ライフサイエンスコーパス: IP

1 IP and DMAP can also competitively inhibit farnesyl diph

2 IP is caused by pathogenic variants that inactivate the

3 IP profiling confirmed Ipk1 as the major IP5 kinase in C

4 IP-10 is an accurate biomarker to screen individuals on

5 IP-10 levels were significantly higher in subjects with

6 IP-10 univariate model demonstrated high classification

7 Plasma interferon-gamma-induced protein 10 (IP-10) levels declined significantly upon dosing with RG

8 and reduced IFN-gamma-inducible protein 10 (IP-10) response to TLR2 stimulation (P = .042).

9 Nasal and serum IFN-inducible protein 10 (IP-10) were measured after doses 1 and 8, then 1 (follow

10 ls of interferon-gamma-inducible protein 10 (IP-10) were quantified by enzyme-linked immunosorbent as

11 kin 10, interferon gamma-induced protein 10 (IP-10), and hepatocyte growth factor differentiated betw

12 ytokine interferon-gamma-induced protein 10 (IP-10/CXCL10) was among the highest released, and high l

13 P-2), interferon gamma inducible protein-10 (IP-10), interferon gamma (IFN-gamma), tumor necrosis fac

14 nes, including GM-CSF, IFN-alpha2, IL-12p70, IP-10 and VEGF, during both acute and chronic stages of

15 he following: (i) vehicle 1 IN and vehicle 2 IP, (ii) HDM IN and vehicle 2 IP, or (iii) HDM IN and C1

16 and vehicle 2 IP, (ii) HDM IN and vehicle 2 IP, or (iii) HDM IN and C188-9 IP.

17 the third ventricle) occurred prior to EX-4 IP injection.

18 -VQ addition per 100g of oil resulted in 50% IP increase.

19 and vehicle 2 IP, or (iii) HDM IN and C188-9 IP.

20 ous transplantation of IFN-gamma-accelerated IP-DPSCs might be a promising new therapeutic strategy f

21 ared with Controls, Paired rats administered IP amphetamine subsequently showed a conditioned locomot

22 reporter gene in a panel of 12 organs after IP injection.

23 ing a possible topography of IP outputs, all IP subnuclei gave rise to descending projections, wherea

24 nic (TNFalpha [tumor necrosis factor alpha], IP-10 [interferon gamma-induced protein 10]), and proinf

25 Animals exposed to amphetamine IP or in the ventral tegmental area (VTA) showed a sensi

26 demonstrate the promise of a gel depot as an IP drug delivery system.

27 Logistic regression was used to build an IP-10-based model able to identify individuals with VL >

28 Here we describe an IP-free one-step method that enables stable genome integ

29 ganics; that is, only those organics with an IP value lower than ca. 9.0 eV can be oxidized in the CP

30 Four additional analytes (IP-10, IL-12/23p40, IFNy, IL-15) were found to be elevat

31 ficantly higher levels of interleukin 18 and IP-10 but lower levels of hepatocyte growth factor than

32 nocyte chemoattractant protein 1[MCP-1], and IP-10) were increased.

33 okine targets in skeletal muscle (IL-1ra and IP-10/CXCL10) were identified from a cytokine array.

34 te chemoattractant protein 1, TNF-alpha, and IP-10 were observed in ZIKV-infected pregnant women carr

35 ds such as RNA IP (RIP) and crosslinking and IP (CLIP) are key starting points for evaluating the mol

36 receptors, with absent antagonism of EP2 and IP, may provide more effective anti-inflammatory activit

37 s within the crystal structures of HOIPs and IP.

38 ines (IL-1beta, IL-12p70) were increased and IP-10 and elafin were decreased.

39 by combining information from both input and IP samples, which could largely improve the testing perf

40 levels of condensed polyphenols than LI and IP apples, while the remaining metabolites assimilated.

41 ntial role for proinflammatory mediators and IP-10 in combination with the vaginal-health-score as pr

42 The entire on-chip process of sonication and IP took only 1 h.

43 and IL-5 and IL-13 expression in vitro, and IP deficiency diminished the stimulatory effect of indom

44 WT and IP(-/-) mice were challenged intranasally with Alternari

45 direct lineage relationship between APs and IPs has been established, the molecular mechanism that c

46 es of creep and relaxation of both HOIPs and IPs are similar, negating prior hypothesis that the pres

47 of donor islets and recipient mice with anti-IP-10 neutralizing monoclonal antibody.

48 for previously described TB markers, such as IP-10, LBP, FCG3B, and TSP4, and for many novel proteins

49 f IP to oxidized triacylglycerol monomers at IP, min/%; time reaching 10% of triacylglycerol dimers a

50 where no feasible LP solution is available, IP-derived reaction deletion sets represent minimal cut

51 , and to investigate the association between IP and Barrett's esophagus.

52 The bidirectional connections between IP and MnR proved to be primarily GABAergic.

53 macrophages, COX-2 expression was blocked by IP, EP2, and EP4 receptor antagonists, indicating a role

54 free-floating in the intraperitoneal cavity (IP) in mice.

55 hase high-performance liquid chromatography (IP-RP-HPLC) and detected by UV spectroscopy as well as O

56 Co-IP-mass spectrometry positioned Psi in an interactome pr

57 Additional co-IP studies in HEK293 cells determined that AQP2 interact

58 olor immunofluorescence (IF) staining and co-IP, we observed a nuclear distribution of BMAL1 and a pr

59 phosphatase inhibitors in mpkCCD14 cells, co-IP with phosphorylation deficient forms of AQP2 expresse

60 lementary methods, coimmunoprecipitation (co-IP) and proximity biotinylation (BioID), to identify vir

61 onal methods for a coimmunoprecipitation (Co-IP)-based workflow from sample preparation for mass spec

62 m considerations for experimental design, Co-IP, preparation of the sample for mass spectrometric ana

63 n extracts containing PiSSK1:FLAG:GFP for Co-IP/MS, we identified two additional SLFs (SLF4 and SLF13

64 it can be successfully used as a general Co-IP method.

65 ) cells and did not co-immunoprecipitate (co-IP) with BMAL1 or CLOCK.

66 tunia inflata for co-immunoprecipitation (Co-IP) and mass spectrometry (MS), and identified PiCUL1-P

67 in cell culture) co-immunoprecipitation (Co-IP) in HEK293 cells stably overexpressing NOX4.

68 Co-immunoprecipitation (co-IP) studies in mpkCCD14 cells uncovered an AQP2/14-3-3 i

69 ChIP) and protein co-immunoprecipitation (co-IP) studies, we show that TopoIIalpha forms protein-prot

70 A (RING1A) as assessed by immunostaining, Co-IP, and mass spectrometry.

71 and Trx2 directly interacts with Mst7 in co-IP assays.

72 We modified the previous Co-IP/MS procedure, including the addition of style extract

73 Consistent with this result, co-IP experiments showed that GpsB complexes with EzrA, Stk

74 interaction could be confirmed by reverse Co-IP and proximity ligation assay, whereas NOX1, NOX2, or

75 cation of several interactions by reverse co-IP, supports the specificity of our screening process.

76 e with the organic solvents used for the COF IP process.

77 ore, the XRCC1 co-immunoprecipitate complex (IP) displayed MMEJ activity in vitro, which was signific

78 he Development of ALLergy (MeDALL; EU FP7-CP-IP; project no: 261357; 2010-2015) studied the complex l

79 The CUF-IP-HILIC-MS/MS method was validated according to the 200

80 CCL11 (Eotaxin-1), CXCL1 (GROalpha), CXCL10 (IP-10), IFN-gamma, IL-4, IL-5, IL-7, IL-12p70, IL-13, IL

81 uction in the serum concentration of CXCL10 (IP-10), CCL22 (MDC), CCL17 (TARC), CCL-2 (MCP-1) and CCL

82 ynergy, production levels of CXCR3 cytokines IP-10 and MIG, depend non-linearly on both IFNgamma and

83 cal upregulation of TLR7-mediated cytokines (IP-10).

84 wild-type (WT) and PGI2 receptor-deficient (IP(-/-)) mice were cultured with IL-33 and treated with

85 receptor (i.e., PGI2 IP receptor-deficient [IP(-/-)]) were compared with wild type (WT) mice.

86 ss IPases (or phytases) that dephosphorylate IPs.

87 The effects of islet-derived IP-10 could be blocked by treatment of donor islets and

88 eatment blindly using a previously described IP-10 kinetic algorithm.

89 -VQ was more effective, since the determined IP increase after its addition was from 5 to 25-fold hig

90 ograde or anterograde tracers into different IP subdivisions or the MnR and additionally examined the

91 As described herein, DMF-IP and P-CLIP-DMF-IP are rapid, automated, and multiplexed methods that ha

92 As described herein, DMF-IP and P-CLIP-DMF-IP are rapid, automated, and multiplex

93 tic particles and digital microfluidics (DMF-IP).

94 tum, and LHb originated from the dorsocaudal IP.

95 y inputs from the contralateral dorsolateral IP, and mainly excitatory inputs from the ipsilateral ro

96 and excitatory neurons, and the dorsomedial IP, which is exclusively inhibitory.

97 In comparison with healthy DPSCs, IP-DPSCs expressed lower colony-forming capacity, popula

98 uction period, min; ki, rate constant during IP, meq/kgmin) of control sample (38.0; 2.0346) were con

99 To facilitate IP chemotherapy of ovarian cancer, we developed an in-si

100 Following IP injection of lipopolysaccharide (LPS), serum levels o

101 owed excellent muscle transduction following IP administration, highlighting its potential as a new m

102 l to reduce the time and effort required for IP sample preparations with applications in the fields o

103 escribe a new IKBKG mutation responsible for IP resulting in an inactive truncated form of NEMO.

104 aled similar metabolic changes over time for IP-, LI- and BIO-samples, mainly decreasing lipid and su

105 recommend to routinely obtain biopsies from IP for histopathology.

106 stored CD4+ Th17 cell function in cells from IP children to levels measured in NIP children.

107 , HDM responses in mice lacking a functional IP receptor (i.e., PGI2 IP receptor-deficient [IP(-/-)])

108 r, they support a cardinal role of GABAergic IP/MnR interconnections in the behavioral response to ni

109 onfirmed cases were more likely to have high IP-10 levels at D0 and had a steeper decline than clinic

110 icantly more marked in barrels with a higher IP and medium grain.

111 macological agonism of the prostaglandin I2 (IP) receptor in pancreatic beta-cells and in glomerular

112 Immunoprecipitation (IP) is a common method for isolating a targeted protein

113 tegrates sonication and immunoprecipitation (IP) for epigenetic assays, such as chromatin immunopreci

114 BP)-RNA interactions by immunoprecipitation (IP)-based methods such as RNA IP (RIP) and crosslinking

115 The combination of immunoprecipitation (IP) with MALDI technology delivers high assay sensitivit

116 Using a series of immunoprecipitation (IP)-tandem mass spectrometry (LC-MS/MS) experiments and

117 However, in our immunoprecipitation (IP) experiments, we did not observe myoferlin binding wi

118 nterfering RNA (siRNA), immunoprecipitation (IP), immunoblots and bioinformatics.

119 dulator to improve the functions of impaired IP-DPSCs, suggesting that autologous transplantation of

120 onses because depletion of NK1.1(+) cells in IP(-/-) mice restored both the HDM-induced lung inflamma

121 For ML375 this was confirmed in IP assays performed after reduction of receptor reserve

122 more likely to have a measurable decline in IP-10 at day 7 than clinically diagnosed cases (48/77 (6

123 ost participants had a measurable decline in IP-10 during the first 7 days of therapy.

124 nfirms the association between a decrease in IP-10 levels during the first week of treatment and a ba

125 Here we investigate the early response in IP-10 levels (between day 0 and day 7 of TB therapy) to

126 igher levels in tumor at 60 min than that in IP route.

127 a brain endothelial-targeted gene therapy in IP.

128 of Th1 cell-recruiting chemokines, including IP-10, through an IFN-gamma-dependent mechanism.

129 (IL-1alpha, IL-8, IL-12(p70)) and increased IP-10.

130 Increasing IP/IV chemotherapy use in clinical practice may be an im

131 alities were used; three polyphenol indices (IP), classified using the NIRS procedure, and three grai

132 expression and suppressed rhinovirus-induced IP-10 secretion, which was associated with increased vir

133 mmadelta T cells, or wild-type mice injected IP with control isotype IgG or gammadelta T cell-depleti

134 suppressive activity, suggesting that intact IP-DPSCs may be inadequate for dentin/pulp regeneration.

135 on of apples from organic (BIO), integrated (IP) and low-input (LI) production.

136 ion signatures distinguishing interpersonal (IP)-related traumas from combat-related traumas.

137 Intraperitoneal (IP) administration can be used to improve therapeutic in

138 Intraperitoneal (IP) chemotherapy is a promising post-surgical therapy of

139 EAP1-C151 dependent manner; intraperitoneal (IP) injection of bixin led to pulmonary upregulation of

140 nship, we hypothesized that intraperitoneal (IP) administration of rAAV produces unique patterns of t

141 ehicle was administered via intraperitoneal (IP) injection.

142 ted intranasally (IN) and intraperitoneally (IP) daily for 3 weeks with the following: (i) vehicle 1

143 Moderate doses of IP ketamine, IP memantine, or intra-PLmPFC memantine increased aggres

144 e exacerbated by allopregnanolone (10 mg/kg, IP) and attenuated by progesterone (30 mg/kg, IP) in SD-

145 tex; furthermore, finasteride (10-100 mg/kg, IP) dose-dependently ameliorated PPI deficits, hyperacti

146 however, only allopregnanolone (5-15 mg/kg, IP) dose-dependently exacerbated TS-like manifestations

147 P) and attenuated by progesterone (30 mg/kg, IP) in SD-subjected, but not control rats.

148 were suppressed by finasteride (25-50 mg/kg, IP), an inhibitor of the main rate-limiting enzyme in al

149 ark anti-tic therapy haloperidol (0.3 mg/kg, IP).

150 trol; 2) sham control + trametinib (1 mg/kg, IP); 3) cecal ligation and puncture; and 4) cecal ligati

151 ns receives bilateral input from the lateral IP, which in turn receives bilateral input from the dors

152 examined the transmitter phenotype of major IP and MnR afferents by combining retrograde tract traci

153 However, structures downstream of the MHb-IP axis, including the median (MnR) and caudal dorsal ra

154 introduce a method for performing microscale IP using magnetic particles and digital microfluidics (D

155 TAAR with DAP and MEPs monitoring and had no IP-SCI.

156 ls (PBMCs) from infection-prone (IP) and non-IP (NIP) children 9-18 months old were examined for thei

157 primary target, the interpeduncular nucleus (IP), have been identified as major players in mediating

158 incipal target, the interpeduncular nucleus (IP).

159 g aortic clamp interval (ACI) nor absence of IP-SCI eliminate the possibility of delayed postoperativ

160 Activity of IP anticancer drugs can be further potentiated by encaps

161 ith elevated CX3CL1 levels in the airways of IP(-/-) mice and treatment with a neutralizing Ab to CX3

162 ge sample volumes, making the application of IP cleanup to precious samples nearly impossible.

163 A previously reported association of IP with Barrett's esophagus was weak, statistically sign

164 ther studies should evaluate the benefits of IP-10 as a triage approach to monitor ART in resource-li

165 In 80% of cases of IP, the deletion of exons 4 to 10 leads to the absence o

166 The correlation of IP with Barrett's esophagus hints to a partly common pat

167 of death decreased by 12% for each cycle of IP chemotherapy completed (AHR, 0.88; 95% CI, 0.83 to 0.

168 bstrates is necessary for rational design of IP modulators.

169 Moderate doses of IP ketamine, IP memantine, or intra-PLmPFC memantine inc

170 gether contributing to the downregulation of IP-10 and other Th1 cell-recruiting chemokines (e.g., CX

171 presence of S. aureus The downregulation of IP-10 by S. aureus was mediated by components of its cel

172 tumor selectivity and antitumor efficacy of IP pro-apoptotic NWs.

173 rmed that donor islet-specific expression of IP-10 contributed to islet inflammation and loss of beta

174 and in vitro immunosuppressive functions of IP-DPSCs to enable dentin/pulp regeneration.

175 Higher plasma levels of IP-10 (P = .0011), TNF-RII (P = .0002), and D-dimer (P =

176 In a mouse model of IP, we administered a single intravenous dose of the ade

177 Th17 responses to S. pneumoniae in PBMCs of IP children can be rescued by addition of Th17-promoting

178 PBMCs of IP children stimulated with heat-killed S. pneumoniae ha

179 study, we examined the stem cell potency of IP-DPSCs.

180 were carefully searched for the presence of IP.

181 to prospectively determine the prevalence of IP and of preneoplasia within IP, and to investigate the

182 The prevalence of IP seems to be underestimated, but preneoplasia within I

183 e best antioxidant performance (the ratio of IP to oxidized triacylglycerol monomers at IP, min/%; ti

184 Taken together with prior studies of IP projections to the raphe, these results form an emerg

185 eneration and in vitro T cell suppression of IP-DPSCs, whereas treatment with tumor necrosis factor a

186 Regarding a possible topography of IP outputs, all IP subnuclei gave rise to descending pro

187 ce and affordability, and extends the use of IP-based mass spectrometry technology to most research a

188 Using a cutoff value of IP-10 >/=44.2 pg/mL, the model identified detectable VL

189 The highest absolute values of IP were reached by extra virgin olive oil, naturally abu

190 shearing chromatin/DNA and mixing/washing of IP beads.

191 Despite the typical absence of IPs in prokaryotes, many of these organisms express IPas

192 e (UPR), further impairing the generation of IPs and leading to microcephaly.

193 croenvironment and increased neurogenesis of IPs.

194 lts suggest that the neurogenic potential of IPs may be boosted in vivo by manipulating Flna-mediated

195 At least one IP was endoscopically identified in 53 patients (14.5%).

196 nd IFN-gamma-inducible protein 10 (CXCL10 or IP-10) remained statistically significant in the multiva

197 with siRNA directed against TNF-alpha, KC or IP-10 to mice suffering from dextran sulfate sodium (DSS

198 Hydroperoxide-based kinetic parameters (IP, induction period, min; ki, rate constant during IP,

199 In particular, IP is often used as the primary means of target purifica

200 cervical esophagus, also termed inlet patch (IP), varies substantially, ranging from 0.18 to 14%.

201 of these compounds and the induction period (IP) were found.

202 ne index (p-An), oxidation induction period (IP), and color were chosen as test parameters.

203 erties of HOIPs and an inorganic perovskite (IP) single crystal by measuring nanoindentation creep an

204 lacking a functional IP receptor (i.e., PGI2 IP receptor-deficient [IP(-/-)]) were compared with wild

205 and functional assays of inositol phosphate (IP) accumulation and Ca(2+) mobilization in a recombinan

206 stingly, we could detect inositol phosphate (IP), inositol 4,5-bisphosphate (IP2 ), inositol 1,4,5-tr

207 myo-Inositol phosphates (IPs) are important bioactive molecules that have multipl

208 The less-phosphorylated IP binding differed in both the myo-inositol ring positi

209 Both enzymes bound these less-phosphorylated IPs in a catalytically competent manner, suggesting that

210 Incontinentia pigmenti (IP) is a genetic disease leading to severe neurological

211 Incontinentia pigmenti (IP; MIM308300) is a severe, male-lethal, X-linked, domin

212 LTNP-C showed lower plasma IP-10 (p=0.019) and higher IFN-gamma (p=0.02) levels tha

213 Lower plasma IP-10 levels were associated with spontaneous clearance

214 e lung and has several 'interaction points' (IPs) which it must overcome to cause infection.

215 thesized via the interfacial polymerization (IP) of terephthalaldehyde and tris(4-aminophenyl)benzene

216 Fungal inositol polyphosphate (IP) kinases catalyse phosphorylation of IP3 to inositol

217 ate of State I and the ionization potential (IP) of the organics; that is, only those organics with a

218 h EMT and angiogenesis to further potentiate IPs' neurogenesis.

219 n between fecal indole and indole-producing (IP) gut microbiota on the outcome of a Cryptosporidium i

220 al fate-restricted intermediate progenitors (IPs) are derived from the multipotent radial glia (RGs)

221 ng in a buildup of intermediate progenitors (IPs) expressing prohemocyte and hemocyte markers.

222 esis by generating intermediate progenitors (IPs), which give rise to projection neurons of all corti

223 atively solve a pair of integer programming (IP) and linear programming (LP) to compute EMs in a sequ

224 onuclear cells (PBMCs) from infection-prone (IP) and non-IP (NIP) children 9-18 months old were exami

225 sis of a new series of potent non-prostanoid IP receptor agonists that showed oral efficacy in the ra

226 Intramembrane proteases (IPs) cleave membrane-associated substrates in nearly all

227 m diseased teeth with irreversible pulpitis (IP-DPSCs) are considered to be suitable for dentin/pulp

228 (DEA) as a hydrophilic ion-pairing reagent (IP-HILIC-MS/MS).

229 role of the eicosanoid PGI2 and its receptor IP during allergic airway sensitization, HDM responses i

230 ndent on signaling through the PGI2 receptor IP.

231 lammation, we identified that PGI2 receptor (IP) signaling was critical for indomethacin-induced, STA

232 MP, and agonists for prostaglandin receptors IP, EP2, and EP4 in infected but not uninfected cPLA2alp

233 ng EP2, EP3, and prostaglandin I2 receptors (IPs); Kb values (in nanomoles) are given in parentheses.

234 PK) activity, which differentially regulated IP-10 expression and subsequent protein release.

235 Remarkably, IP administration of bixin restored normal lung morpholo

236 Remarkably, IP(-/-) mice were less responsive to HDM challenge than

237 hat either block PGI2 production or restrain IP signaling may augment STAT6-independent pathways of a

238 letokine expression in beta-cells and reveal IP-10 as a primary therapeutic target to prevent beta-ce

239 precipitation (IP)-based methods such as RNA IP (RIP) and crosslinking and IP (CLIP) are key starting

240 RNA-IP demonstrated the association of the ARE-mRNAs with TT

241 leus incertus receive input from the rostral IP, which contains a mix of inhibitory and excitatory ne

242 ry inputs from the ipsilateral rostrolateral IP subnucleus.

243 ase the risk of immediate postoperative SCI (IP-SCI).

244 Dose-related increases in nasal and serum IP-10 were observed 24 hours after doses 1 and 8 (>95% c

245 Several IP subnuclei have been described, but their specific pro

246 A single IP administration of PNC-gel extended the survival of tu

247 taxa of common gut microbiota, including six IP taxa (11 subjects) were determined in stool samples c

248 These data indicate that some IP bacteria, or perhaps indole alone, can influence the

249 ecipitations coupled with mass spectrometry (IP-MS) and identified cold inducible RNA-binding protein

250 e show that interaction of Bacillus subtilis IP SpoIVFB with its substrate Pro-sigma(K) depends on pa

251 Surprisingly, IP(-/-) mice had increased numbers of pulmonary CD3(-)NK

252 T-IPs did not include multipotent stem cells or molecular

253 onal analysis demonstrated that most fetal T-IPs expressed genes of and had the potential to develop

254 to the T cell lineage or the migration of T-IPs.

255 yonic thymopoiesis-initiating progenitors (T-IPs) before their entry into the thymus and activation o

256 10 mug of i.p. TDM in light mineral oil (TDM-IP).

257 The responses to TDM-IP, TDM-IV, or TDM-IPIV were consistent between mouse st

258 We demonstrated that IP deficiency increased inflammatory cell infiltration,

259 inputs from MHb and also LHb, we found that IP is reciprocally interconnected mainly with midline st

260 Our findings indicate that IP is closely associated to a distributed network of mid

261 Our data suggest that IP administration emphasizes transduction patterns that

262 alytically competent manner, suggesting that IP hydrolysis has a role in plant pathogenesis.

263 Here we report that IPs' neuron production is enhanced by abrogating filamin

264 The IP receptor agonist MRE-269 increased intracellular 3',5

265 The IP(3)R is a ubiquitous intracellular Ca(2+) release chan

266 The inhibitory effects of PGI2 and the IP signaling pathway on CD4 T cell activation, inflammat

267 del based on a statistical test covering the IP samples only with 2 negative binomial distributions,

268 ants express p32 protein accessible from the IP space.

269 oved engraftment of allogeneic islets in the IP site, but resulted deleterious in the EFP site, proba

270 mma- and delta-tocopherol, as well as in the IP.

271 ship between the dorsal and ventral MHb, the IP, and the secondary efferent targets of this system.

272 The afferent and efferent connections of the IP have hitherto not been systematically investigated wi

273 mplications for the proposed function of the IP in a variety of behaviors, including models of mood d

274 Expression of the IP receptor in podocytes was confirmed in cultured cells

275 In vitro studies showed induction of the IP-10 gene was mediated by calcineurin-dependent NFAT si

276 In each step, the IP identifies a minimal subset of reactions, the deletio

277 lthough prior studies have reported that the IP has ascending projections to ventral forebrain struct

278 sies for histopathology were targeted to the IP, columnar metaplasia of the lower esophagus, gastric

279 edian reversal time [MRT], 1 day) versus the IP site (MRT, 5 days; P < 0.01) in diabetic C57BL/6 mice

280 and p300 histone acetyltransferase with the IP-10 gene required p38 and c-Jun N-terminal kinase mito

281 These IPs likely represent a transitory state in prohemocyte t

282 gnaling module upregulated in men exposed to IP-related traumas, and two modules associated with lipi

283 ase activity upregulated in women exposed to IP-related traumas.

284 earning procedure was used to expose rats to IP or VTA amphetamine either Paired or Unpaired with an

285 Fish treated IP with 10 mug.g(-1) FLX showed an interference in the c

286 Unfortunately, IP is a labor-intensive technique, is difficult to perfo

287 Upon IP administration in mice bearing MKN-45P, SKOV-3, and C

288 Using IP followed by immunoblot assays, we have developed a va

289 ntal stages is the availability of validated IP-quality antibodies.

290 al compound NS-304) with respect to in vitro IP receptor dependent cAMP accumulation assays.

291 tivity, we next set out to determine whether IP receptor agonism similarly affects nephrin phosphoryl

292 sophagus were tested for an association with IP.

293 ng protein was impaired in a male fetus with IP, leading to defective linear ubiquitination.

294 We sequenced the IKBKG gene in patients with IP and performed complementation and transactivation ass

295 s impaired in fibroblasts from patients with IP carrying the truncated NEMO form (as also observed in

296 tive linear ubiquitination) in patients with IP.

297 P xenografts and CT-26 syngeneic tumors with IP linTT1-D(KLAKLAK)2-NWs resulted in significant reduct

298 Regarding cases with adenocarcinoma within IP, some experts recommend to routinely obtain biopsies

299 o be underestimated, but preneoplasia within IP is rare, which does not support the recommendation to

300 prevalence of IP and of preneoplasia within IP, and to investigate the association between IP and Ba