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1 ich was overcome by crossing them to outbred ICR mice.
2 inhibition of capsaicin-induced flinching in ICR mice.
3 munoreactivity (irBC) in the hypothalamus of ICR mice.
4 cells, and lack of neurovirulence in newborn ICR mice.
5 benzo[a]pyrene-evoked forestomach tumors in ICR mice.
6 ague-Dawley rats and antinociception in male ICR mice.
7 ect the gastric mucosa of wild-type, outbred ICR mice.
8 xidative stress and hepatic fibrosis in male ICR mice.
9 liver short hairpin (shRNA) genes to healthy ICR mice.
10 for inbred (BALB/c and C57BL/6) and outbred (ICR) mice.
12 cal determinants for neuroinvasion of normal ICR mice by SPYF virus were also in the E protein, seque
13 utrition (TPN) to genetically normal, immune ICR mice by the i.v. route results in loss of nasal anti
14 or RVV-X (the venom factor X-activator) into ICR mice did not significantly deplete the plasma fibrin
15 lecule MIF antagonist, CPSI-1306, to outbred ICR mice following induction of NIDDM significantly lowe
17 A/H-F/V was transplanted subcutaneously into ICR mice for up to 4 weeks to assess its in vivo effect
18 , were not neurovirulent in 3- to 4-week-old ICR mice inoculated by the intracerebral route, and were
19 Corneal clarity in young adult Swiss (HSD:ICR) mice is restored after Pseudomonas aeruginosa infec
21 s of estren's action on male-orchidectomized ICR mice revealed estren's AR agonist actions on the lev
22 equences of viruses recovered from brains of ICR mice succumbing to encephalitis with the parental SP
24 -treated primary pancreatic islet cells from ICR mice to unravel the protective mechanism of berberin
31 transthoracic echocardiography (TTE), adult ICR mice were injected i.p. with vehicle (10% DMSO) or t
37 67) controlled by the keratin 14 promoter in ICR mice were used to determine the effects of OA on AP-
39 (51)MnCl2 was intravenously administered to ICR mice which were scanned by dynamic and static PET, f
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