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1 n contrast to the mitogenic receptors IL-3R, IL-5R, GM-CSFR, which assemble as alphabeta heterodimers
2             High affinity receptor for IL-5 (IL-5R), a predominant eosinophil maturation factor, is c
3                    Lidocaine does not affect IL-5R expression or IL-5-induced protein tyrosine phosph
4 pha (IL-5Ralpha), a subunit of high-affinity IL-5R, on these cells.
5           Since message for the IL-5R alpha, IL-5R beta, and soluble IL-5R alpha chains was detected
6 op a multiplex assay, in which the IL-1R and IL-5R assays were carried out in the same well with each
7 the structural interactions between IL-5 and IL-5R and the functional consequences of such interactio
8                                     IL-5 and IL-5R drive allergic and inflammatory immune responses c
9 sted of the ectodomains of the GMR-alpha and IL-5R alpha, respectively, fused to the endodomain of IL
10 or with IL-2, increased both IL-5R alpha and IL-5R beta mRNA and decreased soluble IL-5R alpha mRNA.
11  tyrosine phosphorylation of JAK2 kinase and IL-5R beta-chain and inhibited IL-5 priming of leukotrie
12 inophils, IL-5R alpha gene transcription and IL-5R alpha mRNA metabolism can be regulated by mechanis
13 ion, IL-5-induced endocytosis, turnover, and IL-5R signaling were significantly impaired.
14 th IL-5 for binding to the native alpha beta IL-5R on human cells and inhibited IL-5-mediated recepto
15 eotide technology targeting the common betac IL-5R subunit is also being used therapeutically to inhi
16 or SAC, without or with IL-2, increased both IL-5R alpha and IL-5R beta mRNA and decreased soluble IL
17  is composed of an IL-5-binding alpha-chain (IL-5R alpha) and a signal-transducing beta-chain that is
18 example, interleukin-5 receptor alpha chain, IL-5R alpha).
19  their degradation, ubiquitination-deficient IL-5Rs accumulated on the cell surface and displayed blu
20 experiments that in human blood eosinophils, IL-5R alpha gene transcription and IL-5R alpha mRNA meta
21 mmunoreactive major basic protein, Siglec F, IL-5R alpha-chain, and transcripts encoding mouse eosino
22 e B cells, human B cells express message for IL-5R but can respond to IL-5 only if appropriately stim
23                  A soluble form of the human IL-5R alpha-chain (IL-5Ra) that contains the extracellul
24 nding of the role of betac ubiquitination in IL-5R biology is currently lacking.
25 ry injury is associated with the activity of IL-5R-bearing cells.
26 onstrate that cytokine-induced inhibition of IL-5R alpha mRNA accumulation occurs at the level of IL-
27 pha mRNA accumulation occurs at the level of IL-5R alpha gene transcription, whereas enhanced accumul
28             Furthermore, increased levels of IL-5R alpha-chain were expressed by B-1a B cells in SMGs
29 ree cytokines on eosinophils and the loss of IL-5R on airway eosinophils, it is important to take IL-
30 rization reveals that the down-regulation of IL-5R alpha mRNA is specific to IL-3, IL-5, and GM-CSF;
31  was due to their differential regulation of IL-5R mRNA.
32                           The IL-5 receptor (IL-5R) consists of an IL-5-specific alpha subunit that i
33 nd activation of the interleukin-5 receptor (IL-5R) or of the granulocyte-macrophage colony-stimulati
34 lly dependent on IL-5 and the IL-5 receptor (IL-5R), composed of a ligand binding alpha chain (IL-5Ra
35 tokines IL-5, IL-3, and GM-CSF down-regulate IL-5R alpha mRNA while up-regulating alpha-chain mRNAs f
36 for the IL-5R alpha, IL-5R beta, and soluble IL-5R alpha chains was detected in freshly isolated B ce
37 ha and IL-5R beta mRNA and decreased soluble IL-5R alpha mRNA.
38                             As such, soluble IL-5R may be useful in treating diseases such as human a
39 re-clinical and clinical evidence supporting IL-5R as a therapeutic target.
40 mab (MEDI-563), has been developed to target IL-5R and attenuate eosinophilia through antibody-depend
41                        Since message for the IL-5R alpha, IL-5R beta, and soluble IL-5R alpha chains
42  B cells with IL-4 induced expression of the IL-5R alpha-chain, while signaling through membrane Ig s
43 ugh membrane Ig stimulated expression of the IL-5R beta-chain.
44 d not mediate ECA activity by binding to the IL-5R or to CCR3.
45 Lyn kinase is physically associated with the IL-5R betac subunit in unstimulated cells.
46  interaction of the dimeric peptide with two IL-5R alpha chains represents a distinctive mechanism fo
47                        Whereas ubiquitinated IL-5Rs internalized into trafficking endosomes for their

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