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1 ctivation of the transcription factor IRF-3 (IFN regulatory factor 3).
2 TBK1, TAK1, and phosphorylation of TBK1 and IFN regulatory factor 3.
3 IFN production in vitro, acting upstream of IFN regulatory factor 3.
4 infected cells are prevented from activating IFN regulatory factor 3.
5 resulting in inhibition of signaling through IFN regulatory factor 3.
6 molecule STING and the transcription factor IFN regulatory factor 3.
7 at specifically phosphorylates and activates IFN regulatory factor 3.
8 phosphorylation and nuclear translocation of IFN regulatory factor-3.
9 nhancement of reporter activity by predicted IFN regulatory factor 3/7 binding sites in the CAD risk
10 assay; and 3) enhancer activity of predicted IFN regulatory factor 3/7 binding sites within the 9p21
14 umulation without inhibiting dimerization of IFN regulatory factor 3, a protein that is essential for
16 tion and increases LPS-induced NF-kappaB and IFN regulatory factor 3 activation and IL-10 secretion,
17 complex formation and facilitating TBK1 and IFN regulatory factor 3 activation and the induction of
18 n sMLA-induced IkappaB kinase alpha/beta and IFN regulatory factor 3 activation and with restrained e
20 and S1P1 acting through PI3K enhancement of IFN-regulatory factor 3 activation increase IFN-beta exp
24 e ability of NP to suppress translocation of IFN regulatory factor 3 and block activation of the inna
27 which ablates RIG-I signaling of downstream IFN regulatory factor 3 and NF-kappaB activation, attenu
29 hat viperin/cig5 expression was dependent on IFN regulatory factor 3 and NF-kappaB signaling, and tha
32 inhibit the MyD88-independent activation of IFN regulatory factor 3 and production of IFN-beta in re
35 n the activation of the transcription factor IFN regulatory factor 3 and the molecule UNC93B1, indica
36 site 6.1 kb downstream of ifnb1, along with IFN regulatory factor-3 and CREB binding protein only du
37 In contrast, LPS-induced phosphorylation of IFN regulatory factor-3 and expression of IFN-beta or th
38 ts displayed a significantly higher level of IFN regulatory factor-3 and NF-kappaB activity compared
39 l signaling protein, nuclear localization of IFN regulatory factor 3, and augmentation of IFN-stimula
40 inding kinase 1, activation of NF-kappaB and IFN regulatory factor 3, and induction of IL-8 and IFN-b
41 eta depended on the downstream transcription IFN regulatory factor 3, and on activation of NF-kappaB
42 TLR4 pathway in monocytes by activating Syk, IFN regulatory factor 3, and STAT1, which resulted in en
43 ons with the IPS-1 adaptor protein to signal IFN regulatory factor 3- and NF-kappaB-responsive genes.
44 IFN-beta transcription factors NF-kappaB and IFN regulatory factor 3 are not activated for nuclear tr
45 Similarly, phosphorylation of IkappaB and IFN regulatory factor 3 as well as cytokine expression w
46 ll-like receptor 4 signaling, and subsequent IFN regulatory factor 3 binding to IFN-beta promoter.
49 n-induced ER stress augmented recruitment of IFN regulatory factor-3, CREB binding protein/p300, and
51 Polyinosinic-polycytidylic acid activated IFN regulatory factor 3 dimerization and phosphorylation
52 d nonimmune cells that converges on TBK1 and IFN regulatory factor 3 for activation of IFN-beta gene
53 ain-containing adapter inducing IFN-beta --> IFN regulatory factor 3 --> type I IFN is the major axis
54 IFN-beta (TRIF)-dependent phosphorylation of IFN regulatory factor 3 in addition to TIR-containing ad
55 regates that led to activation of STAT-1 and IFN regulatory factor-3 in human macrophages, indicating
56 nslocation of transcription factors RelA and IFN regulatory factor-3 in IFN-alpha-primed HUVECs befor
57 underwent nuclear translocation of RelA and IFN regulatory factor-3 in response to dsRNA, whereas le
58 uated activation of the transcription factor IFN regulatory factor 3, involved in the MyD88-independe
60 The level of IFN induction correlated with IFN regulatory factor 3 (IRF-3) activation, in that IRF-
61 g TLR3 to kinases responsible for activating IFN regulatory factor 3 (IRF-3) and NF-kappaB, transcrip
62 on activated both transcription factors, the IFN regulatory factor 3 (IRF-3) and nuclear factor kappa
63 upt IFN-beta gene transcription induction by IFN regulatory factor 3 (IRF-3) but do so at different p
64 ole of the IKK-related kinase IKKepsilon and IFN regulatory factor 3 (IRF-3) in the activation of ant
66 LP by dsRNA was dependent upon NF-kappaB and IFN regulatory factor 3 (IRF-3) signaling via TLR3 as in
68 ay partially involve limiting the ability of IFN regulatory factor 3 (IRF-3) to function as a transcr
69 its downstream signaling molecules TRIF and IFN regulatory factor 3 (IRF-3) using a dominant-negativ
70 us correlated with an enhanced activation of IFN regulatory factor 3 (IRF-3), NF-kappaB, and ATF-2 in
71 and chemokine genes in a manner dependent on IFN regulatory factor 3 (IRF-3), TANK-binding kinase 1 (
73 ection-induced transcriptional activation of IFN regulatory factor 3 (IRF-3)-responsive mammalian pro
75 IFN promoter-stimulator 1 (IPS-1), activates IFN regulatory factor-3 (IRF-3) and the host IFN-alpha/b
76 rotein expression consistent with a role for IFN regulatory factor-3 (IRF-3) in the expression of IL-
79 ells without inducing the phosphorylation of IFN-regulatory factor 3 (IRF-3), a latent cellular trans
80 promoter by blocking the phosphorylation of IFN-regulatory factor 3 (IRF-3), a transcription factor
82 TRIF)-dependent signaling pathway leading to IFN regulatory factor 3 (IRF3) activation and induction
84 vation of the cellular transcription factors IFN regulatory factor 3 (IRF3) and signal transducers an
85 Consistent with this, the phosphorylation of IFN regulatory factor 3 (IRF3) and the production of IFN
86 genes, and in all systems examined thus far, IFN regulatory factor 3 (IRF3) has been described as an
87 , TLR4, TLR2/4, MyD88, TRIF, MyD88/TRIF, and IFN regulatory factor 3 (IRF3) KO mice, treated them wit
88 H5N1 viruses elicited significantly less IFN regulatory factor 3 (IRF3) nuclear translocation, as
89 ch inhibit IFN induction by targeting either IFN regulatory factor 3 (IRF3) or NF-kappaB, respectivel
92 PEITC preferentially inhibited TLR3-mediated IFN regulatory factor 3 (IRF3) signaling and downstream
93 ntially reduced binding of these factors and IFN regulatory factor 3 (IRF3) to IFN-beta or ISGs promo
95 Data in this study show that cFLIPL inhibits IFN regulatory factor 3 (IRF3), a transcription factor c
96 tly of MyD88, resulting in the activation of IFN regulatory factor 3 (IRF3), a transcription factor r
97 B activation and virus-induced activation of IFN regulatory factor 3 (IRF3), whereas bone marrow-deri
99 I interferons (IFNs), despite maintenance of IFN regulatory factor 3 (IRF3)-dependent IFN-stimulated
100 beta interferon (IFN-beta) promoter and the IFN regulatory factor 3 (IRF3)-responsive IFN-stimulated
110 y the activation of constitutively expressed IFN regulatory factor 3, IRF3, which in turn leads to th
111 infection, type I IFN signaling by STING and IFN regulatory factor 3 is detrimental to the host durin
112 y, we report that signaling through TBK1 and IFN regulatory factor 3 is required for T. cruzi-mediate
113 nse to a self-Ag, DC-intrinsic expression of IFN regulatory factor 3 is required to induce optimal pr
114 after infection with influenza A virus, but IFN regulatory factor-3 is activated and the transcripti
115 of the transcription factors, NF-kappaB and IFN regulatory factor 3, leading to proinflammatory cyto
118 me-edited knockout human cells, we show that IFN regulatory factor 3-mediated IFN induction and downs
119 Functional genetic analyses revealed that IFN regulatory factor 3-mediated pathways that resulted
122 nal IFN were also produced in VLP-inoculated IFN regulatory factor 3 null (IRF3(-/-)) mice, indicatin
124 dependent on its ability to activate either IFN regulatory factor-3 or NF-kappa B but was dependent
125 ining adaptor inducing interferon (IFN) beta-IFN regulatory factor 3 pathway induce less IL-10, and a
126 domain-containing adapter inducing IFN-beta-IFN regulatory factor 3 pathway is required for Ezrin-de
127 we demonstrate the requirement for the TBK1-IFN regulatory factor-3 pathway in host defense against
128 duced activation of the NF-kappaB, MAPK, and IFN regulatory factor-3 pathways and decreased TLR4 degr
129 G aggregation, and TANK-binding kinase 1 and IFN regulatory factor 3 phosphorylation in inflammasome-
132 nt in melanoma 2, DNA-dependent activator of IFN regulatory factor 3, RNA polymerase III, or high-mob
135 protein, MAVS, that activates NF-kappaB and IFN regulatory factor 3 to induce type-I interferons.
136 so required for gene induction driven by the IFN regulatory factor 3 transcription factor activated b
137 RIF-dependent pathways through NF-kappaB and IFN regulatory factor 3 transcription factors, respectiv
139 iates with TANK-binding kinase 1 to regulate IFN regulatory factor 3 translocation and phosphorylatio
140 signaling pathways, IL-15, TNF, IL-1, IL-6, IFN regulatory factor 3, type I IFN receptor, adaptive i
141 Specific interaction between KSHV viral IFN regulatory factor 3 (vIRF3) and the HIF-1 alpha subu
143 he absence of DOK3, the transcription factor IFN regulatory factor 3 was not phosphorylated and could
144 apoptosis could activate NF-kappa B but not IFN regulatory factor-3, yet the activation of NF-kappa
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