ライフサイエンスコーパス: IFN-alphabetaR

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1 IFN-alphabetaR(-/-) mice were also found to exhibit prof

2 IFN-alphabetaR(-/-)Rag(-/-) mice were given injections o

3 Ev knockout mice lacking the IFN-alphabetaR (IFN-alphabetaR(-/-)), the IFN-gammaR (IFN-gammaR(-/-)),

4 nduction of TNF is TLR2/4, MyD88, STAT1, and IFN-alphabetaR independent, but TNF protein release requ

5 protected alpha/beta-IFN-receptor-deficient (IFN-alphabetaR(-/-)) mice from lethality following Sindb

6 s from type I interferon receptor-deficient (IFN-alphabetaR(-/-)) mice, which had reduced apoptosis,

7 wild-type over IFN-alphabetaR gene-deleted (IFN-alphabetaR-/-) mice.

8 Bronchoalveolar lavage fluid from IFN-alphabetaR-/- mice yielded 7- to 8-fold fewer leukoc

9 Foxp3 expression by Tregs transferred from IFN-alphabetaR(-/-) mice was significantly lower than th

10 ansfer of CD4(+)CD45RB(lo)CD25(+) Tregs from IFN-alphabetaR(-/-) mice did not prevent T-cell inductio

11 nt and activation by the human type I IFNAR (IFN-alphabetaR), it is not sufficient to restore this pr

12 nhibition of IFN-alphabeta was manifested in IFN-alphabetaR(-/-) DCs, which lack the positive feedbac

13 ity to decrease Sindbis virus replication in IFN-alphabetaR(-/-) mice or prolong survival of ISG15(-/

14 sma membrane receptor for type I interferon (IFN-alphabetaR(-)(/)(-)) have revealed that IFN signalin

15 TLR2/4-independent pathway that may involve IFN-alphabetaR and STAT1.

16 ally expressed in the presence or absence of IFN-alphabetaR-mediated signaling, further elucidating i

17 h the phenotype was less severe than that of IFN-alphabetaR(-)(/)(-) mice.

18 ible protein-10, and TLR-3 in lung tissue of IFN-alphabetaR-/- mice when compared with wild type.

19 recombinant IFN-alpha following transfer of IFN-alphabetaR(-/-) RB(hi) T cells and CD4(+)Foxp3(+) ce

20 mic medulla secrete IFN-alpha, which acts on IFN-alphabetaR-expressing immature thymocytes to induce

21 pression is MyD88 dependent do not depend on IFN-alphabetaR or STAT1.

22 D45RB(lo) CD25(+) T cells, from wild-type or IFN-alphabetaR(-/-) mice into Rag1(-/-) recipients.

23 ponse genes preferentially in wild-type over IFN-alphabetaR gene-deleted (IFN-alphabetaR-/-) mice.

24 type I IFNs through IFN-alphabeta receptor (IFN-alphabetaR) for protection.

25 ndependent transcriptional responses require IFN-alphabetaR and STAT1, but not IFN-gamma.

26 ed in mice in which T cells were selectively IFN-alphabetaR-deficient.

27 that signal through the common cell surface IFN-alphabetaR.

28 These results indicate that IFN-alphabetaR signaling in both B and T cells plays an

29 h B cells were selectively deficient for the IFN-alphabetaR.

30 ing cells derived from mice deficient in the IFN-alphabetaR, we show that both TLR3 and TLR4 require

31 series of 129SvEv knockout mice lacking the IFN-alphabetaR (IFN-alphabetaR(-/-)), the IFN-gammaR (IF

32 is associated with prolonged survival of the IFN-alphabetaR-/- mice (50% survival at 10.8 +/- 0.6 day

33 Signaling through the IFN-alphabetaR was required for the timely recruitment a

34 to infection, even when the phagocytes were IFN-alphabetaR(+/+).

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