ライフサイエンスコーパス: IFN-gamma production

1 ning), this was not the case for others (eg, IFN-gamma production).

2 psis by activating NK cells and facilitating IFN-gamma production.

3 d counteract the benefits of increased early IFN-gamma production.

4 nvironment, permitting T cell glycolysis and IFN-gamma production.

5 ic effectiveness was critically dependent on IFN-gamma production.

6 T cells but not CD8 T cells were impaired in IFN-gamma production.

7 h22 cells displayed marked plasticity toward IFN-gamma production.

8 tion gene 3 (LAG-3) expression and a lack of IFN-gamma production.

9 mor microenvironment act to reduce MAIT cell IFN-gamma production.

10 lls, which promoted T cell proliferation and IFN-gamma production.

11 -stimulatory signal like CD28 that triggered IFN-gamma production.

12 diabetes development, T-cell expansion, and IFN-gamma production.

13 escribed as a Th1-mediated process involving IFN-gamma production.

14 lished the effect of fenofibrate in reducing IFN-gamma production.

15 chemokine, with a negligible effect on IL-10/IFN-gamma production.

16 ed glycolytic reprogramming was required for IFN-gamma production.

17 P1C)-stimulated and IL-12/18 cytokine-primed IFN-gamma production.

18 nd spleens of infected mice at times of peak IFN-gamma production.

19 se, DNA-HSP65 and CpG/CFP promoted IL-10 and IFN-gamma production.

20 ighest level of lymphocyte proliferation and IFN-gamma production.

21 which neither expressed IL-12 nor stimulated IFN-gamma production.

22 rotein stability are critical in suppressing IFN-gamma production.

23 IL-10Ralpha blockade preferentially restores IFN-gamma production.

24 tially killed by NK cells and trigger potent IFN-gamma production.

25 e of IL-9 or IL-4 enhanced allergen-specific IFN-gamma production.

26 Toll-like receptor ligands further enhanced IFN-gamma production.

27 tely restored Th cell proliferation, but not IFN-gamma production.

28 s to KIR2DL4 and HLA-G did not block NK cell IFN-gamma production.

29 L-12 and stimulated T cell proliferation and IFN-gamma production.

30 ppression of CD4(+) T cell proliferation and IFN-gamma production.

31 -1-like phenotype characterized by IL-10 and IFN-gamma production.

32 aforementioned cellular immune response and IFN-gamma production.

33 chemotaxis of CD74(+)CXCR2(+) NKT cells for IFN-gamma production.

34 allergic phenotype associated with increased IFN-gamma production.

35 Cell-bound factors enhanced NK cell IFN-gamma production.

36 ation by stimulating iNKT cell expansion and IFN-gamma production.

37 vo function for CD43-dependent regulation of IFN-gamma production.

38 ed by hyperactive CD8 T cells and continuous IFN-gamma production.

39 red less peptide than was required to induce IFN-gamma production.

40 of Th1 genes that is sufficient to increase IFN-gamma production.

41 e mediated partially by its ability to blunt IFN-gamma production.

42 ity to activate NK cell immune phenotype and IFN-gamma production.

43 rforin-dependent lysis of infected cells and IFN-gamma production.

44 re induced despite lacking specific IL-2 and IFN-gamma production.

45 2 in NK cells suppressed IL-12/IL-18-induced IFN-gamma production.

46 diabetic intervention approaches modulating IFN-gamma production.

47 like receptor G1(+) effector CD8 T cells and IFN-gamma production.

48 n provides sufficient energy for TLR-induced IFN-gamma production.

49 IFN-DC-mediated NK cell activation and early IFN-gamma production.

50 pSTAT4, nuclear translocation, and impaired IFN-gamma production.

51 G2D-dependent cell-mediated cytotoxicity and IFN-gamma production.

52 coordination of subsequent cytotoxicity and IFN-gamma production.

53 like receptor G1(+) effector CD8 T cells and IFN-gamma production.

54 s and dampened effector T-cell expansion and IFN-gamma production.

55 nction, as measured by CD107a expression and IFN-gamma production.

56 et of SS in NOD mice, in part by suppressing IFN-gamma production.

57 roinflammatory mediators and T cell-mediated IFN-gamma production.

58 nfirmed that 10 of 26 epitopes (38%) induced IFN-gamma production.

59 (+) T-cell proliferation but did not restore IFN-gamma-production.

60 ll responses were observed in the absence of IFN-gamma-production.

61 nd are associated with low-level interferon (IFN)-gamma production.

62 d with excessive Treg cell interferon-gamma (IFN-gamma) production.

63 that correlated with increased type II IFN (IFN-gamma) production.

64 rations in the kinetics of interferon-gamma (IFN-gamma) production.

65 or locus on chromosome region 8q controlling IFN-gamma production after stimulation with live BCG (Ba

66 m-specific in vitro recall gamma interferon (IFN-gamma) production after CHMI, and innate IFN-gamma r

67 egulated autologous T-cell proliferation and IFN-gamma production against a peptide pool consisting o

68 mice was associated with impaired IL-12 and IFN-gamma production and a concomitant increase in IL-4

69 ing, based on significantly higher levels of IFN-gamma production and a remarkable increase in CD8(+)

70 ty in HLA/antigen-matched tumors and induced IFN-gamma production and activation.

71 leading to NK cells that display diminished IFN-gamma production and at least a transiently impaired

72 lindolo[3,2-b]carbazole, potentiates NK cell IFN-gamma production and cytolytic activity.

73 CCR7(+) Dectin-1(-)M1 cells, accompanied by IFN-gamma production and cytolytic function in T cells.

74 f SIY-specific CD8(+) T cells, with enhanced IFN-gamma production and cytotoxic capability.

75 idenced by enhanced proliferation, survival, IFN-gamma production and cytotoxicity toward tumors.

76 from infected wild-type mice with respect to IFN-gamma production and cytotoxicity, and could compara

77 opositivity is associated with lower NK cell IFN-gamma production and degranulation after in vitro re

78 l responses, which correlated with decreased IFN-gamma production and degranulation by Tim-3 KO cells

79 gside STAT4 to coordinate Tfh cell IL-21 and IFN-gamma production and for promotion of the GC respons

80 rate of glycolysis is sufficient to inhibit IFN-gamma production and granzyme B expression.

81 ppressed both T lymphocyte proliferation and IFN-gamma production and had high arginase-I expression.

82 AR-20347 significantly reduced IL-12-induced IFN-gamma production and IL-22-dependent serum amyloid A

83 (SOCS1) protein in T cells, which inhibited IFN-gamma production and killing of CEF-pulsed monocytes

84 s mechanism is required for priming IL-2 and IFN-gamma production and may contribute to fine-tuning T

85 population was fully functional as judged by IFN-gamma production and MHC class I-restricted cytotoxi

86 IFN-gamma production and microbicidal activity were impa

87 nous TPA and MIF to NKT cells promoted their IFN-gamma production and migration, respectively.

88 Tregs in wounded skin attenuated IFN-gamma production and proinflammatory macrophage accu

89 n was shown by a dose-dependent induction of IFN-gamma production and proliferation by the CD4 T-cell

90 data highlight T-bet-independent pathways to IFN-gamma production and reveal a novel role for this tr

91 ity to retinoic acid, resulting in increased IFN-gamma production and subsequent IFN-gamma-mediated s

92 munity against this parasite is dependent on IFN-gamma production and subsequent macrophage activatio

93 In contrast to Bet v 1, Mal d 1 triggered IFN-gamma production and T cells with a different epitop

94 T-bet is a master regulator for IFN-gamma production and Th1 differentiation.

95 demonstrated an inverse correlation between IFN-gamma production and the time from clinical presenta

96 accompanied by low IL-4 and IL-10, but high IFN-gamma productions and reduced regulatory T cells.

97 er studies have shown that gamma interferon (IFN-gamma) production and activation of Th1 cells charac

98 Induced interferon gamma (IFN-gamma) production and NKp46/NKp30 activating recepto

99 -CXCL10 antibodies reduced gamma interferon (IFN-gamma) production and Th17 cell infiltration, as wel

100 ells for increased T-bet expression, optimal IFN-gamma production, and CD4 T cell proliferation.

101 vels were required to block cytotoxicity and IFN-gamma production, and very low levels of PD-1 expres

102 eration, interleukin 2 and interferon gamma (IFN-gamma) production, and phosphorylated STAT1 expressi

103 eptor signaling, as NK cell cytotoxicity and IFN-gamma production are regulated by signal transducer

104 lar flow cytometry was employed to determine IFN-gamma production as a functional readout.

105 A 2-way MLR, adopting IFN-gamma production as a marker of alloresponse, result

106 NA with EP delivery induced antigen-specific IFN-gamma production, as measured by ELISpot assay and I

107 An IFN-gamma production assay and in vivo CTL response stud

108 ty in a human T cell blast proliferation and IFN-gamma production assay.

109 stimulate iNKT cells in vivo, with increased IFN-gamma production at 24 h compared with alphaGalCer,

110 restored OM-specific PBMC proliferation and IFN-gamma production at 5 wpi.

111 ubsets, exhibit impaired cell activation and IFN-gamma production but increased apoptosis compared to

112 This was associated with an increase in IFN-gamma production but not cytotoxicity of NK cells du

113 critical for CD8 T cell activation (promotes IFN-gamma production but not proliferation or granzyme B

114 /-) mice gave rise to T cells with decreased IFN-gamma production, but gained the ability to produce

115 activity also induced spontaneous IL-22 and IFN-gamma production, but these cytokines had also uniqu

116 used apoptosis of Jurkat cells and inhibited IFN-gamma production by activated CD4+ T cells.

117 his was confirmed by in vitro measurement of IFN-gamma production by activated T cells.

118 an essential second signal for induction of IFN-gamma production by activating NK cell receptors tha

119 sin-converting enzyme inhibitors potentiated IFN-gamma production by Ag-specific T cells via C5aR/B2R

120 -) macrophages enhances Th1 polarization and IFN-gamma production by antigen-specific CD4(+) T cells

121 The functional loss of IFN-gamma production by antigen-specific CD4(+) T cells

122 es the ability to suppress proliferation and IFN-gamma production by autologous T cells ex vivo.

123 Identification of excessive IFN-gamma production by blood and lymph node-derived T c

124 We analyzed the role of IFN-gamma production by brain-resident cells in resistan

125 sing bone marrow chimeric mice revealed that IFN-gamma production by brain-resident cells is essentia

126 However, the physiological role of IFN-gamma production by brain-resident cells, including

127 ns through effects on leukocyte recruitment, IFN-gamma production by CD4 and CD8 T cells, and the act

128 IFN-gamma production by CD4(+) and CD8(+) peripheral blo

129 -helper (Th) cell type 1 inflammation (e.g., IFN-gamma production by CD4(+) effector T cells).

130 e were functionally defective in suppressing IFN-gamma production by CD4(+) T cells in coculture.

131 production by macrophages is known to induce IFN-gamma production by CD4(+) T cells, HLA-G5 increased

132 Here we show that IFN-gamma production by CD4(+) T(H)1 cells during mucosa

133 -2) on NK cells accounted for the diminished IFN-gamma production by CD56(bright) NK cells, whereas M

134 rrent study, we examined the role of IL-2 in IFN-gamma production by CD8(+) immune T cells in their s

135 A strong dependency on PI3Kdelta for IFN-gamma production by CD8(+) T cells in vitro was not

136 a potent resistance against reinfection, and IFN-gamma production by CD8(+) T cells is crucial for th

137 s principally reflected profoundly defective IFN-gamma production by circulating gammadelta T cells a

138 tion into the intestinal lamina propria, and IFN-gamma production by colitogenic CD4(+) T cells.

139 ce displayed enhanced capacities to suppress IFN-gamma production by effector T cells, suggesting tha

140 In this study, we investigated IFN-gamma production by freshly isolated NK cells in res

141 However, Eomes is fully dispensable for IFN-gamma production by gammadelta T cells.

142 Cytotoxicity and IFN-gamma production by human gammadelta T cells underli

143 xpression at low O2 levels and the unchanged IFN-gamma production by IL-10-deficient Th1 cells stimul

144 0, IL-6, and TNF-alpha production as well as IFN-gamma production by IL-12p70-mediated activation of

145 tors Nod1 and Nod2 are necessary for optimal IFN-gamma production by iNKT cells, as well as NK cells.

146 Nonetheless, Treg treatment abrogated IFN-gamma production by intraislet CD8(+) and CD4(+) T c

147 After MCMV infection, Ly49D augmented IFN-gamma production by MCMV-specific Ly49H(+) NK cells

148 r release of alarmins, pyroptosis, and early IFN-gamma production by memory CD8 T cells, all of which

149 -gamma only in CD11b(+) cells suggested that IFN-gamma production by microglia, which is the only CD1

150 Here, we have reported that noncognate IFN-gamma production by Mtb antigen-independent memory C

151 from H. polygyrus (HES) was found to dampen IFN-gamma production by mycobacteria-specific CD4(+) T c

152 ongly promote Th1 differentiation, enhancing IFN-gamma production by naive T cells.

153 When activated in vitro, however, IFN-gamma production by naive wild type and tristetrapro

154 2-independent and the events associated with IFN-gamma production by neutrophils are not understood.

155 alectin-9 in mice is associated with greater IFN-gamma production by NK cells and enhanced degranulat

156 gnaling significantly recovered the impaired IFN-gamma production by NK cells from HCV-infected subje

157 ulate that SP-D may constitutively stimulate IFN-gamma production by NK cells, possibly via NKp46.

158 IL-18 and are unable to optimally stimulate IFN-gamma production by NK cells.

159 ry cytokine combinations capable of inducing IFN-gamma production by NK cells.

160 nocytogenes and observed that IS001 enhanced IFN-gamma production by NKT, CD4(+), and CD8(+) T cells

161 ting conditions resulted in higher levels of IFN-gamma production by pTreg compared with thymic Treg,

162 ected phenotype was associated with enhanced IFN-gamma production by T cells and decreased accumulati

163 CD19-hBtk B cells promoted IFN-gamma production by T cells and expression of the im

164 quently, CCL2 deficient MSCs did not inhibit IFN-gamma production by T cells and upon transfer no lon

165 IL-17A production by Th17 CD4 cells, but not IFN-gamma production by Th1 cells.

166 he rate of glycolysis significantly impaired IFN-gamma production by the CD56(bright) subset of cells

167 ent with a neutralizing Ab to CX3CL1 reduced IFN-gamma production by the lung NK cells.

168 and evaluated the mechanistic regulation of IFN-gamma production by these cells in vivo.

169 , commensurate with increased activation and IFN-gamma production by these immature NK cell subsets.

170 IFN-gamma production by these lymphocytes likely plays a

171 hemoattractants for neutrophils, and reduced IFN-gamma production by virus-specific CD8 T cells.

172 Innate gamma interferon (IFN-gamma) production by CD8(+) T cells following exposu

173 different angles, and we discuss how chronic IFN-gamma production can lead to the development of anem

174 were present in the explant, showing a great IFN-gamma production capacity and expressing granzyme B.

175 groups and in particular in relationship to IFN-gamma production capacity.

176 reactivity with a more pronounced effect on IFN-gamma production compared with cytotoxicity.

177 ional Tg Th1 effectors demonstrated enhanced IFN-gamma production compared with polyclonal cells, pro

178 Functionally, Th1 cells with graded IFN-gamma production competence differentially activated

179 In contrast to the dramatic increase in IFN-gamma production, concurrent blockade has a marginal

180 epwise loss of Ag-specific proliferation and IFN-gamma production, corresponding to increasing VL sym

181 feron (IFN)-gamma in vitro, and this reduced IFN-gamma production could be partially reversed by bloc

182 rogram as well as glycolysis and OXPHOS, but IFN-gamma production could be reinstated by retrovirus-m

183 mmatory lymphocytes in diabetes development, IFN-gamma production could represent an attempted limita

184 cytotoxicity increases and pSTAT4-dependent IFN-gamma production decreases in response to endogenous

185 The mTORC1 and IFN-gamma production defects were partially rescued by s

186 the metabolic requirements of murine NK cell IFN-gamma production depending upon the activation signa

187 gans of recipients, but profoundly decreased IFN-gamma production despite normal T-bet and enhanced E

188 cells revealed markedly defective IL-17A and IFN-gamma production, despite preserved T-bet expression

189 ctivated late effectors, regardless of their IFN-gamma production, develop minimal memory.

190 nding T cells and the amount of TNF-alpha or IFN-gamma production did not differ between the three pr

191 uced capacity to prime antigen (Ag)-specific IFN-gamma production during co-culture with naive transg

192 lly, Nod1-deficient T cells exhibit impaired IFN-gamma production during dextran sulfate sodium (DSS)

193 roducing cells and the signals that regulate IFN-gamma production during T. gondii infection.

194 deficient T cells exhibited markedly reduced IFN-gamma production during the early phase of Mtb infec

195 ing MCMV infection but did not show enhanced IFN-gamma production following direct ex vivo cytokine s

196 d mice had a marked increase in both TNF and IFN-gamma production following ex vivo stimulation.

197 o demonstrated a reduced capacity to sustain IFN-gamma production following in vitro activation.

198 nt with recovery of T cell proliferation and IFN-gamma production following stimulation of CD3/CD28.

199 monstrated substantially diminished IL-2 and IFN-gamma production following TCR stimulation of his "u

200 ed recipients initially retained the stunted IFN-gamma production found prior to transfer, and cells

201 of HSCs to suppress the proliferation of and IFN-gamma production from activated T cells, suggesting

202 t secreted factors from tumor tissue reduced IFN-gamma production from MAIT cells.

203 as demonstrated by early and Ag-independent IFN-gamma production, granzyme B expression, and degranu

204 molecule-1, NKp46, and NKG2D) and decreased IFN-gamma production, had a significantly higher risk of

205 These data suggest that IFN-gamma production has antiviral effects in rabies, la

206 attributed the key distal event to excessive IFN-gamma production; however, the proximal events drivi

207 ation (no ND; n = 63), had lower CMP-induced IFN-gamma production in 24-hour cultures pre-cART and 4

208 factor for Th1 cells, which in turn supports IFN-gamma production in a feed-forward manner.

209 induced intracellular Leishmania killing and IFN-gamma production in a macrophage-T cell coculture sy

210 re required to support both cytotoxicity and IFN-gamma production in all NK cells.

211 that, although SOCE is required for NK cell IFN-gamma production in an NFAT-dependent manner, NK cel

212 g by allergen-IgE immune complexes increased IFN-gamma production in B cells of allergic patients dur

213 regulating allergic reactions via modulating IFN-gamma production in B cells.

214 that synergize to induce antigen-independent IFN-gamma production in CD4(+) and CD8(+) TEM cells.

215 ntrinsic TLR pathway as a major regulator of IFN-gamma production in CD4(+) T cells responsible for P

216 tural killer T cells were the main source of IFN-gamma production in co-culture experiments.

217 s associated with enhanced proliferation and IFN-gamma production in CXCR3(+) cells.

218 The developmental mechanism of IFN-gamma production in neutrophils arms the innate immu

219 l death during acute hepatitis by regulating IFN-gamma production in NK and NKT cells and thus functi

220 sulted in significant augmentation of T-cell IFN-gamma production in patients with COPD.

221 ur phenotypic findings, we observed a higher IFN-gamma production in peripheral CD4 memory T cells an

222 ties, and correlated with the enhancement of IFN-gamma production in response to CD48 blockade in HIV

223 , STIM1-deficient T cells displayed enhanced IFN-gamma production in response to elevated levels of I

224 e divided cells did not demonstrate enhanced IFN-gamma production in response to innate cytokine stim

225 d a major locus on chromosome 8q controlling IFN-gamma production in response to stimulation with liv

226 stricted cells, HSV-1 induced markedly lower IFN-gamma production in splenocytes from naive mice.

227 igand nonbinding isoform in NOD mice reduced IFN-gamma production in T effector cells accompanied by

228 tion begins at the onset of T cell entry and IFN-gamma production in the CNS prior to the appearance

229 We also demonstrate that despite normal IFN-gamma production in the heart, ASC(-)/(-) and caspas

230 important autocrine-enhancing role on their IFN-gamma production in the secondary responses to T. go

231 ion of cytotoxicity receptors, and extensive IFN-gamma production in the virtual absence of IL-10.

232 ts in a markedly reduced capacity to inhibit IFN-gamma production in tolerized T cells.

233 Importantly, lenalidomide did not trigger IFN-gamma production in unstimulated NK cells.

234 i-PD-1 mAb restored T cell proliferation and IFN-gamma production in vitro and led to complete resolu

235 b(+) cells suppress T cell proliferation and IFN-gamma production in vitro via NO-dependent mechanism

236 nsferred CTLs enhances T cell activation and IFN-gamma production in vitro, leading to a significant

237 promoted proliferation and gamma interferon (IFN-gamma) production in CD4 and CD8 T cells.

238 tions capable of eliciting gamma interferon (IFN-gamma) production in NK cells.

239 ell maturation defect and leads to increased IFN-gamma production induced by activating receptors.

240 gen target (ESAT-6), taking into account the IFN-gamma production induced by BCG (IFNgamma-ESAT6BCG).

241 Monokine induced by IFN-gamma, MCP-1, and IFN-gamma production induced in T. cruzi-infected infant

242 ammation promotes tumor progression, whereas IFN-gamma production is essential for antitumor immunity

243 These findings further support the idea that IFN-gamma production is essential for HSPC activation an

244 IFN-gamma production, mainly by natural killer cells, wa

245 a pulmonary tularaemia model, POP2 enhances IFN-gamma production, modulates neutrophil numbers, impr

246 Protection against CL was associated with IFN-gamma production, negative LST results, impaired abi

247 of Eomes in CD4(+) T cells did not impact on IFN-gamma production nor increase Th2 or Th17 responses.

248 Impaired metabolism underlies the reduced IFN-gamma production observed in activated RTEs.

249 The deficit in IFN-gamma production observed in ASC(KO) mice was not du

250 The inhibition of IFN-gamma production occurs through blocking the repress

251 ction of 6A6 suppressed DNFB-induced CHS and IFN-gamma production of CD8(+) T cells.

252 ted highly enhanced degranulation, ADCC, and IFN-gamma production of NK cells in response to breast c

253 -2.4 Gy radiation enhances proliferation and IFN-gamma production of PBMC or purified T cells induced

254 itors of JAK1 and JAK3 significantly reduced IFN-gamma production of the T cells.

255 ects of the T cell costimulatory pathway and IFN-gamma production on the pathogenesis of autoimmune p

256 nt IL-13 responses, but had little effect on IFN-gamma production or cell proliferation.

257 ty to retinoic acid contributes to decreased IFN-gamma production, permitting the expression of Foxp3

258 lation and TNF-alpha but not cytotoxicity or IFN-gamma production, potentially favoring the progressi

259 Lower CMP-induced IFN-gamma production pre-cART, but not higher CMP-specif

260 of (64)Cu-PTSM and analyzed cell viability, IFN-gamma production, proliferation, apoptosis, and DNA

261 tory antigen target, when accounting for the IFN-gamma production shared with that induced by BCG (LO

262 creased mitogen-stimulated gamma interferon (IFN-gamma) production suggested immunomodulation, which

263 T cells in vitro decreased proliferation and IFN-gamma production, suggesting additional effects of s

264 However, the observation of impaired IFN-gamma production suggests that the efficacy of such

265 ofibrotic Th17 signaling and proinflammatory IFN-gamma production that contribute to PBC pathology.

266 It induces a short boost of IFN-gamma production that leads to a significant but lim

267 n in group 1 ILCs is required to limit their IFN-gamma production, thereby allowing the development o

268 ened mTOR activity, glycolytic capacity, and IFN-gamma production, thereby allowing tumor progression

269 alpha-GalCer-induced iNKT cell expansion and IFN-gamma production, thereby inhibiting liver regenerat

270 55-overexpressing NK cells exhibit increased IFN-gamma production through distinct cellular mechanism

271 sion in RTEs, driving aerobic glycolysis and IFN-gamma production to the level of mature T cells.

272 cond locus on chromosome region 3q affecting IFN-gamma production triggered by the 6-kDa early secret

273 cose availability does not alter the rate of IFN-gamma production upon TLR-mediated activation.

274 vocal serology and/or borderline ST results, IFN-gamma production was 449 +/- 82 pg/mL in the positiv

275 Increased IFN-gamma production was accompanied by a poor germinal

276 This difference in IFN-gamma production was due to a large production of IF

277 ession was observed on DenV-infected DC, and IFN-gamma production was enhanced in licensed/educated N

278 IFN-gamma production was found to be MyD88- and TLR9-dep

279 In all assay formats, C. burnetii-specific IFN-gamma production was higher (P < .0001) in seroposit

280 -alpha and GM-CSF at a higher level, whereas IFN-gamma production was impaired following IL-2 plus IL

281 Stimulation of NK cell IFN-gamma production was independent of glycolysis or mi

282 IFN-gamma production was measured after in vitro stimula

283 g cells produced less IL-10 but more IL-17A; IFN-gamma production was not affected.

284 ion and respond to heterologous infection by IFN-gamma production was observed in inbred and outbred

285 This IL-2-mediated upregulation of IFN-gamma production was observed in mitomycin C-treated

286 d type I interferon (IFN), respectively, and IFN-gamma production was reduced in response to IL-12 +

287 Moreover, CCL17-induced IFN-gamma production was reduced when Th1-polarized norm

288 Accordingly, NKT cells were less activated, IFN-gamma production was significantly reduced, and leve

289 L-17 compared with the control mice, whereas IFN-gamma production was similar in both groups.

290 gA, IgA antibody-secreting cells (ASCs), and IFN-gamma production were evaluated.

291 (+)IL-10(+)IFN-gamma(+) frequencies and Treg-IFN-gamma production were found in women with current Pl

292 onuclear cells (PBMCs) and interferon gamma (IFN-gamma) production were significantly suppressed in c

293 tic T cell recruitment and interferon-gamma (IFN-gamma) production, were suppressed upon HCV challeng

294 displayed enhanced cytolytic capability and IFN-gamma production when co-cultured with GB cells or p

295 sarcoidosis patients did not rescue IL-2 and IFN-gamma production, whereas restoration of the IL-2 si

296 endogenous ligands to NKT cells, leading to IFN-gamma production, which in turn, stimulated 3T3-L1 a

297 with antigen-presenting cells, resulting in IFN-gamma production, which induced NO and downregulated

298 l viremia, and it differed from the impaired IFN-gamma production, which is typical for NK cells in c

299 y in these cells reduced both viral load and IFN-gamma production, which suggests that targeting CCR4

300 oper T cell activation and gamma interferon (IFN-gamma) production, which are critical for infection