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1 e unresponsive to IL-13, and did not express IL-13 receptor.
2 nal in response to IL-13 and did not express IL-13 receptor.
3 its in several receptor types, including the IL-13 receptor.
4  both chains are essential components of the IL-13 receptor.
5 gulate sPLA2 and MUC2 production through the IL-13 receptor.
6 d against the IL-4Ralpha subunit of IL-4 and IL-13 receptors.
7 nal signaling component of both the IL-4 and IL-13 receptors.
8 bservation that human T cells do not express IL-13 receptors.
9                       Here, we show that the IL-13 receptor alpha 2 (IL-13R alpha 2) is a critical me
10                We demonstrate that increased IL-13 receptor alpha 2 (IL-13Ralpha2) expression is resp
11 (sixfold) reduction in decoy IL-13 receptor (IL-13 receptor alpha-2) expression when compared with in
12 tored in PsKO mice by treatment with soluble IL-13 receptor alpha-2-Fc, the exacerbated fibrotic resp
13 role in pathogenesis of bronchial asthma via IL-13 receptor alpha1 (IL-13Ralpha1) and IL-4 receptor a
14 ested to establish the expression pattern of IL-13 receptor alpha1 (IL-13Ralpha1) on islet-associated
15 as abolished in liver cells lacking Stat3 or IL-13 receptor alpha1 (Il-13ralpha1), which suggests tha
16 umanized mAb, inhibits IL-13 binding to both IL-13 receptor alpha1 and alpha2.
17 e lung was dependent upon recipient IL-5 and IL-13 receptor alpha1 and donor eosinophil C-C chemokine
18 tibodies to TSLPR and ST2, respectively, and IL-13 receptor alpha1 in both scenarios.
19 of recruited M-MDSCs, which highly expressed IL-13 receptor alpha1.
20                             The interleukin (IL) 13 receptor alpha2 (IL13Ralpha2) is a glioma-restric
21                             The interleukin (IL)-13 receptor alpha2 (IL-13Ralpha2) chain is a primary
22                                 Interleukin (IL)-13 receptor alpha2 (IL-13Ralpha2) chain is an essent
23                                              IL-13 receptor alpha2 (IL-13Ralpha2) binds IL-13 with hi
24  during infection and binds to its receptor, IL-13 receptor alpha2 (IL-13Ralpha2), to regulate the pa
25 epair and promotes tubular cell survival via IL-13 receptor alpha2 (IL13Ralpha2)-mediated signaling.
26 as, decreased collagen levels, and increased IL-13 receptor alpha2 gene expression compared to contro
27                              Interleukin-13 (IL-13) receptor alpha2 (IL-13Ralpha2), a high-affinity I
28 s for these peptides are EphA2, interleukin (IL)-13 receptor-alpha2, YKL-40, and gp100.
29 s exhibit divergent expression of functional IL-13 receptor and this expression dictates the responsi
30 tive mAb for AD, dupilumab, targets the IL-4/IL-13 receptor and was approved in early 2017 in the Uni
31 but it also participates in the induction of IL-13 receptors and miR-126a expressed on/in the MDSCs.
32 tment to phosphotyrosine residues on IL-4 or IL-13 receptors and subsequent Tyr641 phosphorylation to
33  levels of expression of the interleukin-13 (IL-13) receptor and downstream effectors of IL-13 signal
34 ytokine receptors, including IL-4, IL-6, and IL-13 receptors as well as CCR3.
35            Our findings also reveal that the IL-13 receptor-associated tyrosine kinase Jak2 is requir
36 th anti-IL-4R mAb, an antagonist of IL-4 and IL-13 receptor binding, or with a control mAb.
37  with 5-10-fold improved binding affinity to IL-13 receptors compared with wild-type IL-13 (wtIL-13).
38 ur studies have characterized the functional IL-13 receptor complex and the downstream signaling even
39 ma, IL-10, TGF-beta, TNF-alpha, and the IL-4/IL-13 receptor complex play important roles.
40 xhibit on their surfaces the interleukin 13 (IL-13) receptor designated IL13Ralpha2.
41 ased surface expression of the high affinity IL-13 receptor IL-13Ralpha2, suggesting that IL-13Ralpha
42 g chain of the high affinity interleukin-13 (IL-13) receptor IL-13Ralpha1.
43 d a significant (sixfold) reduction in decoy IL-13 receptor (IL-13 receptor alpha-2) expression when
44 ) mice have demonstrated a critical role for IL-13 receptor (IL-13R) alpha1 in allergen-induced airwa
45 ave previously shown that the decoy receptor IL-13 receptor (IL-13R) alpha2 attenuates responses of f
46 fication of IL-13Ralpha1, a component of the IL-13 receptor (IL-13R), as a novel ligand of integrin M
47          Moreover, genes encoding IL-15, the IL-13 receptor (IL-13Ra1), and CD14 were suppressed duri
48                       A third, high-affinity IL-13 receptor, IL-13Ralpha2, also exists.
49                              GBMs express an IL-13 receptor (IL13Ralpha2) that differs from the physi
50 at leads us to postulate interactions at the IL-13/receptor interface.
51        Thus, reduced expression of the decoy IL-13 receptor mediated by the elevated type 1 cytokine
52             These results indicate that IL-4/IL-13 receptor-mediated Stat3 signaling may contribute t
53 racellular/transmembrane domains of the IL-4/IL-13 receptor, not the cytoplasmic domains, control sig
54  exotoxin (IL13-PE), which targets and kills IL-13 receptor overexpressing cells.
55 essing IFN-gamma and up-regulating the decoy IL-13 receptor, P-selectin dramatically inhibits the pat
56 ignificantly reduced the numbers of IL-4 and IL-13 receptor-positive mononuclear cells and macrophage
57                           Here, we show that IL-13 receptor (R)alpha2 is a critical down-regulatory f
58 n a manner that is dependent on the IL-4 and IL-13 receptor subunit expression by these cells.
59 ing antibodies against IL-13 or the IL-4 and IL-13 receptor subunit IL-4Ralpha, as well as an antibod
60 3K, bound with 4-fold higher affinity to the IL-13 receptor than wild-type IL-13 but retained no dete
61     IL-13Ralpha2 does not participate in the IL-13 receptor that is up-regulated upon activation of q
62                             For a functional IL-13 receptor, the IL-13Ralpha1 chain forms a productiv
63       Because murine lymphocytes do not have IL-13 receptors, we examined the ability of macrophage/n

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