1                                              IPMT is a dysplastic and likely precancerous lesion that

2  4992 human genes, we screened a total of 13 IPMTs including nine noninvasive and four invasive cases

3 algorithm, and clarified relationships among IPMT, mucinous cystic neoplasms of the pancreas (MCN), a

4  of 4 with malignant and 2 of 11 with benign IPMT died (P < 0.05).

5  codon 12 point mutations are as frequent in IPMT as in ductal adenocarcinoma.

6 ess of carcinogenesis, have been reported in IPMT.

7 l genes are differentially expressed both in IPMTs and pancreatic carcinomas suggests that they may b

8        The most highly up-regulated genes in IPMTs corresponded to three members of the trefoil facto

9 ble to distinguish noninvasive from invasive IPMT preoperatively, complete surgical excision of the d

10  with benign IMPT and in none with malignant IPMT (P < 0.05).

11 f K-ras mutations in the different stages of IPMT was 16.7% in normal epithelium and papillary hyperp

12        Intraductal papillary-mucinous tumor (IPMT) of the pancreatic ducts is increasingly recognized

13  with intraductal papillary mucinous tumors (IPMT) and to analyze their relation to the degree of sit

14 es of intraductal papillary-mucinous tumors (IPMTs) of the pancreas, which represents one of the alte

15 d pathological specimens of 15 patients with IPMT (dilated main pancreatic duct or branch ducts with

16 n situ, and carcinoma) from 16 patients with IPMT and 9 specimens from patients with pancreatic ducta

17 e specimen in 13 (81.2%) of 16 patients with IPMT.

18 e carcinoma in the majority of patients with IPMT.