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1 were black, 37.6% were white, and 16.3% were Hispanic.
2 and 75.1% (n = 151) self-identifying as non-Hispanic.
3 duced DRESS syndrome were African, and 1 was Hispanic.
4 rcent were boys, 78% were white, and 8% were Hispanic.
5 SD), 10) years; 66% were women, and 55% were Hispanic.
6 of participants were white, and 90% were non-Hispanic.
7 0 years, 19% were female, 29% black, and 20% Hispanic.
8 ugh not always significant) for foreign-born Hispanics.
9 s stronger among men, African Americans, and Hispanics.
10 a Natives, and 0.70 (95% CI: 0.60, 0.81) for Hispanics.
11 84% women, 31% non-Hispanic blacks, and 39% Hispanics.
12 as were non-Hispanic black (105 [14.8%]) and Hispanic (1165 [20.0%]) youth compared with non-Hispanic
14 cans, 21.5 kg/m2 (CI, 18.5 to 24.5 kg/m2) in Hispanics, 20.9 kg/m2 (CI, 19.7 to 22.1 kg/m2) in Chines
15 2%, Chinese American 12.0%, black 24.5%, and Hispanic 21.2%) using biplane and short-axis images.
17 f respondents were non-Hispanic white, 32.3% Hispanic, 27.2% non-Hispanic black, and 7.7% other race/
18 panic white, 13.8% non-Hispanic black, 16.5% Hispanic, 4.0% Asian, and 5.1% other), of whom 474 (5.0%
19 hite, 391 (4.9%) were black, 527 (6.6%) were Hispanic, 424 (5.3%) were Asian/Pacific Islander, 63 (0.
20 nce by ethnicity were 45.5% in Asians, 45.3% Hispanics, 44.20% Blacks, 36.7% Whites, and 14.3% in mix
21 consecutive white (1,107), black (361), and Hispanic (479) older adults who had their first medical-
22 ; mean age, 42.9 years; 87.3% women; and 31% Hispanic, 49% non-Hispanic white, and 20% other race/eth
23 The cohort has 296 decedents: 147 Blacks, 64 Hispanics, 49 Whites, 22 Asians, and 14 mixed ethnicitie
25 e; 4 of white race/ethnicity, 1 Asian, and 1 Hispanic), 5 exhibited an autofluorescent AZOOR line in
27 ce of HLA-B*53 carriage in African (20%) and Hispanic (6%) populations, the probability of all 4 pati
29 ts included 371 women (mean age, 28.1 years; Hispanic, 81.6%; mean weight above prepregnancy weight,
30 65.3% (27654) were white, 12.7% (5365) were Hispanic, 9.4% (3976) were African American, and 12.6% (
31 sepsis hospitalizations), black (94.0), and Hispanic (93.5) patients but remained elevated for Asian
33 ational studies have reported that black and Hispanic adults receiving maintenance dialysis survive l
34 ich aims to enroll 1300 individuals (600 non-Hispanic African ancestry, 600 non-Hispanic European anc
36 t in cross-ethnicity analyses among the MESA Hispanic-Americans cohort (OR per triglyceride GRS unit:
38 = 44) of all individuals self-identifying as Hispanic and 75.1% (n = 151) self-identifying as non-His
40 94 women and 8,738 men of European, African, Hispanic and Chinese ancestry, with and without sex stra
42 nt-rated health for US-born and foreign-born Hispanics and 2) assessed whether neighborhood poverty m
45 s (white, black, Mexican American, and other Hispanic), and across three Asian subgroups (Chinese, As
46 (26%) were African American, 1823 (15%) were Hispanic, and 1555 (13%) were other races/ethnicities.
47 an, 11 (14%) were white, 16 (21%) were white Hispanic, and 26 (33%) were other/mixed race/ethnicity.
48 ants (52% white, 23% black, 11% Chinese, 14% Hispanic, and 52% men with a mean age of 67.6 years) at
50 -90.6) in black, 86.0% (95% CI 83.7-88.0) in Hispanic, and 90.7% (95% CI 87.0-93.5) in Asian/Pacific
51 rals produce high SVR rates in white, black, Hispanic, and Asian/Pacific Islander/American Indian/Ala
53 ter US study of Caucasian, African-American, Hispanic, and Chinese-American adults aged 45-84 years.
55 red mortality risk among non-Hispanic black, Hispanic, and non-Hispanic white children who started RR
56 o statins and with drug molecules for black, Hispanic, and non-Hispanic white people, and men and wom
57 s use of IPBR among white, African American, Hispanic, and other minority groups before and after the
58 hinese Americans, 334 African Americans, 252 Hispanics, and 195 South Asians), the prevalence of MAN
59 ethnicities, 10.2% Whites, 4.5% Asians, 3.1% Hispanics, and 2% Blacks; 7.7% children, 7.3% in adults,
60 can Americans, 38.5% (CI, 32.6% to 44.6%) in Hispanics, and 43.6% (CI, 36.8% to 50.6%) in South Asian
63 ach of 4 race/ethnic groups (blacks, Asians, Hispanics, and non-Hispanic whites) were targeted for re
64 ach racial/ethnic group (non-Hispanic black, Hispanic, Asian American and Pacific Islander, and separ
65 American (beta=-2.6 mL/m(2), P<0.0001), and Hispanic (beta=1.1 mL/m(2), P<0.05) ethnicities were ass
66 receive medications (P < .001), as were non-Hispanic black (105 [14.8%]) and Hispanic (1165 [20.0%])
67 eons and the American Cancer Society, on non-Hispanic black (black) and non-Hispanic white (white) pa
69 morbidity and readmission trends between non-Hispanic Black and White patients for the majority of di
70 te abolished the higher risk of death in non-Hispanic black children (hazard ratio, 0.99; 95% CI, 0.8
74 females exist, with Mexican American and non-Hispanic black females at greater risk of ID than non-Hi
75 birth (PTB) prevalence between births of non-Hispanic black individuals and births of non-Hispanic wh
77 the exception of declines among men and non-Hispanic black patients aged 45 to 54 with subarachnoid
78 Residence in urban or poor areas and non-Hispanic black race/ethnicity were all independently ass
79 ] years; 60.7% non-Hispanic white, 13.8% non-Hispanic black, 16.5% Hispanic, 4.0% Asian, and 5.1% oth
80 ces and ethnicities (non-Hispanic white, non-Hispanic Black, American Indian, Asian Indian, Chinese,
84 0 and 2013 for each racial/ethnic group (non-Hispanic black, Hispanic, Asian American and Pacific Isl
85 and race/ethnicity (non-Hispanic white, non-Hispanic black, Mexican American), and covariate-adjuste
87 ic whites, 1.67 (95% CI: 1.20, 2.34) for non-Hispanic blacks, 1.57 (95% CI: 1.17, 2.09) for Asians/Pa
89 ALL was negatively associated with SEP among Hispanics but was positively associated among children o
90 tibility and onset age in 65 independent non-Hispanic Caucasian males in the Collaborative Study on t
93 ng black non-Hispanic children and white non-Hispanic children (80.7%); however, Hispanic children we
94 me direction in CAMP white children and CAMP Hispanic children (combined interaction P = .0065 for re
95 uate the finding of a lower risk of death in Hispanic children (hazard ratio, 0.59; 95% CI, 0.51 to 0
96 ed identical screening rates among black non-Hispanic children and white non-Hispanic children (80.7%
98 Hispanic children were less likely than non-Hispanic children to report vision screening (69.8%).
99 hite non-Hispanic children (80.7%); however, Hispanic children were less likely than non-Hispanic chi
103 in WBCs and total IgE levels in 2 studies of Hispanic children: the Puerto Rico Genetics of Asthma an
104 (mean age: 45 y, range: 18 to 76 y) from the Hispanic Community Health Study / Study of Latinos.
105 mple of Hispanics/Latinos ascertained by the Hispanic Community Health Study and Study of Latinos (HC
106 six Hispanic/Latino background groups in the Hispanic Community Health Study/Study of Latinos (HCHS/S
107 ipants (769 cases, 9,384 controls) of the US Hispanic Community Health Study/Study of Latinos (HCHS/S
110 udied 12 083 participants from the HCHS/SOL (Hispanic Community Health Study/Study of Latinos), a pop
111 (BP) traits in 12,278 participants from the Hispanics Community Health Study/Study of Latinos (HCHS/
112 detrimental effect on the health of US-born Hispanics, comparable to findings for blacks and black-w
114 duals of European, South Asian, African, and Hispanic descent (pan-ancestry, approximately 475 000),
115 related NHW patients, variants present in 19 Hispanic EOAD WES families, and genes with variants in m
116 white race (56.9%) with black race (16.7%), Hispanic ethnicity (15.8%), and Asian race (2.8%) compri
118 relative risk = 1.10; 95% CI, 1.04-1.16) and Hispanic ethnicity (relative risk = 1.08; 95% CI, 1.02-1
121 for baseline characteristics, black race and Hispanic ethnicity remain independent predictors of trea
122 icantly associated with HO-MRSA BSIs whereas Hispanic ethnicity was negatively associated (rate ratio
124 perimenopause persisted after adjustment for Hispanic ethnicity, antiretroviral use, and alcohol (0.1
126 ting for individual age, sex, nonwhite race, Hispanic ethnicity, education, and marital status, as we
128 reased frequency of Ph-like ALL in adults of Hispanic ethnicity, significantly inferior outcomes of a
132 ican American women are more likely than non-Hispanic European American women to be obese and to be d
133 (600 non-Hispanic African ancestry, 600 non-Hispanic European ancestry, and 100 Hispanic) who meet r
134 od, all 3 racial groups (whites, blacks, and Hispanics) experienced substantial increase in hypertens
136 te (from 21.1 % in 2009 to 9.4% in 2015) and Hispanic (from 8.2% in 2009 to 4.7% in 2015) students ov
137 Interaction terms for non-Hispanic black and Hispanic groups compared with the non-Hispanic white gro
142 l segregation, come into play in determining Hispanic health for the US-born in a way that does not o
143 nderstanding the fundamental determinants of Hispanic health, few studies have investigated how metro
146 were significantly associated with black non-Hispanic (incidence rate ratio [IRR], 1.29; 95% CI, 1.17
147 eased substantially across all age groups in Hispanic individuals (up to 3.2% per year), black indivi
148 ting in an estimated 112 000 fewer deaths in Hispanic individuals, 311 000 fewer deaths in black indi
149 tributed to declining premature mortality in Hispanic individuals, black individuals, and Asians and
151 ratio [IRR], 1.29; 95% CI, 1.17 to 1.42) and Hispanic (IRR, 1.11; 95% CI, 1.03 to 1.20) race/ethnicit
155 values for FVC and FEV1 than those in other Hispanic/Latino background groups and also than Mexican
156 subjects and 5,247 control subjects from six Hispanic/Latino background groups in the Hispanic Commun
157 lop spirometry reference equations for adult Hispanic/Latino background groups in the United States.
159 deled as a function of sex, age, height, and Hispanic/Latino background to produce background-specifi
162 e association study of melanin levels in 285 Hispanic/Latino individuals from Puerto Rico, analyzing
167 0(-35) for drinks/week), which replicated in Hispanic/Latinos (OR=0.72, P=4.35 x 10(-7) and beta=-0.2
168 race/ethnicity groups: non-Hispanic whites, Hispanic/Latinos, East Asians and African Americans.
169 a stronger predictor of incident obesity in Hispanics/Latinos (OR = 1.97, 95% CI: 1.73, 2.24; P < 0.
170 opulation-based probability sample of 16,415 Hispanics/Latinos aged 18-74 years living in the Bronx,
172 , ethnically diverse US population sample of Hispanics/Latinos ascertained by the Hispanic Community
173 ly high risk of weight gain and obesity, and Hispanics/Latinos may be especially vulnerable to the ob
174 2 diabetes (T2D) have been conducted in U.S Hispanics/Latinos of diverse backgrounds who are disprop
175 rican-specific alleles associate with CKD in Hispanics/Latinos, but allele frequency varies by Hispan
176 ancestry alleles may contribute to CKD among Hispanics/Latinos, but whether associations differ by Hi
181 n American (OR, 2.82; 95% CI, 1.77-4.50) and Hispanic males (OR, 2.91; 95% CI, 1.75-4.82), and 2 to 5
182 that the current aggregate classification of Hispanics masks heterogeneity in CVD mortality reporting
183 12% of patients were female, 36% black, 13% Hispanic; median age was 37 years, CD4 count 321 cells/m
184 ack women (HR, 0.81, 95% CI, 0.67-0.98), and Hispanic men (HR, 0.61, 95% CI, 0.42-0.89) and women (HR
186 vey, we 1) examined the relationship between Hispanic metropolitan segregation and respondent-rated h
188 252), 33.3% were black (n = 240), 21.8% were Hispanic (n = 157), and 9.9% were of another race/ethnic
189 rquartile range, 1-12 years), one-third were Hispanic (n = 28 [34%]), and two-thirds (n = 55 [67%]) w
191 The findings of a reverse association in Hispanics need to be confirmed and further explained in
192 0.59 [95% CI, 0.36-0.94] for uninsured) and Hispanics (odds ratio, 0.74 [95% CI, 0.60-0.91] for insu
193 the overreporter phenotype: non-white race: Hispanic, odds ratio (OR), 2.4, P = .02; Asian, OR, 3.1,
194 ere white, 151 (42%) were black, 8 (2%) were Hispanic or Latino, 9 (2.5%) were of other race/ethnicit
197 he association was no longer significant for Hispanics (OR, 0.95; 95% CI, 0.78 to 1.17) or blacks (OR
199 rval (CI) 2.9-3.5); less likely to be Black, Hispanic, or Asian versus White/non-Hispanic (ORadj's =
200 e Black, Hispanic, or Asian versus White/non-Hispanic (ORadj's = 0.4-0.6, p < 0.01); more likely to b
201 in whole blood of 573 individuals of diverse Hispanic origin who also had high-density genotype data.
203 years), race (lower in African American and Hispanic participants), geographic location (lower in We
209 46.8 per 100 person-years), Black and Latino/Hispanic people (41.8 and 39.5 per 100 person-years, res
211 comes, approximately 1 in 5 black and 1 in 3 Hispanic persons with a chronic disease continued to lac
212 ses in acquisitions occurred among white and Hispanic persons, but not among black persons, and among
213 wide range of experiences within the diverse Hispanic population and suggests that socioeconomic stat
216 readmissions included age, female sex, black/Hispanic race, prior amputation, Charlson comorbidity in
217 stage (III), younger age (<40 yrs), Black or Hispanic race/ethnicity versus non-Hispanic White (OR 1.
222 n the population is more common in black and Hispanic than in white individuals and is associated wit
223 he annual rate of increase was greater among Hispanics than among non-Hispanic whites (4.2% vs. 1.2%,
225 d stable or decreasing representation of non-Hispanic underrepresented minority critical care fellows
226 3 PUFAs with nonfatal MI risk in Costa Rican Hispanics.We analyzed cross-sectional data from 1932 cas
229 n [SD] age, 31.0 [5.7] years), most were non-Hispanic white (1957 [73.7%]), 98 had panic disorder, 25
230 009 to 10.6 % in 2015) students overall, non-Hispanic white (from 21.1 % in 2009 to 9.4% in 2015) and
231 entative sample of US Black (n = 4,201), non-Hispanic White (n = 20,217) and Hispanic (n = 5,097) gro
233 hnic Cohort study who self-identified as non-Hispanic White (n = 32,103), African American (n = 30,20
234 cific mortality among AIAN (n = 582) and non-Hispanic white (NHW; n = 82,696) enrollees of Kaiser Per
235 Black or Hispanic race/ethnicity versus non-Hispanic White (OR 1.10, 95% confidence interval (CI) 1.
236 ciety, on non-Hispanic black (black) and non-Hispanic white (white) patients, 18-64 years old, diagno
238 th Interview Survey data including 10262 non-Hispanic white adults aged 18 to 60 years without a hist
240 ic Obstructive Pulmonary Disease [COPD]; non-Hispanic white and African American), ECLIPSE (Evaluatio
241 econd most VI is projected to shift from non-Hispanic white children (26.3% in 2015 decreasing to 16.
242 among non-Hispanic black, Hispanic, and non-Hispanic white children who started RRT between 1995 and
243 black children had higher mortality than non-Hispanic white children, which was related to difference
248 ck and Hispanic groups compared with the non-Hispanic white group were not statistically significant.
249 Hispanic black individuals and births of non-Hispanic white individuals in California, according to i
250 and obstruction was demonstrated in all non-Hispanic white individuals in the Pittsburgh Specialized
254 For example, 12 years after detention, non-Hispanic white males had nearly 3 times the odds of educ
256 d 90 [52.3%] in the stent group; and 114 non-Hispanic white patients [65.9%] in the stoma group and 9
257 drug molecules for black, Hispanic, and non-Hispanic white people, and men and women of Asian, Nativ
258 is, male sex, African-American race, and non-Hispanic white race/ethnicity were significantly associa
260 [interquartile range, 9.0] years; 60.7% non-Hispanic white, 13.8% non-Hispanic black, 16.5% Hispanic
261 5.8 years) and 32.8% of respondents were non-Hispanic white, 32.3% Hispanic, 27.2% non-Hispanic black
262 ears; 87.3% women; and 31% Hispanic, 49% non-Hispanic white, and 20% other race/ethnicity) were rando
263 ferences among 14 races and ethnicities (non-Hispanic white, non-Hispanic Black, American Indian, Asi
264 eir sum by age, sex, and race/ethnicity (non-Hispanic white, non-Hispanic black, Mexican American), a
270 also enrolled (race/ethnicity other than non-Hispanic white: 16%; no college degree: 35%; household i
271 ildren (median age, 22 months; 55% male; 61% Hispanic white; 40 per group), 59 completed the trial (2
274 higher in Asians/Pacific Islanders than non-Hispanic whites (aIRR = 2.09); after induction immunosup
275 , 95% CI: 1.73, 2.24; P < 0.001) than in non-Hispanic whites (OR = 1.54, 95% CI: 1.25, 1.91; P < 0.00
278 a genome-wide significant association in non-Hispanic whites between the previously reported SNP rs12
280 2259 participants randomized, 1702 were non-Hispanic whites who completed the trial and had genotype
282 onfidence interval (CI): 1.15, 1.44) for non-Hispanic whites, 1.67 (95% CI: 1.20, 2.34) for non-Hispa
284 ) cohort, in four race/ethnicity groups: non-Hispanic whites, Hispanic/Latinos, East Asians and Afric
285 ageal adenocarcinoma is more frequent in non-Hispanic whites, whereas esophageal squamous cell carcin
289 600 non-Hispanic European ancestry, and 100 Hispanic) who meet rigorous clinical criteria for idiopa
290 4 white, 8 African American, 25 Asian, and 3 Hispanic) with a mean (SD) age of 51.8 (15.9) years (26
291 white, 23 African American, 8 Asian, and 11 Hispanic) with a mean (SD) age of 53.6 (16.2) years (27
292 Among the 230 participants, 152 (66.1%) were Hispanic, with a mean (SD) age of 31.4 (7.3) years.
294 ly specific risk reduction interventions for Hispanic women should focus on awareness of partner risk
295 03 to 2012 ranged from 85 per 100000 for all Hispanic women to 144 per 100000 for Cuban men, but rate
299 th higher metabolic syndrome among black and Hispanic young adults from disadvantaged childhood envir
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