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1 roperitoneal and epididymal white tissue and IBAT, but skeletal muscle 2-DG uptake under the same con
2 CRFR2 revealed significantly elevated basal IBAT thermogenesis and prolonged adrenergic responsivity
3 pinephrine content of the interscapular BAT (IBAT) and the number of sympathetic ganglion cells proje
4 d receive SS inflow from, interscapular BAT (IBAT) in these separate studies suggesting SNS-SS feedba
6 tor-gamma-coactivator1-alpha (PGC-1alpha) in IBAT and increased UCP1protein expression; however, pero
8 se or fructose, leading to SNS activation in IBAT and retroperitoneal fat and enhanced GLUT4 expressi
9 essential role in mediating the increase in IBAT thermogenesis induced by activation of central mela
11 roup of rats, the neural circuit involved in IBAT control, including the location of sympathetic preg
15 by contrast, decreased [(3)H]NE turnover in IBAT, but increased it in epididymal, though not retrope
17 NH2; 0.024nmol) both significantly increased IBAT temperature (T(IBAT)) and pretreatment with the MC4
24 y c-Fos immunoreactivity (IR), we prelabeled IBAT DRG innervating neurons by injecting the retrograde
27 locked the MC4-R agonist-induced increased T(IBAT) in conscious, freely-moving Siberian hamsters.
28 icle-treated hamsters initially maintained T(IBAT), but the ability of the former waned after 2 h bei
32 nability of MSG-treated animals to sustain T(IBAT) in the cold is not due to any obvious MSG-induced
33 significantly increased IBAT temperature (T(IBAT)) and pretreatment with the MC4R antagonist, HS024
36 nover in interscapular brown adipose tissue (IBAT) and retroperitoneal fat increased in response to g
37 evels in interscapular brown adipose tissue (IBAT) from F344 x BN rats ages 3, 12, 18, 24, and 30 mon
38 ivity of interscapular brown adipose tissue (IBAT) in response to physiologic stimuli, such as cold e
39 lyses of interscapular brown adipose tissue (IBAT) thermogenesis by thermal signature analysis and th
40 n affect interscapular brown adipose tissue (IBAT) thermogenesis via its sympathetic nervous system (
45 s of central sympathetic outflow circuits to IBAT, but appears to be extrinsic to the tissue neverthe
46 th components of the CNS outflow circuits to IBAT, with the latter implicated in BAT thermogenesis th
47 ons of MC4-R mRNA and SNS outflow neurons to IBAT that has not been previously reported to be involve
48 co-localization with SNS outflow neurons to IBAT, the subzona incerta (subZI) to test whether IBAT t
51 d-evoked increases in sympathetic outflow to IBAT, the present study compared central nervous system
52 a on the sympathetic nerve activity (SNA) to IBAT evoked by lateral ventricular injection of the mela
55 ibitor of human ileal bile acid transporter (IBAT), in patients with primary biliary cholangitis with
58 was detected in COS cells cotransfected with IBAT and a chimeric molecule having the carboxyl-termina
59 acid efflux in COS cells cotransfected with IBAT and CEA, efflux of [3H]taurocholate was detected in
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