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1 e unresponsive to IL-13, and did not express IL-13 receptor.
2 nal in response to IL-13 and did not express IL-13 receptor.
3 its in several receptor types, including the IL-13 receptor.
4 both chains are essential components of the IL-13 receptor.
5 gulate sPLA2 and MUC2 production through the IL-13 receptor.
6 d against the IL-4Ralpha subunit of IL-4 and IL-13 receptors.
7 nal signaling component of both the IL-4 and IL-13 receptors.
8 bservation that human T cells do not express IL-13 receptors.
11 (sixfold) reduction in decoy IL-13 receptor (IL-13 receptor alpha-2) expression when compared with in
12 tored in PsKO mice by treatment with soluble IL-13 receptor alpha-2-Fc, the exacerbated fibrotic resp
13 role in pathogenesis of bronchial asthma via IL-13 receptor alpha1 (IL-13Ralpha1) and IL-4 receptor a
14 ested to establish the expression pattern of IL-13 receptor alpha1 (IL-13Ralpha1) on islet-associated
15 as abolished in liver cells lacking Stat3 or IL-13 receptor alpha1 (Il-13ralpha1), which suggests tha
17 e lung was dependent upon recipient IL-5 and IL-13 receptor alpha1 and donor eosinophil C-C chemokine
24 during infection and binds to its receptor, IL-13 receptor alpha2 (IL-13Ralpha2), to regulate the pa
25 epair and promotes tubular cell survival via IL-13 receptor alpha2 (IL13Ralpha2)-mediated signaling.
26 as, decreased collagen levels, and increased IL-13 receptor alpha2 gene expression compared to contro
29 s exhibit divergent expression of functional IL-13 receptor and this expression dictates the responsi
30 tive mAb for AD, dupilumab, targets the IL-4/IL-13 receptor and was approved in early 2017 in the Uni
31 but it also participates in the induction of IL-13 receptors and miR-126a expressed on/in the MDSCs.
32 tment to phosphotyrosine residues on IL-4 or IL-13 receptors and subsequent Tyr641 phosphorylation to
33 levels of expression of the interleukin-13 (IL-13) receptor and downstream effectors of IL-13 signal
37 with 5-10-fold improved binding affinity to IL-13 receptors compared with wild-type IL-13 (wtIL-13).
38 ur studies have characterized the functional IL-13 receptor complex and the downstream signaling even
41 ased surface expression of the high affinity IL-13 receptor IL-13Ralpha2, suggesting that IL-13Ralpha
43 d a significant (sixfold) reduction in decoy IL-13 receptor (IL-13 receptor alpha-2) expression when
44 ) mice have demonstrated a critical role for IL-13 receptor (IL-13R) alpha1 in allergen-induced airwa
45 ave previously shown that the decoy receptor IL-13 receptor (IL-13R) alpha2 attenuates responses of f
46 fication of IL-13Ralpha1, a component of the IL-13 receptor (IL-13R), as a novel ligand of integrin M
53 racellular/transmembrane domains of the IL-4/IL-13 receptor, not the cytoplasmic domains, control sig
55 essing IFN-gamma and up-regulating the decoy IL-13 receptor, P-selectin dramatically inhibits the pat
56 ignificantly reduced the numbers of IL-4 and IL-13 receptor-positive mononuclear cells and macrophage
59 ing antibodies against IL-13 or the IL-4 and IL-13 receptor subunit IL-4Ralpha, as well as an antibod
60 3K, bound with 4-fold higher affinity to the IL-13 receptor than wild-type IL-13 but retained no dete
61 IL-13Ralpha2 does not participate in the IL-13 receptor that is up-regulated upon activation of q
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