mean -4 mm Hg (-7, -1; P=0.03) g

221 ine intraocular pressure was +2.6 vs +1.7 mm Hg (P = .52).

222 terone system inhibitor group versus 69.7 mm Hg in the non-renin-angiotensin-aldosterone system inhib

223 ar gradient (from 20.5+/-7.4 to 6.7+/-3.7 mm Hg, P<0.001) and an increase in valve effective orifice

224 groups (6.4 +/- 2.3 mm Hg vs. 5.8 +/- 2.7 mm Hg; p = 0.17), whereas the ViR group had more frequent p

225 A mean DBP less than 70 mm Hg (n=5352) during treatment was associated with greater

226 re 135/85 mm Hg, 133/82 mm Hg, and 128/76 mm Hg, respectively.

227 and the systolic blood pressure was 14.8 mm Hg (95% confidence interval, 14.3 to 15.4) lower in the

228 IOP changed by -1.2 +/- 5.8 mm Hg (P = .18) in the Repositioning group and -3.8 +/- 6.4

229 6.2 mm Hg before surgery and 15.7 +/- 4.8 mm Hg 6 months after surgery (P < .001).

230 tion group and 6.9 mm Hg (95% CI, 5.9-7.8 mm Hg) in the control group; the difference in the reductio

231 reduction was 5.4 mm Hg (95% CI, 4.0-6.8 mm Hg; P < .001).

232 death (1.16, 1.06-1.28) than a DBP 70-80 mm Hg (14 305).

233 y (95% limits of agreement, -3.48 to 8.80 mm Hg).

234 defined as having blood pressure <120/80 mm Hg, fasting glucose <100 mg/dl, glycosylated hemoglobin

235 of 140/90 mm Hg were 135/85 mm Hg, 133/82 mm Hg, and 128/76 mm Hg, respectively.

236 daytime ambulatory BP of at least 135/85 mm Hg and was further divided into masked and sustained hyp

237 linic SBP/DBP of 140/90 mm Hg were 135/85 mm Hg, 133/82 mm Hg, and 128/76 mm Hg, respectively.

238 ars, and mean SBP/diastolic BP was 135/86 mm Hg.

239 mm Hg) in the intervention group and 6.9 mm Hg (95% CI, 5.9-7.8 mm Hg) in the control group; the dif

240 months post-TAVR, with a decrease of -2.9 mm Hg in aortic valve mean gradient, an increase of 0.028 i

241 and DBP were 139.7+/-15.6 and 78.1+/-11.9 mm Hg, respectively.

242 g treatment levels significantly above 90 mm Hg are needed.

243 measures for adults with SBP/DBP <140/90 mm Hg at high risk for CVD may be warranted.

244 ressure control (<140 mm Hg systolic, <90 mm Hg diastolic), angiotensin-converting enzyme inhibitor o

245 fice diastolic blood pressure (DBP) of 90 mm Hg or greater, and a mean 24-h ambulatory SBP of 140 mm

246 ypotension (systolic blood pressure </=90 mm Hg or mean arterial pressure </=65 mm Hg) presenting to

247 corresponding to clinic SBP/DBP of 140/90 mm Hg were 135/85 mm Hg, 133/82 mm Hg, and 128/76 mm Hg, re

248 ertensive medication with SBP/DBP <140/90 mm Hg, 76.6% (95% CI, 75.8-77.5) were eligible for statin t

249 king and untreated blood pressure <140/90 mm Hg, fasting glucose <126 mg/dl, total cholesterol <240 m

250 r diastolic blood pressure of at least 90 mm Hg, or self-reported antihypertensive medication use in

251 al sum increase or decrease in pressures (mm Hg-day) during the follow-up period relative to the base

252 ine PETco2 was 38.9 +/- 0.8 (mean +/- SD) mm Hg (range, 35-43 mm Hg).

253 coastal regions, and their importance to net Hg deposition has been questioned.

254 4, N2O, PM2.5, PM10, NOx, SO2, VOC, CO, NH3, Hg, Pb, Cd, Cr (VI), Ni, As, and dioxins.

255 linear dynamic range of 100.0 pM to 10.0 nM Hg(2+) concentration with R(2) = 0.982.

256 This suggests that complexes of Hg(II) with DOM thiols have similar bioavailability to H

257 and Europe may be important contributors of Hg to Lake Baikal and that, despite the size of Lake Bai

258 ed that sea-salt-induced chemical cycling of Hg (through 'atmospheric mercury depletion events', or A

259 We find that deposition of Hg(0)-the form ubiquitously present in the global atmosp

260 y minor contributions from the deposition of Hg(ii) via precipitation or AMDEs.

261 Trends in atmospheric emissions of Hg suggest that local sources as well as emissions from

262 Ultratrace levels of Hg(2+) have been quantified by undertaking linear sweep

263 Cs allow precise and accurate positioning of Hg-based SECM probes over any sample and enable the depl

264 The role of Hg speciation on Hg bioavailability in marine systems ha

265 In this study, the sorption of Hg(II), Cd(II), and Au(III) onto Bacillus subtilis bioma

266 deposition via precipitation are sources of Hg to the Arctic in its oxidized form (Hg(ii)).

267 sses governing partitioning and transport of Hg in this contaminated river system.

268 is enhanced in summer through the uptake of Hg(0) by vegetation.

269 estigated and compared the effects of DOM on Hg methylation by an iron-reducing bacterium Geobacter s

270 The role of Hg speciation on Hg bioavailability in marine systems has not been teased

271 The Hg(II)-DOM or Hg(II)-DOM-sulfide equilibration times did not significa

272 s were present between prenatal maternal RBC-Hg and %-5mC at any time point.

273 blood %-5hmC for a doubling in prenatal RBC-Hg concentration was -0.013% (-0.029, 0.002), -0.031% (-

274 5mC to %-5hmC for a doubling in prenatal RBC-Hg concentration was 4.70% (0.04, 9.58), 22.42% (7.73, 3

275 issions are transformed to divalent reactive Hg (RM) forms throughout the troposphere and stratospher

276 may provide enhanced Hg deposition, reduced Hg emission and, ultimately, an increase in snowpack and

277 an increase in snowpack and snowmelt runoff Hg concentrations.

278 ty for Hg, and indicate that the sequestered Hg is bound in soil organic matter pools accumulating ov

279 an inland-to-coastal transect show high soil Hg concentrations consistently derived from Hg(0), sugge

280 a uptake of gaseous Hg(0) leads to high soil Hg concentrations, with Hg masses greatly exceeding the

281 at DOM inhibits Hg methylation due to strong Hg-DOM complexation.

282 with those of previous studies, suggest that Hg trends in Arctic freshwater fishes before 2001 were s

283 The Hg(II)-DOM or Hg(II)-DOM-sulfide equilibration times did

284 results favor metacinnabar (beta-HgS) as the Hg-S4 species, which we show is associated with both the

285 ass-balance study, and show that most of the Hg (about 70%) in the interior Arctic tundra is derived

286 To gain insight into this Hg(II) biouptake pathway, we have employed X-ray absorpt

287 The fate of this Hg during and following snowmelt is largely unknown.

288 h DOM thiols have similar bioavailability to Hg(II) complexes with low-molecular-weight thiols.

289 In contrast to Hg, liquid metals based on gallium have low toxicity and

290 Delta(199)Hg was related to Hg levels of fish but we also suggest a relation with ec

291 Sweden that belowground inventories of total Hg are strongly related to soil humus C accumulation (R(

292 tible to amalgam saturation than traditional Hg sphere-caps or thin-films.

293 obilis were a biological vector transporting Hg from freshwater environments into marine ecosystems.

294 gnificant decreasing trend in the lake trout Hg concentrations was found between 2004 and 2015 with a

295 regional atmospheric Hg emissions and water Hg concentrations.

296 ystem restoration are closely linked to what Hg levels we consider natural.

297 IMP-1 levels were higher in Gh compared with Hg group (P <0.05) whereas salivary MMP-8/TIMP-1 molar r

298 -1 molar ratio was lower in Gh compared with Hg group (P <0.05).

299 ) leads to high soil Hg concentrations, with Hg masses greatly exceeding the levels found in temperat

300 Here we present a 320 year Hg deposition history for Central Asia, based on a conti