ライフサイエンスコーパス: IDA

1 IDA also inhibited nitrite production in LPS stimulated

2 IDA is most prevalent among preschool children and women

3 IDA mothers received iron supplements of 125 mg FeSO(4)

4 IDA patients can be treated with iron supplementation, y

5 IDA prevalence was 5.0% +/- 0.4% and 2.6% +/- 0.7% in no

6 IDA random assignment was associated with higher CR rate

7 IDA reduced induction of NOS-2 protein levels, steady st

8 IDA was <5% in most studies in Northern and Western Euro

9 IDA was defined as a low hemoglobin level in the presenc

10 IDA, anthracycline derivative anticancer drug, was adsor

11 IDA-Cu complex acted as an efficient recognition element

12 .5% in CCG-2891 (P <.001) and 5% in CCG-2941 IDA/DNR (P = not significant).

13 Compared with CCG-2891 DNR/DRN, CCG-2941 IDA/IDA increased days in hospital (43 v 36 days; P =.00

14 In course 1 of CCG-2941 IDA/IDA, 11% of patients withdrew compared with 1.5% in

15 received iron supplements of 125 mg FeSO(4) (IDA-Fe; n = 34) or placebo (IDA-PL; n = 30) daily from 1

16 subjects had a low prevalence of ID (2.7%), IDA (1.2%), and depleted iron stores (3%; SF < 12 microg

17 acid (IDA) regenerable sorbents by forming a IDA:Cu(II):Con A sandwich affinity structure that has hi

18 ) and INFLORESCENCE DEFICIENT IN ABSCISSION (IDA) is induced in cauline leaf abscission zones when th

19 tion, INFLORESCENCE DEFICIENT IN ABSCISSION (IDA), suggesting that signalling through IDA and its pro

20 ns in Inflorescence Deficient in Abscission (IDA), which encodes a predicted secreted small protein,

21 ptide INFLORESCENCE DEFICIENT IN ABSCISSION (IDA), which signals through the leucine-rich repeat rece

22 s the INFLORESCENCE DEFICIENT IN ABSCISSION (IDA)-derived peptide and its receptors, HAESA (HAE) and

23 ilotriacetic acid (NTA), iminodiacetic acid (IDA) and desferrioxamine B (DFOB), as well as with Suwan

24 otriacetic acid (NTA) or iminodiacetic acid (IDA) in-house-fabricated columns.

25 ilized on Cu(II)-charged iminodiacetic acid (IDA) regenerable sorbents by forming a IDA:Cu(II):Con A

26 ) (through the bond with iminodiacetic acid (IDA)), as an adsorbent for the chromatographic process t

27 A series of iminodiacetic acid (IDA)-linked polyamides (DpPyPyPy-IDA-PyPyPyDp) were prep

28 ques with information-dependent acquisition (IDA) approaches more and more often.

29 Information-dependent acquisition (IDA) functionality was used to combine s-MRM with enhanc

30 vey scan, information-dependent acquisition (IDA) triggered collection of EPI spectra, and library se

31 ware tool information dependent acquisition (IDA) was used to automatically obtain information about

32 Information Dependent Acquisition (IDA), an extra confirmation tool for samples that contai

33 including information-dependent acquisition (IDA), SWATH (sequential window acquisition of all theore

34 in adults with acute myeloid leukemia (AML), IDA achieves higher remission rates and longer remission

35 An IDA electrode amplifies the signal by recycling an elect

36 da) and blade-on-petiole1 (bop1)/bop2 and an IDA-overexpressing line.

37 In multivariate analysis, IDA random assignment remained associated with a higher

38 A central hydroxyproline residue anchors IDA to the receptor.

39 n deficiency (adjusted OR 8.1; P = .001) and IDA (adjusted OR 5.1; P = .01) at the end of the malaria

40 on the association between achlorhydria and IDA.

41 in the association between achlorhydria and IDA.

42 th AML, younger age, favorable-risk AML, and IDA treatment predict a better long-term outcome.

43 Anemia and IDA are not synonymous, even among low-income minority w

44 ngs in kindled rodents to show that CLON and IDA can have opposing effects on kindling development in

45 At 9 mo, the control and IDA-Fe groups no longer differed.

46 to be a risk factor for iron deficiency and IDA in children in a malaria-endemic environment and thi

47 s displayed increasing prevalences of ID and IDA during pregnancy, which peaked in the middle to late

48 I and produce prevalence estimates of ID and IDA for each target population.

49 sychological outcomes associated with ID and IDA in premenopausal women, as the prevalence of ID and

50 enopausal women, as the prevalence of ID and IDA is often greater in premenopausal women than other p

51 ally representative data suggest that ID and IDA remain a concern in the United States.

52 s were 6-21 mug/L, and prevalences of ID and IDA were 28-85% and 21-35%, respectively.

53 Various definitions for ID and IDA were applied across studies.

54 tion and excess stores, as well as on ID and IDA, is needed.

55 iron status and lower prevalences of ID and IDA, which were dependent on the dose of iron and compli

56 of 2500-2999 g are at higher risk of ID and IDA.

57 evalence of anemia, low iron stores, ID, and IDA in children participating in the Special Supplementa

58 th significant reductions in AI, but IDA and IDA+AI remain unresolved.

59 n-based interventions are needed for IDA and IDA+AI, and monitoring of iron biomarkers reveals a wind

60 nd it interacts with the cytoplasmic ODA and IDA assembly factor DNAAF2 (KTU).

61 scence staining showed an absence of ODA and IDA proteins in cilia from an affected individual, thus

62 (2) CLON retarded and IDA accelerated kindling rate, defined as the number of

63 ons of outer and inner dynein arms (ODAs and IDAs, respectively).

64 Iron-deficiency anemia (IDA) affects millions of persons worldwide, and is assoc

65 semia trait (TT) and iron deficiency anemia (IDA) are the two most common anemia types and can have a

66 ards associated with iron deficiency anemia (IDA) are well known, concerns about risks associated wit

67 Iron deficiency anemia (IDA) has detrimental effects on individuals and societie

68 tablish the cause of iron deficiency anemia (IDA) in a substantial proportion of patients.

69 supplementation for iron deficiency anemia (IDA) in asymptomatic, nonanemic pregnant women could imp

70 e of anemia, ID, and iron deficiency anemia (IDA) in California remains unclear.

71 pylori infection and iron deficiency anemia (IDA) in humans.

72 hydria is a cause of iron deficiency anemia (IDA) in humans.

73 ic disease (ACD) and iron deficiency anemia (IDA) in patients with RA.

74 iron deficiency and iron-deficiency anemia (IDA) in the United States.

75 The prevalence of iron deficiency anemia (IDA) increased over the malaria season (P < .001).

76 early in pregnancy, iron deficiency anemia (IDA) is associated with a > 2-fold increase in the risk

77 Sub-Saharan Africa, iron-deficiency anemia (IDA) is common, and many complementary foods are low in

78 Iron deficiency anemia (IDA) is relatively common in the third trimester of preg

79 Preoperative iron deficiency anemia (IDA) occurs frequently; however if left untreated, incre

80 Iron deficiency anemia (IDA) still remains universally problematic worldwide.

81 deficiency (ID) and iron deficiency anemia (IDA) was 10-32% and 2-5%, respectively, depending on the

82 ron deficiency (ID), iron deficiency anemia (IDA), and other measures of iron nutriture in 1016 elder

83 concentrations were iron deficiency anemia (IDA), anemia of inflammation (AI), and multifactorial an

84 urden is a result of iron deficiency anemia (IDA).

85 5% (n = 5) developed iron deficiency anemia (IDA).

86 hed iron stores) and iron deficiency anemia (IDA; poor iron stores and diminished hemoglobin), affect

87 lence of iron deficiency (ID) and ID anemia (IDA) among toddlers, nonpregnant females, and pregnant f

88 icits despite prompt diagnosis of ID anemia (IDA) by screening of hemoglobin concentrations followed

89 orld, with consequences of ID and ID anemia (IDA) in young children including behavioral and cognitiv

90 nflammation (AI), and multifactorial anemia (IDA+AI).

91 t concentration, and the inter-duplex angle (IDA) between arms.

92 (needle insertion only) in the same animals; IDA significantly reduced thresholds.

93 lone; n = 54) and ODA plus inner dynein arm (IDA) defects (ODA + IDA; n = 18) versus subjects with ID

94 aused by dual loss of the inner dynein arms (IDAs) and outer dynein arms (ODAs), which power cilia an

95 ounded by an interdigitated electrode array (IDA) patterned on a zinc selenide (ZnSe) substrate.

96 electrode E1 of an Au interdigitated array (IDA) electrode device.

97 ionization MS with an interdigitated array (IDA) electrode enabling chemical analysis of electrolyze

98 ed immunoassay with an interdigitated array (IDA) electrode.

99 DE) amperometry and an interdigitated array (IDA) electrode.

100 Carbon interdigitated array (IDA) electrodes with features sizes down to 1.2 mum were

101 to an indium-tin oxide interdigitated array (IDA) microelectrode.

102 latform is based on an interdigitated array (IDA) of electrodes.

103 across a 5 microm gap interdigitated array (IDA) of gold electrodes behaved as "hydrogen sensors".

104 monolayer (SAM)-coated interdigitated-array (IDAs) gold-platinum electrodes.

105 anar (using interdigitated electrode arrays (IDAs)) configurations were investigated.

106 Interdigitated gold microelectrode arrays (IDAs) were first modified with a self-assembled monolaye

107 ne gastritis implies a disease presenting as IDA many years before the establishment of clinical coba

108 es a metal-ion indicator displacement assay (IDA) has been devised.

109 oped by using indicator displacement assays (IDAs), and the protocol was then applied to the vicinal

110 ssible role of diet, in H. pylori-associated IDA.

111 By attaching IDA-bound Cu(2+) to benzenesulfonamide via different cha

112 , Joint Indicator A, to discriminate between IDA, TT, and healthy children in a Chinese population.

113 ion in managing IDA, and distinguish between IDA from the anemia of chronic disease.

114 iated with significant reductions in AI, but IDA and IDA+AI remain unresolved.

115 nd ID were prevalent in this WIC sample, but IDA was uncommon.

116 sm of phosphorescence of Mn-doped ZnS QDs by IDA is a combined dynamic and static quenching.

117 indicate that H. pylori infection can cause IDA and that the composition of the diet influences the

118 ls, P-element-mediated gene silencing caused IDA and ODA defects, proprioception deficits, and steril

119 In addition, metal-charged IDA paper promises to be generally useful for functional

120 CD-affected families afflicted with combined IDA and ODA defects, we found that 6/38 (16%) carried bi

121 orted iminodiacetic acid-copper ion complex (IDA-Cu) immobilized onto gold nanoparticles (GNPs)-modif

122 for anemia with iron-responsive components (IDA, IDA+AI).

123 Strategies to control IDA include daily and intermittent iron supplementation,

124 public health interventions for controlling IDA in low- and middle-income countries, with an emphasi

125 s revising global guidelines for controlling IDA.

126 d cheese whey was passed through the cryogel-IDA-Cu(2+) system.

127 DDA/IDA methods do not exploit valuable information that is

128 a- or information-dependent acquisition (DDA/IDA), where fragmentation mass candidates are selected b

129 the mean hemoglobin level, iron-deficiency (IDA) and non-iron-deficiency anemia (NIDA), and inflamma

130 nfection was associated with iron deficiency/IDA regardless of the presence or absence of peptic ulce

131 Even using several different IDA settings, it was observed that IDA failed to trigger

132 s defects with microtubular disorganization (IDA/CA/MTD; n = 40).

133 ddition of ds-DNA caused formation of ds-DNA/IDA complex and recovered the RTP signal of Mn-doped ZnS

134 xicity prompted replacement of IDA with DNR (IDA/DNR) on days 10 through 14 for the remaining 28 pati

135 cetic acid (IDA)-linked polyamides (DpPyPyPy-IDA-PyPyPyDp) were prepared and constitute polyamides jo

136 vailable for download at http://lyle.smu.edu/IDA/EMMSA/, implemented in JAVA and supported on MS Wind

137 No energetic contribution of either IDA or triethylene glycol spacer was found (at least in

138 An interdigitated array electrode (IDA) is fabricated on an IR-transparent substrate for us

139 sults support the hypothesis that C. elegans IDA-1 is the functional homolog of IA-2 and phogrin in m

140 In C. elegans, IDA-1-expressing cells comprised a subset of those expre

141 ds (IDA, MMDF (multiple mass defect filters)-IDA, SWATH, or MS(All)) using an ultrahigh-performance l

142 eceived high DNR doses (cure rate, 27.4% for IDA v 15.9% for DNR; P = .049).

143 with serum ferritin higher than expected for IDA, mutations involving iron trafficking and regulation

144 panded and improved treatment modalities for IDA.

145 Iron-based interventions are needed for IDA and IDA+AI, and monitoring of iron biomarkers reveal

146 MYND10 is a cytoplasmic protein required for IDA and ODA assembly and that its variants cause ciliary

147 current screening and treatment strategy for IDA fails to achieve this goal for 3 reasons.

148 ce that routine prenatal supplementation for IDA improves maternal or infant clinical health outcomes

149 k Force on screening and supplementation for IDA in pregnancy.

150 s of DRM detected in microsomal samples from IDA, SWATH, and MS(All) methods, respectively.

151 eastfed infants with a birth weight >2500 g, IDA is uncommon before 6 mo, but male infants and those

152 The gold IDA was patterned on the ZnSe substrate using a photolit

153 es of isotope dilution for data analysis (GS-IDA).

154 t delivery, and 18% of women (n = 11/61) had IDA.

155 inase (LRR-RK) HAESA and the peptide hormone IDA.

156 The prevalence of ID, IDA, and depleted iron stores was unaffected by the pres

157 in Europe, we describe the prevalence of ID, IDA, iron repletion, and excess stores with the use of p

158 anemia with iron-responsive components (IDA, IDA+AI).

159 apped Mn doped ZnS quantum dots/ Idarubicin (IDA) nanohybrids were used as novel room temperature pho

160 Randomized comparisons of idarubicin (IDA) with daunorubicin (DNR) show that in adults with ac

161 alf received the alpha2 antagonist idazoxan (IDA; 0.33 nmol).

162 Binding of idazoxan (IDA) to imidazoline receptors of the I2 subtype in astro

163 Here, we identified IDA-like 6 (IDL6), a gene that is prominently expressed

164 or identifying ID and of 97% for identifying IDA.

165 rhydria) was 44% in patients with idiopathic IDA and 1.8% in healthy controls.

166 s was demonstrated for Pb(II)-PSS and Pb(II)-IDA systems.

167 Benzenesulfonamide and iminodiacetate (IDA)-conjugated Cu(2+) independently interact at the act

168 lfonamide prong and a cupric iminodiacetate (IDA-Cu(2+)) prong separated by linkers of different leng

169 The chelator moiety, iminodiacetate (IDA), was linked to the polymerizable phosphatidylethano

170 ealed that the attachment of iminodiacetate (IDA)-Cu(2+) to benzenesulfonamide (via a triethylene gly

171 GNPs acts as a substrate to immobilize IDA-Cu coordinative complex and its interaction with Lys

172 in a statistically significant difference in IDA incidence at term, favoring supplementation (risk ra

173 ormone by binding to an Arg-His-Asn motif in IDA.

174 was to determine the effects of diet-induced IDA on intrauterine growth and neonatal iron status in a

175 incidence and severity of H. pylori-induced IDA.

176 The genes that encode HAE/HAESA-LIKE2, IDA, NEVERSHED, and MAPK KINASE4 and 5 are all necessary

177 ctice guidelines for diagnosing and managing IDA should include the design of an algorithm that is in

178 cs, which will provide direction in managing IDA, and distinguish between IDA from the anemia of chro

179 t compounds were analyzed with four methods (IDA, MMDF (multiple mass defect filters)-IDA, SWATH, or

180 Cells were incubated with 1-100 microM IDA, and at 24 h NOS-2 expression assessed.

181 eved after a 8 h preincubation in 100 microM IDA.

182 ered for MS(2) acquisition with IDA and MMDF-IDA methods, respectively.

183 Ni2+-IDA paper was used to identify a PCNA- point mutant that

184 ickel-charged iminodiacetic acid paper (Ni2+-IDA paper).

185 and macromolecules were transferred to Ni2+-IDA paper.

186 hildren aged 9 to 48 months with nutritional IDA (assessed by history and laboratory criteria) that w

187 ion in infants and children with nutritional IDA.

188 A plus inner dynein arm (IDA) defects (ODA + IDA; n = 18) versus subjects with IDA and central appara

189 with ciliary outer and inner dynein arm (ODA+IDA) defects.

190 utations in SPAG1 cause PCD with ciliary ODA+IDA defects and that exome sequencing is useful to ident

191 o had defined ciliary ultrastructure had ODA+IDA defects.

192 unrelated affected individuals (62 with ODA+IDA defects, 35 with ODA defects, 1 without available ci

193 esults suggest that the sequential action of IDA, HAE and HSL2, and a MAP kinase cascade regulates th

194 l iron measures) were 16.2% and 8.8%, and of IDA (ID with low hemoglobin) were 3.4% and 3.2% on the b

195 transferrin receptor (sTfR), a biomarker of IDA, on a photonic crystal (PC) biosensor was establishe

196 achlorhydria can be an independent cause of IDA.

197 ion and, after excluding all other causes of IDA, 64% to 75% of such patients are permanently cured b

198 med achlorhydria preceded the development of IDA in time.

199 on, which persisted after the development of IDA.

200 ry gene expression, we tested the effects of IDA on astroglial nitric oxide synthase type-2 (NOS-2) e

201 It is unknown how expression of IDA in the abscission zone leads to HAESA activation.

202 Helicobacter pylori, and hereditary forms of IDA is recommended.

203 Analysis of the function of IDA-1 should contribute to our understanding of the func

204 The long-term impact of IDA was still observed in patients younger than age 65 y

205 Second, brain ID, independently of IDA, is responsible for long-term neurological deficits.

206 Management of IDA is based on supplementation combined with effective

207 approach for the diagnosis and management of IDA.

208 ity of substituting 4 mg of DNR with 1 mg of IDA in intensive-timing daunorubicin-based induction the

209 , starting iron treatment after the onset of IDA is less effective than prevention.

210 ired MS(2) spectra decreased in the order of IDA, SWATH, and MS(All) methods.

211 The predictors of IDA (8%) were male sex (AOR: 7.6; 95% CI: 2.5, 23.0), bi

212 ection was associated with the prevalence of IDA (prevalence odds ratio (POR) = 2.6, 95% CI: 1.5, 4.6

213 Also, the detection rate of IDA compared to SWATH was investigated in a forced coelu

214 5 patients, toxicity prompted replacement of IDA with DNR (IDA/DNR) on days 10 through 14 for the rem

215 ary outcomes included complete resolution of IDA (defined as hemoglobin concentration >11 g/dL, mean

216 Proportion with a complete resolution of IDA was higher in the ferrous sulfate group (29% vs 6%;

217 The PTP catalytic site consensus sequence of IDA-1, like IA-2 and phogrin, diverged and would not be

218 Toxicity of IDA/DNR was not different from that of DNR/DNR in CCG-28

219 However, the 5- to 11-year-old IDA/CA/MTD group had more lobes of bronchiectasis (media

220 inescence profile in the LECs constructed on IDAs, showing that the charge injection in such devices

221 the mother does not have iron deficiency or IDA.

222 Overall, IDA-based methods acquired qualitatively better MS(2) sp

223 Plants overexpressing IDA in a hae hsl2 background have abscission defects, in

224 125 mg FeSO(4) (IDA-Fe; n = 34) or placebo (IDA-PL; n = 30) daily from 10 wk to 9 mo postpartum.

225 d, N-(phosphonomethyl)iminodiacetic acid (PM-IDA), in aqueous solution.

226 On charging with nickel ions, the PM-IDA functionalized iron oxide nanoparticles exhibited hi

227 der optimal conditions, RTP intensity of QDs/IDA nanohybrids increased linearly with the concentratio

228 t difference in serum sTfR values between RA-IDA and RA-ACD (40.2 +/- 14.0 versus 23.9 +/- 5.3 nmoles

229 ncrease in serum sTfR values seen between RA-IDA and RA-ACD.

230 on uptake at 4 hours was more evident for RA-IDA (3.96 +/- 1.73 versus 1.66 +/- 0.66; P = 0.03) than

231 ivided as RA-ACD (marrow iron present) or RA-IDA (marrow iron absent) on the basis of visible reticul

232 rategies to increase iron intakes and reduce IDA at this age.

233 4% to 6% of patients with obscure refractory IDA have celiac disease, and autoimmune gastritis is enc

234 ic evaluation with unexplained or refractory IDA, screening for celiac disease, autoimmune gastritis,

235 50% of patients with unexplained refractory IDA have active H pylori infection and, after excluding

236 nd management of unexplained, or refractory, IDA.

237 Here we show that this process requires IDA, HAE, and HSL2.

238 In the root, IDA is strongly auxin-inducible and dependent on key reg

239 hroblasts obtained from patients with simple IDA showed a significant increase in surface TfR express

240 althy volunteers, and 6 patients with simple IDA.

241 s whereas currents measured for the smallest IDA electrodes, that were larger than the simulated curr

242 nded particulate reagent-iminodiacetate (SPR-IDA) as resin materials which are embedded in an ether-b

243 Limited evidence supports IDA treatment with preoperative IV iron.

244 ral function and is 3 times more common than IDA in toddlers.

245 However, we found no credible evidence that IDA caused gastritis or that IDA preceded the developmen

246 different IDA settings, it was observed that IDA failed to trigger relevant compounds.

247 e evidence that IDA caused gastritis or that IDA preceded the development of achlorhydria.

248 These results show that IDA inhibits NOS-2 activity and protein expression in gl

249 Here we show that IDA is sensed directly by the HAESA ectodomain.

250 The IDA-Cu(2+) cryogel system was also able to capture lacto

251 nown that the complexity of a sample and the IDA settings might prevent important compounds from bein

252 inities of known inhibitors by attaching the IDA-Cu(2+) groups to interact with the surface-exposed h

253 mical recycling of the PAP/PQI couple by the IDA electrode lowered the limit of detection to 90 ng/mL

254 ified to enable the substrate containing the IDA to be used for electrooptical measurements in transm

255 the child responsiveness scale than did the IDA-PL group (P = 0.007-0.032), whose iron status remain

256 tly greater in the control group than in the IDA groups (P = 0.028 and 0.009, respectively).

257 Median body mass index was lower in the IDA/CA/MTD group (46th percentile) versus the ODA groups

258 Median FEV1 was worse in the IDA/CA/MTD group (72% predicted) versus the combined ODA

259 ordinates to the active site Zn(2+) ion; the IDA-Cu(2+) prong of the tightest-binding inhibitor, BR30

260 electrode spacing was developed to model the IDA electrode response.

261 The fabrication of the IDA and its unique sampling geometry for transmission sp

262 pretation of the impedance components of the IDA microelectrode system.

263 electrochemical cell and the behavior of the IDA microelectrode, an equivalent circuit, consisting of

264 distinguish between the binding modes of the IDA-linked polyamides.

265 t span the gap between the electrodes of the IDA.

266 The collection efficiency depends on the IDA electrode spacing, which ranged from around 2.7 to 1

267 10 muL of 1 muM solution of dopamine on the IDA.

268 It was found that the IDA linker exerts a unique influence over the DNA bindin

269 dies and the binding of E. coli cells to the IDA microelectrode surface increased the electron-transf

270 Upon the recognition of the Lys to the IDA-Cu, the peak current decreased due to the hindered e

271 As an example, we used the IDA peptide HAE/HSL2 receptor signaling system known to

272 er with a following chemical reaction at the IDAs with larger gaps whereas currents measured for the

273 In this IDA approach, a sulfur mustard simulant (the analyte) is

274 ON (IDA), suggesting that signalling through IDA and its proposed receptors, HAESA and HAESA-LIKE2, m

275 ts, indicating HAE and HSL2 are epistatic to IDA.

276 that SWATH's detection rate was superior to IDA, which failed to trigger approximately 10% of the an

277 A trench IDA electrode was incorporated into a poly(dimethyl silo

278 appropriate animal model for third-trimester IDA in women.

279 eloping brain is already iron deficient when IDA is diagnosed owing to the prioritization of availabl

280 ented onset of spontaneous seizures, whereas IDA precipitated onset of spontaneous seizures in 100% o

281 To determine whether IDA affected glial inflammatory gene expression, we test

282 degree of inhibition was diminished whether IDA was added after LPS plus cytokine mixture.

283 different among the three regimens: 14% with IDA/IDA, 7% with IDA/DNR, and 9% with DNR/DNR.

284 he three regimens: 14% with IDA/IDA, 7% with IDA/DNR, and 9% with DNR/DNR.

285 ere not triggered for MS(2) acquisition with IDA and MMDF-IDA methods, respectively.

286 No genotypes were associated with IDA before the malaria season.

287 Reinfection was associated with IDA for high reinfection intensities only.

288 Crystal structures of HAESA in complex with IDA reveal a hormone binding pocket that accommodates an

289 PC2 homolog KPC-2 (C51E3.7), consistent with IDA-1 being a component of neuropeptide-containing dense

290 than in uninfected gerbils, consistent with IDA.

291 n contrast to NE, continuous incubation with IDA was required to achieve suppression.

292 associated with NIDA (P=0.019) but not with IDA (P=0.29).

293 ust 2011 and November 2014, 72 patients with IDA were assigned to receive either IV iron or usual car

294 The percentages with IDA were 2% in Sweden, 4% in Mexico, 5-11% in Honduras,

295 astrocytes and C6 cells, preincubation with IDA dose-dependently inhibited nitrite accumulation (IC5

296 ed potential benefits of early response with IDA.

297 cts (ODA + IDA; n = 18) versus subjects with IDA and central apparatus defects with microtubular diso

298 and genotype groups, but worse in those with IDA/CA/MTD ultrastructural defects, most of whom had bia

299 ot C6 cells, longer preincubation times with IDA yielded significantly greater suppression, and maxim

300 e-risk AML; P < .001) and those treated with IDA (16.6% v 9.8% with DNR; P = .018).