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1 and 12 months (as assessed by means of 3-day Holter monitoring).
2 adequacy of MRC was assessed by using serial Holter monitoring.
3 ery 3 to 6 months by clinic review and 7-day Holter monitoring.
4 ticipants did not have at least 1 PAC during Holter monitoring.
5 VT was assessed using Holter monitoring.
6 mia with an event transmitter and ambulatory holter monitoring.
7 den of ectopy was quantified through 24-hour Holter monitoring.
8 puted tomography perfusion scan, and 24-hour Holter monitoring.
9 ocardiography, upright exercise testing, and Holter monitoring.
10 ity of life and for increased arrhythmias by Holter monitoring.
11 re), exercise testing, echocardiography, and Holter monitoring.
12 ause they did not have AECG ischemia on 48-h Holter monitoring.
13 ssion (n = 10 [27%], including by ECG [14%], Holter monitoring [11%], or signal-averaged ECG [14%]) w
15 AF rhythm control was determined using 7-day Holter monitoring and AF severity scale questionnaire.
16 use of signal-averaged electrocardiography, Holter monitoring and assessment of left ventricular fun
17 rcise tests, exercise thallium scintigraphy, Holter monitoring and electrophysiologic study findings
18 hocardiography, surface electrocardiography, Holter monitoring and exercise testing, when applicable,
20 by assessing heart rate variability on 24-h Holter monitoring and plasma norepinephrine levels durin
21 After ablation, patients underwent repeated Holter monitoring and reassessment of cardiac function.
22 cidence of ineffective capture using 12-lead Holter monitoring and to assess whether this affects res
23 gned to 24-h ambulatory electrocardiography (Holter) monitoring and who had a normal LVEF and no hist
31 aged ECG, exercise testing, cardiac imaging, Holter-monitoring, and selective provocative drug testin
33 ncreas transplant recipients underwent 24-hr Holter monitoring before and again at 6 and 12 months po
35 studies, 12-lead electrocardiograms, 24-hour Holter monitoring, blood tests, and completion of Minnes
37 The maximum reduction in mean heart rate by Holter monitoring during the first 6 h in ozanimod-treat
38 ination, which included electrocardiography, Holter monitoring, echocardiography, bicycle ergometry,
39 cluding ECG, 2-dimensional echocardiography, Holter monitoring, exercise tolerance testing, and ajmal
41 on cardiac ultrasonography in 100 of 195, on Holter monitoring in 2 of 192; and on hypercoagulable pa
42 etermined before and after ablation by 7-day Holter monitoring; intermittent ECG event monitoring was
43 verse events at regular scheduled visits and Holter monitoring; key efficacy measures were annualized
44 were evaluated by electrocardiography (ECG), Holter monitoring, late-enhancement cardiac magnetic res
47 ties on the basis of electrocardiography and Holter monitoring, of whom 20 (48%) had abnormal results
48 s in patients with organic heart disease, 2) Holter monitoring or telemetry in patients known to have
51 cal abnormalities on electrocardiography and Holter monitoring precede detectable structural abnormal
52 Patients underwent 48-h electrocardiographic Holter monitoring quarterly to detect brief subclinical
56 were evaluated by electrocardiography (ECG), Holter monitoring, signal-averaged ECG, and cardiac magn
60 d corrected QT intervals were recorded; 24 h Holter monitoring was utilized to gauge efficacy of trea
61 The clinical course, electrocardiograms, and Holter monitoring were available for review in 114 subje
62 aphy (SPECT) perfusion scanning, and 24-hour Holter monitoring were performed at baseline and follow-
64 axar significantly reduced early ischemia on Holter monitoring without a significant increase in majo
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